Chest Pain: EMS Review

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Chest Pain EMS Implications Wayne Guerra MD, MBA Porter/Littleton/Parker Adventist EMS Pain is inevitable; suffering is optional.

description

Chest pain and implications for EMS. Review the history, physical and treatment of chest pain. Learn the most important causes of chest pain in the EMS setting and see great EKG examples of MI and the EKG mimics of cardiac ischemia.

Transcript of Chest Pain: EMS Review

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Chest Pain EMS Implications

Wayne Guerra MD, MBAPorter/Littleton/Parker Adventist EMS

Pain is inevitable; suffering is optional.

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Before I refuse to take your questions, I have an opening statement.Ronald Reagan

Wayne Guerra MD, MBA

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Objectives

• Improve EMS History and Physical• Expand and Focus Differential Diagnosis• Review EMS Treatment• Avoid Pitfalls• Learn A Zebra or Two

Experience is something you don't get until just after you need it.

Wayne Guerra MD, MBA

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The Father of Medicine

patient confidentiality

physicians record their findings and their medicinal methods

rejected the superstition and magic

460 BC to 380 BC

Wayne Guerra MD, MBA

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To EMS or Not EMS?

• Private transport: 35 minutes• EMS: 39 minutes• Definitely EMS

http://heartdisease.about.com/cs/heartattacks/a/chestpainEMS.htmWayne Guerra MD, MBA

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www.iTriageHealth.comWayne Guerra MD, MBA

Differential Diagnosis of Chest Pain is Vast

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Possible Causes

• ACS– MI– Unstable Angina

• PE• Aortic Dissection• Esophageal Rupture• Tension Pneumothorax

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History

• Establishing Prevalence• Who, What, Where, How, Why• Chest pain

– Typical– Atypical (One study of 430,000 33% had no CP!)

JAMA. 283(24):3223–3229, 2000Wayne Guerra MD, MBA

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“Atypical” Chest Pain

• Females• Diabetics• Elderly

– 14% in < 65 years– 21 % in 65-74 years– 32% in >= 75 years

Circulation, III: e435-e437, 2005 Wayne Guerra MD, MBA

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“Atypical” Chest Pain

• 23% burning (indigestion) • 5% as sharp and stabbing• 6-9% as positional or pleuritic• 26% SOB

Arch Intern Med. 145: 65-69, 1985. Wayne Guerra MD, MBA

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“Atypical” Chest Pain

• Back, shoulder, neck• Abdomen• N/V

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“Atypical” Chest Pain

• Diaphoresis• Syncope• Palpitations • “Weakness”• “Falls”

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History

• Past Medical History• Medications (Bring all into ED)

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History

• Past Surgical History• Recruit others for information• Recent illnesses• Social history

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Physical

• Vitals are vital• Heart• Lungs • Pulses• Abdomen• Neuro

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BP Discrepancy

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Diagnostics

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EKGGet One You Can Read!

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Stratergery For A Good Tracing

• Do before transport• Give pain medications if possible• Encourage patient to relax• Ensure good lead contact

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Importance of CVD an ACS• Second most common complaint in ED• 2-5% ACS missed

http://bit.ly/hkAZEWayne Guerra MD, MBA

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EKG Mimics of MI

• Left Ventricular Hypertrophy• Early Repolarization• Acute Pericarditis• Left Ventricular Aneurysm

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LVH With Strain• ST elevation and depression• T wave changes

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Early Repolarization

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Acute Pericarditis

PR depression

ST ElevationWayne Guerra MD, MBA

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LV Aneurysm

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ECG FindingAcute Pericarditis Myocardial

InfarctionEarly Repolarization

ST-segment shape Concave upward Convex upward Concave upward

Q waves Absent Present Absent

Reciprocal ST-segment changes

Absent Present Absent

Location of ST-segment elevation

Limb and precordial leads

Area of involved artery

Precordial leads

ST/T ratio in lead V6

>0.25 N/A <0.25

Loss of R-wave voltage

Absent Present Absent

PR-segment depression

Present Absent Absent

EKG Changes: Mimics of MI

Dave Sanko: ACS and 12 Lead Review Wayne Guerra MD, MBA

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AMI Localization

aVF inferiorIII inferior V3 anterior V6 lateral

aVL lateralII inferior V2 septal V5 lateral

aVRI lateral V1 septal V4 anterior

Dave Sanko: ACS and 12 Lead Review Wayne Guerra MD, MBA

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Acute Anterior MI

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Acute Ant-Lat MI

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Acute Inferior MI

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Acute Posterior MI

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Acute Right Ventricular MI

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Cardiac Alert

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Acute MI and LBBB

1) ST-segment elevation measuring 1 mm in the same direction with the QRS in any lead.2) ST-segment depression measuring 1 mm in any of the V1 through V3

leads.

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Unstable angina

• Includes non-Q wave MI• New pattern of angina• Angina at rest• 30 day death rate: 3.5%• 30 day MI rate: 8.5%

http://bit.ly/PlrtS Wayne Guerra MD, MBA

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Pulmonary Embolus

• 650,000 cases annually• 3rd most common cause of death• 1st or 2nd most common unexpected death• 10% die within 60 minutes

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Pulmonary EmbolusIncreased Risk

• Virchow’s Triad– Venous stasis– Hypercoagulability– Inflammation

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Pulmonary Embolus

• Pleuritic CP: 74%• Risk factors:

– Pregnancy and post partum– BCPs– Malignancy– Surgery– Immoblization– Inherited hypercoagulability

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Pulmonary Embolus

• Signs– Tachypnea (>16) 92%– Rales 58%– Tachycardia (>100) 44%– Fever (>100 ⁰F) 43%– Diaphoresis 36%– Signs of DVT 32%

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S1Q3T3

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Aortic Dissection

• Characteristic description

• Increased risk• BP differential• Murmur• Can be associated with

acute MI

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Aortic Dissection

• EMS treatment• ED treatment• Hospital treatment

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Esophageal Rupture

• Baron von Wassenaer• Boerhaave’s syndrome (Spontaneous)• Most are iatrogenic

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Esophageal Rupture

• Forceful vomiting• 50% have GERD• Severe chest/epigastric pain• Other sxs depending on time

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Esophageal Rupture

• Physical– Subcutaneous emphysema (60%)– Mackler triad (vomiting, CP, SubQ emphysema)– Tachycardia/tachypnea– Hamman sign (crunching sound over heart)– Decreased breath sounds

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Esophageal Rupture

• EMS Treatment– O2– IV fluids– Pain meds– Check lactate (May appear septic)

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Tension Pneumothorax

• One way valve• Pathophysiology

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Tension Pneumothorax

• Sudden CP & SOB• Tachys• Hypos• breath sounds• Tracheal deviation• JVD• Sub-Q emphysema

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Tension PneumothoraxEMS Treatment

• O2• Needle thoracostomy• IVFs• +/- intubation

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Needle Thoracostomy

• Iodine prep• 14/16 Ga catheter,

4.5cm minimum• Just superior 3rd rib 1-2

cm from sternum• Listen for hissing sound• Flutter valve or

stopcock

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Case 1

• 32 yo female with crushing cp, sob and diaphoresis

• Meds/PMH/Soc Hx/Past Surg Hx all negative• 120/70, 90, 18, RA Sat=97%

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Case 1

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Case 1: EKG After 1 NTG

Diagnosis?Wayne Guerra MD, MBA

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Prinzmetal's Angina

• Coronary artery spasm• Typically occurs at rest• 2/3rds have CAD• Spasm can be induced during angiogram• Rx with nitrates and Ca channel blockers

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Case 2

• 50 yo male pressure like chest pain • PMH: DM, Htn, Elevated cholesterol• Meds: Insulin, HCTZ, Tenormin, Lipitor• 130/70, 70, 18, RA Sat=96%• Exam: nl

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EKG with CP

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Pain Free After 1 NTG

Diagnosis?Wayne Guerra MD, MBA

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Wellens Syndrome• Isoelectric or minimally ST followed by

concave or straight ST and a symmetrically inverted T wave

• Most common V2-V3, and V4-V6• Highly suggestive critical LAD stenosis

http://bit.ly/15RAYb

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Wayne Guerra MD, MBA