Diabetic foot & ankle

19
Diabetic Foot & Ankle Dr. Vijay Kumar Loya JR, JIPMER

Transcript of Diabetic foot & ankle

Page 1: Diabetic foot & ankle

Diabetic Foot & Ankle

Dr. Vijay Kumar Loya JR, JIPMER

Page 2: Diabetic foot & ankle

EPIDEMOLOGY Diabetic foot ulcers were found in 4.54% newly diagnosed patients(Sinharay K et. al, 2012)

Prevalence of DFU is about 14.3% in Indian population (according to a study done in Northern India, Shahi SK et. al)

Page 3: Diabetic foot & ankle

RISK FACTORS

NEUROPATHY

SENSORY

MOTOR

AUTONOMIC

PVD DELAYED BONE HEALING ALTERED IMMUNE FUNCTION

Page 4: Diabetic foot & ankle

Large sensory fibres – protective sensation is lostSmall fibres can lead to increased pain sensation, allodynia.Sensory disturbances show a length related pattern with stocking & glove distribution due to dying back distal axonopathy Motor neuropathy can cause claw toes from intrinsic muscle weakness & equinus contracture of Achilles tendon→ stress on fore-foot→high focal pressure & skin breakdown.Sweat gland dysfunction allows the skin to dry & crack→microbe entry

Page 5: Diabetic foot & ankle

DIAGNOSISPHYSICAL EXAMINATIONEMG/NCSLOSS OF PROTECTIVE SENSATION IS THRESHOLD AT WHICH NEUROPATHIC ULCERS/CHARCOT ARTHOPATHY OCCUR

Page 6: Diabetic foot & ankle

PVD ABI is unreliable in diabetics as calcification can lead to high results masking the severity of disease.

Toe pressure or TcpO2 better indicator. Angiography is gold standard, but requires IV contrast infusion.

ABI >0.45 & TcpO2 >40mm Hg necessary for ulcer to heal.

Page 7: Diabetic foot & ankle
Page 8: Diabetic foot & ankle
Page 9: Diabetic foot & ankle
vijay loya
Page 10: Diabetic foot & ankle

TREATMENTNON-OPERATIVETCC – TOTAL CONTACT CASTINGREMOVABLE DIABETIC BOOTSNEGATIVE PRESSURE WOUND THERAPYHYPERBARIC WOUND THERAPYEXTRA CORPOREAL SHOCK WAVE TREATMENT

Page 11: Diabetic foot & ankle
Page 12: Diabetic foot & ankle
Page 13: Diabetic foot & ankle

TCC is the gold standard for off-loading of plantar ulcerations.

ii. Patients with grade 3 or greater ulcers should undergo incision and drainage and antibiotic therapy, with wound improvement before TCC application.

iii. Casts should be changed every 2 to 4 weeks until erythema and edema have resolved and the temperature of the affected limb has decreased and become similar to that of the contralateral limb. Ulcers should be evaluated and debridement should be performed at the time of cast changes.

iv. Radiographs should be repeated every 4 to 6 weeks, or more often if there is an acute change.

v. TCC commonly continues for up to 4 months; when the active disease phase is complete, the patient can be fitted with a Charcot restraint orthotic walker, later followed by a custom shoe with orthoses.

Page 14: Diabetic foot & ankle
Page 15: Diabetic foot & ankle

OPERATIVE TREATMENT – URGENT SURGICAL INDICATION – NECROTISING FASCITTIS/ GANGRENE/ DEEP ABSCESSLESS URGENT INDICATIONS ARE - COMPROMISED SOFT TISSUE ENVELOPE

NEED TO AVOID PROLNGED ANTIBOTICS LOSS OF MECHANICAL FUNCTION BONE INVOLVEMENT THAT IS LIMB THREATENING

Page 16: Diabetic foot & ankle

OPERATIVE TREATMENTDEBRIDEMENTEXOSECTOMY OR REALIGNMENT ARTHODESISAMPUTATION AT APPROPRIATE LEVELRESECTION ARTHOPLASTYACHILLES TENDON Z-PLASTY

Page 17: Diabetic foot & ankle
Page 18: Diabetic foot & ankle
Page 19: Diabetic foot & ankle

THANK YOU