Superficial thickness
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Transcript of Superficial thickness
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Superficial thickness
First-degree
Full thicknessThird- or fourth-
degree
Partial thickness —
deepSecond-degree
Partial thickness — superficial
Second-degree
Epidermis involvement
Dermis and underlying
tissue
Deep (reticular) dermis
Superficial (papillary
Erythema minor pain, lack of blisters
Hard, leather-like eschar, purple fluid, no sensation
(insensate)
Whiter appearance, with decreased pain. Difficult to
distinguish from full thickness
Blisters, clear fluid, and pain
Nomenclature
Traditional nomenclature
Depth Clinical findings
BURN
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Require only symptomatic treatment.
Usually heals in 7 days.
No blister formation.
Skin is red, painful, and tender.
Involves only the Epidermal layer of the skin.
Leaves no scars.
I= 1st DEGREE BURN
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The deeper layer of the dermis, hair follicles, sweat glands and sebaceous glands are spared
II= 2nd DEGREE BURN1- SUPERFICIAL – PARTIAL THICNESS The burn extends to the dermis
The epidermis and superficial (papillary layer) dermis are injured
Blistering of the skin
Exposed dermis is red, moist at blister base
Very painful to touch
Good perfusion of dermis with intact capillary refill
Heal in 14 – 21 days
Scaring usually minimal
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II= 2nd DEGREE BURN2- DEEP – PARTIAL THICKNESS
Extends into the deep reticular layer of the dermis
There is damage in the hair follicles, sweat glands, & sebaceous glands
Caused by steam, hot liquids, flameSkin may be blisteredThe burned areas don’t blanchNo capillary fillingAbsent pain sensationHealing takes 3 W – 2 monthsScaring is commonSurgical debridement & skin grafting may be necessary to obtain maximum function
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III= 3rd DEGREE BURN
Involves the entire skin thickness
All epidermal and dermal layers are destroyed
Caused by flame, hot oil, steam and contact e’ hot object
Skin is charred, pale, painless, leathery
Injuries will not heal spontaneously
Surgical repair and grafting are necessary
Injuries will leave significant scaring
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9%
Front18%
Back18%
9%9%
18%18%
ESTIMATION OF BURN %(ADULT)
RULE OF 9 & ESTIMATION OF BURN SIZE
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CLASSIFICATIONS OF BURN ACCORDING TO SEVERITY
1= MINOR
Partial thickness burn less 15% of BSA in the 10 – 50 – year old age group
Partial thickness burn less than 10% BSA in children under 10 and adults more than 50
Full thickness burn of less than 2% BSA in any one with out associated injuries
These burn imply out patient treatment
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2 =MODERATE
CLASSIFICATIONS OF BURN ACCORDING TO SEVERITY / cont
Partial thickness burn of 15 – 25 % BSA in 10 – 50 year – old age group
Partial thickness burn of 10 – 20 % BSA in children under 10 or adults over 50
Full thickness burn of less than 10% in any one
Partial thickness burn of the hands, face, feet, perineum, or circumferential burn of an extremity are excluded
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3 =MAJOR
CLASSIFICATIONS OF BURN ACCORDING TO SEVERITY / cont
Partial thickness burn greater than 25% BSA in the 10 – 50 – year – old age groupPartial thickness burn greater than 20% BSA in children younger than 10 and adults over 50Full thickness burn greater than 10% BSA in any oneBurn involving face, feet, hands and perineumBurn complicated by inhalation burnBurn crossing major jointsCircumferential burns of an extremityElectrical burnsBurn complicated by # or other traumaBurns in infants & elderly
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FLUID REPLACEMENT
PARKLAND FORMULA
use crystalloids
ADULTS
RL 4ml X patient Wt ( kg ) X % BSA over initial 24 h
½over the 1st 8 hrs from the time of burn
½over the subsequent 16 hrs
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FLUID REPLACEMENT
PARKLAND FORMULA
use crystalloids CHILDREN
they have an increased body surface area – to - wt ratio, so they have greater fluids requirement
RL 3 ml X patient Wt ( kg ) X % BSA over initial 24 hrs plus maintenance fluid
½over the 1st 8 hrs from the time of burn
other ½ over the subsequent 16 hrs
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Maintenance
Based upon the child’s wt
100 ml/kg for the 1st 10 kg
then 50 ml/kg up to 20 kg
then 2o ml/kg for any wt above 20kg
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DIGOXIN TOXICITY• S/S• Acute & chronic• DX• FAB
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HYPERKALEMIA • Is life threatening Hyperkalemia present ?• ECG changes• high risk ( RF,On dialysis, medication)• Serum K > 5.5mEq/l
IV 0.9%Nacl
YDOC > Alkalinizing agents > Sodium bicarbonateIncrease pH, which results in a temporary K shift from the ECS to ICS• Adult: 1 mEq/kg slow IV push not to exceed 50-100 mEq• Children: 1-2 mEq/kg IV over 5-10 min; repeat in 10 min prn
Stabilize the myocardium > IV Calcium gluconate • Adult: 10 mL of 10% sol IV over 10 min (under ECG monitor)•Children: 100 mg/kg (1 mL/kg) of 10% sol IV over 10 min; not to exceed 10 mL
Shift K into the cells
DW & InsulineAd: 5-10 U simple insuline and 25-50 g D (50-100 ml 50% DW)children: 0.5g Dw 25% and 0.1u/kg IV slowly
Albuterol (Ventolin) NEB
Adult: 5 mg mixed with 3 mL NS high-flow Nebulizer q20min Children: 2.5 mg/dose with 3 mL isotonic saline nebulized
Enhance elimination of K
N
N or HighLow
Pt volume status Kayexalate: 25-50 g mixed with 100 mL of 20% sorbitol PO/PR
Is urine output present?
N
NO
RESPONSE
Y
Attempt loop diuretics(lasix 40-100mg)
Hemodialysis
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Simple febrile seizures are: Temperature greater than 38 C Age – 6 months to 6 years isolated Generalized tonic – clonic seizures lasting less than 15 minutes do not recur within 24 hours or within the
same febrile illness No CNS infection or inflammation No systemic metabolic abnormality No history previous afebrile seizure
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HYPOGLYCEMIA
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1- Serum glucose level < 50 mg/dl.2- Symptoms consistent with the diagnosis.3- History of DM4- Patient with altered mental status
Yes
Is patient responsive ? No
• 1g/kg/IV 50% D/W• IV infusion of 10% D/w• 200 mg hydrocort when (adrenal crisis, no response to treatment)
IV line Present ?
• Glucagon: 1gm IM,SC• NG tube >> sweet Drink
Y
N
• Asess and monitor patient Response• Check RBS q 15 min
• continue to monitor• Search for causes• Ensure patient safety
• Establish IV access• 1g/kg/IV 50% D/W• Continuous infusion of 10% D/w• Glucagon: 1gm IM,SC
Recheck RBS in 15 min
• 20 – 30 gms of oral carbohydrate• Assess response
Is patient NPO
Yes
Yes
NO
Is patientResponsive
Retreat with 1gm/kg
CarbohydratePO, IV
• Monitor Pt• If eating, feed meal within 30 min• Evaluate etiology• Educate Pt how to prevent future episode
Is RBS greaterThe 70 mg/dl
Yes
Recheck RBS in 30 min
Is RBS greater than
60 mg/dl
No
Is patient NPO
• Retreate with 20-30gm carbohydrate• continue monitoring Pt
No
Yes
NO
YEs
No
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S/S OF HYPOGLYCEMIA
NEUROGLYCOPENICSYMPTOMS
HYPEREPINEPHRINEMICsymptoms
-Alternation in LOC -Lethargy, confusion
- Agitation- Unresponsiveness
- Seizures- Focal neurological deficits
- Coma
-Anxiety, nervousness -Irritability
-Nausea, vomiting -Palpitation, tremor
- Sweating ,- Change in pupils size
-Salivation -Bradycardia
Due toHypoglycemia
Due torelease of adrenaline
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HYPOTHERMIA
IT IS DEFINEDAS A CORE TEPERATURE LESS THAN 35C ( 95F )
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1. T wave inversion2. PR, QRS, QT prolongation3. Muscle tremor artifacts4. Osborne ( J ) wave5. Dysrhythmias
- Sinus bradycardia - AF or Flutter - Nodal rhythm - AV block - PVC,s - VF - Asystole
ECG CHANGES IN HYPOTHERMIA
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J wave: it is a positive reflection at the junction of the QRS and S – T segment.
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CORE TEPERATURE LESS THAN 35C ( 95F )Primary: Secondary
Resulting from a medical illnessSee D/D
Radiation, Conduction, Convection& Evaporation
Mechanisms of heat loss
Due to environmental exposure, with no underlying medical condition
REWARMING TECHNIQUES
1. Passive warming:Removal from cold environmentUse of insulating blankets
GENERAL & SUPPORTIVECARE
2. Active external warmingRadiant heat, Warmed blanketWarm water immersion, heated objects
3. Active core re – warming= warm NS (42*C) throughNG tube, folly’s catheter, peritonial catheter. Rectal tube= Warm IV fluids
ABC Assessment, RR, pulse oxymetry, effort
V/S: core temperature, HR, BP
adequateYes
No
O2
INTUBATIONLMA, ETT
CORE TEMP < 35*C
Warm IV fluids, ECG monitoring, soft handling
TREATMENT OF THE CAUSE (See D/D)
Treatment of dysrhythmiasaccordingly
YES
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Paracetamol poisoning1. toxic dose
2. Paracetamol level
3. Activated charcoal
4. Lavage
5. NAC IV dose
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Time of ingestion
< 2 hours > 8 hours 2 – 8 hours
Activated charcoal
< 150 mgs/kg >150mgs/kg < 150 mgs/kg >150mgs/kg
NAC
Treat possible , probable and high risk group with NAC.
Paracetamol level at presentation
Paracetamol level at 4 hours
PROTOCOLS FOR NAC ADMINISTRATION(Do not Delay NAC treatment while waiting for paracetamol level)
• ORAL:72 h treatment= Loading dose: 140 mg/kg, Subsequent Doses: 70mg/kg q 4h for 17 doses.• IV: 20 h treatment= Loading dose:150 mg/kg over 15 min, Subsequent Doses: 50 mg/kg over 4h followed by 100mg/kg over 16h
Paracetamol Poisoning
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Patient assessment in ACLS
And care priority
ABC: when patient either responsive of not with intact circulation
CAB: when patient not responsive with no pulse
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Antidotes for Toxins in Emergency Overdose Patients
Antidote Toxin Dose and comments Naloxone Opiates Naloxone2 mg; less to avoid narcotic withdrawal,
more if inadequate response; same dose in children
Bicarbonate
Tricyclics 44–88 mEq in adults; 1–2 mEq/kg in children; IV push, not by slow infusion
Flumazenil Benzodiazepines 0.2 mg, then 0.3 mg, then 0.5 mg, up to 5 mg; not to be used if patient has signs of TCA toxicity; not
approved for use in children but probably safe Calcium Calcium channel
blockers1 g calcium chloride IV in adults, 20–30 mg/kg/dose in children, over a few minutes with continuous monitoring
Glucagon b-blockers, calcium channel
Blockers
5–10 mg in adults, then infusion of same dose per hour
Physostigmine Anticholinergics 1–2 mg IV adults, 0.5 mg in children over 2 min for anticholinergic delirium, seizures, or dysrhythmias
Atropine Organophosphates, carbamates
Test dose 1–2 mg IV in adults, 0.03 mg/kg in children. Titrate to drying of pulmonary secretions
N-acetylcysteine
Acetaminophen 140 mg/kg, then 70 mg/kg q4h; IV form still investigational
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Antidotes for Toxins in Emergency Overdose Patients
Antidote Toxin Dose and comments Ethanol Methanol, ethylene
glycolLoading dose 10 ml/kg of 10%; maintenance dose
0.15 ml/kg/hr of 10%; double rate during dialysis Fomepizole Methanol, ethylene
glycol15 mg/kg every 12 hr
Pyridoxine. Isoniazid 5 g in adults, 1 g in children, if ingested dose unknown. Antidote may cause neuropathy
Thiamine Ethylene glycol chronic ethanol ingestion
100 mg IV
Digoxin-specific FAB
fragments.
Digitalis glycosides 10–20 vials if patient in ventricular fibrillation. Otherwise, dose is based on serum digoxin concentration or amount ingested
Sodium nitrite Cyanide, H2S 10 ml of 3% (300 mg; 1 ampule) in adults; 0.33 ml/kg in children, slowly IV
Deferoxamine Iron15 mg/kg/hr IV, higher doses reported to be safe
protamine sul Heparine
Vitamine k warfarine
Specific anti venom
Snakes & yellow scorpions
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Odors in Overdose History ODOR POSSIBLE INTOXICANT
Bitter almond Cyanide
Fishy Zinc or aluminum phosphide
Fruity ethanol, acetone, chloroform
Garlic Arsenic, organophosphates
Glue Toluene, solvents
Pears Chloral hydrate, paraldehyde
Rotten eggs Hydrogen sulfide, NAC
Shoe polish Nitrobenzene
wintergreen Methyl salicylate
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Activated charcoal does not adsorb:A. ironB. lithiumC. sodiumD. leadE. cyanideF. hydrocarbonsG. causticsH. alcohols
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Causes and Differential Diagnoses of Potentially Catastrophic Illness Presenting with Vaginal Bleeding
Assess ABC, V/S, Start IV, Blood for Lab, Cross Match, Cardiac Monitoring, O2 therapy, history
Pain Assessment ( PQRST method) provoke, paliate, quality, radiation, site, time (onset-duration-frequency)
Ectopic pregnancy Abruptio placentae Placenta previa Postpartum hemorrhage Uterine rupture
D I F E R E N T I A L D I A G N O S I S
P H Y S I C A L F I N D I N G S
Anxiety/diaphoresis,Tachycardia,
Tenderness on pelvic and lower abdominal
exam. Vaginal bleeding
Hypovolemic shock, Hypotension, Abdominal and uterine tenderness. Variable dark red uterine
bleeding
Hypovolemic shock, Hypotension, Gravid
uterus, abdominal exam usually benign
Enlarged, doughy uterus (uterine atony). Vaginal bleeding without uterine
bleeding (vaginal lacerations
Hypovolemic shock, Hypotension Tender
abdomen with guarding
PELVIC US FINDINGAdnexal mass Ovarian
massPlacental separation Low lying placenta NORMAL, Uterine mass Peritoneal fluid
Lower abd pain, always present .Variable character. sudden onset,. Occasional
radiation into back or flank. unilateral
Uterine tenderness and irritability. Intermittent or
steady abdominal cramping. Back pain
Usually very little pain.Often minimal. Can be
severe with uterine inversion.
Uterine pain without contraction
Supporting History
First-trimester pregnancy, amenorhea,
irregular mensprior ectopic pregnancy, PID, STD, tubal ligation.
Over 20 w gestationIncreased maternal age,
HTN smoking, abdominal trauma.
Over 20 weeks gestation. Incidence
increased with multiparity and prior
C-section.
Difficult, traumatic delivery. Previous CS
retained placnta Multiple gestations, hydramnios, multiparity. uterine atony
Previous cesarean section.
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4321
SpontaneousTo voiceTo painNone
EYE OPENING
54321
OrientedConfusedInappropriate wordsIncomprehensive wordsNone
VERBAL RESPONSE
654321
Obeys commandLocalizes painWithdraw (pain)Flexion (pain)Extension (pain)non
MOTOR RESPONSE
G.C.S
15MAXIMUM SCORE
Severe < 8Moderate 9 – 12Mild 13 - 15
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How to choose appropriate ET Tube size for age??
(Age/4)+4
(16/age)+4
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Snake bite
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human bite
Child abuse
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Anion gap calculation and metabolic acidosis
Na+ − (Cl- + HCO3−)
• 8 to 16 mEq/L plasma when not including [K+]
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1. Lactic acidosis2. Ketoacidosis
Diabetic ketoacidosisAlcohol abuse
3. Toxins:MethanolEthylene glycolPropylene GlycolLactic acidUremiaAspirinPhenforminIronIsoniazidCyanide
4. Renal failure, causes high anion gap acidosis by decreased acid excretion and decreased HCO3
− reabsorption. Accumulation of sulfates, phosphates, urate, and hippurate accounts for the high anion gap.
High anion gap metabolic acidosis
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2[Na+] + [Glucose]/18 + [ BUN ]/2.8
Normal human reference range of osmolality in plasma is about 285-295 milli-osmoles per kilogram.
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High quality CPR
1. Rate >> at least 100 compression/min
2. Depth >> at lest 2 ½ inches
3. Allow chest recoil
4. Do not interupt compression
5. Do not hyperventilate
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TRIAGECOMPONENT
Focused PhysicalEvaluation
Across the roomassessment
Triage history & Physical assessment
Triage decision
-Visual assess -General appearance
Acuity levels1,2,3,4,5
Subjective Objective
ChiefComplaint
DescriptionOf symptoms
Hx of past &Present illness
AMPLEAssessment Of the Presenting event
Include:• Appearance• ABC, VS• Physical exam• GCS• AVUP
Pain Assessment“PQRST” Method
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Patient dying? Intubated/apnic/pulseless
OR Unresponsive 1
N
Y
Shouldnt wait??High risk situation Or
Confused/lethargic/ disorientation?Or Severe pain/distress
How many different resources are needed?
N
2None One
Two or more
45
A
D
C
B
2-8y >140 >30
3m-2y >160 >40
>8y >100 >20 Age HR RR SaO2<90%
Danger zone vitals
E
S
I
ALGORITHM
Y
3
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ESI ResourcesRESOURCES NOT RESOURCES
Labs (blood, urine) History & physical(including pelvic)
ECG, X-rays, CT-MRI-ultrasoundangiography
Point-of-care testing
IV fluids (hydration) Saline or heplock
IV, IM or nebulized medications PO medications
Specialty consultation Phone call to PCP
1. Simple procedure (laceration repair, Foley cath)2. Complex procedure(conscious sedation)
Simple wound care (dressings, recheck) Crutches, splints, slings
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Toxidromes (a toxic “fingerprint)
Refers to the collection of signs and symptoms.
It include grouped, physiologically based abnormalities of vital signs General appearance Skin, eyes, mucous membranes Lungs, heart, abdomen Neurologic examination
Helpful in establishing a diagnosis when the exposure is not well defined.
Certain clinical findings may narrow the etiologic possibilities
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Common Toxic Syndromes (Toxidromes) Anticholinergic Common
signs
Delirium with mumbling speech, tachycardia, dry flushed skin, dilated pupils, myoclonus, slightly elevated temperature, urinary retention, decreased bowel sounds. Seizures and dysrhythmias may occur in
severe cases.
Common causes
Antihistamines, antiparkinsonians, atropine, scopolamine, amantadine, antipsychotics, antidepressants, antispasmodics, mydriatics, muscle relaxants.
Sympathomimetic
Common signs
tachycardia (or bradycardia with pure a-agonist), hypertension, hyperpyrexia, diaphoresis, Delusions, paranoia, piloerection, mydriasis,
hyperreflexia. Seizures, hypotension, and dysrhythmias. Common causes
Cocaine, amphetamine, methamphetamine and its derivatives, ephedrine, pseudoephedrine. In caffeine and theophylline overdoses, similar findings, except for the organic psychiatric signs, result from
catecholamine release.
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Common Toxic Syndromes (Toxidromes) Opioid/sedative/ethanol Common
signs
Coma, respiratory depression, miosis, hypotension, bradycardia, hypothermia, pulmonary edema, decreased bowel sounds, hyporeflexia, needle marks. Seizures may occur after overdoses of some narcotics
(e.g., propoxyphene) Common causes
Narcotics, barbiturates, benzodiazepines, glutethimide, methyprylon,
methaqualone, meprobamate, ethanol, clonidine, guanabenz
Cholinergic Common
signs
Confusion, central nervous system depression, weakness, salivation, lacrimation, urinary/fecal incontinence, gastrointestinal cramping, emesis, diaphoresis, muscle fasciculations, pulmonary edema, miosis,
bradycardia/tachycardia, seizures Common causes
Organophosphate and carbamate insecticides, physostigmine,
edrophonium, some mushrooms
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HR ?
FAST SLOW
What is ventricular rate ? (correspond to pulse rate)
NORMAL
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NARROW OR WIDE??
0.10 sec
0.17 sec
P
ST
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Teaching Moment When an impulse originates anywhere in the
atria or above the ventricles:1. SA node2. Atrial cells
3. AV node4. Bundle of His
and then is conducted normally through the ventricles, the QRS will be narrow (0.08 - 0.12 s).
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Classification of Arrhythmias
Slow pulse rateBradyarrhythmias
Fast pulse rateTachyarrhythmias
Depending On HR/min &QRS Duration/sec
SlowNarrow
Slow Wide
Fast Narrow
Fast Wide
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Interventions to treat Rhythm disturbances
1. Mechanical
Vagal maneuvers
Pericardiocentesis
Chest decompression
Valsalva maneuver
2. Pharmacological
Adrenaline
Amiodarone
Adenosine
Mg sulphate
Lidocain
Atropine
3 .Electrical
Defibrilation
Synch cardioversion
Pacing
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Treatment mode selection depends on the patient’s general status:
1. Presence or absence of pulse
2. Patient’s stability
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Pulse present??YESNO
Stablepatient??
YESNO
mechanical
Pharma
Failed
FailedElectrical
Shockable rhythm ??
YESNO
VF &Pulsless VT
CPR
AsystolePEA
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When you decide to use the electrical intervention,
please remember the 3 S’s
1.Sedate the patient when needed (Medazolam)
2.Select the mode (DC or synchronous mode)
3.Select the dose (Jules)
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Rhythm Disturbances“Arrhythmias“
May be shockablewhen
• Pulse is present • Unstable patient
Non shockable(No pulse)
Shockable(No pulse)
Ventricular Fibrillation
Pulseless V T
Ventricular Tachycardia Asystole
PEASupraventricular Tachycardia
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ARRHYTHMIAS
Svt Vt Vf/pulseless VT Bradycardia Asystole
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BRADYCARDIA S/S MANAGEMENT
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YES
Is It 2nd Degree II AV BlockOr 3rd Degree AV Block
NO YES
Observe
Prepare For Trans-venous Pacer
Use TCP AS Bridge Device
BRADYCARDIA
ALGORITHM
Serious Signs & Symptoms? Signs of poor perfusion caused by Bradycardia
5 T’s 5 H’s
Assess CAB, Secure Airway, V/SStart IV, History, Physical Exam,
12 Lead ECG, Cardiac Monitoring Pulse Oxymetry, O2 therapy
No
• Atropine 0.5 Q3 – 5 min
• TCP
• Dopamine 2 – 10 ug/kg/min
• Epinephrine 2 – 10 ugm/min
Intervention Sequence:
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Toxins Tamponade, cardiacTension pneumothoraxThrombosis, MI / PETrauma (Increased ICP)
HypovolemiaHypoxiaHydrogen ion (acidosis)Hyper/hypokalemiaHypothermiaHypoglycemia
Attach to monitor or defibrillatorobtain IV access rapid NS infusion
take blood samples for Lab
PEA ALGORHYTHMUnresponsive patient
CAB: No pulse >> start CPR 30:2(MOVE )
Epinephrine 1 mg IV
Continue CPR
Quick exam and history for cause5H, 6Ts
PROBLEMS
HypoxiaVentilation
HypothermiaIncrease core tempe
Tension PneumothoraxNeedle Decompression
Tamponade CardiacPericardiocentesis
HypovolemiaVolume Infusion
Hydrogen ion (acidosis)NaHCo3
Hyper >> Ca Gluconate Insuline/Glucose, Na Hco3
Tablets (Drug Over Dose)lavage, Charcoal, Anti Dote
Thrombosis, coronary MI(MONA, HIBA, PTCA, CABG)
Epinephrine 1 mg IV EO cycle ORm1 dose of Vasopressin 40Uto replace 1st t or 2nd dose of EPI
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Hand and Foot infections
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F B
I NGESTION
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Notice anything else?
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•A case of iron
deficiency?
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ThanksWith my
best wishes
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If asystole or PEA go to protocol --------------------- If pulse present, start post CPR care
Give 5 cycles of CPR (2 min)
Shockable
Check rhythmShockable rhythm?
Give 5 cycles of CPR (2 min)
Continue CPR while charching defibrillator Give one shock Resume CPR Epinephrine 1 mg Q 3-5 min IV/IO/ET Vasopressin 40u IV/IO/ET
IV/IO, ETT
Give 1 shock Biphasic device 200J Monophasic device 360JResume CPR immediately
Check rhythmShockable rhythm? No
SHOCKABLE
No
Continue CPR Give one shock Amiiodaron 300 mg IV/IO once then Consider additional 150 mg IV/IO
Continue CPR Give one shock Lidocaine 1 – 1.5 mg/kgm IV/ IO/ET, then 0.5 – 0.75 mg/kgm max 3 doses
Continue CPR Give one shock Procainamide 17 mg/kg IV Infusion Over 30 min Na Hco3 1mEq/kg
Continue CPR Give one shock Mg Sulphate 1 – 2 g IV/IO In TDP or Refractory VF
Pulseless arrest BLS algorithm: call for help Give O2 Attach monitor/Defibrillator when available
VF/pulless VTAsystole/PEA
Non Shockable
Shockable
Check rhythmShockable rhythm?
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