^The presentation is solely for educational purposes only and not … · 2020. 5. 13. ·...
Transcript of ^The presentation is solely for educational purposes only and not … · 2020. 5. 13. ·...
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“The presentation is solely for educational purposes only and not for commercial activity.
the ownership and copyright to the materials remain with the actual owner of the content. No claim for the originality of content is made. ”
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MECHANICAL Asphyxia-
(Part2:Drowning and Suffocation)
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Mechanical asphyxia
Compression of neck
Hanging Strangulations
Submersion of mouth & nostrils
under fluid
Drowning
means other than
compression at neck &
drowning (Suffocation)
Compression & mechanical
fixation of chest( Crush
asphyxia /traumatic asphyxia)
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Drowning
voilent asphyxial death , wherein the entry of air into the lungs is prevented by water or any other fluids due to submersion of mouth & nostril
Complete submersion of whole body is not necessary.
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Types of drowning
Drowning
Typical/wet
Fresh water drowning
Salt water drowning
Atypical
Dry drowning
Immersion syndrome
Secondary drowning
Shallow water drowning
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Mechanism of drowning
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Classification of drowning
A. Typical Drowning :also k/a Wet Drowning
i. Fresh water drowning
ii. Sea water drowning.
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Pathophysiology of fresh Water drowning
FRESH WATER
(hypotonic as compared to alveolar capillaries)
Water from alveoli enters alveolar capillaries & thus enters circulation
Dilution of blood (Haemodilution)
RBC’S swell & burst
K + levels elevated
Cardiac arrthymias
Death(4-5mins)
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Pathophysiology of Sea Water drowning
SEA WATER
(hypertonic as compared to alveolar capillaries)
Water is drawn into alveoli from circulation
Haemoconcentration of blood
RBC’S crenated
Pulmonary odema
Myocardial dysfuction
Death(8-12mins)
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B. Atypical drowning
1.Dry drowning :Rare , 10-15% cases
fall into water column
water entering nasopharynx/larynx
laryngeal spasm
Asphyxia
Death *on autopsy no water in lungs
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2.Immersion syndrome/ Hydrocution/cold water drowning/submersion inhibition :
Seen in cold & temperate environment.
Causes:
• cold water stimulating the nerve endings of surface of the body
• Water striking epigastrium
• Cold water entering eardrums ,nasal passages ,pharynx & larynx.
• Falling or diving into water with feet's first or duck driving by inexperienced.
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Duck diving- Head first
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3. Near-drowning/post-immersion syndrome/secondary drowning:
survival beyond 24hrs of a submersion episode
Death is caused by complications or sequelae:
ARDS
Pneumonia
Sepsis
hypoxic-ischaemic encephalopathy
cerebral odema
DIC
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4.Shallow water drowning/submersion of unconscious
Seen in persons:
epilepsy
heart disease
drunkenness
sustains a head injury during fall
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Symptoms
• Recalling of memory of past events.
• Mental confusion with auditory & visual hallucinations
• Tinnitus , vertigo
• Chest pain etc.
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Fatal period
Fresh water: 4-5 mins
Sea water: 8-12 mins
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Treatment
• Artificial respiration with close chest cardiac massage
• Defibrillators
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Causes of death
1. Asphyxia
2. Ventricular fibrillation
3. Vagal inhibition
4. Laryngeal spasm
5. Concussion / head injury
6. Apoplexy
7. Secondary causes
• septic aspiration pneumonia
• Sudden bursting of an aneurysm etc
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Postmortem findings
1. External signs of drowning
2. Internal signs of drowning
3. Biochemical & biophysical test for drowning
4. Analysis of diatomaceous material
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External signs When freshly removed from water:
1. Body & clothes wet, sand & mud stains may be +nt.
2. Body surface pallid & cold
3. Face: pale,bloated & discolored with putrefaction
4. Eyes: half open or closed, conjunctiva suffused,petechial haemorrhages +nt.
5. Tongue swollen & protuded
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6. P/M lividity: free flowing – no lividity, in stagnant water- pink color , confined to head ,neck & front of upper part of chest,extremities
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7. Froth : from mouth & nostrils
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Froth in antemortem drowning
• White
• Copious
• tenacious
• persistent
• Fine
• leathery
• Ocassionally blood tinged
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• Foam consist of fine bubbles & do not collapses on touching with pointed knife
• Foam production is a vital process
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Mechanism:
Fluid in respiratory tract provokes production of mucus
Mucus+water + air+ surfactant--------------- is churned into tenacious foam by voilentrespiratory mov. Made by victim during course of drowning.
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Foam also seen in…….
1. Opium poisoning
2. Organo-phosphorous poisoning
3. Strangulation
4. Epileptic attacks
5. Acute pulmonary odema
6. Electric shock
7. Putrefaction.
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8. Cutis anserine/ goose skin/goose flesh/goose bumps/horripilation:
Puckered ,granular appearance of skin
Medicolegal importance:
Seen in both antemortem& postmortem.
(postmortem due to rigormortis in erector pilli muscle.)
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9.Maceration of skin/ washer women’s hands & feets :
d/t imbibition of water in outer layer of skin
• Usually involves fingertips, palms, back of hands & soles
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• Skin whitened
• swollen
• Sodden
• wrinkled
• corrugated.
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• Later on epidermis loosened followed by nails & from hands & feet can be detached in glove & stocking fashion
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First seen in fingertips – 3-4 hrs
Entire hands – 24hrs.
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Maceration helps in estimating approx. duration of immersion.
wrinkling of skin= 1-2 hrs
bleaching of cuticle = 12 hrs
bleaching , corrugation & soddening = 24 hrs
cuticle separation from palms of hands & soles of feet = 48 hrs
easily peeled off = 3-4 days
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Floatation of body summers= 24hrswinters = 2-3 days
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10. Gravel, mud,sand,weeds or aquatic vegetation held firmly in clenched hands – cadaveric spasm
vital proof of antemortem drowning
indicates place of submersion.
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11.Scrotum & penis gets retracted in contact with cold water.
12. Rigor mortis appears early
13.Antemortem injuries
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Two important Antemortem features
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Internal signs
• voluminous, bulky ,water-logged ,over-inflated , filling the entire chest cavity & over-lapping the heart
• indentations of ribs
• - lung surface shows mottled appearance
• sub-pleural haemorrhagesk/a paltauf’s haemorrhagesseen.(anteriorly)
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- - cut surface of lung- copious amt. of frothy blood-stained liquid seen. Continous Column of froth seen
This overall picture of lungs is k/a emphysema aquosum.
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Odema aquosum:
Person Unconcious at time of drowning- mere flooding of lungs with water ,columns of froth not continous
Hydrostatic lung :
dead person thrown in water . d/t hydrostatic forces water enters lungs.
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2.larynx, trachea, bronchi:
+nce of sand, mud, aquatic vegetation, algae, diatoms in trachea & lower bronchi.
Fine white froth in trachea & bronchi lumen
Mucosa of larynx, trachea & bronchioles red & congested.
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3. Heart : rt .full lf. Empty
4. Stomach & intestines : +nce of some disagreeable material like muddy water, liquid manure, aquatic vegetation etc. which could not have swallowed voluntarily is highly suggestive of antemortemdrowning.
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Middle ear:
+nce of water & haemorrhages in middle ear & mastoid air cells
strongly suggests antemortem drowning.
6. brain, liver,spleen , kidneys – congested
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Biochemical & biophysical test for drowning
Gettler’s test
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Gettler’s test
1921 by Alexender Gettler- chloride content s of both sides of heart .
Normal chloride content of right & left side of heart is same
= 600 mg/100ml
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Difference b/w 2 chambers not more than 5mg/100ml
A difference of 25mg/100ml --- inducates death d/t drowning.
Fresh water drowning:
Chloride of lf. Side of heart < rt. Side
Salt water drowning
Chloride of lf. Side of heart > rt. Side
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Limitations of test:
laryngeal spasm
vagal inhibition
patent Foramen ovale
saline content of drowning media approximates that of blood.
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1944 Mortiz suggested magnesium levels
Mg left side of heart > right side.- sea water drowning
1955 Freimuth et al.
Specific gravity of plasma of 2 sides
-ve difference of both side – drowning/non drowning
+ve – other than drowning.
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Diatom test
Diatoms :
1904 Revenstrof first attempted to use diatoms as a test for drowning. (1896 by Hoffmann)
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Diatoms / bacillariophycaes
Unicellular algae with silicious exoskeleton –frustules
Chemically inert, indestructible, resistant to strong acids & resists putrefaction.
15000 species (1/2 in fresh water & ½ in sea water)
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Principle of test
Victim falls in water with diatom
diatoms carried to pulm. Parenchyma
enters blood stream (alveolar capillaries ) via tears of alveolar walls which occurs during forceful inspiratory & expiratory
efforts
diatoms to distant organs via systemic circulation
bone marrow , bone , kidney .
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Method
2-5 gm of tissue /40 gm of bone marrow
Place in Kjeldahl flask , add con HNO3
Heat for 1-2 hrs
Transparent yellow fluid with supernatant disc of fat
Yellow fluid centrifuged
Centrifuged Deposit examined on a slide while still wet.
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Comparison of no. nature & distribution of diatoms in visceral organs / marrow with those observed in alleged medium of submersion.(Drowning medium also collected as control)
Interpretation
+nce of diatoms in distant organ & marrow –suggestive proof of antemortem drowning .
Absence of diatoms does not exclude drowning
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Limitations
1. Drowning media does not contains diatoms
2. Drinking water containing same diatom
3. Since ubiquitous in nature may enter circulation via GIT
- Contaminated foods(salads, sea foods etc)
- Via Resp. tract- +nt in air in some paints ,building plasters, dusts, chalk powder etc.
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Medicolegal importance
1. Accidental – common
Also seen in precipitate labor.
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2.Suicidal common in India, specially in females
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3.Homicidal also not rare.
seen in infanticide
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Scuba divers –
defective equipment(rapid ascent / decent)
Causes air embolism, pneumothorax or interstitial emphysema
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SUFFOCATION
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Defination ….
Form of asphyxia caused by lack of oxygen in an atmosphere or by mechanical obstruction of the air passage by means other than constriction of neck & drowning .
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LACK OF OXYGEN
ENVIRONMENTAL OBSTRUCTION OF COMPRESSION OF
SUFFOCATION AIR PASSAGES CHEST
EXT. AIR PASSAGE INT. AIR PASSAGE
SMOTHERING CHOKING
OVERLAYING GAGGING
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1. Environmental suffocation • High altitudes
• Underground chambers
• Underground sewage entrapment
• Entrapment
• Domestic circumstances
• Carbon-monoxide & CO2 poisoning .
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Smothering
Mechanical occlusion of ext. air passage (Nose & mouth) by hands , cloths, plastic bags, duct tape etc.
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• Abrasion ,bruises & lacerations
• crescent shaped nails marks*
• # of nasal cartilage with bleeding
• frenular tears, abrasions, contusions, lacerations seen over inner aspects of lips,
• teeth loose & dislocated & tearing of gums.
• Associated struggle marks
Autopsy findings:Externally
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Internally :
+nce of sand ,dust, mud, cotton wool, flour, barley grains etc. in mouth ,nostrils
+nce of such material in the deeper respiratory passage intermixed with fluid & mucus - highly significant
Lungs show congestion, odema, & areas of haemorrhage & collapse with intervening emphysema
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Medicolegal aspects
1. Accidental smothering :
• common in alcoholics,
• epileptics ,
• newborn born with membranes covering mouth & nose (cul-de-sac),
• children while playing with plastic bag.
• Auto-erotic asphyxias etc.
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OVERLAYING
infant & mother sharing same bed
Mother roll over the child or occlusion of air passages by her breast while feeding.
In infants < 5mths & seen upto 2years.
MLI- SIDS & infanticide case.
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2. Homicidal smothering:Adults very rare, & difficult
unless victim weak,
stupefied by drugs or alcohol, or
great physical disparity between victim & assailant .
3. suicidal smothering :By owns hands is impossible
Tying plastic bag over mouth & nostril & applying multiple knots
Or tying a pillow in front of face with application of knots.
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2.Choking Form of asphyxia caused by mechanical occlusion
within various sites of upper air passages by foreign objects
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objects
1. Metallic coin, edible fruit seed, toffees, candies etc.
2. Food particle- bolus of rice, fish bone etc.
3. Tongue of the person in an epileptic attack may fall back onto the posterior pharyngeal wall
4. Sucking piece of balloon while inflating.
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Cause of death in choking
Asphyxia
Vagal inhibition
Laryngeal spasm
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Autopsy finding
Specific findings:
+nce of foreign object in the resp. tract
If victim epileptic, tongue must be specially examined for its position,& +nce & -nce of bruising or bite marks
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Medicolegal importance
1. Usually accidental
Café coronary : eg .of accidental choking where a bolus of
food produces complete obstruction of the larynx.
it mimics heart attack.
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Cause:
suppressed gag reflex due to gross intoxication with alcohol or drugs .
reflex cardiac arrest as a consequences of stimulation of laryngeal nerve endings.
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Clinical findings :
apparently healthy individual suddenly turns blue while eating
Autopsy findings: bolus of unchewed food or such other material found impacted in larynx.
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Gagging
Preventing air entry into the respiratory tract by stuffing gag material (clothes,paper balls etc.) into the mouth.
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Cause/mechanism
Gag material tightly stuffed into the mouth & obstructing breathing through the back of the throat.
Saliva,mucus or odema fluid moistens & air pockets in cloth & paper collapses
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Traumatic asphyxia.
Form of asphyxia resulting from trauma of the chest leading to forceful compression of the chest preventing respiratory movements.
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Causes
1. Penetrating injuries- stab injury
2. Non-penetrating injury- runover motor vehicle accidents, steering vehicle impact injury on chest , collapse of mine or building etc.
3. Indirect compression- victims thighs & knees are drive against the chest resulting in so called ‘jack-knife’ position.
4. Stampede by crowd-----also k/a RIOT CRUSH
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Autopsy findings:
Predominantly seen over level of obstruction of chest
Face congested & livid
Medicolegal importance:
Usually accidental
Rarely suicidal
Could be homicidal------- eg. Burking.
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Burking
Combination of –
smothering/palmar strangulation
traumatic asphyxia
This was used by 2 criminals William Burke & William Hare- who used to kill victims to sell their bodies to the Anatomy dept. of Edinburg Medical School (19th century) of Scotland.
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Victim first intoxicated with alcohol---- then pinned to the ground ---- Burke used to sit on the chest of the victim & cover his mouth & compress the neck with one hand---while other partner Hare pulled the body of victim round the room by feets.
16 murders over period of 10 mnths & bodies were
sold to Dr.Robert Knoxx.