gingival and periodontal diseases

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Gingival and periodontal diseases BY MOHD MOHSIN JAMAL UNDER DR. ANSHUL AGGARWAL DEPT. OF ORAL MEDICINE AND RADIOLOGY DR. ZIAUDDIN AHMAD DENTAL COLLEGE A.M.U

Transcript of gingival and periodontal diseases

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Gingival and periodontal

diseasesBYMOHD MOHSIN JAMAL

UNDERDR. ANSHUL AGGARWAL

DEPT. OF ORAL MEDICINE AND RADIOLOGY

DR. ZIAUDDIN AHMAD DENTAL COLLEGEA.M.U

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Gingival diseases

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Gingival Diseases Gingivitis is inflammation of the gingival

tissue. Gingivitis is characterized by areas of redness

and swelling, and there is a tendency for the gingiva to bleed easily.

Gingivitis is limited to the epithelium and gingival connective tissues. It is important to note that there is no tissue recession or loss

of connective tissue or bone.

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Gingivitis

Gingivitis associated with poor oral hygiene is usually classified asa. Initial lesionb. Early lesionc. Moderate lesiond. Advanced lesion

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Stages of gingivitisstage Initial

stageEarly stage

Established stage

Time (days)

2-4 4-7 14-21

Blood vessels

Vascular dilatation

Vascular proliferation

Vascular proliferation,Blood stasis

Junctional &Sulcular epi.

Infiltration by PMNs

Same as stage 1,

Same but more advanced

Predominant immune cells

PMNs Lymphocytes

Plasma cells

Collagen Perivascular loss

Increased loss

Continuous loss

Clinical findings

Gingival fluid flow

Erythema,Bleeding on probing

Changes in color, texture, size

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Plaque removal

may progress

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Acute gingival diseases

Primary herpetic gingivostomatitis Recurrent aphthous ulcer Acute necrotizing ulcerative gingivitis (vincent

infection) Acute candidiasis (thrush, c)

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Primary herpetic gingivostomatitis

Caused by Herpes simplex virus type 1 Age-Children younger than 6 yrs, but also may

be seen in adolescents and adults. Primary infection is asymptomatic Location- lesions mainly involve hard palate,

attached gingiva and oral mucosa. Manifestations include blister outside the lip so

disease commonly called recurrent herpes labialis.

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….contd. Characteristic oral finding:

a. Diffuse erythematous involvement of gingiva.

b. Initial stage in characterized by discrete spherical gray vesicles.

c. Lip- excoriation involving lip become hemorrhagic

d. Course is self limited to 7-10 days.

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Oral symptoms:a. Generalized sorenessb. Ruptured vesicles – focal site of painc. Infants show irritability and refusal to eatd. Pain upon swallowing

Extra oral symptoms:a. Cervical lymphadenopathyb. Fever ( 101- 105℃)c. Generalized malaise, irritability

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Treatment

Symptomatic & supportive. Application of mild anesthetic such as

dyclonine hydrochloride(0.5%) Bed rest , soft diet are recommended during the

febrile stage & the person should be kept well hydrated.

Pyrexia - paracetamol suspension and secondary infection of ulcers may be prevented using chlorhexidine.

In severe case, systemic acyclovir(200 mg daily for 5 days).

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Recurrent aphthous ulcer

Characterized by painful ulceration on the oral mucosa

Occurs between school age and adults Recurrent ulceration with painful discrete and

confluent lesions. Lesions are round to oval crateriform base,

raised and reddened margins.

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Clinical features: Occur between second and third decade of life. Buccal and labial mucosa tongue and gingiva

are commonly involved. Symptoms- lesions are typically very painful. Signs- begins as single or multiple superficial

erosion covered by grey membrane, surrounded by localized area of erythema.

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Treatment

Symptomatic treatment Topical corticosteroid triamcinolone 3-4 times

daily by rinse and expectorate method. Nutritional diet. Maintenance of oral hygiene.

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Acute necrotizing ulcerative gingivitis

Characterized by sloughing of gingival tissue Predisposing factors: Local: poor oral hygiene, pre-existing gingivitis

and smoking Systemic: Emotional stress Nutritional deficiency –Vit B and C

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Clinical features Characteristic lesions are punched out, crater

like depression at the crest of interdental papillae

Surface of gingival craters is covered by pseudomembranous slough.

Linear erythma.

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Treatment Perform debridement under local anesthesia. Remove pseudo membrane. Patient counselling should include specific oral

hygiene instructions, instruction on proper nutrition,

For any signs of systemic involvement, the recommended antibiotics are:

Amoxicillin, 250 mg 3 x daily for 7 days and/or Metronidazole, 250 mg 3 x daily for 7 days

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Gingival enlargement

Inflammatory enlargementa. Chronic inflammatory enlargementb. Acute inflammatory enlargement

Drug induced gingival enlargement Vitamin C deficiency associated gingival

enlargement

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Chronic inflammatory gingival enlargement

Long standing gingivitis in young patient sometimes results in chronic inflammatory gingival enlargement, which may be localized or generalized.

Etiology: Prolonged exposure to plaque Factors that favor plaque accumulation and

retention. Chronically dried gingiva in mouth breathing

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Clinical features Characterized by slight ballooning of interdental

papilla and marginal gingiva. In early stage , it produces a life preserver-

shaped bulge around the involved teeth. Treatment: Removal of local irritants Oral hygiene maintenance

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Acute inflammatory enlargement

Gingival abscessIs a localized, painful rapidly expanding lesion that is usually of sudden onset

Etiology:a.Irritation from foreign substanceb.Tooth brush bristle c.Piece of apple cored.Lobster shell fragment –embedded in to gingiva

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Clinical feature:a. Localized, painful, rapidly expanding lesionb. Limited to the marginal gingiva or interdental

papillaec. Early stage: red swelling with smooth shiny

surfaced. With in 24 hours to 48 hours- lesion will be

fluctuant. Management: Incision and drainage

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Drug-induced gingival enlargement

Drug-induced gingival enlargement: Anticonvulsant Immunosuppressant cyclosporine Calcium channel blocker

Clinical and microscopic features of enlargement caused by different drugs are similar.

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Clinical features The growth starts as a painless, beadlike

enlargement of the interdental papilla and extends to the facial and lingual margins.

As the condition progress, marginal and papillary enlargement units and may develop into a massive tissue fold.

May interfere with occlusion.

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Treatment modalities

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Ascorbic Acid Deficiency Gingivitis

Associated with Vit C deficiency Involves marginal and papillary gingiva in the

absence of local predisposing factors Complains of severe pain and spontaneous

hemorrhage Treatment: Complete dental care, improved

dental hygiene, and supplementation with Vit C – improves gingival conditions

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Eruption Gingivitis

Gingivitis associated with tooth eruption. Tooth eruption usually does not cause gingivitis,

however inflammation associated with plaque accumulation around erupting tooth.

perhaps secondary to discomfort caused by brushing these friable areas, may contribute to gingivitis.

Treatment: Complete dental care, improve oral hygiene.

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Fig:- Medication-induced gingivitis (From Perry D, Beemsterboer P, Taggart E: Periodontology for the dental hygienist, Philadelphia, 2001, Saunders.)

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Fig:- Pregnancy gingivitis (From Perry D, Beemsterboer P, Taggart E: Periodontology for the dental hygienist, Philadelphia, 2001, Saunders.)

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Periodontal diseases

Periodontal disease is an infectious disease process that involves inflammation. Periodontal diseases involve the structures of the periodontium.

Periodontal disease can cause a breakdown of the periodontium resulting in loss of tissue attachment and destruction of the alveolar bone.

Introduction

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Prevalence of Periodontal Disease

Periodontal diseases are the leading cause of tooth loss in adults.

Almost 75% of American adults have some form of periodontal disease, and most are unaware of the condition.

Almost all adults and many children have calculus on their teeth.

Fortunately, with the early detection and treatment of periodontal disease, most people can keep their teeth for life.

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Systemic Conditions: Links to Periodontal Disease

Certain systemic conditions increase the patient’s susceptibility to periodontal disease, and periodontal disease may actually increase a patient’s susceptibility to certain systemic conditions. Cardiovascular disease Preterm low birthweight Respiratory disease

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Fig:- Structures of the periodontium: junctional epithelium, gingival sulcus, periodontal ligaments, and cementum

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Periodontal Diseases Infectious diseases that are the leading

cause of tooth loss in adults. Nearly 75% of American adults suffer from

various forms of periodontal disease and most are unaware of it.

Almost all adults have calculus on their teeth.

With the early detection and treatment of periodontal disease, it is possible for most people to keep their teeth for a lifetime.

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Classification:-Periodontal problems

PERIODONTAL CONDITIONS WITH LOSS OF CONNECTIVE TISSUE ATTACHMENT Early-onset periodontitis

a. Localized aggressive periodontitisb. Generalized aggressive periodontitis

Prepubertal periodontitis associated with systemic disease

a. Papillon-Lefevre syndrome b. Ehlers-Danlos syndromec. Chediak-Higashi syndromed. Leucocyte adhesion deficiency syndromee. Neutropenia

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Periodontitis

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…contd.

It is inflammatory disease of gingiva and deeper tissues of periodontium.

Characterized by pocket formation and destruction of supporting alveolar bone.

Periodontal probing for attachment loss and bitewing radiograph are often used to clinically confirm the diagnosis.

In its classification of periodontitis, the American Academy of Periodontology categorized the early-onset form under Aggressive Periodontitis.

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Page and colleagues believe that there are four different forms of

periodontitis : prepubertal, juvenile, rapidly progressing and

adult.

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COMMON FEATURES OF LAP AND GAP

Aggressive forms of periodontal disease have been defined based on the following primary features (Lang et al. 1999)a. Non-contributory medical historyb. Rapid attachment loss and bone destructionc. Familial aggregation of cases

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Localized Aggressive periodontitis(LAP):

Clinical features: characterized by “localized loss of attachment and

bone around permanent incisors and first permanent molars”

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….contd.

Prevalence is 1% It is linked to presence of Actinobacillus

actinomycetemcomitans and successful treatment outcomes correlate well with eradication of bacteria.

Treatment : local measures in combination with systemic antibiotic therapy.

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Generalized aggressive periodontitis (GAP):

It occurs in adolescents and teenagers. Characterized by generalized interproximal

attachment loss affecting at least three permanent teeth other than incisor and first molar.

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Radiographs showing the severe generalized nature of disease

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Causes of Periodontal Diseases Dental plaque is the major factor in causing periodontal

disease. Dental calculus provides a surface for plaque to attach.

Subgingival calculus Supragingival calculus

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Fig:- Buildup of bacterial plaque on the teeth affects the gingival tissues

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Fig:- Periodontal disease (Courtesy Dr. Edward J. Taggart, San Francisco, CA.)

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Treatment:

A combined regimen of regular SRP with 2-week course of systemic tetracycline therapy (250 mg, four times daily) .

Aa is sensitive to tetracycline, which also has the ability to be concentrated up to 10 times in gingival crevicular fluid when compared with serum.

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….cont. A combination of metronidazole (250 mg) &

amoxicillin (amoxicillin) (375 mg), three times a day for 8 days, in association with subgingival scaling, has also been found to be effective.

A more radical approach is to undertake flap surgery so that better access is achieved for root cleaning, and the superficial, infected connective tissues are excised.

An antimicrobial regimen can also be implemented in conjunction with a surgical approach.

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