The Biology and Origins of Language Part 1. Body Size, Brains and Stuff Smaller Body Size Short...
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![Page 1: The Biology and Origins of Language Part 1. Body Size, Brains and Stuff Smaller Body Size Short lifespan R selected Brain functions integrated Reliance.](https://reader036.fdocuments.us/reader036/viewer/2022081414/551408ea550346ec488b4cc1/html5/thumbnails/1.jpg)
The Biology and Origins of Language
Part 1
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Body Size, Brains and Stuff
Smaller Body Size• Short lifespan• R selected• Brain functions
integrated• Reliance on instinct• Stimulus bound
learning• Rigid response to
environment• Less adaptable to
change• Short neurons lead to
short reaction time
Larger Body Size• Long lifespan (for
learning)• K selected• Brain functions
specialized• Reliance on learning• Transferable learning• Flexible response to
environment• More adaptable to
change• Long neurons lead to
delayed reaction time
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r and K selection
“r” - Turtles lay & abandon many eggs
“K” - Chimps care for offspring for 5-7 years
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Animal vs. Human Brains• Animals have much less
cortex (gray matter) and much shorter nerve fibers than humans.
• The Cortex is where higher brain functions like thinking take place.
• The non-cortical brain is involved in reflexes and emotional reactions.
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Brain to Body Ratio
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Brain Specialization
Of all animals, only humans and apes have been found to have lateralized brains
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Language Parts of the BrainWernicke’s Area• Receives speech from primary auditory area and decodes
speechBroca’s Area• develops speech and sends it to the motor cortexGeschwind’s Territory• at the junction of the auditory, visual, and somatosensory
cortexes• neurons in this lobule can process different kinds of stimuli
(auditory, visual, sensorimotor, etc.) simultaneously. • doesn’t fully develop until about five years of age• classifying, labeling, thinking abstractly, forming concepts.Arcuate fasiculus• Bundle of neurological connections between three areas
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Left Cortex Language Areas
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Inferior parietal lobule AKA Geschwind’s Territory
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Aphasia
DEFINITIONis a loss of the
ability to produce and/or comprehend language
Two forms of Aphasia:
GRAMMAR PROBLEMS
Damage to Broca’s Area results in the inability to complete grammatically complete sentences.
PROBLEMS IN MEANING
Damage to Wernieke’s Area results in speech that has a natural-sounding rhythm and normal grammatical patterns but is meaningless.
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How do we know what parts of the Human Brain Control
Language?
Videos on Damage to Human Brains
Physical Injury
Stroke Patients
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Language Activity Brain Scans
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Genes and Language
Two links between specific genes and language:
1. The FoxP2 Gene
2. ASPM and Microcephalin Genes
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FoxP2• Discovered through one family(the “KE”
family), half of whom had a defect in that gene and could not speak
• The KE family was of Pakistani origin living in Britain
• In 37 members in 4 generations, 15 suffered specific language impairment
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FoxP2
• Patterns of inheritance indicated standard dominant/recessive inheritance, not sex chromosome inheritance.
• Fox P2n is Located on a short segment of chromosome 7
• each of us inherits two copies of the FOXP2 gene: one from our mother, and one from our father
• both copies must be intact for our language functions to be normal.
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FoxP2• responsible for producing a protein called a
transcription factor.• transcription factors bind to DNA molecules to
turn other genes off and on.• Broca’s area and the caudate nuclei (regulates
motor control) are smaller than in normal people
• trouble in identifying some elementary sounds of language,
• trouble in understanding sentences and using grammar
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ASPM and Microcephalin • These genes are associated with increased brain
size
• Mutations at these sites cause primary microcephaly, a developmental defect with severe reduction in the cerebral cortex—(planning, abstract reasoning and other higher brain functions).
• Microcephalin was more involved in evolution of
primate brain size (prosimians to monkeys)
• ASPM more involved in late evolutionary step leading to humans.
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ASPM and Microcephalin
Both genes are thought to affect brain development.
Older versions of these genes are found in tonal language populations. (Half of the world’s language are tonal including many in Asia, Southeast Asia and Sub Saharan Africa).
New versions of these genes are found in non-tonal language populations.