1 Tonsillectomy, and Adenoidectomy. 6/8/[email protected] History Celsus 50 A.D. Caque...
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Transcript of 1 Tonsillectomy, and Adenoidectomy. 6/8/[email protected] History Celsus 50 A.D. Caque...
04/18/23 [email protected] 2
HistoryHistory
• Celsus 50 A.D.
• Caque of Rheims
• Philip Syng
• Wilhelm Meyer 1867
• Samuel Crowe
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EmbryologyEmbryology
• 8 weeks: Tonsillar fossa and palatine tonsils develop from the dorsal wing of the 1st pharyngeal pouch and the ventral wing of the 2nd pouch; tonsillar pillars originate from 2nd/3rd arches
• Crypts 3-6 months; capsule 5th month; germinal centers after birth
• 16 weeks: Adenoids develop as a subepithelial infiltration of lymphocytes
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AnatomyAnatomy
TonsilsTonsils• Plica triangularis• Gerlach’s tonsil
AdenoidsAdenoids• Fossa of Rosenmüller• Passavant’s ridge
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Blood SupplyBlood Supply
TonsilsTonsils• Ascending and descending
palatine arteries
• Tonsillar artery
• 1% aberrant ICA just deep to superior constrictor
AdenoidsAdenoids• Ascending pharyngeal,
sphenopalatine arteries
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Common Diseases of the Common Diseases of the Tonsils and AdenoidsTonsils and Adenoids
• Acute adenoiditis/tonsillitis
• Recurrent/chronic adenoiditis/tonsillitis
• Obstructive hyperplasia
• Malignancy
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Acute AdenotonsillitisAcute Adenotonsillitis
Etiology
• 5-30% bacterial; of these 39% are beta-lactamase-producing (BLPO)
• Anaerobic BLPO
GABHS most important pathogen because of potential sequelae
• Throat culture
• Treatment
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Microbiology of Microbiology of AdenotonsillitisAdenotonsillitis
Most common organisms cultured from patients with chronic tonsillar disease (recurrent/chronic infection, hyperplasia):
• Streptococcus pyogenes (Group A beta-hemolytic streptococcus)
• H.influenza
• S. aureus
• Streptococcus pneumoniae
Tonsil weight is directly proportional to bacterial load.
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Acute AdenotonsillitisAcute Adenotonsillitis
Differential diagnosisInfectious mononucleosisMalignancy: lymphoma, leukemia, carcinomaDiptheriaScarlet feverAgranulocytosis
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Medical ManagementMedical Management• PCN is first line, even if throat culture is negative for
GABHS• For acute UAO: NP airway, steroids, IV abx, and immediate
tonsillectomy for poor response• Recurrent tonsillitis: PCN injection if concerned about
noncompliance or antibiotics aimed against BLPO and anaerobes
• For chronic tonsillitis or obstruction, antibiotics directed against BLPO and anaerobes for 3-6 weeks will eliminate need for surgery in 17%
• Co-amoxiclav or clindamycin or PCN+Rifampin
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Obstructive HyperplasiaObstructive Hyperplasia
• Adenotonsillar hypertrophy most common cause of SDB in children
• Diagnosis:snoring, restless sleep, FTT, daytime symptoms… poor mentation, decreased attn span, poor scholastic performance, dysphagia, nocturnal enuresis, chronic mouth breathing
• Indications for polysomnography• Interpretation of polysomnography• Perioperative considerations
• Interpretation of polysomnography:
Indications for surgical intervention
>1 apnea in 1 hour
Central apnea + o2sat<90%
O2sat<92%
CO2>53
CO2>45 in more than 60% of test time
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Unilateral Tonsillar Unilateral Tonsillar EnlargementEnlargement
Apparent enlargement vs true enlargement
Non-neoplastic: • Acute infective• Chronic infective • Hypertrophy• Congenital
Neoplastic
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Other Tonsillar PathologyOther Tonsillar Pathology
• Hyperkeratosis, mycosis leptothrica
• Tonsilloliths
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Indications for Tonsillectomy; Indications for Tonsillectomy; Historical EvolutionHistorical Evolution
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Indications for TonsillectomyIndications for Tonsillectomy
Paradise study • Frequency criteria: 7 episodes in 1 year or 5
episodes/year for 2 years or 3 episodes/year for 3 years
• Clinical features (one or more): T 38.3, cervical LAD (>2cm) or tender LAD; tonsillar/pharyngeal exudate; positive culture for GABHS; antibiotic treatment
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Indications for TonsillectomyIndications for Tonsillectomy
AAO-HNS:• 3 or more episodes/year• Hypertrophy causing malocclusion, UAO• PTA unresponsive to nonsurgical mgmt• Halitosis, not responsive to medical therapy• UTE, suspicious for malignancy• Individual considerations
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Also:1.Recurrent tonsillitis in patient with heart valve dis2.In patient with febrile convulsion3.Unresponsive carrier 4.Obstructive IMN unresponsive to medical therapy5.Carrier of diphtheria6.Chronic tonsillitis with never-ending sore throat7.OSAS8.FTT9.Disphagia10.phonation disorders11.Malocclusion12.Craniofacial growth abnormalities13.Corpulmonale14.Suspicious malignancy
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Pathophysiology of corpulmonale in OSA:
1.URT obstruction2.Alveolar hypovantilation3.Chronic hypoxia and hypercapnea4.Respiratory acidosis5.Vasoconstriction of alveolar capillaries6.Right atrial dilatation7.Pulmonary hypertension 8.Corpulmonale and CHF
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Pathophysiology of enuresis:1.SDB 2.REM abnormalities3.Irregularities in ADH secretion
Pathophysiology of FTT:1.SDB2.REM abnormalities3.Irregularities in GH secretion, may sometimes stop totally
Both return to normal following A&T
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ADHD (Attention Deficit Hyperactivity Disorder) is the mostCommon psychiatric diagnosis in children.
In direct correlation with Sleep Disordered Breathing(SDB)
Pathophysiology uncertain, Maybe due to abnormal sleep pattern
Neurocognitive development delays:Healthy sleeping pattern: important component of brain growthFrontal cortex growth continues up to pubertyMost critical age: 3-7
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Indications for AdenoidectomyIndications for Adenoidectomy
Paradise study • 28-35% fewer acute episodes of OM with adenoidectomy in
kids with previous tube placement
• Adenoidectomy or T & A not indicated in children with recurrent OM who had not undergone previous tube placement
Gates et al • Recommend adenoidectomy with M & T as the initial
surgical treatment for children with MEE > 90 days and CHL > 20 dB
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Indications for AdenoidectomyIndications for Adenoidectomy
Obstruction:• Chronic nasal obstruction or obligate mouth breathing
• SDB with FTT, cor pulmonale
• Dysphagia
• Speech problems
• Severe orofacial/dental abnormalities
Infection:• Recurrent/chronic adenoiditis (3 or more episodes/year)
• Recurrent/chronic OME (+/- previous BMT)
Also:
• Chronic sinusitis: first step
biofilm in 95% adenoid specimen vs. 2% in SDB
• Chronic recurrent AOM
• COM
• Chronic otorrhea
• VT and persistent discharge
• Suspecious malignancy
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PreOp Evaluation ofPreOp Evaluation of Adenoid Adenoid DiseaseDisease
• Triad of hyponasality, snoring, and mouth breathing
• Rhinorrhea, nocturnal cough, post nasal drip
• “Adenoid facies”• “Milkman” & “Micky
Mouse”• Overbite, long face,
crowded incisors
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PreOp Evaluation of Adenoid PreOp Evaluation of Adenoid DiseaseDisease
Differential diagnoses• Allergic rhinitis
• Sinusitis
• GERD
• For concomitant sinus disease, treat adenoids first
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PreOp Evaluation of Adenoid PreOp Evaluation of Adenoid DiseaseDisease
Evaluate palate• Symptoms/FH of CP or
VPI• Midline diastasis of
muscles, bifid uvula• CNS or neuromuscular
disease
• Preexisting speech disorder?
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PreOp Evaluation of Adenoid PreOp Evaluation of Adenoid DiseaseDisease
Lateral neck films are useful only when history and physical exam are not in agreement.
Accuracy of lateral neck films is dependent on proper positioning and patient cooperation.
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PreOp Evaluation of Adenoid PreOp Evaluation of Adenoid DiseaseDisease
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PreOp Evaluation of Tonsillar PreOp Evaluation of Tonsillar DiseaseDisease
History• Documentation of episodes by physician
• FTT
• Cor pulmonale
• Poststreptococcal GN
• Rheumatic fever
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PreOp Evaluation of Tonsillar PreOp Evaluation of Tonsillar DiseaseDisease
TONSIL SIZE• 0 in fossa• +1 <25%
occupation of oropharynx
• +2 25-50%• +3 50-75%• +4 >75%
Avoid gagging the patient
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PreOp Evaluation of Tonsillar PreOp Evaluation of Tonsillar DiseaseDisease
Down syndrome• 10% have AtlantoAxial laxity
• Obtain lateral cervical films (flexion/extension) when positive findings on history, PE
• If unstable, need neurosurgical evaluation preoperatively
• Large tongue and small mandible… difficult intubation
• Prone to cardiac arrhythmias/hypotension during induction
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PreOp Evaluation for PreOp Evaluation for Adenotonsillar DiseaseAdenotonsillar Disease
Coagulation disorders• Historical screening
• CBC, PT/PTT, BT, vWF activity
• Hematology consult
• von Willebrand’s disease
• ITP
• Sickle cell anemia
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Principles of Surgical Principles of Surgical ManagementManagement
Numerous techniques:
• Guillotine
• Tonsillotome
• Beck’s snare
• Dissection with snare (Scissor dissection, Fisher’s knife dissection, Finger dissection
• Electrodissection
• Laser dissection (CO2, KTP)
• Coblation
• RadioFrequency
… Surgeon’s preference
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Post Operative ManagmentPost Operative Managment
Criteria for Overnight Observation• Poor oral intake, vomiting, hemorrhage• Age < 3• Home > 45 minutes away• Poor socioeconomic condition• Comorbid medical problems• Surgery for OSA or PTA• Abnormal coagulation values (+/- identified disorder)
in patient or family member
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ComplicationsComplications
#1 Postoperative bleeding
Other:• Sore throat, otalgia, uvular swelling
• Respiratory compromise
• Dehydration
• Burns and iatrogenic trauma
• Dental problems and trauma
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Rare ComplicationsRare Complications
• Velopharyngeal Insufficiency• Nasopharyngeal stenosis• Atlantoaxial subluxation/ Grisel’s syndrome• Regrowth• Eustachian tube injury• Depression• Laceration of ICA/ pseudoaneursym of ICA
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Management of HemorrhageManagement of Hemorrhage
• Ice water gargle, afrin
• Overnight observation and IV fluids
• Dangerous induction
• ECA ligation
• Arteriography
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Case study
• 13 year old female referred by PCP for frequent throat infections
• “She’s always sick. She’s been on four different antibiotics this year.”
• You call her pediatrician… he is out of town and his nurse can’t find the chart
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Case study
• No known medical problems, no prior surgical procedures
• Takes motrin for menustrual cramps
• No personal history of bleeding other than occasional nose bleeds and extremely heavy periods.
• Family history unknown. Patient is adopted.
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Case study• Physical exam is unremarkable. • Mom breaks down in tears when you tell her you
do not have enough documentation of illness to warrant T & A. “I had to go on welfare because I’ve missed so much work from her being out sick.”
• You hesitate. She adds, “Her grades have dropped from all A’s to all F’s. If she misses any more school, she’ll be held back.”
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Case study• You confirm with her pediatrician that she has had
4 episodes of tonsillitis this year and agree to T & A.
• Because of her history of epistaxis and menorrhagia, you order a PT, PTT, CBC, BT.
• She has a mild microcytic anemia and prolonged bleeding time.
• You order vWF activity level and consult hematology
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Case study
• She has a subnormal level of vWF, which responds to a DDAVP challenge (rise in vWF and Factor VII greater than 100%).
• You advise her to stop taking motrin.
• Before surgery, she receives desmopressin 0.3 microg/kg IV over 30 min and amicar 200mg/kg.
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Case study
• She receives the same dose of DDVAP 12 hours postoperatively and every morning.
• Amicar is given 100mg/kg PO q 6 hr.
• Before each dose of DDAVP, serum sodium is drawn. Sodium levels drop to 130.
• Desmopressin is discontinued and substituted with cryoprecipitate.
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Case study
• Patient presents to the ER on POD # 7 complaining of intermittent bleeding from her mouth.
• You order cryoprecipitate, draw a Factor VII level and CBC, and call her hematologist.
• Hemoglobin has dropped from 11.9 to 9.6.
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Case study
• PE reveals no active bleeding; an old clot is present
• You establish IV access, admit the patient for overnight observation, have her gargle with ice water, and administer crypoprecipitate
• No further bleeding occurs, patient is discharged the next day