MEDICINAL EMBRYONIC PHYTOTHERAPY (MEPTM...creatinine . Increased Nigrum (bud concentrations of...

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MEDICINAL EMBRYONIC PHYTOTHERAPY (MEPTM) SELECTIVE TOXIC METALS CHELATION

WITH EMBRYONIC PLANT EXTRACTS (EPEs)

© Dominique Richard 2018 Organic trace minerals are not deposited in the body like inorganic forms of these elements. Research shows that organic plant-derived trace ionic minerals naturally chelate or remove the inorganic minerals from the body. This is called Oligotherapy and Lithotherapy and are part of Biotherapy methodology. Toxic metal may occupy the site of the enzyme but are void of any activity in that site, thus enzyme "clinkers" are formed, which the body cannot utilize. Nutrient "Oligo-elements" can displace a toxic metal on the surface of the cells, as well as the toxic metals, which occupy the sites of activity in the enzyme, thereby activating the enzyme where it can be utilized by the body. The Oligo-elements also can serve as energy for the body and to aid in the detoxification process of the toxic metal if present. Minerals and oligo or trace elements regulate all organic processes in the body and are indispensable to life.

METALS & SOURCE

ACUTE CLINICAL PRESENTATION

CHRONIC EXPOSURE & POTENTIAL

MULTISYSTEM DISEASE CLINICAL

PRESENTATION

Toxic Concentration

Blood

EPEs Selective Chelator

ALUMINUM (AI) Atomic number

13

Aluminum cans and foils.

Antacids

Aspirin

Baby powder

Baking powder

Cigarette filters

Coffee

Some commercial

cheeses.

Cookware

Cosmetics

Dental amalgams and crowns.

Deodorants

A Tulane University

study showed that the

presence of aluminum

makes the brain more

permeable to aluminum

and other neurotoxins.

Many vaccines contain aluminum adjuvant, an ingredient necessary for stimulating a strong immune response and immunity. Aluminum adjuvant is typically made of Al hydroxide and/or Al phosphate nanoparticles.

Abnormal Blood Results could include:

Alter function of the blood–brain barrier (BBB).

Alzheimer’s disease (AD).

Anemia

Asthma.

Attention-deficit/hyperactivity disorder (ADHD).

Brittle bones

Dementia

Depression

Dermatitis

Headache.

Increases estrogen-related gene expression in breast cancer.

Insomnia

Serum, Plasma, or Whole Blood < 9 μg/L Patients on dialysis: < 40 μg/L

AI is a bio-persistent and potent neurotoxin that strongly stimulates neuro inflammation causing brain injuries.

Induce blockade of critical macrophage functions such as migration and xeno/autophagic disposition of particles, as previously reported for infectious particles.

Antioxidants prevent aluminum-induced toxicity.

Ash – Fraxinus Excelsior (buds) A

Beech – Fagus Sylvatica (buds) A essential adjuvant

Bilberry – Vaccinium Myrtillus (young shoots) P

Black Currant – Ribes Nigrum (buds) P

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Some Infant formulas.

Toothpaste

Vaccines

White flour

Extremely low or elevated liver enzymes.

Nanomolar amounts of aluminum induces expression of the pro-inflammatory systemic biomarker C-reactive protein (CRP) in human brain microvessel endothelial cells (hBMECs), (Alexandrov et al., 2015).

The Flarend study also showed that the aluminum spread to numerous organs, and tissues was the same for both adjuvants (kidney > spleen > liver > heart > lymph node > brain), (Flarend et al., 1997).

The chemokine (C-C motif) ligand 2 (CCL2) also known as monocyte chemoattractant protein 1 (MCP1) is elevated in AI toxicity.

Blood serum Elevation of MCP-1 in Autism.

Elevated interleukins IL-6, IL-17 and TNF-α.

Could have a low level of iron and zinc.

Irritable bowel syndrome (IBS).

Joint pain.

Kidney malfunction

Muscle pain.

Neurobehavioral disorders in children.

Poor memory.

Pulmonary fibrosis.

Parkinson’s disease (PD)

Seizures

AI cause brain damage at dosages human infants receive from vaccines.

Macrophagic myofasciitis (MMF) lesion detected in patients with myalgic encephalomyelitis/chronic fatigue/syndrome (CFS), (Gherardi et al., 2015).

Al accumulation alter iron metabolism and induce peroxidative injury (Abreo et al., 2004).

METALLOESTROGENS include: Arsenite (oxidation of arsenic). Metalloestrogens are an emerging class of inorganic xenoestrogens with potential to add to the estrogenic burden of the human malignant breast cancers (Darbre, 2006).

Black Elder – Sambucus Nigra (buds) P

Black Poplar – Populus Nigra (buds) P

Bramble – Rubus Fruticosus (young shoots) P

California Poppy – Eschscholzia Californica (buds) P

Cramp Bark – Viburnum Opulus (embryonic bark) P

Crab Apple – Malus Sylvestris (buds) P

Dog Rose – Rosa Canina (young shoots) P

Eyebright – Euphrasia Officinalis (bud of flowers) P

Fig – Ficus Carica (buds)

Giant Redwood – Sequoiadendron Giganteum (young shoots) P high in silicon (silica), antagonize the metabolism of aluminum.

Grape Vine – Vitis Vinifera (buds) P

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Horsetail – Equisetum Arvense (young shoots) P high in orthosilicic acid (silica) antagonize the metabolism of aluminum.

Mountain Elm – Ulmus Glabra (buds) A

Propolis Bend 1:5 P

Rhodiola – Rhodiola Rosea (embryonic roots) P

Virginia Creeper – Ampelopsis Veitchii (young shoots) P

ARSENIC (As) Atomic number

33

Drinking water

Insecticides

Non-organic source worse than organic

foods

Seafood

Skin products

Acute arsenic poisoning is associated initially with:

Abdominal pain

Nausea

Severe diarrhea

Vomiting

Encephalopathy and peripheral neuropathy are reported.

"Rice-water" diarrhea,

Multiple organ dysfunction syndrome (MODS)

Fatal: cardiovascular collapse and hypovolemic shock.

Abdominal discomfort

Acrocyanosis (intermittent)

Anorexia

Arrhythmias

Blackfoot disease (gangrene with spontaneous amputation).

Bone marrow depression

Both sensory and motor neuron peripheral neuropathy.

Cancers: bladder, kidney, liver, lung, lymphatic, prostate and skin cancers.

Carotid atherosclerosis

Whole Blood < 10 µg/L optimum

Considered Acceptable:

Inorganic Arsenic value: 2 – 23 µg/L

Elemental arsenic and arsenic compounds are classified as “toxic” and “dangerous for the environment and human’s health.

Occupational exposure in wood preservation.

If a large amount of arsenic is swallowed by humans, in a form that is readily absorbed, it can cause rapid

Arnica - Arnica Montana (bud of flowers) A/P





Cramp Bark – Viburnum Opulus

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Cerebral infarction

Chronic Fatigue Syndrome (CFS).

Chronic obstructive pulmonary diseases (COPD).

Cirrhosis

Colitis

Death if exposed to high levels.

Decreased intelligence

Diabetes

Diarrhea

Difficulty breathing

DNA damage

Elevated liver enzymes

Encephalopathy

Esophagitis

Fatty degeneration

Gastritis

Headaches

Hemolysis

Hepatomegaly

Hyperkeratosis

Hypopigmentation

Hypertension

Increased risk of infections

Ischemic heart disease

Lens opacity

Leucopenia low white blood cells (WBCs)

Malabsorption

Melanosis

Microcirculation abnormalities.

poisoning and death. The gut, the heart and the nervous system are affected. Those who survive acute poisoning may develop pigment spots in the skin and damage to the red blood cells, bone marrow, liver, nerves and brain.

Long-term exposure to high levels of arsenic in drinking water can cause thickening and pigment spots in the skin, and cancer of the skin, lungs, bladder or kidney. Exposure in the workplace – mainly via the air breathed in – can cause lung cancer. Smoking further increases the risk.

Long-term ingestion of arsenic, mainly from drinking of contaminated well water, has caused a disease called “Blackfoot disease” in Taiwan. Blood vessels of the leg and foot become damaged, resulting in coldness, loss of feeling and eventually gangrene in the foot.

METALLOESTROGENS include: Arsenic. Metalloestrogens are an emerging

(embryonic bark) A

Fig – Ficus Carica (buds) P

Giant Redwood – Sequoiadendron Giganteum (young shoots) P


Holly – Ilex Aquifolium (young shoots) A


Propolis Blend 1:5 P

Wheatgrass – Triticum Aestivum (germinating seeds) A

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Nausea

Numbness

Pericarditis

Permanent liver and kidney damages.

Portal hypertension without cirrhosis.

Pulmonary insufficiency (emphysematous lesions).

Raynaud’s syndrome.

Rhino-pharyngo-laryngitis.

Shortage of red blood cells (RBCs).

Tracheobronchitis

Tremors

Vomiting

Weight loss

class of inorganic xenoestrogens with potential to add to the estrogenic burden of the human malignant breast cancers (Darbre, 2006).

BISMUTH (Bi) Atomic number

83

Renal failure, acute tubular necrosis.

Renal toxicity.

In industry, bismuth metal is used to manufacture alloys, catalysts, ceramics, cosmetics, magnets, paints, pharmaceuticals, semiconductor, and x-ray contrast media.

Poisonings from this source are seldom if ever reported.

In contrast, toxicity from the medicinal use is not uncommon, and may even occur in epidemic form.

Pepto-Bismol (Bismuth subsalicylate) and found in some

Diffuse myoclonic

encephalopathy.

Skin a lichen planus-like

rash.

Mouth stomatitis with a blue-black gum line.

Inflammation and inclusion bodies in the liver, kidney, and bone are characteristic.

In the brain, the lesions will cause encephalitis.

Foul breath “tellurium breath.”

Confusion, disorientation

and black stools.

Whole blood Considered Normal: <10 μg/L

Urine (random or 24-hour) 0.3−4.6 μg/L

In appropriate cases, a simple flat plate of the abdomen will reveal the bismuth in the intestine because it is almost as dense as lead.

A simple, reliable, and fast bedside test is the modified Reinsch's test.

It can identify mercury, arsenic, antimony, and bismuth. Use 10 to 15 g of gastric contents or tissue


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cosmetics: lipsticks, powders, and fingernail enamels (DiPalma et al., 2001).

Bismuthia, a rare dermatological condition that results from the prolonged use of bismuth.

Stomatitis and may progress to malaise, nausea, weight loss, and depression.

homogenate. Add 3 ml of concentrated hydrochloric and insert a copper wire spiral. Heat gently for two hours. A silvery deposit is mercury; shiny black is bismuth; dull black is arsenic; and purple is antimony.

Confirmatory tests such as the Gutzeit test can confirm each deposit, and they can even be quantified. (Kaye et al., 1980).

CADMIUM (Cd) Atomic number

48

Cigarette smoke is the major source of cadmium toxicity, whether first or second-hand smoke.

Cadmium is absorbed much more readily by inhalation than by ingestion.

Other sources:

Crabs

Evaporated milk

Fungicides

Mussels

Organ meats

Oysters

Some rice

Well water

Pneumonitis (oxide

fumes) Flu like symptoms including chills, fever, and muscle ache sometimes referred to as "the cadmium blues.

Pneumonitis

Pulmonary edema.

Tracheo-bronchitis.

Anosmia

Cancers: breast, lung and prostate (Luevano, & Damodaran, 2014).

Cardiovascular disease: coronary heart disease, stroke, and peripheral arterial disease (Tellez-Plaza et al., 2013).

Emphysema

Hearing loss

Hyperchloremia

Hypertension

Hyperuricemia (gout and arthritis).

Increased fracture risk due to demineralization.

Inhibit apoptosis and DNA repair, and induce carcinogenesis (Luevano, & Damodaran, 2014).

Joint pain

Acute poisoning 1000- 3000 μg/L and 100-400 μg/L and higher in fatal events

Whole blood

Nonsmoker: 0.3−1.2 μg/L,

Smoker: 0.6−3.9 μg/L

Chronic cadmium exposure in a range of 25-50 μg/L or 25-75 μg/g creatinine

Increased concentrations of urinary beta-2 microglobulin can be an early indicator of renal dysfunction.

Proteinuria and/or ≥15 μg/g creatinine.

METALLOESTROGENS include: Cadmium. Metalloestrogens are an emerging class of inorganic








Dandelion – Taraxacum Officinale

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Kidney problem

Liver damage

Osteomalacia (softening of bones).

Osteoporosis

Ototoxic (hearing).

Prostate

Proteinuria (excess protein in urine, possible kidney damage).

Renal lithiasis

Renal tubular (hypophosphatemia), causing muscle weakness and sometimes coma.

Tinnitus

Weakened immune system allowing bacteria, viruses, yeast and parasites to proliferate.

Zinc deficiency.

xenoestrogens with potential to add to the estrogenic burden of the human malignant breast cancers (Darbre, 2006).

(embryonic roots) P

Dogwood – Cornus Sanguinea (buds) A

European Alder – Alnus Glutinosa (buds) P

Fig – Ficus Carica (buds) P

Giant Redwood – Sequoiadendron Giganteum (young shoots) P


Grey Alder – Alnus Incana (buds) P


Lilac – Syringa Vulgaris (buds) P


Oriental Plane Tree – Platanus Orientalis (buds) P


Silver Birch – Betula

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Verrucosa (buds) P

St John’s Wort – Hypericum Perforatum (bud of flowers) A

Virginia Creeper – Ampelopsis Veitchii (young shoots) A

Betulinic Acid Concentrate (BCA) P

CHROMIUM (VI)

Hexavalent chromium is

toxic.

Water insoluble chromium (III)

compounds and chromium metal

are not considered a health hazard

The acute toxicity of chromium (VI) is due to its strong oxidative properties.

GI hemorrhage, hemolysis, acute renal failure (Cr6+

ingestion).

Chromium salts (chromates) are also the cause of allergic reactions such as dermatitis.

Ulceration of the skin referred to "chrome ulcers"

A known human carcinogen associated with pulmonary diseases and lung cancer.

Genotoxicity to the binding to the DNA.

Hemolysis, renal and liver failure.

Lung cancer (inhalation).

Metallosis metal prosthesis.

Premature dementia.

Pulmonary fibrosis.

METALLOESTROGENS include: Chromium. Metalloestrogens are an emerging class of inorganic xenoestrogens with potential to add to the estrogenic burden of the human malignant breast cancers (Darbre, 2006).

The LD50 for chromium (VI) ranges between 50 and 150 mg/kg.

Plasma 0.1−2.1 μg/L

Whole blood chromium concentrations are in the range of 2.0 µg/100 mL to 3.0 µg/100 mL

Low risk group: <3.0 ng/mL

Moderate risk group: 3.0−10.0 ng/mL

High risk group: >10.0 ng/mL

Valerian – Valeriana Officinalis (embryonic roots) Chromium Total is 230±0.00 mg/g (Pilerood & Prakash, 2011). Beware




Propolis Blend 1:5 P

Oligotherapy: A

Bilberry – Vaccinium Myrtillus (young shoots)

Black Currant – Ribes Nigrum (buds)

Cowberry – Vaccinium Vitis Idaea (young shoots)

Cramp Bark – Viburnum

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Opulus (embryonic bark)

Dandelion – Taraxacum Officinale (embryonic roots)

Dog Rose – Rosa Canina (young shoots)

Elm – Ulmus Campestris (buds)

Eyebright – Euphrasia Officinalis (buds of flowers)

Grape Vine – Vitis Vinifera (buds)

Hawthorn – Crataegus Oxyacantha (buds)

Hazel – Corylus Avellana (buds)

Hornbeam – Carpinus Betulus (buds)

Horse Chestnut – Aesculus Hippocastanum (buds)

Horsetail – Equisetum Arvense (young shoots)

Juniper – Juniperus Communis (young shoots)

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Linden Tree – Tilia Tomentosa (buds)

Maize – Zea Mais (embryonic germinating seed-rootlets)

Mistletoe – Viscum Album (young shoots)

Purple Coneflower – Echinacea Purpura (embryonic roots)

Raspberry – Rubus Idaeus (young shoots)

St. John’s Wort – Hypericum Perforatum (buds of flowers)

Sweet Almond – Prunus Amygdalus (buds)

Virginia Creeper – Ampelopsis Veitchii (young shoots)

Walnut – Juglans Regia (buds)

Wayfaring Tree – Viburnum Lantana (buds)

White Willow – Salix Alba (buds)

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Yarrow – Achillea Millefolium (young shoots)

COBALT (Co) Atomic number

27

A key constituent of cobalamin vitamin B-12. And caution needs to be exercise with B12 chronic supplementation.

Cobalt compounds are used as pigments in paints.

Medications such as cobaltous chloride

Allergic dermatitis.

In 1966, the addition of cobalt compounds to stabilize beer foam in Canada led to a peculiar form of toxin-induced cardiomyopathy, which came to be known as beer drinker cardiomyopathy.

Pulmonary fibrosis, cough, and dyspnea.

Chronic cobalt ingestion has caused far reaching serious health problems at doses far less than the lethal dose.

A major cause of contact dermatitis.

Congestive heart failure (CHF)

Dementia

Fatigue

Goiter

Hearing loss

Impaired concentration.

Insulin inhibitor

Mental difficulties

Metallosis metal prosthesis.

Neurological abnormalities

Pericardial effusion

Pneumoconiosis when inhaled.

Polycythemia vera

Thyroid dysfunction

Tremors

Vertigo

Visual problems

METALLOESTROGENS include: Cobalt. Metalloestrogens are an emerging class of inorganic xenoestrogens with potential to add to

Plasma <1.0 μg/L

Cobalt is an essential element for life however, only in minute amounts. The LD50 value for soluble cobalt salts has been estimated to be between 150 and 500 mg/kg.

Immediately dangerous to life or health (IDLH) value is 20 mg/m3.

The National Institute for Occupational Safety and Health (NIOSH) has set a recommended exposure limit (REL) of 0.05 mg/m3, time-weighted average.

Antagonists: iron and manganese.

Synergistics:

Copper

Folic acid

Iodine

Iron deficiency

Molybdenum

Vitamin C-Complex (Black Current and Dog Rose)

Zinc



Propolis Blend 1:5 A

Oligotherapy: A

Cedar of Lebanon – Cedrus Libani (young shoots)

Cramp Bark – Viburnum Opulus (embryonic bark)

Dandelion – Taraxacum Officinale (embryonic roots)




Horsetail – Equisetum Arvense

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the estrogenic burden of the human malignant breast cancers (Darbre, 2006).

(young shoots)



Betulinic Acid Concentrate (BAC).

COPPER (Cu) Atomic number

29

Beer

Chocolate

Copperware

Copper IUD

Copper Pipe

Copper Supplement.

Denture

Fungicide

Icemaker

Insecticides

Liver organ meat.

Milk

Nuts

Oysters

Swimming Pools

Water

Blue vomitus.

Coma

GI irritation hemorrhage.

Hematemesis (vomiting of blood).

Hemolysis

Hemolytic anemia.

Hypotension

Jaundice

Melena (black "tarry" stools).

Multiple organ dysfunction syndrome (MODS) when ingested.

Metal fume fever (MFF) inhaled.

Alzheimer’s disease (AD).

Arteriosclerosis

Cirrhosis

Cluster headache

Emotional problems.

Excess Zinc intake.

Hyperactivity

Liver disease

Migraine

Parkinson’s disease (PD).

Schizophrenia

The generation of reactive oxygen species (ROS) that damage DNA.

Vineyard sprayer’s lung when inhaled.

Wilson disease (hepatic and basal ganglia degeneration). Specific for this is Cramp Bark – Viburnum Opulus (embryonic bark) P

Copper in the blood exists

in two forms: bound to ceruloplasmin (85–95%), and the rest "free"

Value 72−166 μg/dL

METALLOESTROGENS include: Copper. Metalloestrogens are an emerging class of inorganic xenoestrogens with potential to add to the estrogenic burden of the human malignant breast cancers (Darbre, 2006).

Arnica - Arnica Montana (bud of flowers) A protects liver cells from copper-induced oxidative stress.




Giant Redwood – Sequoiadendron Giganteum (young shoots) A

Grape Vine – Vitis Vinifera (buds) A

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Worsening symptoms of Alzheimer's disease (AD).


Olive – Olea Europaea (young shoots) P


Virginia Creeper – Ampelopsis Veitchii (young shoots) P

Propolis Bend 1:5 P

IRON (Fe) Atomic number

26

Alcoholism increase iron.

Cast iron

Iron supplement

Multivitamins

Multiple blood transfusions.

Cardiac depression

Dehydration and lethargy if not treated adequately.

Dysmetabolic Iron Overload Syndrome (DIOS) is characterized by an elevated serum ferritin with a normal transferrin-iron saturation percentage.

Person with DIOS will likely also have an elevated gamma-glutamyl transferase (GGT) possibly due to a fatty liver. Individuals with DIOS are helped by phlebotomy, diet and exercise.

Ferrotoxic Disease: Quantitative Effects of Iron Excess and Antioxidant Inadequacy on Health.

The FeGGT test is helpful in determining

Abdominal pain

Adrenal function problems.

Amenorrhea

Blurry vision.

Cellular poison.

Chronic fatigue

Chronic hepatitis C or alcoholism.

Cysts and tumors.

Depression

Diabetes

Early onset of neurodegenerative disease.

Elevated blood sugar

Elevated liver enzymes

Excessive alcohol intake

Fatigue

Hair loss

Heart attack or heart failure.

Less than 350 mcg/dL is associated with minimal toxicity.

350-500 mcg/dL with mild to moderate toxicity.

Greater than 500 mcg/dL with serious systemic toxicity.

Iron overload, however, is deadly. Most physicians believe it is rare and mainly hereditary. In fact, it is far more common and more dangerous than many people imagine.

Chelation with EPEs or phlebotomy are the two methodology use for the treatment of iron overload.

African iron overload, sickle




Black Elder – Sambucus Nigra (buds) A


Boxwood – Buxus Sempervirens (young shoots) A


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the iron status and GGT status. This test can be found: http://www.healtheiron.com/

This test check for 4 factors: Serum Ferritin (SF), Transferrin Saturation% (TS%), GGT and Hemoglobin (Hgb).

GI hemorrhage bloody stool.

Hemosiderosis: is described as a general increase in tissue iron without fibrotic changes.

Hemochromatosis: is more serious and describes tissue iron accumulation with fibrotic changes occurring in the liver, spleen, and pancreas.

Metabolic acidosis

Nausea

Pain in the stomach

Stomach lining becomes ulcerated.

Vomiting of blood

Hereditary hemochromatosis (all types).

Hepatic cirrhosis

Hepatomegaly

Hypogonadism

Hypopituitarism

Hypothyroidism

Impotence

Inability to eliminate enough iron from the body.

Infertility

Irregular heart rhythm

Iron in excess replaces other vital minerals causing enzyme dysfunction.

Iron in excess stimulates bacterial growth.

Joint pain

Liver and Heart failure.

Liver cancer

Lowered libido

Metabolic acidosis, which in turn cause damages to the brain and the liver.

Numbness or tingling in the arms or legs.

Osteoarthritis

Osteoporosis

Skin color changes (bronze, ashen-gray green).

Slow wound healing.

Splenomegaly

Suicidal ideology.

The carcinogenicity of

cell disease, thalassemia, X-linked sideroblastic anemia, enzyme deficiencies (pyruvate kinase; glucose-6-phosphate dehydrogenase) and a very rare protein transport disorders called Aceruloplasminemia and Atransferrinemia.

None of these conditions should be confused with polycythemia vera (PV), which is not an iron disorder, but a condition where the bone marrow produces too many blood cells (red, white and platelets). People with PV have abnormally high levels of hemoglobin and are at risks of a stroke or progressing to the development of acute myelogenous leukemia (AML). Part of the therapy for PV are phlebotomies.

Please note: Grape Vine buds can potentially extend to other root causes of iron-overload associated with the following hematological conditions such as primary and secondary haemochromatosi







Nigella – Nigella Sativa (germinating seeds) P

Oak – Quercus Pedonculata (buds) A only with copper deficiency.

Olive – Olea Europaea (young shoots) P

Propolis Bend 1:5 P

Sweet Chestnut – Castanea Vesca (buds) A

Virginia Creeper – Ampelopsis

http://www.healtheiron.com/

http://www.healtheiron.com/

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iron is still under debate.

s, hemochromatosis, and hemochromocytosis without the need for routine phlebotomy. Furthermore, iron-overload is often over express in some types of cancer malignancies. What is even more curious and interesting is? How can a plant like that of Grape Vine with a high concentration of iron 18.08 mg/g (Sousa et al., 2014), can possibly reduce iron-overload the results of its total chemical composition and not that of any one single compound? Here is once more is demonstrated the bipolar/biphasic modulatory nature of Grape Vine buds in the treatment for both iron-overload as well as ferritin anemia. These effects may be mediated by alterations in lead toxicokinetics via iron metabolic pathways regulated by the hemochromatosis gene (HFE) product and body iron stores (Richard, 2018).

Veitchii (young shoots) A

LEAD (Pb) Atomic number

82

Abdominal pain

Ataxia

Abdominal pain

Acid Blood pH

No amount of lead is safe. Eliminating all lead

Ash – Fraxinus

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Airborne lead particulates from industrial processing.

Aviation lead fuel

Batteries

Ceramics

Cosmetics: kohl (83% lead).

Cigarette smoke

Drinking water

Herbal Medicine from medicines China, India, Mexico and Vietnam.

Ayurvedic remedies including Guglu (reports of 14,000 ppm lead), Sundari Kalp (pill, reports of up to 96,000 ppm lead), and Jambrulin (reports of 44,000 ppm lead (Gunturu et al., 2011).

Household dust

Gasoline

Lead crystal

Lead-based paints.

Lead-glazed pottery.

Solder

Water from lead pipe

Well water

Food source with high amounts of lead that can cause iron anemia include:

Cardiovascular disease: ischemia.

Contaminated soil

Encephalopathy

Headache

Intellectual disability

Nausea

Obtundation

Pregnancy complications: miscarriage, stillbirth, premature birth and low birth weight.

Seizures

Vomiting

Weakness

Weight loss

Toxic exposure to lead through alternative sources such as herbal medicine remains a significant and poorly recognized public health problem.

Adrenal dysfunction

Anemia

Arthralgia

Attention deficit hyperactivity disorder (ADHD).

Behavioral problems

Blue line on the gum

Body aches and pain

Cataracts

Cognitive deficits

Coma

Convulsion

Delirium

Decreases libido

Decreased sperm count

Depression

Delayed reaction times.

Diarrhea or constipation.

Dysendocrinia

Emotional disturbance

Encephalopathy

Foot-drop/ wrist-drop

Hair loss

Hallucinations

Hearing loss

Headaches

Hypertension

Impotence

Infertility

Insomnia

exposure in our environment is our best course of action. The main reason why they are no safe level of lead is that it is a cumulative toxicant

Blood lead levels below 5 µg/dL

Lead level ≥ 10 μg/dl was significantly associated with anemia, decreased iron absorption and hematological parameters affection. High blood lead levels were associated with low serum iron and ferritin. Lead level in drinking water was found to be higher than the permissible limits (Hegazy et al., 2010).

Lead can cross the placenta – accounts for 50% of fetal absorption

Blood Brain Barrier (BBB) is not developed until age 6 thus damages the nervous system, in addition children absorb 50% more lead, whereas

adults absorb 10-15%. Lead toxic side effects of which are largely irreversible.

METALLOESTROGENS include: Lead. Metalloestrogens

Excelsior (buds) A






Caraway – Carum Carvi (germinating seeds) A

Cedar of Lebanon – Cedrus Libani (young shoots) A

Cowberry – Vaccinium Vitis Idaea (young shoots) A



Dandelion – Taraxacum

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Fresh collard greens may contain up to 30 micrograms of lead (50 times higher than what the California proposition 65 stipulates acceptable).

Dry roasted mix nuts may contain up to 20 mcg.

Brussels sprouts may contain up to 15 mcg.

Sweet potatoes may contain up to 16 mcg of lead.

Spinach may contain up to 15 mcg of lead.

Irritability

Iron anemia

Muscle and joint pain

Kidney damage and failure.

Loss of appetite

Loos of coordination

Memory loss

Learning difficulties.

Miscarriage

Nephropathy

Nerve disorders

Pain and tingling

Peripheral neuropathy.

Personality changes

Reduced IQ

Slurred speech

Sperm dysfunction lower sperm counts and motility.

Thyroid dysfunction

Tremor

Unusual taste

Vision blindness

are an emerging class of inorganic xenoestrogens with potential to add to the estrogenic burden of the human malignant breast cancers (Darbre, 2006).

Officinale (embryonic roots) P


Elecampane – Inula Helenium (embryonic roots) A

Fig – Ficus Carica (buds) A


Grey Alder – Alnus Incana (buds) A


Nigella – Nigella Sativa (germinating seeds) P


Propolis Blend 1:5 A

Rosemary – Rosmarinus Officinalis (young shoots) P

MANGANISM or

MANGANESE (Mn)

Atomic number 25

Metal fume fever (MFF) inhaled.

Excess levels may

result in iron deficiency anemia.

Interferes with the metabolism of B1

Manganism:

Anosmia

Diminished IQ scores

Hallucinations

No clear reference standard

Exposure to ambient manganese air concentrations in excess of 5 micrograms


Crab Apple – Malus

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Mainly industrial pollution and welding.

Recent study found 40% of welders exhibit signs of idiopathic Parkinson's disease (PD).

Small amounts ubiquitous in air, water, and food.

Major food sources are wheat, nuts, and liver organ meat.

thiamine, copper metallo-enzymes.

Estrogen hormone replacement therapy (HRT) may raise serum manganese concentration.

Higher concentrations have been found in severe hepatitis and post hepatic cirrhosis, in dialysis patients and in patients suffering from myocardial infarct (MI).

High level decreases dopamine levels.

Hypertension

Impaired memory

Increased hyperactive and oppositional behaviors.

Kidney failure

Loss of appetite

Neuropsychiatric and motor disturbances, reduced response speed, irritability, mood changes, and compulsive behaviors.

Oxidative stress, mitochondrial dysfunction, glutamate-mediated excitoxicity, and aggregation of proteins.

Resemble idiopathic Parkinson's disease (PD) tremors and stiff muscles.

Neurotoxic Mechanism: interaction of manganese with iron metabolism, zinc, aluminum and copper.

Based on various studies, disturbed iron metabolism could be the major cause of the neurotoxic action of manganese. It participates in Fenton reactions and induces

Mn/m3 can lead to Mn-induced symptoms.

Sylvestris (buds) P




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oxidative damage as seen in occupational battery makers, and welders with fewer children and bone malformation of their offspring.

MERCURY (Hg) Atomic number

80

Mercury exists in three basic forms:

Elemental,

Inorganic

Organic

Dental amalgams are the most common source for elemental mercury vapor, which is a stable monoatomic gas.

Inorganic mercury, which is a divalent compound, is the toxic species found in human tissue after the conversion from other forms.

Organic mercury in the form of methyl and ethyl mercury is primarily from fish, sea mammals, and thimerosal vaccines. Although dental amalgams have historically been the major source of human exposure, fish and sea mammals are becoming an increasing

Elemental (inhaled):

Diarrhea

Fever

Vomiting

Inorganic salts (ingestion):

Caustic gastroenteritis.

Mercury induces mitochondrial dysfunction with reduction in adenosine triphosphate (ATP), depletion of glutathione, and increased in lipid peroxidation. Increased oxidative stress and reduced oxidative defense are common.

The overall vascular effects of mercury include increased oxidative stress and inflammation, reduced oxidative defense, thrombosis, vascular smooth muscle dysfunction, endothelial dysfunction, dyslipidemia, and immune and mitochondrial dysfunction. The clinical consequences of mercury toxicity include hypertension, coronary heart disease, myocardial

Acrodynia (pink disease). Specific for this condition is Beech – Fagus Sylvatica (buds) P

Alteration of enzyme function.

Alteration of tertiary structure.

Alteration of the genetic code.

Altered cell membrane permeability.

Amyotrophic Lateral Sclerosis (ALS, or Lou Gehrig’s Disease). Mercury has been found to accumulate preferentially in the primary motor function related areas involved in ALS—such as the brain stem, cerebellum, rhombencephalon, dorsal root ganglia, and anterior horn motor neurons, which enervate the skeletal muscles.

Alzheimer's disease (AD).

Anxiety

Autism infants or adult’s onset.

Blindness and deafness.

Background exposure "normal" limits: 10 μg/L (whole blood); 20 μg/L (24-h urine).

Chronic fatigue syndrome (CFS) has shown to improve with amalgam removal – streptococcus mutans can methylate mercury, mercury accumulates in RBC membranes reducing oxygen transport, it inactivates sulfhydryl group cellular enzymes, and CoA thus cellular respiration. The improvement in CFS is almost instantaneous by the removal of dental amalgam.

Noteworthy the posterior pituitary hormone (oxytocin) is decreased in dentist and was found to contain 800 times more mercury in their pituitary, which could explain the high suicide rate among dentists. Studies have shown that low levels of pituitary function are associated with



Beech – Fagus Sylvatica (buds) P and essential adjuvant.

Bilberry – Vaccinium Myrtillus (young shoots) A


Black Elder – Sambucus Nigra (buds) A



Cedar of Lebanon – Cedrus Libani (young shoots) A

Chaste Tree – Vitex Agnus Castus (young

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environmental source of potential mercury toxicity

Canned tuna fish

Contaminated food by polluted water from industrial waste or fungicides.

Dental Amalgam

Seafood

Shellfish

Mercury, as thiomersal or thimerosal, is widely used in the manufacture of mascara and vaccines. Thiomersal is used as a preservative in vaccines, immunoglobulin preparations, skin testing antigens, antivenins, ophthalmic and nasal products, and tattoo inks. Thiomersal is very toxic by inhalation, and when in contact with the skin (EC hazard symbol T+), with a danger of cumulative effects.

Mercury is used in thermometers, barometers, manometers, sphygmomanometers, float valves, mercury switches, mercury relays and fluorescent lamps.

infarction, cardiac arrhythmias, reduced heart rate variability, increased carotid intima-media thickness and carotid artery obstruction, cerebrovascular accident, generalized atherosclerosis, and renal dysfunction, insufficiency, and proteinuria.

Mercury diminishes the protective effect of omega-3 fatty acids. Mercury inactivates catecholaminei-0-methyl transferase, which increases serum and urinary epinephrine, norepinephrine, and dopamine. This effect will increase blood pressure and may be a clinical clue to mercury-induced metal toxicity.

Mercury toxicity should be evaluated in any patient with hypertension, coronary heart disease, cerebral vascular disease, cerebrovascular accident, or other vascular disease. Specific testing for acute and chronic toxicity and total body burden should be performed.

Mercury from various sources, including elemental mercury from earth sources or inhaled mercury vapor, methyl and ethyl mercury are converted by

Blue line along the gum.

Brain damage

Chronic fatigue

Cognitive problems.

Contribution to antibiotic resistance.

Contribution to autoimmune disease.

Depression

Diabetes (mercury bonds to insulin) causing glycemic regulation homeostatic.

Diarrhea

Digestion problems and malabsorption.

Dizziness

Drowsiness,

Emotional instability.

Endocrine imbalances (dysendocrinia).

Gingivo-stomatitis.

Hallucinations

Hashimoto’s

Headache

Hunter-Russell syndrome.

Hypertension

Immunotoxic causing immune suppression.

impulses

Infertility

Inhibition of DNA repair

Insomnia

mercury poisoning with symptoms of depression and suicidal thoughts.

METALLOESTROGENS include: Mercury. Metalloestrogens are an emerging class of inorganic xenoestrogens with potential to add to the estrogenic burden of the human malignant breast cancers (Darbre, 2006).

The vascular biological side effects of mercury were studied. Numerous toxic effects of mercury have been demonstrated in vitro studies of animal and human. Mercury increases free-radical production; inactivates antioxidant defenses; binds to thiol-containing compounds; binds to selenium, forming seleno-mercury complexes, reducing selenium availability for GPx activity; inactivates glutathione, catalase, and superoxide dismutase; increases oxidation of low-density lipoprotein (OxLDL) complexes. Thrombosis is potentiated by increased platelet

shoots) P for pituitary mercury toxicity.

Cowberry – Vaccinium Vitis Idaea (young shoots) A

Crab Apple – Malus Sylvestris (buds) P highest in catalase CAT).


Dandelion – Taraxacum Officinale (embryonic roots) P

Dog Rose – Rosa Canina (young shoots) A


Elecampane – Inula Helenium (embryonic roots) A



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Volcanoes, are responsible for approximately half of atmospheric mercury emissions.

biomethylation to inorganic divalent mercury, the toxic type in human organs and tissues. Divalent mercury is soluble and stable in water and undergoes biomethylation to methyl mercury, which is found in high concentrations in certain fish and sea mammals. It is this source that is becoming the major source of human exposure to mercury. Mercury is the most dangerous of all the toxic metals. It will modify the distribution and retention of other toxic metals. Mercury has no known physiologic role in human metabolism, and the human body has no mechanisms to actively excrete mercury. Mercury thus accumulates during life so that the average 165-lb person has a total body burden of about 13 mg of mercury. Mercury has a high affinity for sulfhydryl groups, various enzymes and amino acids, N-acetyl cysteine (NAC), alpha lipoic acid (ALA), and glutathione (GSH), which provide about 10% to 50% of the plasma protein antioxidant capacity. Both NAC and ALA, as well as cysteine, are precursors for glutathione, which is the most potent

Interference in nerve

Interference with endocrine function.

Irritability

Kidney damage.

Lack of coordination.

Leukemia

Low hemoglobin.

Memory loss.

Mental retardation.

Metallic taste (parageusia).

Mimicking Multiple Sclerosis (MS).

Minamata disease.

Nausea

Nephrotic syndrome.

Nervousness

Neurasthenia

Neuropsychiatric

Paresthesia

Parkinson disease (PD).

Rheumatoid arthritis (RA).

Suicidal

The role of mercury toxicity in hypertension, cardiovascular disease, and stroke.

Tremors

Source of Selenium:

Ash – Fraxinus Excelsior (buds)

aggregation, increases in Factor VIII, platelet Factor IV, and thrombin. Increasing catalase (CAT) can potentially be beneficial in the detoxification of mercury and thereby may reduce hypertension (Houston, 2011).

From the oligo-elements composition of a specific plant, the end fate of its biological activity can be deduced.

If a plant has a higher content of selenium coexisting with xyloglucans, then it is more likely to possess antioxidant and antitumor activities. This again helps define new pharmacological indications. Understanding interactive synergistic effects that are attributed not to one compound, but to an entire phytochemical composition, and how to achieve the desired end biological activities, will give an advanced edge in the design of combinatorial treatments.

Fig – Ficus Carica (buds) A





Propolis Bend 1:5 P

Rosemary – Rosmarinus Officinalis (young shoots) P

White Birch – Betula Pubescens (flower male-catkins) A

White Willow – Salix Alba (buds) P

Betulinic Acid Concentrate (BCA) P

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intracellular antioxidant and protects against oxidative stress, inflammation, and cardiovascular disease. This mercury-induced reduction in oxidant defense and increase in oxidative stress increase the risk for CVD and cerebrovascular accidents (CVA).

Selenium antagonizes some of the adverse effects of mercury by forming a seleno-mercury complex in tissue that is less toxic. Higher intake of selenium reduces mercury-related CVD and CVA (Houston, 2011).


Bramble – Rubus Fructicosus (young shoots)


Crab Apple – Malus Sylvestris (buds)


Dandelion – Taraxacum Officinale (embryonic roots) P Contains a total of 199.94 mg/kg (Păduret et al., 2016).







Hedge Maple – Acer Campestre (buds)



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Maize – Zea Mais (germinating seeds)


Nigella – Nigella Sativa (germinating seeds)



Rosemary – Rosmarinus Officinalis (young shoots)


Sweet Chestnut – Castanea Vesca (buds)



Wheat Grass– Triticum Aestivum (germinating seeds)

White Birch – Betula Pubescens (flower male-catkins)


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NICKEL (Ni) Atomic number

28

Most toxic nickel compound is Cyclopentadienyl nickel nitrosyl, (C5H5)NiNO.

Auto exhaust

Baking powder

cookware

Dental bridges

Fertilizers

Hydrogenation of oils

Jewelry

Nickel air

Nickel plating particulates

Stainless steel

Tap water

Nickel from tobacco smoke and skin contact with jewelry, shampoos, detergents, and coins.

The major source of nickel exposure is oral consumption, as nickel is essential to plants.

Nickel is found naturally in both food and water and may be increased by human pollution.

Nickel-plated faucets may

Allergic dermatitis.

Nickel carbonyl: myocarditis,

ALI, encephalopathy

Blisters and exudate in severe cases.

Chest pain

Coughing

Dry patches of skin that

may resemble a burn.

Headaches

Itching that may be

severe

Nausea

Rash or bumps on the

skin

Redness or changes in skin color.

Vertigo

Vomiting

Weakness

Activation of hypoxia inducible

factor (HIF-1)

and the up-regulation of hypoxia-inducible genes are due to depleted intracellular ascorbate levels caused by nickel.

Allergies

Asthma, nasal and sinus problems.

Breast and Lung cancers.

Bronchiectasis

Decrease in birth weight of offspring.

Nasopharyngeal tumors.

Occupational (inhaled):

Pulmonary fibrosis.

Reduced sperm count.

Still births.

Weight loss

Excessive exposure: ≥8 μg/L (blood)

Severe poisoning: ≥500 μg/L (8-h urine).

METALLOESTROGENS, Nickel. Metalloestrogens are an emerging class of inorganic xenoestrogens with potential to add to the estrogenic burden of the human malignant breast cancers (Darbre, 2006).





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contaminate water and soil.

Mining and smelting may dump nickel into waste-water.

Nickel–steel alloy cookware and nickel-pigmented dishes may release nickel into food.

A less-common form of chronic exposure is through hemodialysis as traces of nickel ions may be absorbed into the plasma from the chelating action of albumin.

The average daily exposure does not pose a threat to human health.

Nickel is not a cumulative poison, but larger doses or chronic inhalation exposure may be toxic, even carcinogenic, and constitute an occupational hazard.

Nickel compounds are classified as human carcinogens based on increased respiratory cancer risks observed in epidemiological studies of sulfidic ore refinery workers.

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Acute respiratory distress syndrome (ARDS).

SELENIUM (Se)

Atomic number 34

Selenosis occurs above 400 μg.

Tolerable Upper Intake Level Selenates and selenites are very toxic, having an oxidant effect of action similar to that of arsenic trioxide.

It is found in anti-dandruff shampoo.

Apple cider vinegar.

Hydrogen selenide exposure in industrial workplace.

Photography toner,

Pineapple juice

Yeast

Yogurt

The thyroid gland and every cell that uses thyroid hormone use selenium, which is a cofactor for the three of the four known types of thyroid hormone deiodinases, which activate and then deactivate various thyroid hormones and

Acute respiratory distress syndrome (ARDS).

Cardiac failure

Caustic burns

Diarrhea

Facial flushing

Fatigue

Garlic breath

Hair loss

Irritability

Kidney failure

Lightheadedness

Mood swings

Mottled teeth

Muscle tenderness

Tremors

Myocardial infarction (MI)

Nausea

Nervous system

abnormalities

Pneumonitis

Hypotension

Rashes

Severe gastrointestinal symptoms.

Severe neurological symptoms.

Vomiting

Chronic toxic dose of selenite for human beings is about 2400 to 3000 μg.

Cirrhosis of the liver.

Depression

Dermatitis

Epistaxis

Gastrointestinal

Disturbances

Hemiplegia

Multiple Chemical Sensitivity (MCS) can be one of the causes.

Pallor

Paresthesia

Pulmonary edema and death.

Red skin

Red stained finger nails, teeth, and hair

Mild toxicity: [Se] >1mg/L

Serum Serious Toxicity: >2 mg/L

Increased dietary intake of selenium reduces the effects of mercury toxicity although it is effective only at low to modest doses of mercury.

In target tissues, T4, the most abundant circulating thyroid hormone, can be converted to T3 by selenium-containing enzymes known as deiodinases. In this manner, thyroid hormones regulate a number of physiologic processes, including growth, development, metabolism, and reproductive function.


Oligotherapy with plants that contains (Se) include:

Ash – Fraxinus Excelsior (buds)


Bramble – Rubus Fructicosus (young shoots)


Crab Apple – Malus Sylvestris (buds)




Eyebright – Euphrasia Officinalis

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their metabolites; the iodothyronine deiodinases are the subfamily of deiodinase enzymes that use selenium as the otherwise rare amino acid selenocysteine.

(buds of flowers)




Hedge Maple – Acer Campestre (buds)







Nigella – Nigella Sativa (germinating seeds)

Purple Coneflower – Echinacea

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Purpura (embryonic roots)


Rosemary – Rosmarinus Officinalis (young shoots)


Sweet Chestnut – Castanea Vesca (buds)



Wheat Grass– Triticum Aestivum (germinating seeds)

White Birch – Betula Pubescens (flower male-catkins)



SILVER (Ag) Atomic number

47

Very high doses

Bone marrow suppression.

Argyria: blue-grey discoloration of skin, nails and mucosa that is

Asymptomatic workers have mean [Ag] of 11 μg/L Serum and

Beech – Fagus Sylvatica (buds) A

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The National Center for Complementary and Alternative Medicine (NCCAM) States the following: Reviews of the scientific literature on colloidal silver have concluded that: Silver has no known function in the body. Silver is not a nutritionally essential mineral or a cure-all and should not be promoted as such.

Claims that there can be a “deficiency” of silver in the body and that such a deficiency can lead to disease are unfounded. Claims made about the effectiveness of colloidal silver for numerous diseases are unsupported scientifically. In fact, Colloidal silver can have serious side effects.

Hemorrhage

Hepatorenal necrosis

Pulmonary edema

caused by silver salts, including silver nitrate, silver arsphenamine, silver chloride and possibly silver iodide. The estimated amount of silver accumulation over a one-year period that is required to produce argyria is 1 to 5 grams.

Irreversible neurologic toxicity associated with poor outcome

Mild forms of Argyria are sometimes mistaken for cyanosis.

2.6 μg/L (spot urine)

Colloidal silver (a colloid consisting of silver particles suspended in liquid) and formulations containing silver salts were used by physicians in the early 20th century, but their use was largely abandoned in the 1940s following the discovery of safer and more effective antibiotics. In 1999, the U.S. food and drug association (FDA) banned colloidal silver sellers from claiming any therapeutic or preventive value for the product.

essential adjuvant


Crab Apple – Malus Sylvestris (buds) P for the detoxification of colloidal silver prolonged intake.



Silver Fir – Abies Pectinata (young shoots)

THALLIUM (Tl) Atomic number

81

Man-made sources of thallium pollution include gaseous emission of cement factories, coal-burning power plants, and metal sewers. The main source

Early Onsets:

Autonomic instability

Coma

Diarrhea

Multiple organ dysfunction syndromes (MODS).

Painful neuropathy

Vomiting

Late Findings:

Thallium is a suspected human carcinogen.

Alopecia

Birth defects

Mees lines

Residual neurological

symptoms

Toxic: >3 μg/L (blood).



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of elevated thallium concentrations in water is the leaching of thallium from ore processing operations.

Later Onsets:

Ataxia

Athetosis

Coma

Cranial nerve palsies.

Death

Headache

Insomnia

Seizures

Tremor

Acute agitation

Aggression

Anxiety

Apathy

Confabulation

Confusion

Delirium

Depression

Diplopia: abnormal color vision, and impairment of visual acuity.

Hallucinations

Hypertension

Internal and external ophthalmoplegia and nystagmus.

Loss of the lateral half of the eyebrows.

Noninflammatory keratitis, lens opacities, and optic atrophy due to toxic optic neuropathy.

Paresthesia

Personality changes.

Pleuritic chest pain

Psychosis

Scaling of the palms and soles and acneiform or pustular eruptions of the face.

Seventh nerve palsy.

Skin lesions on eye lids.

Ptosis

Tachycardia

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TITANIUM (Ti) Atomic number

22

Dental implants and prosthesis such as hip balls and sockets (joint replacement).

These devices are made from a blend of several different metals, including chromium, cobalt, nickel, titanium and molybdenum. When the metal parts rub against each other, they release microscopic amounts of these metal particles into the blood and surrounding tissues.

Metallosis & Metal Poisoning. Some individual has allergies to these metals. It is highly recommended to test for metal allergies before having any of the prosthesis replacement.

Ceramic (Zirconia) is the newly recommended material for dental implants, crowns, knee and hip replacements that is non-allergenic and in facts so biocompatible that it increases osteogenesis and prevents bone loss.

Biocompatibility: zirconium is an inert material that seldom cause allergic reactions.

Metal-free: no corrosion, no galvanism effect, no metallic taste, no electronic disturbances.

Zirconia dental implants are exceptionally strong even stronger than titanium.

Ceramic dental implants are so biocompatible that soft tissue actually grows up around the abutment. This means that the gums continue to grow without the concerns of receding gums that accompany titanium implants.

Whereas metal-on-metal hip implants release metal

Titanium implants can elevate serum levels, which correlates to lung cancer.

Osteoporosis

Platelet suppression.

A New Zealand study showed higher incidence of death from cancer among hip replacement patients.

If there is pitting edema associated with metallosis it will be necessary to either increase the doe of Dandelion or instead use in its place Betulinic acid concentrate (BCA) 100-150 drops tid, PRN, QD. Taken first by itself into ½ glass water and DO NOT SIP and drink quickly.

Whereas, Dandelion can be added to the other plants and SIP over a period of at least 5 minutes is best.

Depending on blood results for chromium, cobalt, nickel choose appropriate protocol for the chelation and detoxification of these metals.

A General Metallosis Protocol could include the following plants all given together into ½ glass filtered water 30 minutes prior to each meal 3 times daily.

Beech – Fagus Sylvatica (buds) 1:10 15 gtts, tid, QD. 1:20 would require more drops 25 gtts, tid, QD.

Black Currant – Ribes Nigrum (buds) 1:10 15 gtts, tid, QD.

Crab Apple – Malus Sylvestris (buds) 25 gtts, tid, QD.

Oriental Plane Tree – Platanus Orientalis (buds) 1:10 15 gtts, tid, QD.

Propolis Blend 1:5, 15 gtts, tid, PRN, QD.

When liver enzymes are found elevated or when known liver or renal dysfunction would require the addition of Dandelion – Taraxacum Officinale (embryonic roots)


This protocol should be given long- term (6 months to even one year until all toxic level have returned back to normal values.

Metallosis tends to be a chronic condition. However, once the prosthesis has been replaced with Ceramic (Zirconia), and with the help of EPEs you can eventually corrects the toxicity of the metal poisoning and prevent the likelihood of developing any further disease from this highly toxic condition and consequences.

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particles into the body, causing metallosis or metal poisoning.

Symptoms include bone and tissue death (necrosis), implant failure and severe pain.

1:10 15-50 gtts, tid, PRN, QD depending on severity. Especially if constipation is present.

Choose between one of the next two plants

Black Elder – Sambucus Nigra (buds) when immunoglobulin is found to be low: 15 gtts, tid, QD.

OR

Lilac – Syringa Vulgaris (buds) in individuals with arteriosclerotic biofilm plaque formation: 15 gtts, tid, QD. 1:20 strength give 15 gtts, tid, QD. and in 1:10 8 gtts, tid. QD.

ZINC (Zn) Atomic number

30

It is the only metal, which appears in all enzyme classes.

Most zinc is found in the brain, muscle, bones, kidney, and liver, with the highest concentrations in the prostate and semen and part of the eye.

Common ingredient of denture adhesive which may contain between 17 and 38 mg of zinc per gram. Disability and

MFF (oxide fumes);

Abdominal pain and/or nausea from (ingestion)

Diarrhea

Vomiting

Zinc pennies pose a risk of zinc toxicosis, which can be fatal.

The free zinc ion is a powerful Lewis acid up to the point of being corrosive. Stomach acid contains hydrochloric acid, in which metallic zinc dissolves readily to give corrosive zinc chloride. It can cause damage to the stomach lining through the high solubility of the zinc

Person taking 100–300 mg of Zinc daily may suffer induced copper deficiency

Anemia

Excessive absorption of zinc suppresses copper and iron absorption.

Neurological degeneration.

Osteoporosis

Linked to alterations of blood lipoprotein levels, with increased levels of LDL and decreased levels of HDL seen.

Normal range: 0.6-1.1 mg/L (plasma) 10-14 mg/L (red cells)



Propolis Bend 1:5 P

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even deaths from excessive use of these products have been reported.

ion in the acidic stomach.

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