Vertigo - Ministry of Healthmohs.gov.mm/ckfinder/connector?command=Proxy&lang=en&type=… ·...
Transcript of Vertigo - Ministry of Healthmohs.gov.mm/ckfinder/connector?command=Proxy&lang=en&type=… ·...
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DR SAN MAY THEIN
Lecturer/Consultant ENT Surgeon
Otorhinolaryngology,Head and Surgery Department
North Okkalapa Teaching and General Hospital
Vertigo Clinical Work Up for Vertigo
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Introduction
Balance disorders are common and may be
encountered by a variety of specialists.
Although rarely life threatening, they may be
associated with significant morbidity.
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dizziness is one of the most common complaint
four types
Vertigo (most prevalent type)
Lightheadedness
Pre syncope
Dysequilibrium
It is important to make distinction between true
vertigo from other dizzy sensation.
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Vertigo
Vertigo - derives from a Latin word ‘vertex’ that
means ‘to turn around’.
Vertigo - an illusion of movement either of self
(subjective vertigo) or the environment (objective
vertigo) due to disturbance in peripheral or central
vestibular system
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Dizziness - a lay term & means non specific
lightheadness, faintness, or giddiness & is common
in many general medical disorders s/a CVS,
anemia, GI d/s & endocrine d/o.
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Anatomy & Physiology
Vestibular sense o/g consists of three semicircular
canals, the saccule & utricle.
These are membranous tubes within the dense
temporal bones.
The membranes are fluid filled & have cells with
cilia which bends as the fluid moves relative to
them.
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This excites or depresses the nerve cells & alters
the tonic input into the brain.
Semicircular canals are at right angles to each
other & detect changes in angular acceleration.
Utrical & saccule have otoconia embedded in a gel
overlying the cilia & detect linear acceleration.
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Central connection of vestibular N
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Balance - visual input 70%
- proprioception 15%
- vestibular system 15%
The brain stem computerized these three inputs &
with the help of cerebellum maintains the balance
& coordination of head & body.
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Causes of Vertigo
Peripheral vestibular causes
Central vestibular causes
Other causes
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Peripheral vestibular causes
- acute labyrinthitis
- acute vestibular neuronitis
- BPPV
- cholesteatoma
- otosclerosis
- herpes zoster oticus
- Ménière's disease
- perilymphatic fistula
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Central vestibular causes
- CP angle tumour
- CVA
- Migraine, Epilepsy
- Multiple sclerosis
Other causes
- cervical vertigo
- drug-induced vertigo
- psychological
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Epidemiology
Studies show that about one third of cases of
dizziness are vertigo.
Prevalence estimate for vertigo are 4.5%.
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because patients with dizziness often have
difficulty in describing their symptoms,
determining the cause can be challenging
an evidence-based approach using knowledge of
key historic, physical examination and
radiologic findings for the causes of vertigo can
help establish a diagnosis and consider
appropriate treatments in most cases
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History
It is often difficult to describe their sensation
eg.spinning sensation
must determine whether pt truly has vertigo versus
another type of dizziness
next task is to determine whether pt has a
- peripheral (or)
- central cause of vertigo
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Full description of sensation
- timing & duration
- provoking factors
- associated symptoms
- previous H/O trauma, medical d/s, drug h/o
& alcohol ingestion
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Typical duration of symptoms for different causes of vertigo
Duration of episode
Suggested diagnosis
A few seconds Peripheral cause; a/c vestibular
neuronitis, Ménière's disease
Several seconds – few minutes BPPV, perilymphatic fistula
Several minutes – 1 hr TIA, perilymphatic fistula
Hours Ménière's , perilymphatic
fistula, Migraine, Acoustic
neuroma
Days A/c vestibular neuronitis,
stroke, migraine, multiple
sclerosis
Weeks Psychogenic
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Provoking factors for different causes of vertigo
Provoking factors Suggested diagnosis
Changes in head position A/c labyrinthitis, BPPV, CP angle
tumour, multiple sclerosis,
perilymphatic fistula
Recent upper resp viral illness A/c vestibular neuronitis
Stress Psychiatric, migraine
Immunosuppression Herpes zoster oticus
Changes in ear pressure, head
trauma, straining, loud noise
Perilymphatic fistula
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Associated symptoms for different causes of vertigo
Symptom Suggested diagnosis
Aural fullness Acoustic neuroma, Ménière's d/s
Ear or mastoid pain Acoustic neuroma, acute middle ear d/s
Facial weakness Acoustic neuroma, herpes zoster oticus
Focal neurological findings CVA, CP angle tumour, multiple sclerosis
Headache Acoustic neuroma, migraine
Hearing loss Ménière's d/s, perilymphatic fistula,
acoustic neuroma, cholesteatoma,
otosclerosis, CVA, Herpes zoster oticus
Imbalance A/c vestibular neuronitis, CP angle
tumour
Nystagmus Peripheral or central vertigo
Photophobia Migraine
Tinnitus A/c labyrinthitis, acoustic neuroma,
Ménière's d/s
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Causes of vertigo associated with hearing loss
Diagnosis Characteristics of hearing loss
Acoustic neuroma Progressive, unilateral, sensorineural
Cholesteatoma Progressive, unilateral, conductive
Herpes zoster oticus Subacute to acute onset, unilateral
Ménière's d/s Sensorineural, initially fluctuating affecting
lower frequencies, later – progressive,
affecting higher frequencies
Otosclerosis Progressive, conductive
Perilymphatic fistula Progressive, unilateral
TIA Sudden onset, unilateral
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Distinguishing characteristics of peripheral versus central causes of vertigo
Feature Peripheral vertigo Central vertigo
Nystagmus Combined horizontal and
torsional inhibited by
fixation of eyes onto object
Purely vertical,
horizontal or torsional,
not inhibited by
fixation of eyes
Imbalance Mild to moderate, able to
walk
Severe, unable to stand
still or walk
Nausea, vomiting May be severe Varies
Hearing loss & tinnitus Common Rare
Non auditory neurologic
symptoms
Rare Common
Latency following
provocative diagnostic
maneuver
Longer (up to 20 seconds) Shorter (up to 5
seconds)
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Physical examination
An otological & neurological exam: is mandatory in
all cases of vertigo.
A general medical exam: may be required if the
symptoms dictate.
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Specific clinical exam of vestibular
pathology
Exam of ext ear & TM together with clinical T of
auditory acuity & tuning fork T
Vestibulo-occular T
Vestibulo-spinal T
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Vestibulo-occular T
Most essential part of investigation of vestibular
system
Caloric Test
Hallpike maneuver
Electronystagmography ( ENG )
Rotation Test
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Vestibulo-occular T
Caloric T
Corner stone of Ivt for any vestibular pathology
Classic bithermal calorics utilize H2O at 30-44C in two
heated tanks about 1m a/b T couch
Pt recline on T couch at 30D a/b horizontal ( lat
semicircular canal vertical)
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A/f checking that ext canal are clear of wax & debris &
TM intact, cold H2O (30C) run into Lt ear, via a siphon
tube & 14G canula, for 40S
Stop watch is used to record the time period from start
to nystagmus stops with pt fixing on a point on ceiling
Repeat Rt ear & then both ear with warm H20 (44C)
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Frenzel’s glasses to remove optic fixation
Measuring of nystagmus electrically
(electronystagmography)
Interpretation
Cold stimulation
fast phase of nystagmus opposite side
Warm same side
Most common abnl - canal paresis
- directional
preponderance
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Unilat canal paresis
Response of one side is reduced or (-) compare with
opposite site
lesion of peripheral vestibular system
( eg. Horizontal canal or vestibular N )
Directional preponderance
Non specific enhancement of nystagmus in one
particular direction
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It suggest pathology, but is usually non-localizing
( may arise from any part of peripheral or central
vestibular system)
Hallpike T
Positional nystagmus is best elicited
Pt - sitting on a bed & procedure is explained
(reassurance)
Pt - keep eyes open & looks straight ahead
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Head - held firmly b/t examiner’s hands & turn 45D to
Lt or Rt
Pt - rapidly laid backwards, head over edge of bed,
30D b/l horizontal
Eyes - observed for nystagmus
If neither occur a/f 30 S then pt - return to upright
position & again asked for vertigo & exam; for
nystagmus
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Difference b/t nystagmus of central &
peripheral origin
central peripheral
Latent period ( - ) ( + ) 2-20 s
vertigo ( - ) ( + )
fatigue ( - ) ( + )
Direction Variable Undermost ear
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Rotation T
Nystagmus - induced by acceleration & deceleration
in a rotating chair
Disadv - stimulating both labyrinth simultaneously
Limited clinical application
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Vestibulo-spinal T
Romberg T
Unterberger T
Gait Testing
Posturography
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Romberg T
To assess pt ability to stand
Feet together, arms by the side, with eyes open &
then closed
Pt fall towards the side of peripheral vestibular
lesion
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Unterberger T
Pt - marching up & down on the spot for 30 sec
With eyes closed & arms outstretched in front, with
hands clasped together
Body rotation of >30D , or forward or backward
displacement of > 1 m - abnormal
NL pt - less tendency to deviate to Rt or Lt than pt
with peripheral or central vestibular d/o.
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Gait Testing
Assess by watching pt walk normally with eyes open
& then closed
Hemiplegic gait, cerebellar ataxic gait, parkinsonian
shuffle, or high stepping gait with loss of
proprioception
With eyes closure pt with uncompensated vestibular
lesion will veer towards the affected side
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Posturography
Recording of postural sways
Most commonly used are force platforms
Effects of various sensory modalities upon balance to
be identified
Some claimed - various pathological conditions to be
differentiated
Their use is prohibited in both research & clinical
practice d/t high cost
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Other investigation
Laboratory evaluation
electrolytes
glucose
blood counts
thyroid function
identify the aetiology of vertigo in fewer than 1%
of pt with dizziness
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Radiologic studies
MRI for CNS lesion
Conventional radiographs or CT for cervical
vertigo
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