Tricuspid Valve Disease

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Tricuspid Valve Disease Dr M C Mayet

description

Tricuspid Valve Disease. Dr M C Mayet. Introduction. Rarely affected in isolation Primarily extracardiac symptoms Closely related to right venticular function Influenced by left sided pathology Difficult to evaluate. Anatomy. Changes in tricuspid annulus area. Tricuspid orifice. - PowerPoint PPT Presentation

Transcript of Tricuspid Valve Disease

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Tricuspid Valve Disease

Dr M C Mayet

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Introduction

• Rarely affected in isolation• Primarily extracardiac symptoms• Closely related to right venticular function• Influenced by left sided pathology• Difficult to evaluate

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Anatomy

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Changes in tricuspid annulus area

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Tricuspid orifice

12.1%

15.3%

16.6%

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Saddle shape of the tricuspid annulus AS- anteroseptal commissure AM- midpoint base anterior leaflet AP- anteroposterior commissure PM- midpoint base of posterior leaflet SP- posteroseptal commissure SM- midpoint base of septal leaflet

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Leaflets

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Anatomical relationships

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PathophysiologyTraditional

• Organic• Functional

Carpentier

• type I—normal leaflet motion with annular dilation

• type II—increased leaflet movement due to leaflet prolapse secondary to chordal rupture or elongation

• type III—reduced leaflet motion due to leaflet thickening, fused commissures, or leaflet tethering

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Organic tricuspid valve disease

• Infective endocarditis• Rheumatic fever• Degenerative (myxomatous)• Traumatic• Postinfarction• Carcinoid• Appetite suppressing drugs• Endocardial fibroelastosis• Lupus erythematosus• Tumors (myxoma)• Mediastinal fibrosis

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Rheumatic

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Tricuspid + ass. rheumatic mitral disease

• incidence varies widely• Echocardiographic 6% • Postmortem 33%• Surgical findings 11%

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Organic tricuspid valve disease

• Infective endocarditis• Rheumatic fever• Degenerative (myxomatous)• Traumatic• Postinfarction• Carcinoid• Appetite suppressing drugs• Endocardial fibroelastosis• Lupus erythematosus• Tumors (myxoma)• Mediastinal fibrosis

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Functional tricuspid regurgitation

Normal Pathological

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Functional tricuspid regurgitation

Normal annulus

Dilated annulus

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Clinical features- tricuspid regurgitation

Right sided failure• ascites• hepatosplenomegaly• pulsatile liver • pleural effusions • peripheral edema Late stages• cachexia• cyanosis• Jaundice Atrial fibrillation

Jugular venous distention

• s wave/fused c & v waves

• prominent y descent Cardiac examination • S3

• P2

• pansystolic murmur

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Clinical features- tricuspid stenosis ↑ right atrial pressure• distention of the jugular

veins • ascites • pleural effusion• peripheral edema Jugular venous pulse • prominent a waves ↓cardiac output

cardiac murmur • mid-diastolic• increases with

inspiration• is heard maximally

along the left sternal border

• opening snap

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Diagnosis

• Symptoms usually minimal & overshadowed by left-sided symptoms

• Extracardiac symptoms- venous engorgment• Disease mostly silent• Diagnosis previously considered difficult

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Apical 4-chamber transthoracic echocardiography

Normal TV annulus

Normal mitral valve

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TTE

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Essential preoperative info• Presence of tricuspid valve disease • Organic, functional, or mixed • Degree of regurgitation• Direction of the regurgitant jet • Pulmonary artery peak and mean pressures • Transvalvular pressure gradients • Maximum & minimum annulus diameter • Systolic shortening • Leaflet thickness, mobility, billowing of the leaflet body, &

location of the prolapsing free edge toward the RA• Patent foramen ovale

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Critical annulus diameter

No regurg.

Mild regurg.

Severe regurg.

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Surgical indications

• Vast majority- easily repaired• Most lesions (unless severe) not adressed• Unpredictable regression following left sided

repair• Problem- lack of detailed preoperative search

for Tricuspid disease

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Ignoring Tricuspid valve disease

• Require a 2nd operation• Postoperative issues• 66% need TV surgery late after Mitral valve

surgery

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Specific surgical indications

• Ass. Left sided valvular lesions• Rheumatic polyvalvular lesions• Traumatic insuffiencies• Infective endocarditis• Functional regurgitation• Cardiac transplant• Ischaemic/idiopathic cardiomyopathy

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Surgical approach

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Commisurotomy

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Valve repair

• Repair manouvres are needed less often than for Mitral disease

• Non-rheumatic- often repaired• Infective endocarditis- vegetectomy

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Tricuspid annuloplasty

• Selectively reduce the length of the annulus• Techniques Bicuspidization Partial purse-string reductionRings or bands

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Bicuspidization

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DeVega

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Rings & bands

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Rings & bands

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Rings & bands

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Tricuspid replacement

• Mitral or tricuspid homografts• Stented porcine or pericardial valves• Mechanical prosthesis• Excision without replacement

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Tricuspid replacement

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Homograft replacement

• Mitral valve• Sizing- intertrigonal distance• Annuloplasty ring• Beating heart

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Prosthetic valve choice

• Age• Anticoagulation considerations • Young woman- childbearing years• Social issues must be considered

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Conclusions

• Results have been poor in the past• Only severe lesions were treated• Valve replacement- exceptional cases• The superior diagnostic tools and surgical

techniques available today will enlarge the field of tricuspid surgery

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THANK YOU