Week 3 SportInMedicine - 30 Agustus 2012
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Transcript of Week 3 SportInMedicine - 30 Agustus 2012
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Musculoskeletal in sports medicine
John Butarbutar
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Sports Medicine
• Etiology, diagnosis, treatment and prevention of disorders and injuries of athletes, as well as with the ideal methods of athletic training
Salter, Textbookof Disorders and Injuries of teh musculoskeletalSystem
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Muscle disorder & injury
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Hamstring strain
Composed of • biceps femoris• semitendinosus • semimembranosus
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History
A noncontact injury , either acute or insidious onset. Strain injuries frequently are seen in athletes who run, jump, and kick. Avulsion injuries are seen in patients who participate in water-skiing, dancing, weight lifting, and ice-skating. The patient may report a popping or tearing sensation.
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Physical
• Tenderness over the site of injury• Ecchymosis• A palpable defect may be felt with severe
strains, but swelling and the deep location of the muscle may obscure this finding in the acute stage.
• Pain with passive extension of the knee and with resisted knee flexion
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Imaging
• Radiographs can rule out an avulsion injury from the ischial tuberosity
• When a confirmation or grading of a hamstring strain is necessary, magnetic resonance imaging (MRI) is the most sensitive test
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3 grades of severity of hamstring injuries.
• First-degree strain is the result of stretching of the musculotendinous unit and involves tearing of only a few muscle or tendon fibers.
• Second-degree injury refers to a more severe muscle tear without complete disruption of the musculotendinous unit.
• Third-degree injury refers to a complete tear of the musculotendinous unit.
The American Medical Association (AMA)
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treatment
• Protection, rest, ice, immobilisation , gradual mobilization, muscle stretching and strengthening
• Surgery
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Test
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Etiology
– Strength – Flexibility– Warm up– Fatique
Worrell, T.W., & Perrin, D.H. (1992)Journal of Orthopaedic and Sports Physical Therapy, 16, 12-18.
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Motor units
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2- The Mechanism of Force Generation in Muscle
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Fiber Type ComparisonType I Type IIa Type IIb
ShorteningSpeed
slow fast fast
Energy System oxidative oxidative,glycolytic
glycolytic
Size small large largeForceProduction
low high high
AerobicCapacity
high medium low
AnaerobicCapacity
low medium high
Fatigability low medium high
Tissue
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Greatest Riska) 2-joint musclesb) muscles that limit ROMc) muscles used eccentrically
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Which consequences do you expect ?• Myasthenia gravis: autoimmune disease where antibodies
against the Ach receptors are produced• Muscular dystrophy: some proteins forming the muscle fibers
are abnormal. • Curare binds to the Ach receptor without activating them.
What are the effect of curare on the skeletal muscle? • The botulism toxin prevents the release of the
neurotransmitter into the synapse. • Nerve gas inhibits acetylcholinesterase present in the
synapse.
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• Rigor mortis: why does the body stiffen shortly after death?
• How do muscles become fatique after several contraction ?
• What are the different effects between high intensity and long sustained exercise to the muscle ?
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Tendon disorder and
injury
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Achilles Tendon Injury
• Common site for injury and rupture • Symptoms include posterior heel pain with
stiffness• The tendon is thickened• Neovascularization might be involved in the
pain mechanism
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mechanism
• Pushing off with the weight bearing forefoot while extending the knee (53%)
• Sudden unexpected dorsiflexion of the ankle (17%)
• Violent dorsiflexion of a plantar flexed foot (10%)
Arner and Lindholm ,Acta Chir Scandinavia ,1959
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Examination
• Diffuse edema, bruise, palpable gap, 2-6 cm proximal to the insertion
• Calf squeeze test• Knee flexion test
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presentation
• Sudden pain in the affected leg (feels like struck or kicked )
• Audible snap• Weakness of affected ankle
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imaging
• MRI and US are methods of choice• Treatment• Rest, acitivy modification, NSAIDs,physical
therapy, surgical debridement, eccentric muscle training, bracing, heel inserts(Tasto et al 2003, Koenig et al 2004)
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treatment
• Conservative with cast or splint• surgery
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Pain mechanism
• Overuse and degeneration• Local hypoxia, micro trauma, impaired wound
healing may contribute• Pathology linked to degradation of collagen
and hypercellularity• Tendon contains no inflammatory cell
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Tendon healing
• Occurs through extrinsic and intrinsic processes
• Divided into 3 phases– Inflammation (Day 0-7)– Repair (Day 3-60)– Organisation and remodelling (Day 28-180)
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Organisation (28-180 days)
• Final stability acquired during this phase by the normal physiological use of the tendon
• Accompanied by cross linking between fibrils further increasing tendon tensile strength
• Complete regeneration never achieved– Defect remains hypercellular– Thinner collagen fibrils
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• What is pain mechanism in rotator cuff tendinosis?
• How do you examine lateral epicondylitis, de quervainn, biceps tendonitis ?
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Ligament disorders and injury
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Anterior cruciate ligament rupture
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Injury Mechanism
• 50-70% are noncontact injuries
• These usually involve planting, cutting and jumping
• This also commonly involves imbalances of body weight and malalignment of body position
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History
• Noncontact injury• Audible pop• Immediate
hemarthrosis
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Examination• Hemarthrosis• Lachman Test– Done at 20-30 degrees
of knee flexion
• Pivot shift Test• Drawer test
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Lachman Test
1+ 0-5 mm of anterior displacement - sometimes with an end point 2+ 5-10 mm of anterior displacement - with no end point 3+ 10 mm of anterior displacement - with no end point
Comparison to other side is important
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Xray Findings
• Tibial Spine Avulsion Fracture 8%
• Radiology. 1993 Jun;187(3):821-5.
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MRI Findingsany discontinuity or signal change in the ligament is
indicative of ACL tear;
indirect signs of ACL tear: always look for signs of additional injury (meniscal tear, PCL tear,
LCL tear);
Pivot shift injury: combination of signal changes in lateral femoral condyle and posteror lateral tibial plateau:
abnormal slope of ACL
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MRI Findings
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MRI Findings
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Treatment Options
• Surgery: Reconstruction– Restores stability, prevents further injury
• Conservative Treatment– older patients w/ isolated ligament injury who are
willing to moderate their activity will find non-operative treatment to be satisfactory in the majority of cases (over 80%)
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Ligaments
• Made of dense fibrous tissue with the fibers in roughly parallel lines in the direction of functional need
• In most cases pliable but inelastic• Bind bones to bones• Supply passive support and guidance to joints
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Ligaments
• May be so integrated into the joint capsule that they are indistinguishable as separate structures
• Reinforce joint stability• Generally loose enough to allow free joint
movement within a desirable range• Designed to prevent movement in a range
which would be damaging
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Ligaments
• Blood supply– Poor in the ligament itself– Ligaments depend on diffusion to supply inner
fibers with nutrients– Vascular damage during injury can be particularly
bad for the healing of ligaments– Blood flow is facilitated by passive movements
after injury or surgery
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Ligament Stress-Strain Curve
toe elastic plastic
failureyield point
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Sensory Receptors in Ligaments
• May supply feedback to muscle tissue • Allow muscles to be recruited to assist in joint
stability
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Failure Mechanisms
• Ligamentous Failure – The ligament itself fails– Characteristic of fast loading rates
• Bone Avulsion – The bone itself fractures or fails beneath the junction of ligament and bone– Characteristic of slow loading rates
• Failure at the Ligament-Bone Junction– The least common type of failure