Shigellosis by Nelson Munthali (DNC/RN)

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GROUP 5 MEMBERS 1. Alex Mbewe 2. Monica Banda 3. Rosella Munyenyembe 4. Andrew Moyo 5. Nelson Munthali 6. Mtisunge Wandale 7. Jacqualine Ntaba

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Transcript of Shigellosis by Nelson Munthali (DNC/RN)

Page 1: Shigellosis by Nelson Munthali (DNC/RN)

GROUP 5 MEMBERS1. Alex Mbewe2. Monica Banda3. Rosella Munyenyembe4. Andrew Moyo5. Nelson Munthali6. Mtisunge Wandale7. Jacqualine Ntaba

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PRESENTS SHIGELLOSIS

By ALEX ‘ SIAL’ MBEWE

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BROAD OBJECTIVE

By the end of this presentation, learners should acquire knowledge on how to manage a patient with shigellosis.

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SPECIFIC OBJECTIVESO Definition of shigellosisO Causes of shigellosisO Types O How it is spreadO PathophysiologyO Clinical manifestationsO Medical managementO Nursing managementO Complications

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Definition

OThis is an acute bacterial infection of the lining of the intestines (especially large intestines)

CausesShigellosis is caused by a group

of bacteria called shigella (gram- negative organism)

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Types

1. Shigella sonei – also called group D. it is responsible for most of the cases

2. Shigella flexineri Also called group B

3. Shigella dysenteriae Can lead to outbreaks in developing

countries

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SPREADO Shigellosis is spread through fecal-

oral routeO People with shigellosis release it

through the stoolsO It spreads from one infected person

to contaminate water or food or directly to another person.

O Outbreaks are associated with poor sanitation, contaminated food or water and crowded living conditions

O Common among travelers in developing countries and workers or residents of refuge camps

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CLINICAL MANIFESTATIONS

OUsually develop about 1-7 days (average 3 days) after you come into contact with the bacteria

OAcute (sudden) abdominal pain or cramping

OAcute (sudden) feverOBlood, mucus or pus in stoolsOCrampy rectal painONausea and vomiting

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OWatery diarrhoeaOAbdominal tendernessODehydration with fast heart rate

and low BpO Loss of appetite

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Diagnostic tests

OStool cultureOWhite blood cells in stoolsOElevated blood cell count (FBC)

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PATHOPHYSIOLOGYO Once ingested, the bacteria survives the

gastric environment of the stomach and progresses to large intestines

O There, they attach to and penetrate the epithelial cells of the intestinal mucosa.

O After invasion, they multiply intracellulary and spread to neighboring epithelial cells, resulting in tissue destruction.

O It produces toxins that can attack the lining of the large intestines, causing swelling, ulcers on the intestinal wall and bloody diarrhoea.

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Pathophysiology cont….

OSeverity of diarrhoea sets apart shigellosis from regular diarrhoea and it is usually associated with bloody or pus stained diarrhoea.

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MEDICAL MANAGEMENTO The goal is to replace fluids and

electrolytesO Advise patient on dietO Self measure to avoid dehydration like

drinking electrolyte solution to replace fluids e.g. ORS

O Antibiotics only in severe cases e.g. ampicillin and ciprofloxacin 250mg BD IV-they shorten the length of illness

O Antidiarrhoea agents e.g. Loperamide 2mg BD

O I.V fluids 2-3 litres/24hrs e.g. R/LO Stop taking diuretics

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NURSING MGTOASSESSMENT - History of stool pattern and

associated symptomsO FrequencyODurationOCharacterOConsistency of stools

O history of medication use of other drugs known to cause

diarrhoea e.g. laxativesOSocial history

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NURSING MGT CONT……

OFamily historyORecent travel, stress, health

and family history of illnessOEating habits, appetite, food

intolerance especially milk and other dairy products

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Objective data Lethargy Sunken eye balls Fever Pallor Dry mucous membranes Poor skin turgor Parienal irritational Malnutrition Concentrated urine

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Physical examinationOVital signs and weight measurementOPatients’ skin is inspected for signs

of dehydrationOPoor turgor and dryness and area of

breakdown of the skinOAbdomen

• Distension• Bowel sounds• Palpate for tenderness

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Nursing diagnosisODiarrhoea r/t acute infectious

process evidenced by frequent loose and liquid stools

OFluid and electrolyte imbalance r/t diarrhoea and vomiting

ONutritional imbalance; less than body requirements r/t loss of appetite, nausea, vomiting evidenced by weight loss

OAltered thermoregulation hyperthermia r/t to the infection as evidenced by rise of temperature to 38 degrees celsius

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OAltered comfort (abdominal pain) r/t increased peristalsis evidenced by patient’s verbalization and facial expression

ORisk for anemia related to blood in stools

ORisk for altered skin integrity related to dehydration

ORisk for Hypovolemic shock r/t loss of fluids due to diarrhoea

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Interventions O Commence IV fluids as ordered e.g. R/L – to

replace lost fluids and correct electrolyte balanceO Catheterize – to monitor input and output and balance fluidsO Enforce strict IP measures to avoid cross infectionO Provide small and frequent food to normalize

nutritional status and reduce peristalsis movement

O Administer prescribed antipyretics e.g. panadol 1g tds po. This will act on the prostagrandin of the hypotharamus hence it will reduce fever.

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O Administer analgesics e.g. panadol 1g po tds to reduce pain .

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complicationsO Intestinal perforationODehydrationOHypoglycemiaOComaORectal prolapseOHypovolemic shockOBacteremiaOPeritonitis

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ReferencesO Lewis S.M., Heitkemper M.M and Dirksen

S.R. (2010). Medical surgical nursing assessment and management of clinical problems.(7thed) St Louis:C.V.Mosby

O Smeltzer S.C., Bare B.G and Hinke J.L (2010). Brunner & suddarth’s textbook of medical surgical nursing.(12th ed). Philadelphia:J.B Lippincott

O www.mayoclinic.com