PEDIATRIC Advanced Life Support Neva Batayola, MD Pediatric Critical Care.
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Transcript of PEDIATRIC Advanced Life Support Neva Batayola, MD Pediatric Critical Care.
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PEDIATRIC Advanced
Life Support
Neva Batayola, MDPediatric Critical Care
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What is PALS all about?
Evaluating and recognizing an infant or child with respiratory compromise, circulatory compromise, or cardiac arrest
Giving timely and appropriate treatment or interventions
Applying effective team dynamics, observing individual roles and responsibilities during pediatric resuscitation
Providing optimal post resuscitation management
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Pediatric Chain of Survival
Berg, M. D. et al. Circulation 2010;122:S862-S875
prevention Early CPR EMS Rapid PALS IntergratedPost-cardiacArrest care
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BLS: foundation of saving lives
Fundamental aspects: immediate recognition of sudden cardiac
arrest ( unconsciousness) activation of emergency response system
( call 911 ) early performance of CPR (C A B steps) rapid defibrillation (AED) when appropriate
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NEW OLD
CPR: ABC IS FOR BABIES. NOW IT’S C-A-B!
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High quality CPR…
Chest compressions of appropriate rate and depth. "Push fast": push at a rate of at least 100 compressions per minute. "Push hard": push with sufficient force to depress the chest (at least 1/3 of the AP diameter of the chest or approximately 1½ in. = 4 cm in infants and approximately 2 in. = 5 cm in children)
allowing complete recoil of the chest after each compression
minimizing interruptions in compressions
avoiding excessive ventilation
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High quality CPR = Effective PALS
the cornerstone of a system of care that can optimize outcomes beyond return of spontaneous circulation (ROSC).
Return to a prior quality of life and functional state of health is the ultimate goal of a resuscitation system of care.
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Pathway to Pathway to pediatric cardiac pediatric cardiac arrestarrest
AHA Pediatric Advanced Life Support. 2006AHA Pediatric Advanced Life Support. 2006
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Assessment: Key to Pediatric Management
AHA Pediatric Advanced Life Support AHA Pediatric Advanced Life Support Manual 2006Manual 2006
Life threatening
Not life threatening
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What WeWhat WeHad…Had…
Assess-Categorize-Decide-Act Model Assess-Categorize-Decide-Act Model
Pediatric Advanced Life Support 2006Pediatric Advanced Life Support 2006
General Assessment ( P A T )Primary AssessmentSecondary AssessmentTertiary Assessment
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The PAT & the Primary, Secondary & Tertiary Surveys
AHA Pediatric Advanced Life Support. 2006AHA Pediatric Advanced Life Support. 2006
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WhatWhat’s ’s NEWNEW
……
Evaluate-Identify-Intervene Sequence Pediatric Advanced Life Support 2010Evaluate-Identify-Intervene Sequence Pediatric Advanced Life Support 2010
E v a l u a t e Primary assessment Secondary assessment Diagnostic tests
I d e n t i f y
I n t e r v e n e
I n i t i a l I m p r e s s i o n
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The Initial Impression
A modification of the PAT, the goal of which is to help one quickly recognize a child at risk for deterioration and prioritize actions and interventions
The first quick (within seconds) “from the doorway” visual and auditory observation of the child’s consciousness, breathing and color
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C – B - C Initial Impression
Consciousness Unresponsive, irritable, alert
Breathing
Increased work of breathing, absent or decreased respiratory effort, or abnormal sounds heard without ausculation
Color Pallor, mottling, cyanosis
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Initial Impression: DECISION & ACTION POINTS
Unresponsive and not breathing or only gaspingCall for helpCheck pulse (-) pulse, start CPR beginning with compressions
if with ROSC begin E-I-I sequence (+) pulse rescue breathing
HR<60 & poor perfusion despite adequate oxygenation/ventilation chest
compressions & ventilations
HR>60 begin EII sequence
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Initial Impression: DECISION & ACTION POINTS
Findings normal or non-urgent, child breathing adequately
begin E-I-I sequence
Always be alert to a life-threatening situation. If at any point you identifya life-threatening problem, call for
help and begin lifesaving interventions.
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The E-I-I Sequence: Evaluate
Clinical Assessment What It Is
Primary Assessment Rapid, hands-on ABCDE approach evaluating respiratory, cardiac & neurologic function; includes vital signs & pulse oximetry
Secondary AssessmentFocused medical history & physical exam
Diagnostic TestsLaboratory, radiographic & other advanced tests that help to identify the child’s physiologic condition & diagnosis
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Pediatric Primary Assessment
rapid ordered, stepwise hands-on evaluation of cardiopulmonary and neurologic function to prioritize treatment
Includes vital signs & O2 saturation by pulse oximetry
Airway, Breathing, Airway, Breathing, Circulation, Disability, Circulation, Disability, ExposureExposure
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Pediatric Primary Assessment
AIRWAYopen?movement of the chest/abdomen?air movement and breath sounds?
Decide if: Clear – open / unobstructed Maintainable – simple measures not maintainable - advanced
interventions
AHA Pediatric Advanced Life Support.2010AHA Pediatric Advanced Life Support.2010
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Pediatric Primary Assessment
BREATHING
Respiratory rate (RR)Normal, Irregular, Fast, Slow, Apnea
Respiratory effortNormal, Increased, Inadequate
Chest expansion & air movement (TV)Normal, Decreased, Unequal, Prolonged expiration
Lung and airway sounds Pulse oximetry (SaO2)
Normal, HypoxemicAHA Pediatric Advanced Life Support.2010AHA Pediatric Advanced Life Support.2010
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CIRCULATION Heart Rate (HR) & rhythm Pulses (central & peripheral) CRT Skin color and temperature Blood Pressure (BP); in
children <3 yrs, attempt only once
Level of consciousness Urine output
Pediatric Primary AssessmentPediatric Primary Assessment
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DISABILITY AVPU Pediatric Response
Scale (cerebral cortex fxn) GCS Pupillary response Blood sugar
EXPOSURE
Hypo/hyperthermia Evidence of trauma
or injury Rash
Pediatric Primary AssessmentPediatric Primary Assessment
Decreased LOCLoss of muscle toneIrritability, lethargy, agitationGeneralized seizuresPupil dilatation
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Pediatric Secondary Assessment
Focused history Signs and symptoms Allergies Medications Past Medical History Last Meal Events
Detailed PE
SS
AA
MMPP
LL
EE
Focused medical hx Focused medical hx using SAMPLE using SAMPLE mnemonic and a mnemonic and a thorough head-to-toe thorough head-to-toe P.E.P.E.AHA Pediatric Advanced Life Support. AHA Pediatric Advanced Life Support.
20102010
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Diagnostic Tests Assessment of respiratory and circulatory
abnormalities
ABG, VBG, Hb, Blood sugarPulse oximetry, CXRCapnography (ETC02), exhaled C02 Sv02 saturation, arterial lactateCVP, 2DEcho, ECG, PEFRInvasive arterial pressure monitoring
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The E-I-I Sequence: IDENTIFY Type Severity
Respiratory Upper Airway Obstruction Respiratory Distress
Lower Airway Obstruction Respiratory Failure
Lung Tissue Disease
Disordered Control of
Breathing
Circulatory Hypovolemic Shock Compensated Shock
Distributive Shock Hypotensive Shock
Cardiogenic Shock
Obstructive Shock
Cardiopulmonary Failure
Cardiac Arrest
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The E-I-I Sequence: INTERVENE
Positioning to maintain a patent airway Activating ERS or calling a code Starting CPR Obtaining the code cart & monitor Placing the pt on a cardiac monitor & pulse oximeter Administering oxygen Supporting ventilation Starting medications & fluids (e.g., nebulizer
treatment, IV/IO fluid bolus)
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Let’s look at a scenario…
You are on duty at the ER and the nurse asks you evaluate a 10-yr-old with difficulty
breathing 15 min after eating.
Initial impression: anxious, with increased inspiratory effort and stridor, with pale skin
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IDENTIFY the problem
Respiratory distress or respiratory failure
INTERVENE
Open airway if needed, give 100% O2 via non-rebreathing mask in tolerated, attach to
monitor, apply pulse oximeter
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EVALUATE – Primary Assessment
Airway: inspiratory stridor Breathing: RR 30/min, deep suprasternal retractions,
nasal flaring, poor aeration on auscultation, SP02 90% room air
Circulation: HR 130/min, peripheral pulses normal, CRT 2 sec, BP 115/75 mmHg
Disability: somewhat anxious Exposure: T 37ºC
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IDENTIFY
Respiratory distress vs respiratory failure;
Upper Airway Obstruction
Assess response to 02; analyze cardiac rhythm
INTERVENE
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EVALUATE – Secondary Assessment: SAMPLE History
Signs and symptoms: difficulty breathing 15 min after eating a cookie
Allergies: Peanuts Medications: None Past medical history: previously healthy Last meal: had only a cookie since breakfast Events: difficulty of breathing began within several min
of eating a cookie
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EVALUATE – Secondary Assessment: P.E.
Vital signs after 02: HR 120/min RR 20/min SP02 98% at 100% 02 BP 115/75 mmHg
HEENT: stridor at rest Heart & Lungs: no murmur, breath sounds course, CRT 2 sec Abdomen: normal Extremities: no edema Back: normal Neurologic: somewhat anxious
IDENTIFY
Respiratory distress vs respiratory failure; Upper Airway Obstruction
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IDENTIFY
Respiratory distress vs respiratory failure; Upper Airway Obstruction
Allow position of comfort; consider specific interventions for UAO (eg. Racemic epinephrine, IV/IM dexamethasone, helium-02 mixture, etc.; consider vascular access IV/IO; prepare for
endotracheal intubation
INTERVENE
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EVALUATE – Diagnostic Tests
ABG / VBG, electrolytes, BUN/creatinine, glucose, CBC with differential
Imaging as appropriate
RE-EVALUATE – IDENTIFY – INTERVENE after each intervention
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Identification of Respiratory Problems
By severity
1. respiratory distress
2. respiratory failure By type
1. upper airway obstruction
2. lower airway obstruction
3. lung tissue disease
4. disordered control of breathing
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Respiratory distress
Clinical state characterized by abnormal respiratory rate (tachypnea) or effort (increased or inadequate)
Ranges from mild to severe
Signs: tachypnea, increased/inadequate respiratory effort, abnormal airway sounds, tachycardia, pale cool skin, alteration in consciousness
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Respiratory Failure Inadequate ventilation, insufficient oxygenation, or both
Signs:- ↑RR, signs of distress (eg, ↑respiratory effort:
nasal flaring, retractions, seesaw breathing, or grunting)
- inadequate respiratory rate, effort, or chest excursion (eg, diminished breath sounds or gasping), especially if mental status is depressed
- Cyanosis with abnormal breathing despite supplementary oxygen
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Upper airway obstruction
Foreign body aspiration Epiglottitis Croup Anaphylaxis Tonsillar hypertrophy Mass compromising the airway lumen
(abscess, tumor) Congenital airway abnormality (congenital
subglottic stenosis)
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Lower airway obstruction
Obstruction of the lower airways (lower trachea, bronchi, bronchioles)
Asthma, bronchiolitis
Tachypnea, expiratory/inspiratory/biphasic wheezing, increased respiratory effort, prolonged expiratory phase
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Lung tissue disease
Heterogenous group of clinical conditions affecting the lung at the level of gas exchange, characterized by alveolar and small airway collapse or fluid-filled alveoli
Pneumonia (bacterial, viral, chemical), pulmonary edema (CHF, ARDS), pulmonary contusion, toxins, vasculitis, infiltrative disease
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Disordered control of breathing
Abnormal breathing pattern producing signs of inadequate respiratory rate, effort, or both
Neurologic disorders (seizures, CNS infections, head injury, brain tumor, hydrocephalus, neuromuscular disease)
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Initial management of respiratory distress or failure AIRWAY
position of comfortopen airway (head tilt-chin lift, modified jaw thrust)clear airway (suction, remove FB)consider OPA, NPA
BREATHINGmonitor Sp02, provide 02, assist ventilationinhaled medication as neededendotracheal intubation if needed
CIRCULATIONmonitor HR, rhythm, BPestablish vascular access as indicated
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Bag-Mask Ventilation
Appropriate face mask (extending from bridge of the nose to cleft of the chin)
Self inflating ventilation bag Bag size: 400-500 ml infant/young child
1000 ml older child/adolescent Position: neutral or sniffing E-C clamp technique
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Bag-Mask Ventilation
Breathing: EC clamp technique
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Tracheal Tube- size and depth
Uncuffed tube size:<1yr 3.5mm ID1-2 yr 4.0mm ID>2 yr 4 + (Age/4)
Cuffed tube size:<1yr 3.0 mm ID1-2 yr 3.5 mm ID>2 yr 3.5 + (Age/4)
ETT depth (lip):
ETT size x 3
Uncuffed tube size:<1yr 3.5mm ID1-2 yr 4.0mm ID>2 yr 4 + (Age/4)
Cuffed tube size:<1yr 3.0 mm ID1-2 yr 3.5 mm ID>2 yr 3.5 + (Age/4)
ETT depth (lip):
ETT size x 3AHA, Basic Life Support Textbook,2007
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Shock
Results from inadequate blood flow and oxygen delivery to meet tissue metabolic demands
Typical signs of compensated shock include TachycardiaCool and pale distal extremitiesCRT >2 sec despite warm ambient tempWeak peripheral vs central pulsesNormal systolic blood pressure
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Identification of Shock
By severity (effect on BP)Compensated shockHypotensive
By typeHypovolemic (diarrhea, vomiting, hge, burns)Distributive (septic, anaphylactic, neurogenic)Cardiogenic (CHD, myocarditis, arrhythmias, sepsis)Obstructive (cardiac tamponade, tension
pneumothorax, ductal-dependent lesions, massive PE)
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Blood Pressure
Typical SBP 1-10 y.o. (50th percentile)
90 + (age in yrs x 2) mmHg
Hypotension (5th percentile)
term neonates <60mmHg
up to 12 months <70mmHg
1-10 yrs: 70 + (age in yrs x 2 ) mmHg
>10 yrs <90mmHg Typical MAP: 55 + (age in yrs x 1.5) mmHg
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COMPENSATED SHOCK
HYPOTENSIVE SHOCK
CARDIAC ARREST
Possibly Hours
Potentially Minutes
AHA Pediatric Advanced Life Support Manual 2011AHA Pediatric Advanced Life Support Manual 2011
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Shock management
Optimizing 02 content of the blood Improving volume & distribution of
cardiac output Reducing 02 demand Correcting metabolic derangements Identifying and reversing the underlying
cause of shock
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10 steps of goal-directed management of pediatric shock
1. Recognize shock at time of triage
2. Transfer pt immediately to shock/trauma room and amass resuscitation team
3. Begin Oxygen and establish IV access using 90 sec for peripheral attempts
4. If unsuccessful after 2 peripheral attempts, consider IO
5. Palpate for hepatomegaly; auscultate for rales
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10 steps of goal-directed management of pediatric shock
6. If liver is up and if no rales are present, push 20ml/kg boluses of isotonic saline up to 60ml in 5-10min until improved perfusion or liver comes down or patient develops crackles. Give blood if with unresponsive hemorrhagic shock
If liver is down, beware of cardiogenic shock. Consider inotropic support ( PGE1 to maintain ductus arteriosus in all neonates).
7. If CRT>2 sec and/or hypotension persists during fluid resuscitation, begin IO / peripheral Epinephrine
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10 steps of goal-directed management of pediatric shock
8. If at risk for adrenal insufficiency give hydrocortisone as bolus (50mg/kg) and then as infusion titrating between 2-50 mg/kg/day
9. If continued shock, intubate and support ventilation mechanically.
10. Direct therapy to goals: CRT < 3sec, normal BP for age, improving shock index.
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Therapeutic End Points
RESUSCITATION TO CLINICAL GOALS IS THE FIRST PRIORITY!
Normal mental status Normal pulses (no differential between peripheral & central) Equal central and peripheral temperatures/warm extremities CRT < 2 sec Normal HR & BP for age Urine output > 1cc/kg/hr ↓ serum lactate (<2mmol/L) Reduced base deficit Central venous 02 sat (SvO2) > 70%
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Hemodynamic Support
Dopamine – 1st line vasopressor for fluid-refractory hypotensive shock with low SVR (10-20mcg/k/min); increase myocardial contractility after preload restoration.
Epinephrine – 1st line inotrope for fluid refractory, dopamine-resistant nonvasodilatory shock (0.02-1mcg/k/min, to as high as 2-3 mcg/kg/min in severe cases)
Norepinephrine – 1st line pressor agent for fluid refractory, dopamine-resistant vasodilatory (“warm”, hyperdynamic) shock (0.03-1.5mcg/k/min)
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Phosphodiesterase inhibitors
for catecholamine-refractory low cardiac output and high SVR
milrinone 50-75 mcg/kg iv loading 60 min0.375-0.75 mcg/kg/min continuous infusion
increases contractility & improves diastolic function by decreased degradation of cAMP and increased intracellular calcium release
Pediatric Critical Care Medicine 2005; 6:195-199
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Pediatric Critical Care Medicine 2001, 2:24-28
Phosphodiesterase inhibitors
Amiodarone (inodilator)5 mg/kg iv 30 min
5-10 mcg/kg/min infusion
improves myocardial depression and does not increase SVR or the metabolic demands of the heart
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Dobutamine (2-20mcg/kg/min)
not to be used alone in severe shock
increases cardiac contractility and decreases PVR (afterload)
Vasodilator therapy (Nitroprusside/NTG) for epinephrine-resistant low CO and elevated SVR, normal blood pressure (afterload unloader)
may need simultaneous inotropic support
always augment volume (preload)
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Vasopressin
Endogenous levels decrease in vasodilatory shock
potent vasoactive agent in the treatment of vasodilatory shock in adults and children
Dose: 0.0005-0.002 U/kg/minvarying doses in studies
Pediatr Crit Care Med 2008 Vol. 9, No. 4 Vasopressin in pediatric vasodilatory shock: a multicenter randomized controlled trial. Choong K. et al., Am J Crit Care Med. 2009 Oct 1;180(7):632-9. Epub 2009 Jul 16.
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PALS Pulseless Arrest Algorithm.
Kleinman M E et al. Pediatrics 2010;126:e1361-e1399
©2010 by American Academy of Pediatrics
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PALS Bradycardia Algorithm.
Kleinman M E et al. Pediatrics 2010;126:e1361-e1399
©2010 by American Academy of Pediatrics
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PALS Tachycardia Algorithm.
Kleinman M E et al. Pediatrics 2010;126:e1361-e1399
©2010 by American Academy of Pediatrics
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PALS means TEAMWORK
Resuscitation = medical expertise and mastery of skills = multiple tasks
Teamwork divides the tasks while multiplying the chances of
success
Successful resuscitation = effective communication and team dynamics
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If you want to be on the team & make a difference…
Learn the science of PALS and learn it well
Understand your role and the role of every member of your team in resuscitation
Understand how teamwork increases the chances of resuscitation success
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The Resuscitation Team
Team leader Airway Compressor IV / IO meds Monitor / Defibrillator Observer/ Recorder
Team leader
airway
comressor
Observer/ recorder
IV/IV/IO meds
Monitor/
defibrillator
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Elements of effective resuscitation team dynamics
Closed-loop communication Clear messages Clear roles and responsibilities Knowing limitations Knowledge sharing Constructive intervention Reevaluation and summarizing Mutual respect
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THANK YOU