Tension Pneumothorax by Ankur
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Transcript of Tension Pneumothorax by Ankur
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Dr. Ankur Agrawal
Dept. of Respiratory
Medicine
PGIMS Rohtak
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Pneumothorax
Defined as accumulation of air in the pleural spacewith secondary collapse of surrounding lung
Due to disruption of parietal or visceral pleura
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Classification
Spontaneous pneumothorax
Primary - no identifiable pathology
Secondary - underlying pulmonary disorder
Catamenial Traumatic
Blunt or penetrating thoracic trauma
Iatrogenic
Postoperative Mechanical ventilation
Thoracocentesis
Central venous cannulation
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Primary Spontaneous Pneumothorax
Usually occurs in young healthy adult men 85% patients are less than 40 years old Male : female ratio is 6:1 Bilateral in 10% of cases Occurs as result of rupture of an acquired subpleural bleb Blebs have no epithelial lining and arise from rupture of
the alveolar wall Apical blebs found in 85% of patients undergoing
thoracotomy Frequency of spontaneous pneumothorax increases after
each episode Most recurrences occur within 2 years of the initial episode
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Secondary Spontaneous Pneumothorax---
--- underlying lung/pleural disease
Emphysema
Chronic bronchitis
Asthma TB
Necrotizing lung infections
ARDS
Pneumocystis jiroveci pneumonia ILD
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Catamenial pneumothorax Catamenial pneumothorax refers to the development
of pneumothorax at the time of menstruation. represents 3-6% of spontaneous pneumothorax in
women. Typically, it occurs in women aged 30-40 years with a
history of pelvic endometriosis (20-40%). It usually affects the right lung (90-95%) and occurs
within 72 hours after the onset of menses. The recurrence rate in women receiving hormonal
treatment is 50% at 1 year. T/t- ocp, danazol, lupron [GnRH agonist]
,hystrectomy and b/l oophorectomy
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Traumatic Pneumothorax
Open Chest wall is penetrated : outside air enters pleural space
Closed Chest wall is intact Eg. Fractured rib
Iatrogenic
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Common causes of IatrogenicPneumothorax
Positive-pressure ventilation
Central venous catheter placement
Thoracentesis
Tracheostomy Nasogastric tube placement (inadvertent insertion of the
NG tube into tracheobronchial tree)
Bronchoscopy(esp if transbronchial biopsy performed)
Pericardiocentesis
Transthoracic needle aspiration
Cardiopulmonary resuscitation.
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Tension PneumothoraxA condition in which air continuously leaks out of the
lung into the pleural space
This results in lung collapse of the affected side, andthen pressure on the mediastinum, the other lung andthe great vessels
E T
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Et o ogy o Tens on
Pneumothorax
Trauma(blunt or penetrating): disruption of the parietal or visceralpleura.
Fractures: most prevalent as a result of rib fractures, however alsoseen in displaced thoracic spine fractures.
Barotrauma: ventilator dependent patients on large volume PEEPmay rupture peripheral alveoli sacs secondarily disrupting thevisceral pleura. Index of suspicion is raised when larger peak airwaypressures are needed to achieve a specific tidal volume.
Iatrogenic: secondary to trauma induced by
Bronchoscopy Chest compressions during CPR
Central venous catheter placement
Conversion of Simple Pneumothorax -> Tension Pneumothorax
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Pathophysiology of Tension
Pneumothorax
Disruption of the lung parenchyma or parietalpleura acts like a one way valve. During inspiration air
is drawn into the pleural space. During expiration thetissue f lap/valve prevents air from escaping.Subsequent inspirations additively draw more air intothe pleural space causing increasing intrapleural
pressures greater than atmospheric pressurethroughout expiration and even during inspirationresult in collapse of ipsilateral lung and deviation ofmediastinal structures contralaterally.
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Tension PneumothoraxEach time we inhale,
the lung collapses further. Thereis no place for the air to
escape..
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Tension Pneumothorax Each time we inhale,the lung collapses further. There
is no place for the air toescape..
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Tension Pneumothorax
Heart is beingcompressed
The trachea ispushed to
the good side
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Symptoms Dyspnea
Cough
Pleuritic chest pain Nerve endings at pleural capsule
Sense of impending doom
Sudden onset
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SignAsymmetric chest expansion Deviated trachea Diminished breath sounds unilaterally Hyper-resonance unilaterally Decreased tactile fremitus
Cyanosis Profuse diaphoresis Marked tachycardia Hypotension
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Etiology of symptomsHypoxia
- Decreased PaO2
- Perfusion of atelectatic lung- VQ mismatchDecrease venous return
- increase intrathorasic pressure
-decrease cardiac output
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LAB STUDIESABGs: Often seen in tension pneumothorax is a
varying degree of acidemia, hypercarbia, and hypoxia.The reduction in PaO2 is caused by alveolarhypoperfusion secondary to atelectasis, lowventilation/perfusion ratios, and anatomic shunts
Cardiac Enzymes: necessary to r/o acute MI andresulting cardiogenic shock, must have serial readingto accurately r/o acute MI
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Findings on CXR:
Large radiodense lung field
Absent lung markings on ipsilateral side
Contralateral deviation of trachea and mediastinal
structures If tension pneumothorax involves left lung the left
hemidiaphragm may be depressed/flattened. The liverprevents this radiographic finding on the right side
Imp -Laboratory and diagnostics may confirm thediagnosis of a tension pneumothorax (i.e. ABG, CXR)however the diagnosis lies predominantly on clinicalpresenting symptoms
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D/D Myocardial infarction Pulmonary infarction Pleural infection Perforated peptic ulcer Generalized emphysema Massive emphysematous bulla
Congenital lung cyst Diaphragmatic hernia COPD, asthma
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Treatment of Tension
PneumothoraxABC to be maintained
High Flow oxygen
Treat for S/S of Shock If Open Pneumothorax and occlusive dressing present
apply occlusive dressing
Monitor Cardiac Rhythm
Needle Decompression of Affected Side
Tube Thoracostomy
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NEEDLE CHEST DECOMPRESSION Locate 2d intercostal space at midclavicular line
Insert 14-gauge catheter-over-needle into chest cavityover superior edge of rib
Listen for gush of air and observe for improvement ofsymptoms
Tape catheter in place with cap or valve in place to
prevent re-entry of air May also place Asherman chest seal over catheter
Dress open chest wound if present
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NEEDLE CHEST
DECOMPRESSION
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Tube Thoracostomy Identify and prepare the area w/ Betadine at ICS 4 or 5 along the mid-axillary or anterior axillary line
Anesthetize the area (subcutaneous tissue, intercostal muscles) withLidocaine. Some physicians use opioid analgesia or a combination of an
opioid + Benzo. Make a 2 cm incision Insert a large blunt clamp over superior aspect of rib (preventing
damage to the neurovascular bundle that lies on the inferior border ofthe rib). Apply gentle pressure until the parietal pleura is pierced.
Open clamp to establish a tract for the chest tube.
Bluntly dissect w/ finger. Clamp proximal end of tube tangentially w/ Clamp. Insert tube over
superior aspect of rib into pleural space. Insert the chest tube past the last hole. Suture chest tube w/ Silk
sutures.
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Monitor patient continuously with arterial O2saturationwatch for sudden desaturations
F/U CXR may be ordered to assess re-expansion oflung and resolution of pneumothorax. Important: re-expansion pulmonary edema may occur with rapidlung re-expansion s/p tube thoracostomy. This is apotential life threatening situation which can lead tocardiovascular collapse.
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Pulmonary edema occur most commonly unilateral,on the collapsed lung side but can become bilateral
REPE is due to increased permeability of pulmonarycapillary that are damaged by mechanical stress anddecrease surfactant.
Other theories include ischemic reperfusion injury,
free radical injury and decrease lymphatic flow Chances of REPE are high when pneumothorax
present for more than 3 days or lung expanded withmore than -20cm H2O pleural pressure
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Onset of symptoms can be immediate or within 24 hr
Pt. will present with severe persistent cough, chestpain, hypoxia, tachycardia, tachypea and hypotension
Corticosteroid, diuretics, bronchodilator, and highflow oxygen is given and sometimes pt may needmechanical ventilator support
If pt survive the first 48 hr recovery is usually complete
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Keep chest tube on water seal. Chest tube may beremoved when indication for placing it has resolved.F/U CXR must be ordered immediately s/p chest tube
removal and 24 hrs post-removal to assess for presenceof a reoccurring pneumothorax.
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