Textbook Discussion
-
Upload
jandz-nietes -
Category
Documents
-
view
221 -
download
0
Transcript of Textbook Discussion
-
7/29/2019 Textbook Discussion
1/3
TEXTBOOK DISCUSSION
A. Definition
Cerebrovascular diseases
Cerebrovascular disease encompasses a number of disorders involving vessels in the cerebral
circulation. These disorders include stroke and transient ischemic attacks (TIAs), aneurysmal
subarachnoid hemorrhage, and arteriovenous malformations.
Stroke is the syndrome of acute focal neurologic deficit from a vascular disorder that injures
brain tissue. The term brain attack has been promoted to highlight that time-dependent tissue
damage occurs and to raise awareness of the need for rapid emergency treatment, similar to
that with heart attack.
There are two main types of strokes: ischemic and hemorrhagic.
Ischemic Stroke
Ischemic strokes are caused by cerebrovascular obstruction by thrombosis or emboli. A
common classification system identifies five stroke subtypes: ischemic penumbra in evolving
stroke, transient ischemic attack, large vessel stroke, small vessel stroke, and cardiogenic
embolic stroke.
Hemorrhagic Stroke
The most frequently fatal stroke is a spontaneous hemorrhage into the brain substance. Withrupture of a blood vessel, hemorrhage into the brain tissue occurs, resulting in edema,
compression of the brain contents, or spasm of the adjacent blood vessels. In the most
common situation, hemorrhage into the basal ganglia results in contralateral hemiplegia, with
initial flaccidity progressing to spasticity. The hemorrhage and resultant edema exert great
pressure on the brain substance, and the clinical course progresses rapidly to coma and
frequently to death.
B. Signs and symptoms
According to textbook As manifested by patient
Weakness of face -
Weakness of the arm + August 2, 2013
Unilateral numbness -
Vision loss in one eye -
Language disturbance + August 2, 2013
Aphasia + August 2, 2013
Sudden, unexplained imbalance or ataxia + August 2, 2013
A. Definition
Unstable Angina (NonST-Segment Elevation Myocardial Infarction)
Unstable angina is considered to be a clinical syndrome of myocardial ischemia ranging
between stable angina and myocardial infarction. It most frequently results from
-
7/29/2019 Textbook Discussion
2/3
atherosclerotic plaque disruption, platelet aggregation, and secondary hemostasis.In contrast
to stable angina, the pain associated with unstable angina has a more persistent and severe
course and is characterized by at least one of three features: (1) it occurs at rest usually lasting
more than 20 minutes; (2) it is severe and described as frank pain and of new onset; and (3) it
occurs with a pattern that is more severe, prolonged, or frequent than previously experienced.Persons who have no evidence of serum markers for myocardial damage are considered to
have unstable angina, whereas a diagnosis of nonST-segment elevation myocardial infarction
is indicated if a serum marker of myocardial injury is present.
B. Signs and symptoms
According to textbook As manifested by patient
Chest pain or discomfort + August 2, 2013
Pain in arms, neck, jaw, shoulder or back
accompanying chest pain
-
Nausea + August 2, 2013
Fatigue -
Shortness of breath -
Excessive sweating -
Dizziness -
A. Definition
Hypertensive Cardiovascular Disease
Uncontrolled and prolonged elevation of BP can lead to a variety of changes in the myocardial
structure, coronary vasculature, and conduction system of the heart. These changes in turn can
lead to the development of left ventricular hypertrophy (LVH), coronary artery disease (CAD),
various conduction system diseases, and systolic and diastolic dysfunction of the myocardium,
complications that manifest clinically as angina or myocardial infarction, cardiac arrhythmias
(especially atrial fibrillation), and congestive heart failure (CHF).
Thus, hypertensive heart disease is a term applied generally to heart diseases, such as LVH,coronary artery disease, cardiac arrhythmias, and CHF, that are caused by the direct or indirect
effects of elevated BP. Although these diseases generally develop in response to chronically
elevated BP, marked and acute elevation of BP can lead to accentuation of an underlying
predisposition to any of the symptoms traditionally associated with chronic hypertension.
B. Signs and Symptoms
According to textbook As manifested by patient
High blood pressure + August 2010
Enlarged heart and irregular heartbeat -
Fluid in the lungs or lower extremities + August 10, 2013
Unusual heart sounds -
-
7/29/2019 Textbook Discussion
3/3
A. Definition
Type 2 Diabetes
The two main problems related to insulin in type 2 diabetes are insulin resistance and impaired
insulin secretion. Insulin resistance refers to a decreased tissue sensitivity to insulin. Normally,insulin binds to special receptors on cell surfaces and initiates a series of reactions involved in
glucose metabolism. In type 2 diabetes, these intracellular reactions are diminished, thus
rendering insulin less effective at stimulating glucose uptake by the tissues and at regulating
glucose release by the liver. The exact mechanisms that lead to insulin resistance and impaired
insulin secretion in type 2 diabetes are unknown, although genetic factors are thought to play a
role.
To overcome insulin resistance and to prevent the buildup of glucose in the blood, increased
amounts of insulin must be secreted to maintain the glucose level at a normal or slightly
elevated level.
B. Signs and symptoms
According to textbook As manifested by patient
Fatigue -
Irritability + August 10, 2013
Polyuria -
Polydipsia -
Slow healing of skin wounds -
Vaginal infections -Blurred vision + August 10, 2013
Increased hunger -
Weight loss -
Areas of darkened skin + August 10, 2013
SCHEMATIC DIAGRAM
Predisposing Factors Precipitating Factors
Obesity Increased carbohydrate and fat intake
Race/Ethnicity Lack of exercise
History of GDM History of smoking
Delivery of babies over 9Ilbs History of alcoholism
Family history
Gender: Female
Age>45: 64 years old