Opener A “Smart Drug”. Figure 17.1 One Gene, One Enzyme (Part 1)
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Transcript of Opener A “Smart Drug”. Figure 17.1 One Gene, One Enzyme (Part 1)
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Opener A “Smart Drug”
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Figure 17.1 One Gene, One Enzyme (Part 1)
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Figure 17.1 One Gene, One Enzyme (Part 2)
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Figure 17.2 Hemoglobin Polymorphism
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Figure 17.3 Genetic Diseases of Membrane Proteins (Part 1)
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Figure 17.3 Genetic Diseases of Membrane Proteins (Part 2)
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Figure 17.3 Genetic Diseases of Membrane Proteins (Part 3)
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Figure 17.3 Genetic Diseases of Membrane Proteins (Part 4)
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Figure 17.4 Prion Proteins
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Figure 17.5 A Fragile-X Chromosome at Metaphase
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Figure 17.6 Strategies for Isolating Human Genes
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Figure 17.7 RFLP Mapping (Part 1)
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Figure 17.7 RFLP Mapping (Part 2)
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Figure 17.8 5-Methylcytosine in DNA Is a “Hot Spot” for Mutagenesis (Part 1)
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Figure 17.8 5-Methylcytosine in DNA Is a “Hot Spot” for Mutagenesis (Part 2)
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Figure 17.9 The CGG Repeat in the Fragile-X Gene Expands with Each Generation
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Figure 17.10 Genetic Screening of Newborns for Phenylketonuria (Part 1)
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Figure 17.10 Genetic Screening of Newborns for Phenylketonuria (Part 2)
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Figure 17.11 DNA Testing by Allele-Specific Cleavage
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Figure 17.12 DNA Testing by Allele-Specific Oligonucleotide Hybridization (Part 1)
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Figure 17.12 DNA Testing by Allele-Specific Oligonucleotide Hybridization (Part 2)
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Figure 17.13 A Cancer Cell with Its Normal Neighbors
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Figure 17.14 Dividing Cells Are Especially Susceptible to Genetic Damage
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Figure 17.15 Oncogene Products Stimulate Cell Division
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Figure 17.16 The “Two-Hit” Hypothesis for Cancer
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Figure 17.17 Tumor Suppressor Gene Products Inhibit Cell Division and Cancer
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Figure 17.18 Multiple Mutations Transform a Normal Colon Epithelial Cell into a Cancer Cell (1)
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Figure 17.18 Multiple Mutations Transform a Normal Colon Epithelial Cell into a Cancer Cell (2)
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Figure 17.18 Multiple Mutations Transform a Normal Colon Epithelial Cell into a Cancer Cell (3)
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Figure 17.19 Strategies for Killing Cancer Cells
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Figure 17.20 Gene Therapy: The Ex Vivo Approach (Part 1)
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Figure 17.20 Gene Therapy: The Ex Vivo Approach (Part 2)
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Figure 17.21 Two Approaches to Sequencing DNA
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Figure 17.22 The Human Genome
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Figure 17.23 Is This the Future of Medicine?
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Figure 17.24 Proteomics (Part 1)
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Figure 17.24 Proteomics (Part 2)
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Table 17.1 Human Cancers Known To Be Caused by Viruses