RECOGNITION & TREATMENT OF SHOCK IN ANIMALS EMERGENCY PROCEDURES.
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Transcript of RECOGNITION & TREATMENT OF SHOCK IN ANIMALS EMERGENCY PROCEDURES.
![Page 1: RECOGNITION & TREATMENT OF SHOCK IN ANIMALS EMERGENCY PROCEDURES.](https://reader036.fdocuments.us/reader036/viewer/2022062519/5697c01a1a28abf838cced57/html5/thumbnails/1.jpg)
RECOGNITION & TREATMENT OF SHOCK
IN ANIMALS
EMERGENCY PROCEDURES
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SHOCK: RECOGNITION AND TREATMENT
SHOCK is inadequate tissue perfusion resulting in poor oxygen deliveryCardiogenicDistributiveObstructiveHypovolemic
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Shock• TYPES OF SHOCK:–Cardiogenic: results from heart failure • ↓ blood pumped by heart• HCM, DCM, valvular
insufficiency/stenosis
–Distributive: blood flow maldistribution (Vasodilation)• Sepsis, anaphylaxis →↓arteriole
resistance →loss of fluid from vessels to interstitial spaces →↓BP→ ↓ blood return to heart
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SHOCK: RECOGNITION & TREATMENTTYPES OF SHOCK:–Obstructive - physical obstruction in circulatory system• HW disease → heart pumping against
the adult worm blockage• Gastric torsion →↓blood return to
heart
–Hypovolemic - decreased intravascular volume• Most common in small animals• Blood loss, dehydration from
excessive vomiting/diarrhea, effusion of fluid into 3rd spaces
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HYPOVOLEMIC SHOCK
Pathophysiology of hypovolemic shock:
↓blood vol →↓venous return, ↓vent filling →↓stroke vol, ↓CO →↓BP
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HYPOVOLEMIC SHOCK Stage I: Compensation
◦ Baroreceptors detect hypotension (↓BP)a. Sympathetic reflex—(Epi, Norepi, cortisol
released from adrenals)- ↑ HR, contractility- Constriction of arterioles (↑BP) to skin (cold,
clammy), muscles, kidneys, GI tract; not brain, heart
b. Renin (kidney)→angiotensin (blood)→aldosterone (adrenals) reflex- ↑ Na+ and water retention → ↑ intravascular vol
(↑BP)
◦ PE findings Tachycardia Prolonged cap refill time Pale mm
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HYPOVOLEMIC SHOCK• Pathophysiology of hypovolemic shock
• Stage II: Decompensation– Tachycardia–Delayed cap refill time–Muddy mm (loss of pink color, more
brown than pink)–BP IS DROPPING–Altered mental state
• Stage III: Irreversible shock– PE findings worsen– cannot revive– death will occur
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HYPOVOLEMIC SHOCK Treatment: the goal of therapy is
to improve O2 deliveryO2 supplementation
Face maskO2 cage/hoodsTranstracheal/nasal insufflation
Venous accessCephalicSaphenousJugularIntraosseous
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Oxygen supplementation
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FLUID ADMINISTRATION
CEPHALIC CATHETER PLACEMENT
INTRAOSSEOUS CATHETER PLACEMENT
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CONTRAINDICATED IN PATIENTS WITHSEPSIS,FRACTURES, OR INFECTED BONES
INTRAOSSEOUS CATHETER PLACEMENT
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SHOCK • Treatment–Fluid resuscitation (O2 delivery is improved by ↑CO)1. Crystalloids • Isotonic solutions (electrolytes: Na+, Cl-, K+,
bicarbonate)– Examples (body fluid=280-300 mOsm/L)» Lactated Ringer’s (273 mOsm/L)»Normal saline (0.9%) (308 mOsm/L)
– Dose: Dog 50-90 ml/kg/hr Cat 40-60 ml/kg/hr
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SHOCK• Treatment:• 1. Crystalloids continued• Hypertonic solutions—when lg vol of fluid cannot
be administered rapidly enough– Examples—7.5% saline– Causes fluid shift from intercellular space→
intravascular space →↑vascular vol →↑venous return → ↑CO
– Also causes vasodilation → ↑ tissue perfusion– Dose: 4-6 ml/kg over 5 min
• Hypotonic solutions should never be used for hypovolemic shock– Examples—5% Dex in water (252 mOsm/L)
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SHOCK Treatment
Fluid resuscitation (O2 delivery is improved by ↑CO)2. Colloids—Large molecular wt solutions that do not leave
vascular systemBetter blood volume expanders than crystalloids50-80% of infused volume stays in blood vesselsExamples
Whole bloodPlasma Dextran 70, Hetastarch, Vetstarch
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SHOCK• Treatment (continued)
– SympathomimeticsUse only after adequate fluid administration if BP and
tissue perfusion have not returned to normal• Dopamine (Inotropin®)– 0.5-3.0 μg/kg/min
» Dilation of renal, mesenteric, coronary vessels– 3.0-7.5 μg/kg/min
» ↑ contractility of heart» ↑ HR
– >7.5μg/kg/min
» Vasoconstriction • Dobutamine (Dobutrex®)– ↑ contractility of heart (min effect on HR)
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SHOCK• Monitoring Hemodynamic/metabolic sequelae of shock are continually
changing– Physical Parameters• Respiratory– Color of mm– RR – Breathing efforts smooth?– Breathing pattern regular?– Auscultation normal?
• Cardiovascular – HR normal?– ECG normal?– Color of mm– Cap refill time (1-2 sec)– Urine production? (1-2 ml/kg/hr)– Weak pulse? → ↓stroke volume
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SHOCK Monitoring
Physiologic Monitoring ParametersO2 Saturation
Pulse oximetry—noninvasiveNormal: Hb saturations (SpO2)>95%
oSpO2<90%--serious hypoxemiaArterial BP—a product of CO, vascular capacity,
blood volume
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SHOCK• Monitoring –Laboratory Parameters• Hematocrit (PCV)– Increase →dehydration–Decrease →blood loss
• Electrolytes– Proper balance needed for proper cell
function– Fluid therapy may alter the balance;
supplement fluid as needed
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SHOCK• Monitoring• Arterial pH and blood gases– PaCO2 tells how well patient is ventilating» PaCO2 <35 mm Hg → hyperventilation
» PaCO2 >45 mm Hg → hypoventilation
– PaO2 Tells how well patient is being oxygenated» PaO2 <90 mm Hg → hypoxemia
– pH tells acid/base status of patient–<7.35 → acidosis–>7.45 → alkalosis