Vertigo

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VERTIGO -- Prashiddha Dhakal MBBS,KUSMS

description

Vertigo --- Perception of false rotatory movements

Transcript of Vertigo

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VERTIGO

-- Prashiddha Dhakal MBBS,KUSMS

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Vertigo

• “Illusion” or “Hallucination” of movement.

Abnormal sense of motion

between patient & surrounding

PLUS

Loss of balance

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• Patient often complain of DIZZINESS/GIDDINESS which is a vague term & mayn’t always mean vertigo.

• Most dizzy patients can be placed in to one of four categories:

1. True Vertigo (50%)2. Pre- syncope3. Dysequillibrium4. Vague lightheadedness

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True Vertigo

•Loss of balance

PLUS

Abnormal sense of motion between patient & surrounding

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Pre-syncope

• Transient sensation that a faint in about to occur.

• May present as nausea ,weakness, SOB or change in vision.

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Dysequilibrium

• Loss of balance

• No sense of faintness.

• No abnormal sense of motion.

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Vague lightheadedness• Head discomfort due to:

1.Psychiatric disorders

2.Hyperventilation syndrome

3.Encephalopathies

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How do we maintain equilibrium?

Visual input

Proprioceptiual

input

Vestibular input

labyrinths.

equilibrium

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Sensory cortex

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Causes of Vertigo

Central Peripheral

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Evaluation of a patient with vertigo

1. Clinical tests

2. Laboratory tests

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Clinical tests of vestibular function

1. Spontaneous Nystagmus

• Nystagmus- Involuntary,

rhythmic, oscillatory

movements of eyes.• Procedure--• May be horizontal,

vertical or rotatory• Direction of nystagmus—

direction of fast component

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• Nystagmus of peripheral origin:

Suppressed by- Looking at a fixed pointEnhanced by- Darkness or Frenzel glasses (+20 D), both of which abolish optic

fixation

Frenzel Glasses

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2. Fistula Test

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• Normally, the test is NEGATIVE• POSITIVE--- Erosion of horizontal semicircular

canal(Cholesteatoma), Abnormal opening in oval window(Poststapedectomy fistula) or round window (Rupture of round window membrane)

• FALSE NEGATIVE--- When cholesteatoma covers the fistula

• FALSE POSITIVE--- Congenital Syphilis & Meniere’s disease (25% cases)

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3. Romberg Test

• Peripheral lesion- Patient sways to the side of lesion

• Central lesion- Patient doesn’t sway

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• Sharpened Rhomberg test If the Rhomberg test is

normal, this test is performed.Inability to perform this test

indicates vestibular impairment.

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4. Past-Pointing

Past pointing, Falling and the Slow component of nystagmus are all in the same direction

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5. Hallpike Manoeuvre (Positional Test)

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6. GaitPeripheral lesion- Patient deviates to affected

side.

7. Test of cerebellar function

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LAB TESTS OF VESTIBULAR FUNCTION

• A. caloric test• 1. modified kobrak test• 2. fitzgerald-hallpike test• 3. cold air caloric test• B. Electronystagmograph• C. optokinetic test• D. rotation test• E. galvinic test• F. posturography

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Caloric test

• Test of lateral semicircular canal• Induce nystagmus by thermal stimulation• Each labyrinth can be tested separately

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Modified kobrak test

• Ear is irrigated with icewater for 60 s• First with 5 ml then with 10,20 and 40• If no response to 40 ml ,it indicates dead

labyrinth

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Fitzgerald-hallpike test

• ears are irrigated for 40 s alternately with water at 30 and 44 degree celcious

• Time taken is recorded in calorigram• If no nystagmus, repeated at 20 degree

celcious for 4 min• Depending on response of caloric test • a.canal paresis• b.directional preponderance

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• A.canal paresis• indicates response illicted from particular

canal is less than that of opposite side• depressed function of ipslateral

labyrinth,vestibular nerve or vestibular nuclei

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b. Directional perponderance

• Duration of nystagmus irrispective to rt or left is considered.

• Right beating nystagmus is caused by LC and RW

• Left beating nystagmus is caused by RC and LW

• TOTAL RESPONSE = RW+RC+LW+LC

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FORMULA FOR RB AND LB NYSTAGMUS

• Right beating nystagmus=(LC+RW)/TR X100%

• Left beating nystagmus=(RC+LW)/TR X100%

• IF nystagmus >25 to 30% in one ear than other ,then it is called directional perponderance

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• DP=RT BEATING – LEFT BEATING/TR

• RVR(relative vestibular reduction)=(RC+RW-LC-LW)/TR

• RVR normally is <25%• RVR > 40 % seen in vestibular neuritis

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• 3. cold air caloric test• Done when there is perforation of tympanic

membrane• Dundas grant tube • Ethyl chloride is used to cool air

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Electronystagmography

• Recoding of nystagmus by caloric,positional,rotational or optokinetic stimulus

• Corneoretinal potential recorded

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C. OPTOKINETIC TEST

• EYE movement ellicted by tracking of moving field

• POSITIVE IN brainstem and cerebral lesion

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E. Rotational test

• Patient seated in Brany’s revolving chair• Rotated 10 turns in 20 second• Normally nystagmus is for 25 to 40 second• Used for pt with congenital anomalies where

calorie test is not possible•

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F. GALVANIC TEST

• VESTIBULAR TEST• Differentiate from end organ lession from

vestibular nerve• 1 mA current passed in one ear• Normally person sways towards anodal

current

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G. POSTUROGRAPHY

-- evaluate vestebular function by measuring postural stability

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VESTIBULAR NEURITIS

• A type of peripheral vestibular disorder • A common cause of spontaneous vertigo• Definition

– disorder in which there is sudden,– spontaneous, isolated, total or subtotal loss of

afferent vestibular input from one labyrinth

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• Etiology– Viral infection of vestibular nerve

• Selective neuron loss in vestibular ganglia

– Virus• Herpes Simplex virus type 1 (latent infection)

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• Clinical manisfeststions– Acute spontaneous vertigo– Nausea, Vomitting– Postural imbalance– SYMPTOMS ARE TYPICALLY AGGRAVATED BY HEAD

MOVEMENT AND MINIMIZED BY KEEPING HEAD STILL AND EYES CLOSED

– Symptoms gradually subside over the following days

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• In acute phase– Spontaneous horizontal torsional nystagmus

• Unidirectional• Quick phase towards unaffected side• Suppressed by visual fixation

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• Patients charcteristically rotate towards the affected side when attempting to march on the spot with their eyes closed – POSITIVE FUKUDA / UNTENBERGER TEST

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• Diagnosis– Clinical diagnosis– Investigations

• Subjective visual Horizontal (SVH) test• Electronystagmography• Caloric test• Contrast MRI

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• Differential Diagnosis– Cerebellar infarction

-labyrinthine infarction -autoimmune inner ear disease -minner’s disease

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Outcome and complication

• Symptoms gradually subsides• Often patient complains of• -oscillopsia• -postural imbalance

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Management

theraputic -corticosteroids(methylprednisolone) -antiviral(valacyclovir)Vestibular rehabilation therapy

Early mobilization

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Thank You