clinical meeting 19.12.13

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1ST ANNUAL CLINICAL MEETING DIVISION OF CARDIAC SURGERY Shisong Cardiac Center Organizing Committee: Mr. Marcel Fanka Tanlanka Program (5 minutes for each presentation) Moderators: Dr. Jacques Cabral Tantchou Tchoumi; Dr. Jean Claude Ambassa The Role of Extracorporeal Circulation in Cadiac Surgery Mr. Fanka Tanlanka Marcel ; Discussants: Sr. Juliet Berinyuy; Hilary Ayong Surgical Techniques for the Establishment of Extracorporeal Circulation Mr. Julius Peter Mbiydzenyuy ; Discussant: Mr. Roger Tachea The Use of Inotropes in Cardiac Surgery Mr. Thierry Yunishe ; Discussant: Sr. Ruth; Mr. Gerard Management of Cardiac Arrythmias after Open-Heart Surgery Mr. Justin Bika; Discussant: Sr. Isodora One -Year Experience in Cardiac Surgery at The Shisong Cardiac Center Dr. Charles Mve Mvondo ; Discussants: Dr. Jean Claude Ambassa, Dr Jacques Cabral Tantchou Tchoumi Open Discussion and Cardiologists Point of View Conclusions Dr. Charles Mve Mvondo Thursday 19, December 2013; 07.30 to 8.30 am; Relax Area, ICU. Cardiac Center.

Transcript of clinical meeting 19.12.13

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1ST ANNUAL CLINICAL MEETING DIVISION OF CARDIAC SURGERYShisong Cardiac CenterOrganizing Committee: Mr. Marcel Fanka Tanlanka

Program(5 minutes for each presentation)

Moderators: Dr. Jacques Cabral Tantchou Tchoumi; Dr. Jean Claude Ambassa

The Role of Extracorporeal Circulation in Cadiac SurgeryMr. Fanka Tanlanka Marcel ; Discussants: Sr. Juliet Berinyuy; Hilary Ayong

Surgical Techniques for the Establishment of Extracorporeal CirculationMr. Julius Peter Mbiydzenyuy ; Discussant: Mr. Roger Tachea

The Use of Inotropes in Cardiac SurgeryMr. Thierry Yunishe ; Discussant: Sr. Ruth; Mr. Gerard

Management of Cardiac Arrythmias after Open-Heart SurgeryMr. Justin Bika; Discussant: Sr. Isodora

One -Year Experience in Cardiac Surgery at The Shisong Cardiac CenterDr. Charles Mve Mvondo ; Discussants: Dr. Jean Claude Ambassa, Dr Jacques Cabral Tantchou Tchoumi

Open Discussion and Cardiologists Point of View

ConclusionsDr. Charles Mve Mvondo

Thursday 19, December 2013; 07.30 to 8.30 am; Relax Area, ICU. Cardiac Center.

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The Role of EThe Role of ExtraxtraCCorporeal orporeal CCirculation in Cardiac Surgeryirculation in Cardiac Surgery

Presentation: Fanka Tanlaka MarcelPresentation: Fanka Tanlaka Marcel• Discussant : Sr Juliette BerinyuyDiscussant : Sr Juliette Berinyuy• Contributions : Ayong Hilary GahContributions : Ayong Hilary Gah

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What is Extracorporeal Circulation ?

A medical equipment that provides Cardiopulmonary bypass, (temporary mechanical circulatory support) to the stationary heart and lungs)

• Heart and Lungs are made “functionless temporarily” , in order to perform surgeries

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The Physiology of ECC

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What key things does ECC do during Cardiac Surgery ?

• Provides bloodless field for the Surgeon• Artificial Pump• Artificial lungs• Myocardial protection• Organs function• Blood gas, chemistry , anticoagulation

state

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“to return the patient to the normal

physiologic state in spite of the insults that may transpire’’

Goal of the Perfusionist

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Factors Inspiring Evolution• Coagulation Disorders• Biocompatibility• Infection Control• Blood transfusion• Haemolysis• Biochemistry & BGA• Post operative complications

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Future Prospects1. Material science2. Pumping systems3. Oxygenating systems4. Monitoring ( computer Science)5. Blood Salvaging6. Circulatory assistance7. Artificial heart ( remains the ultimate goal

of the Bioengineer)

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Thanks for your very fervent attention...

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Surgical Techniques for the Establishment of

Extracorporeal Circulation

Mr. Julius Peter Mbiydzenyuy ; Discussant: Mr. Roger Tachea

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Introduction • Most operations require the use of CPB. • Basic techniques of arterial and venous

cannulation are similar in both CHD & adult cardiac surgery.

• Some modifications are necessary to accommodate the multiple anatomical variations that may be encountered in congenital defects

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The procedure• Collection of Cannulae • Reception and fixation of CPB Circuit

to the Sterile camp• Standard median sternotomy or

Thoracotomy• Purse Strings• Cannulation

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Venous cannulation

• Monocaval (Double stage)• Bicaval

– IVC– SVC

• Tricaval in rare cases of CHD– IVC– SVC– LSVC

• Femoral

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Cannulation for Cardioplegia

• Antegrade – Ascending Aorta – aortic root

• Retrograde – Coronary sinus• Selective - Coronary ostia

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Cannulation for Heart Venting

Left Heart Venting

Aortic Vent

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Conclusion

The techniques which are described in this presentation are used routinely in our institution in all patients, whatever the complexity of the pathologies and the age of the patients.

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Thanks for your very fervent attention

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The Use of Inotropes in Cardiac Surgery

Mr. Thierry Yunishe ; Discussant: Sr. Ruth GOOH; Mr.

Gerard Kindzeka

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The use of Inotropes in Cardiac Surgery

• Drugs that affect the force of contraction of myocardial muscle

• Positive or negative

• Term “inotrope” generally used to describe positive effect

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Main Goal

Tissue perfusion & oxygenation

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Basic principles - Inotropes

MAP = CO x SVR

CO = HR x SV

Preload Contractility After load

+VE INOTROPES

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Drug Classification

• Sympathomimetics– Naturally occurring– Synthetic

• Other inotropes– cAMP dependent– cAMP independent

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Sympathomimetics

• Naturally occuring– Epinephrine– Norepinephrine– Dopamine

• Synthetic– Dobutamine– Dopexamine– Phenylephrine– Metaraminol– Ephedrine

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Other inotropes

• cAMP dependent– Phosphodiesterase inhibitors

• cAMP independent– Digoxin– Calcium

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Phosphodiesterase inhibitors

• Non-selective– Aminophylline

• Selective • Enoximone• milrinone • Levosidan

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Receptor Sites• Adrenergic

Receptors

• Alpha 1• Alpha 2• Beta 1• Beta 2

• Dopamergic receptors

• D1• D2• D3• D4• D5

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Main classes of Adrenoceptor

receptors 1

• Located in vascular smooth muscle• Mediate vasoconstriction

2

• Located throughout the CNS, platelets• Mediate sedation, analgesia & platelet

aggregation

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Main classes of Adrenoceptor receptors

1• Located in the heart• Mediate increased contractility & HR

2• Located mainly in the smooth muscle of bronchi• Mediate bronchodilatation• Located in blood vessels

– Dilatation of coronary vessels– Dilatation of arteries supplying skeletal muscle

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Epinephrine (Adrenaline)• Stimulates & receptors

– Predominantly effects at low doses and effects at high doses

• Clinical uses– Cardiac arrest– Anaphylaxis– Low cardiac output states– Upper airway obstruction– Combination with local anaesthetics

• Side effects– Dysrhythmias– Increase in myocardial oxygen consumption

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Norepinephrine

• Predominantly stimulates 1 receptors• Most commonly used vasopressor in

critical care• Very potent• Administered by infusion into a central vein• Uses

– Hypotension due to vasodilatation– Septic shock

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Dopamine

• Effect dose dependent– Direct

• Low dose - 1

• High dose - 1

– Indirect• Stimulates norepinephrine release

• D1 receptors– Vasodilatation of mesenteric & renal circulation

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Dobutamine

• Synthetic• Predominantly 1

• Small effect at 2

• Uses– Low cardiac output states– Cardiogenic shock

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Thanks for your kind attention

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Management of Cardiac Arrhythmias after Open-Heart

Surgery

Mr. Justin Bika; Discussant: Sr. Isidora Jaff

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• SA Node• Inter-nodal and inter-atrial pathways• A-V Node• Bundle of His• Perkinje Fibers

Conduction System

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• Normal– Heart rate = 60 – 100 bpm– PR interval = 0.12 – 0.20 sec– QRS interval <0.12– SA Node discharge = 60 – 100 / min– AV Node discharge = 40 – 60 min– Ventricular Tissue discharge = 20 – 40

min

Physiology

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• Cardiac cycle– P wave = atrial depolarization– PR interval = pause between atrial and

ventricular depolarization– QRS = ventricular depolarization– T wave = ventricular depolarization

Physiology

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Arrhythmias

• Definition: Heart rhythm problems (arrhythmias) occur when the electrical impulses in your heart that coordinates your heartbeats don't function properly, causing your heart to beat too fast, too slow or irregularly.

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Arrhythmias

● Arrhythmias may cause sudden death, syncope, heart failure, dizziness, palpitations or no symptoms at all.

● There are two main types of arrhythmia:bradycardia: the heart rate is slow (< 60 b.p.m). tachycardia: the heart rate is fast (> 100 b.p.m).

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Pathophysiology of Arrhythmias.• Arrhythmias:- Heart is beating too fast, - Heart is beating too slow, - Heart is beating irregularly.• Two types of arrhythmias; Bradycardia & Tachycardia• Bradycardia; Heart is beating too slow. Two causes: 1) SA node is either slowed or absent. 2) Blockage of conduction at the AV node 3) types of Heart blocks)• Tachycardia; Heart is beating too fast, Causes: 1) Increased Pace maker Activity from the SA node 2) Re-entry Tachycardia 3) Delayed Repolarization

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P & P Ward experience & Mgtw.r.t types arrhythmias .

• Sinus node Dysrhythmias:• Sinus bradycardia• Sinus tachycardia• Atrial Dysrhythmias :• Premature Atrial Complex (PAC)• Atrial Flutter• Atrial fibrillation• Junctional dysrhythmias• junctional rhythm• Ventricular Dysrhythmias:• Premature Ventricular Complex (PVC)• Ventricular Tachycardia• Ventricular Fibrillation• Ventricular Asystole• Conduction Abnormalities:• First-Degree Atrioventricular Block• Second-Degree Atrioventricular Block, type 1• Second-Degree Atrioventricular Block, type 2• Third-Degree Atrioventricular Block

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THANKS FOR YOUR FERVENT ATTENTION

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One -Year Experience in Cardiac Surgery at The Shisong Cardiac Center

Dr. Charles Mve Mvondo ; Discussants: Dr. Jean Claude Ambassa, Dr Jacques Cabral

Tantchou Tchoumi

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Open Discussion

1. Cardiologists Point of View

2. General Discussions

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Conclusion “CARDIAC SURGERY CAN BE SIMPLY AWEFULLY CARDIAC SURGERY CAN BE AWEFULLY SIMPLY”

Thank you so much. Happy Christmas & prosperous New Year 2014

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Let’s Have a common Picture outside

to commemorate this day