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I. DEGENERASI DAN NEKROSIS
• 1. Degenerasi melemak
• 2. Degenerasi hidropik
• 3. Degenerasi hyalin• 4. Sel nekrosis
• 5. Autolosis post mortum
• 6. Apoptosis• 7. Inclusion odies
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MEMBUAT DIAGNOSA
MORFOLOGIS
1 SEVERITY
2 DISTRIBUTION
3 DURATION
4 LESION TYPE
5 TISSUE INVOLVED
1 NAMA ORGAN
2 TIPE LESI
3 DURASI
4 DISTRIBUSI
5 DERAJAT
KEHEBATAN
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MEMBUAT DIGNOSA
MORFOLOGISS!"!#!#I$%
DIS$#I&'$I()
D'#A$I() *!SI() $%+! $ISS'!I)"(*"!D
S!"!#! ,%+(+*ASIA -!#!&!**I
S!"!#! +!#I*(&'*A# A-'$! A$$% D!/!) *I"!#
S!"!#! 0'*$I(-A* -,#()I- $'&!#-'*('S ,!+A$I$IS
0(D!#A$! 0'*$I(-A* A-'$! )!-#($I- ,!+A$I$IS
0I*D (-A* S'&A-'$! $'0(# SI)
S!"!#! DI'S! -,#()I- $'0(# "'*"A"A/I)A
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Degenerasi melemak pada hepar
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degenerasi hidropik
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aryorrheis lymphocytes lien.
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Sel nekrosis piknosis
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Degenerasi hyalin
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Apoptosis cytoarchitecture ,! stain
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II. Kalsifkasi danPigmentasi
• 1. anthrakosis
• 2. hemosiderosis
• 3. melanosis• 4. Icterus
• 5. kalsiikasi metastatik
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Anthrakosis pigmen karon pada septa al8eoli
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III. Bentuk – bentuk
nekrosis
• 1. nekrosis koagulasi
• 2. nekrosis li9ue9kti
• 3. nekrosis caseosa
• 4. at necrosis
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)ekrosis koagulasi
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)ekosis
koagulasi
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D. Gangguanemodinamik !
sirkulasi• 1. ongesti hiperemia
• 2. +erdarahan hemoragi
• 3. (edema
• 4. $hromus
• 5. Inark
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Arterial thromi lapisan
gelap platelet irin
lapisan terang.
&ercampur dengan eritrosit
dan leukosit dalam lapisan
:ahn.
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INFARK
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". bentuk radang ! eksudates in
a#ut in$amasi
• 1. radang irinous serous akut• 2. radang catarrhal
• 3. radang purrulent suppurati8e
• 4. ulcera
&entuk radang kronis ;
1. #adang granulomatous caseosa
G. BENTUK_-BENTUK RADANG.
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1
2 3
4 56
7
<
Gambarkan bentuk sel radang dalam jaringan :
Sel radang !lim!r"!nuklear #mn$ :
Sel radang mononuklear 1 SD 7 ;
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!udation microscopic in acute inlammation
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!udation microscopic in acute inlammation.
Seen here is 8asodilation =ith eudation that
has led to an outpouring o luid =ith irin into
the al8eolar spaces along =ith +0)>s.
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A -hronic inlammation in the
lung sho=ing all three
characteristic histologic
eatures; 1 collection ochronic inlammatory
cells ? 2 destruction o
parenchyma normal al8eoli are
replaced y spaces lined y
cuoidal epithelium
arrowheads), and 3replacement y connecti8e
tissue irosis arrows).
B, &y contrast in acute
inlammation o the lung acute
ronchopneumonianeutrophils ill the al8eolar
spaces and lood 8essels are
congested.
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#adang serousirinous
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0ild acute dius katharrha pn
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0oderate dius suacute suppurati8e pneumonia
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I/'#! 1@22 P#$%&'(%)* $)+&),-) #. )),%$ 0, ))$0') (,* 0))$0')
0$-)0- -)'' 0,& N#%) %(% ('%#&8 )*&-)* #90*(%0)
:#$:#'(%0#, (,* ATP '))'$ () ( -),%(' #'); 0$-)0( -(, -(&$) *0)-%
)(,) *((8) ER; ),*#:'($0- )%0-&'&< =K;
-)(%0,) >0,($)< LDH; '(-%(%) *)*#8),($)< RNP; 0#,&-')#:#%)0,
=#(8&'(%0) (,* '0+&).(-%0) ,)-#$0$ A; K0*,) 0,.(-% )900%0,8 -#(8&'(%0) ,)-#$0$; ?0% '#$$ #.
,&-')0 (,* -'&:0,8 #. -%#:'($ &% ?0% :)$)(%0#, #. ($0- #&%'0,)$ #. 8'#)&'( (,* %&&'(
(-0%)-%&) B; A .#-&$ #. '0+&).(-%0) ,)-#$0$ 0, %) >0*,) -(&$)* .&,8(' 0,.)-%0#, T) .#-&$ 0$
.0'')* ?0% ?0%) -)''$ (,* -)''&'( *)0$; -)(%0,8 ( ),(' ($-)$$ %(% #'0%)(%)$ %) ,#('
(-0%)-%&)
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,ere are t=o pulmonary granulomas. /ranulomatous inlammation
typically consists o mitures o cells including epithelioid
macrophages giant cells lymphocytes plasma cells and
irolasts. $here may e8en e some neutrophils.
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B(0,; ':#-%0- :)0($-&'( -&..0,8
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'l
ulcer
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'lceraulcer
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