MED 1.3 HEENT

13
TRANSCRIBED BY: FRED, GEORGE, RON, BILL, GINNY Page 1 of 13 Medicine Faculty “What a slut time is. She screws everybody.” — John Green, The Fault in Our Stars HEENT 1.3 9 June 2014 THE HEAD COMMON OR CONCERNING SYMPTOMS Headache o Most common symptom (30% of the general population) o Careful evaluation for life-threatening causes: meningitis, subdural or intracranial hemorrhage, mass lesion, etc. o Elicit full description Is the headache one-sided or bilateral? Severe with sudden onset? Steady or throbbing? Continuous or intermittent? o Most important attributes are severity and chronologic pattern Severe and sudden onset t/c subarachnoid hemorrhage or meningitis o Primary Headaches No identifiable underlying cause Migraine o Most frequent cause of headache (80%) o Unilateral Tension o Bilateral, often arise in the temporal areas Cluster o Unilateral, may be retro-orbital o Secondary Headaches Arise from other condition Change in vision: hyperopia, presbyopia, myopia, scotomas Diplopia Hearing loss Vetigo Nosebleed Sore throat Swollen glands Goiter THE HEAD EXAMINATION HAIR Note its quantity, distribution, and pattern loss Check for loose flakes of dandruff o e.g. fine hair hyperthyroidism coarse hair hypothyroidism SCALP Look for scaliness, lumps, nevi, or other lesions o e.g. pigmented nevi, pillar cysts redness and scaling seborrheic dermatitis, psoriasis SKULL Observe size and contour Note deformities, depressions, lumps, or tenderness Recognize irregularities in normal skull o e.g. enlarged skull hydrocephalus, Paget’s disease tenderness/ step-offs common after trauma SKIN Observe and note color, pigmentation, texture, thickness, hair distribution, and lesions o e.g. acne in many adolescents hirsutism in women polycystic ovary syndrome FACE General appearance of the face Not patient’s facial expression and contours Observe for asymmetry, involuntary movements, edema, and masses Determine if face abnormality is a local, systemic, or neurological manifestation FACE ABNORMALITIES IN DIFFERENT DISEASES Acromegaly Enlargement of both bone and tissue due to increased growth hormone production Head is elongated with bony prominence of the forehead, nose, and lower jaw Soft tissue of the nose, lips, and ears are enlarged Facial features generally coarsened e.g. in pituitary adenoma Myxedema May be caused by decreased thyroid hormone levels Face is dull and puffy Edema is generally pronounced around the eyes, and does not pit with pressure Hair and eyebrows are dry, coarse, and thinned Skin is dry e.g. in severe hypothyroidism Nephrotic Syndrome Face is edematous and pale Swelling first appears in the morning around the eyes Eyes may become slit-like when edema is severe TOPIC OUTLINE I. The Head A. Common or Concerning Symptoms B. The Head Examination C. Face Abnormalities in Different Diseases II. The Eye A. The Eye Examination B. Visual Field Defects C. Observation of External Structures D. Abnormalities of the Eyes III. Testing Extra-Ocular Movement A. Strabismus or Squint B. Nystagmus C. Lid Lag IV. Test for Convergence V. Inspection Using the Diagnostic Instruments: Ophthalmoscope A. Using the Ophthalmoscope (*Bates) B. Inspection of Other Retinal Structures Using the Ophthalmoscope (*Bates) C. Optic Disc Abnormalities VI. Notes on Ophthalmologic Examination VII. Red Eyes VIII. Ear Examination A. External Examination B. Use of Otoscope IX. Nose Examination X. Assessment of Frontal & Maxillary Sinuses A. Techniques in the Examination of the Sinuses B. Infection of the Nasal Cavity C. Foreign Bodies in the Nose D. Nose Bleeding XI. Examination of the Mouth & Pharynx A. Techniques in the Examination of the Mouth B. Techniques in the Examination of the Pharynx XII. Examination of the Neck A. Lymph Nodes B. Trachea C. Thyroid

description

Med

Transcript of MED 1.3 HEENT

  • TRANSCRIBED BY: FRED, GEORGE, RON, BILL, GINNY

    Page 1 of 13

    Medicine Faculty

    What a slut time is. She screws everybody. John Green, The Fault in Our Stars Paulo Coelho

    HEENT

    1.3 9 June 2014

    THE HEAD COMMON OR CONCERNING SYMPTOMS

    Headache o Most common symptom (30% of the general population) o Careful evaluation for life-threatening causes: meningitis,

    subdural or intracranial hemorrhage, mass lesion, etc. o Elicit full description

    Is the headache one-sided or bilateral? Severe with sudden onset? Steady or throbbing? Continuous or intermittent?

    o Most important attributes are severity and chronologic pattern Severe and sudden onset t/c subarachnoid hemorrhage

    or meningitis o Primary Headaches

    No identifiable underlying cause Migraine

    o Most frequent cause of headache (80%) o Unilateral

    Tension o Bilateral, often arise in the temporal areas

    Cluster o Unilateral, may be retro-orbital

    o Secondary Headaches Arise from other condition

    Change in vision: hyperopia, presbyopia, myopia, scotomas

    Diplopia

    Hearing loss

    Vetigo

    Nosebleed

    Sore throat

    Swollen glands

    Goiter

    THE HEAD EXAMINATION HAIR

    Note its quantity, distribution, and pattern loss

    Check for loose flakes of dandruff o e.g. fine hair hyperthyroidism

    coarse hair hypothyroidism

    SCALP

    Look for scaliness, lumps, nevi, or other lesions o e.g. pigmented nevi, pillar cysts

    redness and scaling seborrheic dermatitis, psoriasis

    SKULL

    Observe size and contour

    Note deformities, depressions, lumps, or tenderness

    Recognize irregularities in normal skull o e.g. enlarged skull hydrocephalus, Pagets disease

    tenderness/ step-offs common after trauma

    SKIN

    Observe and note color, pigmentation, texture, thickness, hair distribution, and lesions o e.g. acne in many adolescents

    hirsutism in women polycystic ovary syndrome

    FACE

    General appearance of the face

    Not patients facial expression and contours Observe for asymmetry, involuntary movements, edema, and

    masses

    Determine if face abnormality is a local, systemic, or neurological manifestation

    FACE ABNORMALITIES IN DIFFERENT DISEASES

    Acromegaly

    Enlargement of both bone and tissue due to increased growth hormone production

    Head is elongated with bony prominence of the forehead, nose, and lower jaw

    Soft tissue of the nose, lips, and ears are enlarged

    Facial features generally coarsened

    e.g. in pituitary adenoma

    Myxedema

    May be caused by decreased thyroid hormone levels

    Face is dull and puffy

    Edema is generally pronounced around the eyes, and does not pit with pressure

    Hair and eyebrows are dry, coarse, and thinned

    Skin is dry

    e.g. in severe hypothyroidism

    Nephrotic Syndrome

    Face is edematous and pale

    Swelling first appears in the morning around the eyes

    Eyes may become slit-like when edema is severe

    TOPIC OUTLINE

    I. The Head A. Common or Concerning Symptoms B. The Head Examination C. Face Abnormalities in Different Diseases

    II. The Eye A. The Eye Examination B. Visual Field Defects C. Observation of External Structures D. Abnormalities of the Eyes

    III. Testing Extra-Ocular Movement A. Strabismus or Squint B. Nystagmus C. Lid Lag

    IV. Test for Convergence V. Inspection Using the Diagnostic Instruments:

    Ophthalmoscope A. Using the Ophthalmoscope (*Bates) B. Inspection of Other Retinal Structures Using the

    Ophthalmoscope (*Bates) C. Optic Disc Abnormalities

    VI. Notes on Ophthalmologic Examination VII. Red Eyes VIII. Ear Examination

    A. External Examination B. Use of Otoscope

    IX. Nose Examination X. Assessment of Frontal & Maxillary Sinuses

    A. Techniques in the Examination of the Sinuses B. Infection of the Nasal Cavity C. Foreign Bodies in the Nose D. Nose Bleeding

    XI. Examination of the Mouth & Pharynx A. Techniques in the Examination of the Mouth B. Techniques in the Examination of the Pharynx

    XII. Examination of the Neck A. Lymph Nodes B. Trachea

    C. Thyroid

  • TRANSCRIBED BY: FRED, GEORGE, RON, BILL, GINNY

    Page 2 of 13

    HEENT

    Cushings Syndrome Increased adrenal hormone

    causes round or moon face with red cheeks

    Excessive hair growth in the mustache and sideburn areas

    Parotid Enlargement

    Has swellings anterior to the ear lobes and above angle of jaws

    Gradual unilateral enlargement suggests neoplasm

    Acute enlargement is seen in mumps

    Bilateral asymptomatic enlargement suggests obesity, DM, and cirrhosis

    Parkinsons Disease Decreased facial mobility

    and blunt expression

    Mask-like face

    Decreased blinking and characteristic stare

    Facial skin is oily

    Drooling may occur

    Since neck and upper trunk tend to flex forward, patient seems to peer upward toward the observer

    THE EYE

    Start inquiry about eye and vision problems with open-ended questions o How is you vision? o Have you had any trouble with your eyes?

    Ask if there is blurring of vision. If yes, is the onset sudden or gradual? If sudden and unilateral, is the visual loss painless or painful?

    As if the patient wears glasses.

    Ask about pain in or around the eyes, redness, and excessive tearing or watering.

    Check for diplopia or double vision o Horizontal Diplopia

    Images display side by side o Vertical Diplopia

    Images are on top of each other

    THE EYE EXAMINATION IMPORTANT AREAS OF EXAMINATION

    Visual acuity

    Visual fields

    Conjunctiva and sclera

    Cornea, lens, pupils

    Extraocular movements

    Fundi (includes optic disc, retina, retinal vessels)

    VISUAL ACUITY

    First part of the eye exam is an assessment of acuity

    Can be done with either a standard Snellen chart hanging on a wall, with the patient standing at a distance of 20ft or a specifically designed pocket card held at 14in

    Each eye is tested independently (e.g. one is covered while the other is used to read)

    Patient who use glasses should put them on, and the results are referred to as best corrected vision

    If they have no complaints, rapidly skip down to the smaller characters

    Record visual acuity expressed as 2 numbers (e.g. 20/20)

    The numbers at the end of the line provide an indication of the patients acuity compared with normal subjects; the larger the denominator, the worse the acuity o e.g. 20/200 means that the patient can see at 20ft what a

    normal individual can at 200ft

    o First number indicates the distance of the patient from the chart, and the second, the distance at which a normal eye can read the line of letters

    If the patient is unable to read any of the lines, a gross estimate of what they are capable of seeing should be determined (e.g. ability to detect light, motion, or number of fingers placed in front of them)

    Acuity is only tested when there is a new specific, visual complaint

    PINHOLE TESTING

    Determine if a problem with acuity is due to refractive error (thus correctable with glasses)

    Patient is instructed to view the Snellen chart with the pinholes up, and then again with them in the down position

    If deficit corrects with the pinholes in place, the acuity issue is related to a refractive error

    Pinholes allow the passage of light which is perpendicular to the lens and thus does not need to be bent prior to being focused on the retina

    VISUAL FIELDS

    Entire area seen by an eye when it looks at a central point

    Center of the circle represents the focus of gaze; the circumference is 90 from the line of gaze

    The fields extend farthest on the temporal sides, normally limited by the brows above, the cheeks below, and the nose medially

    The normal visual field for each eye extends out from the patient in all directions, with an area of overlap directly in front

    Field cuts specific regions where the patient has lost their ability to see o Occurs when the transmitted visual impulse is interrupted at

    some point in its path from the retina to the visual cortex in the back of the brain

    You would, in general, only include a visual field assessment if the patient complained of loss of sight; in particular blind spots or holes in their vision

    Visual fields can be assessed as follows:

    1. The examiner should be nose to nose with the patient, separated by approximately 8-12in.

    2. Each eye is checked separately. The examiner closes one eye and the patient closes the opposite. The open eyes should then be staring directly at one another.

    3. The examiner should move their hand out towards the periphery of his/her visual field on the side where the eyes are open. The finger should be equidistant from both persons.

    4. The examiner should then move the wiggling finger in towards them, along an imaginary line drawn between the two persons. The patient and examiner should detect the finger at more or less the same time.

    5. The finger is then moved out to the diagonal corners of the field and moved inwards from each of these directions. Testing is then done starting at a point in front of the closed eyes

  • TRANSCRIBED BY: FRED, GEORGE, RON, BILL, GINNY

    Page 3 of 13

    HEENT

    6. The wiggling finger is moved towards the open eyes. 7. The other eye is then tested

    Meaningful interpretation is predicated upon the examiner having normal fields, as they are using themselves for comparison.

    CONFRONTATION

    Starts in the temporal fields because most defects involve these areas

    Imagine the patients visual field encircling the front of the patients head. 1. Ask patient to look with both eyes into your eyes. 2. Place your hands about 2ft apart, lateral to the patients ear. 3. Ask the patient to point at your fingers as soon as they are

    seen. 4. Slowly move the wiggling fingers of both your hands along the

    imaginary circle and toward the line of gaze until the patient identifies them.

    5. Repeat this pattern in the upper and lower temporal quadrants.

    Usually, a person sees both sets of fingers at the same time.

    VISUAL FIELD DEFECTS

    1. Horizontal Defect

    Occlusion of a branch of the central retinal artery may cause a horizontal (altitudinal) defect. Shown is the lower field defect associated with occlusion of the superior branch of this artery

    2. Blind Right Eye (right optic nerve)

    Optic nerve lesion, and of course of the eye itself, produces unilateral blindness

    3. Bitemporal Hemianopsia (optic chiasm)

    Lesion at the optic chiasm may involve only the fibers that are crossing over to the opposite side; since these fibers originate in the nasal half of each retina, visual loss involves the temporal half of each field

    4. Left Hemianopsia (right optic tract)

    Optic tract lesion interrupts fibers originating on the same side of both eyes; visual loss in the eyes is therefore similar (homonymous) and involves half of each field (hemianopsia)

    5. Homonymous Left Superior Quadrantic Defect (right optic radiation, partial)

    Partial lesion of the optic radiation may involve only a portion of the nerve fibers, producing, for example, a homonymous quadrantic effect

    6. Left Homonymous Hemianopsia (right optic radiation)

    Complete Interruption of fibers in the optic radiation produces a visual defect similar to that produced by a lesion of the optic tract

    If you suspect a temporal defect in the visual field 1. Ask the patient to cover the right eye, and with the left eye,

    to look into your eye which is directly opposite (right eye) 2. Slowly move your wiggling fingers from the defective are

    toward the better vision

    OBSERVATION OF EXTERNALL STRUCTURES

    Position and Alignment of the Eye o Ocular Symmetry

    Occasionally, one of the muscles that control eye movement will be weak or foreshortened, causing one eye to appear deviated medially or laterally compared with the other

    o Eyelid Symmetry Both eyelids should cover approximately the same amount

    of eyeball Damage to the nerves controlling these structures can

    cause the upper or lower lid on one side to appear lower than the other (CN 3 and 7)

    Eyebrows o Inspect eyebrows quantity and distribution

    e.g. scaliness seborrheic dermatitis Eyelids

    o Note position in relation to eyeballs o Inspect width of palpebral fissures, edema and color of lids,

    lesions, condition and direction of lashes, adequacy with which the eyelids close e.g. red inflamed lids blepharitis

    Lacrimal Apparatus o Inspect for swelling o Look for tearing or dryness

    e.g. excessive tearing impaired drainage of tears Sclera

    o Observe color and vascular pattern o Normal sclera is white and surrounds the iris and pupil

    e.g. icteric sclera liver or blood disorder, yellowish sclera in the case of hyperbilirubinemia; can easily be confused with a muddy-brown discoloration common among older African Americans

    Conjunctiva o Thin transparent membrane covering of the sclera o Reflects back onto the underside of the eyelids o Normally, its invisible except for the fine blood vessels that run

    through it o Check for inflammation and vascular pattern o By applying pressure and pulling down and away on the skin

    below the lower lid, you can examine the conjunctival reflection e.g. severe anemia pale conjunctiva

    conjunctivitis infected or inflamed; appear quite red

    subconjunctival hemorrhage blood accumulate

    under the conjunctiva when small

    blood vessels rupture relatively due

    to trauma; generally self limited, and

    does not affect vision

    Cornea o Inspect cornea of eye for opacities, clarity, foreign body, ulcers,

    erythema/exudate

    Iris o With the light shining directly from the temporal side, look for a

    crescentric shadow on the medial side of the iris o If shadow is present, it could be an indication of glaucoma

    Pupils

    o Inspect size, shape, and symmetry o If the pupils are large (>5mm), small (

  • TRANSCRIBED BY: FRED, GEORGE, RON, BILL, GINNY

    Page 4 of 13

    HEENT

    Pupillary inequality of

  • TRANSCRIBED BY: FRED, GEORGE, RON, BILL, GINNY

    Page 5 of 13

    HEENT

    There are six cardinal directions

    Fig6. Six Cardinal Directions of Gaze

    CN 4 o Innervates the superior oblique muscle o Allows you to move either eyeball down or inward

    CN 6 o Innervates the lateral rectus muscle o Allows you to move either eyeball laterally

    CN 3 o Innervates the remaining extra-ocular muscles as well as the

    upper eye lid o Allows eyeball movement in all remaining directions as well as

    lifting of the upper lid o The dilation is due to disruption of the parasympathetic fibers

    which run along the outside of CN3

    Disorders of eye movement can also be due to problems with the extra-ocular muscles themselves

    STRABISMUS OR SQUINT

    Deviation of the eyes from their normally conjugate position

    May be classified into 2 groups: 1. Non-paralytic Strabismus

    the deviation is constant in all directions of gaze

    caused by an imbalance in ocular muscle tone

    has many causes, may be hereditary, and usually appears early in childhood

    deviations are further classified according to direction:

    Convergent Strabismus (Esotropia)

    Divergent Strabismus (Exotropia)

    COVER-UNCOVER TEST

    A cover-uncover test may be helpful. Here is what you see in the right monocular esotropia

    Fig8. Cover-Uncover Test

    2. Paralytic Strabismus

    the deviation varies depending on the direction of gaze

    usually caused by weakness or paralysis of one or more extraocular muscles

    determine the direction of gaze that maximizes the deviation

    A. Left 6th Nerve Paralysis

    Fig9. Left 6th Nerve Paralysis

    In CN 6 Paralysis, the eyes are CONJUGATE in RIGHT lateral gaze but not in LEFT lateral gaze.

    B. Left 4th Nerve Paralysis

    Fig10. Left 4th Nerve Paralysis

    NYSTAGMUS

    a fine rhythmic oscillation of the eyes, analogous to a tremor in other parts of the body

    possible causes: impairment of vision in early life, disorder of the labyrinth and the cerebellar system, drug toxicity

    occurs normally when a person waches a rapidly moving object

    a few beats of nystagmus on extreme lateral gaze are normal

    may be present in all directions of gaze

    Stabilize patient's head

    Ask the patient to follow your finger to a specific direction (i.e., to the right)

    Observe for fast oscillations toward the opposite direction (fast nystagmus)

    Observe for slow oscillations toward the same direction (slow nystagmus)

    Bring your finger within field of binocular vision and look again; record

    Assess for nystagmus in other directions of gaze

  • TRANSCRIBED BY: FRED, GEORGE, RON, BILL, GINNY

    Page 6 of 13

    HEENT

    LID LAG observed as the eyes move from up to down

    usually seen in hyperthyroidism in lid lag of hyperthyroidism, a rim of sclera is visible above the iris with downward gaze

    If you suspect lid lag, as the patient to follow your finger again as you move it slowly from up to down in the midline

    PLEASE SEE TABLES ON STRABISMUS AND RED EYES

    TEST FOR CONVERGENCE Ask the patient to follow your finger or pencil as you move it in toward

    the bridge of the nose.

    The converging eyes normally follow the object to within 5 cm to 8 cm of the nose

    Clinical Notes: o Hyperthyroidism Poor Convergence

    Fig7. Test for Convergence

    INSPECTION USING THE DIAGNOSTIC INSTRUMENTS: OPHTHALMOSCOPE

    The ophthalmoscope magnifies the normal retina about 15 times

    and the normal iris about 4 times. The optic disc actually measures about 1.5 mm.

    The optic disca yellowish orange to creamy pink oval or round structure that may fill your field of gaze or even exceed.

    Dont DILATE the eyes better posterior view of the retina However, to better view PERIPHERAL structures, use mydriatic

    drops to dilate the pupils

    USING THE OPHTHALMOSCOPE (Bates)

    Clinical Notes: o Absence of the red reflex:

    o Cataracts or Opacity of the Vitreous Humour o Detached Retina o Children: Retinoblastoma

    o The opthalmoscope magnifies the retina about 15 times and the iris about 4 times. o If the lens is surgically cut, its magnifying lens is lost. Retinal

    structures look much smaller while the fundus is much bigger.

    (+) for lid lag if a rim of sclera is visible above the iris

    Ask the patient to follow your finger as you move it slowly from up to down in the midline

    Normal = lid should overlap the iris slightly

    Darken the room. Switch on the ophthalmoscope until you see the large round beam of light.

    To better examine the sclera, conjunctiva, pupil, cornea or iris, start with the lens identified by a green 4 or 6.

    Grasp the handle with your hand (R hand for R eye, L hand for L eye) such that your middle finger is resting on the lower, front aspect of

    the head of the opthalmoscope. This gives you mobility.

    Bring your right eye up to the viewing window. The patient's glasses should be taken off. It's OK to leave contacts in place.

    Place your left hand on the patient's forehead and gently pull the top lid with your thumb.

    Place your left hand on the patient's shoulder.

    Try to keep both of your eyes open while doing the exam.

    Start approximately 15 in from the patient and approach from about 15 or 20 degrees to the left of center/ laterally. Check to

    make sure you see through the aperture.

    Shine the light beam on the pupil and look for the orange glow in the pupilthe RED REFLEX. Note any opacities interrupting the red

    reflex.

    When you look through the viewing window, the outer structures of the eye should come into sharp focus. If not, slowly move closer or further from the patient until these structures become clear. Change

    the lens to get a better focus.

    Place the thumb of your other hand on the patient's eyebrow to help you keep steady. Lower the brightness to make the exam more

    comfortable for the patient and to avoid HIPPUS (spasm of the pupil)

    Inspect the optic disc and the retina.

  • TRANSCRIBED BY: FRED, GEORGE, RON, BILL, GINNY

    Page 7 of 13

    HEENT

    INSPECTION OF OTHER RETINAL STRUCTURES USING THE OPHTHALMOSCOPE (Bates)

    A. The Optic Disc

    OPTIC DISC ABNORMALITIES A. Papilledema:

    Pinkish, hyperemic

    Loss of venous pulsations

    Disc vessels are more visible

    Physiologic Cup not visible

    Seen in intracranial mass, lesion or hemorrhage

    B. Glaucomatous Cupping

    Enlarged physiologic cup extending to the edge of the disc

    Retinal vessels sink and may be displaced laterally

    C. Optic Atrophy

    Whitish in color

    Tiny disc vessels absent

    Optic neuritis, multiple sclerosis, temporal arteritis

    Locate the optic disc. It is a round, yellowish orange structure. If you do not see it at first, follow a blood vessel centrally unti you

    do.

    Bring the optic disc into sharp focus by adjusting the lens.

    Normal: Focused at 0 diopters

    Blurred: Rotate the lens disc to find the sharpest focus

    Myopia : Rotate COUNTERCLOCKWISE

    Hyperopia: Rotate CLOCKWISE

    Inspect the optic disc. Note the following:

    Sharpness/ Clarity of Disc Outline - Nasal Portion may be blurred which is normal

    Color - Yellowish orange to creamy pink. Size of central cup, if present ; yellowish white in color Symmetry of the eyes and fundus.

    *Detect Papilledema:

    Papilledema - swelling of optic disc and anterior bulging of the physiologic cup due to increased Intracranial Pressure. This often signals serious disorders of the brain.

    Follow the vessels peripherally in each of four directions, noting their relative sizes and the character of the arteriovenous. Distinguish

    arteries from veins.

    Identify any lesions of the surrounding retina and note their size, shape, color, and distribution.

    As you search the retina, move your head and instrument as a unit, using the patients pupil as an imaginary fulcrum.

    Inspect the fovea and surrounding macula by directing your light beam laterally or by asking the patient to look directly into the light

    In younger people, the tiny bright reflection at the center of the fovea helps to orient you.

    Shimmering light reflections in the macular area are common in young people.

    SEE NEXT

    Lesions of the retina can be measured in terms of disc diameters from the optic disc.

    Inspect the anterior structures.

    Look for opacities in the vitreous or lens by rotating the lens disc progressively to diopters of around +10 or +12.

  • TRANSCRIBED BY: FRED, GEORGE, RON, BILL, GINNY

    Page 8 of 13

    HEENT

    NOTES ON OPHTHALMOLOGIC INSPECTION FROM LECTURE FROM BATES GENERAL You can only see small portions at a time Compare both eyes for symmetry RED REFLEX Seen as you approach the patient while looking through

    the ophthalmoscope --

    FUNDUS Normally yellow or pink Should be compared with the fundus of the other eye for symmetry

    In older adults, the fundi lose their youthful shine and light reflections

    Inspect fundi for colloid bodies causing alterations in pigmentation called drusen

    VASCULAR SUPPLY

    Branch away from the optic disc To distinguish arteries from veins:

    ARTERIES VEINS

    Color Light red Dark red

    Size Smaller Larger

    Light Reflex

    Bright Inconspicuous or absent

    Note the presence of venous pulsations.

    In a normal person, pulsations in the retinal veins as they emerge from the central portion of the disc may or may not be present.

    In older adults, the arteries look narrowed, paler, straighter and less brilliant.

    OPTIC DISC (NASAL SIDE)

    Has a clear border

    Optic cup o Has blood vessels

    Vein = dark red Artery = light red

    o Check ratio of blood vessels o Wide optic cup = Glaucoma

    Note:

    The sharpness or clarity of the disc outline.

    The nasal portion of the disc margin may be somewhat blurred, a normal finding.

    The color of the disc, normally yellowish orange to creamy pink.

    White or pigmented crescents may ring the disc, a normal finding.

    The size of the central physiologic cup, if present. It is usually yellowish white.

    The cup-to-disc ratio is usually 1:2 or less o Increased cup:disc ratio seen in open angle glaucoma

    MARGINS Sharp

    Well defined

    MACULA (FOVEA CENTRALIS) | (CENTRAL SIDE)

    Central Vision/Color

    Difficult to examine DO NOT focus light directly on the macula or the patient will feel pain

    CLASSIFICATION CONJUNCTIVITIS SUBCONJUNCTIVAL

    HEMORRHAGE CORNEAL INJURY

    OR INFECTION ACUTE IRITIS GLAUCOMA

    ILLUSTRATION

    PATTERN OF REDNESS

    Conjuctival infection: diffuse dilatation of conjunctival vessels with redness that tends to be maximal peripherally Dr. Guzman: Redness from conjunctiva spreading in an upward direction

    Leakage of blood outside of the vessels, producing a homogeneous, sharply demarcated, red area that fades over days to yellow and then disappears Dr. Guzman: due to rubbing of the eyes; usually not pathologic

    Ciliary injection: dilation of deeper vessels that are visible as radiating vessels or a reddish violet flush around the limbus. Ciliary injection is an important sign of these three conditions but may not be apparent. The eye may be diffusely red instead. Other clues of these more serious disorders are pain, decreased vision, unequal pupils, and a less than perfectly clear cornea.

    PAIN Mild discomfort rather than pain

    Absent Moderate to severe, superficial

    Moderate, aching, deep

    Severe, aching, deep

    VISION Not affected except for temporary mild blurring

    Not affected Usually decreased Decreased Decreased

    OCULAR DISCHARGE

    Watery, mucoid, or mucopurulent

    Absent Watery or purulent Absent Absent

    PUPIL Not affected Not affected Not affected unless

    iritis develops May be small and, with time, irregular

    Dilated, fixed

    CORNEA Clear Clear Changes depending on

    cause Clear or slightly clouded

    Steamy, cloudy

    SIGNIFICANCE

    Bacterial, viral, and other infections; allergy; irritation

    Often none. May result from trauma, bleeding disorders, or a sudden increase in venous pressure, as from cough.

    Abrasions, and other injuries; viral and bacterial infections

    Associated with many ocular and systemic disorders

    Acute increase in intraocular pressure-an emergency

    EAR EXAMINATION

    External structures: Briefly examine the outer structures, paying particular attention to any skin changes suggestive of cancer (e.g basal cell, melanoma, squamous cell), a common asymptomatic abnormality affecting this sun exposed area. If the patient has pain, try to identify its precise location. Infection within the external canal, for example, may cause discharge from the ear as well as pain on manipulation of any of the external structures.

  • TRANSCRIBED BY: FRED, GEORGE, RON, BILL, GINNY

    Page 9 of 13

    HEENT

    EXTERNAL EXAMINATION (Auricle and Mastoid) Size, Shape, Symmetry, Color, Position, Tenderness, Color

    USE OF OTOSCOPE OTOSCOPE Speculum

    Position Patient Scope

    AURICLE Up and Back CANAL Discharge

    Scaling Redness Lesions Foreign Bodies Cerumen

    TYMPANIC MEMBRANE

    Landmarks Color Contour Perforations

    The otoscope allows you to examine the external canal, the

    structure that connects the outside world with the middle ear, as well as the ear drum and a few inner ear structures. Proceed as follows:

    1. Put the otoscopic head on your oto-opthalmoscopic grip. It should easily twist into position.

    2. Turn on the light source. 3. Place one of the disposable specula on the end of the scope. 4. Grasp the scope so that the handle is either pointed directly

    downward or angled up and towards the patient's forehead. Either technique is acceptable. The scope should be in your right hand if you are examining the right ear.

    5. Place the tip of the specula in the opening of the external canal. Do this under direct vision (i.e. not while looking through the scope).

    6. Gently grasp the top of the left ear with your left hand and pull up and backwards. This straightens out the canal, allowing easier passage of the scope.

    7. Look through the viewing window with either eye. Slowly advance the scope, heading a bit towards the patient's nose but without any up or down angle. Move in small increments. Try not to wiggle the scope too much as the external canal is quite sensitive. I find it helpful to extend the pinky and fourth fingers of my right hand and place them on the side of the patient's head, which has a stabilizing effect.

    8. As you advance, pay attention to the appearance of the external canal. In the setting of infection, called otitis externa, the walls becomes red, swollen and may not accommodate the speculum. In the normal state there should be plenty of room. If wax, which appears brownish, irregular and mushy, obscures your view, stop and go to the other side. Do not try to extract it until/unless you have had specific training in this area! There are pharmacologic means of softening wax, which may then be easily irrigated from the canal.

    EXTERNAL EXAMINATION (Auricle and Mastoid)

    OTOSCOPIC EXAMINATION TYMPANIC MEMBRANE

    COLOR When healthy, it has a grayish, translucent appearance.

    STRUCTURES BEHIND IT

    The malleus, one of the bones of the middle ear, touches the drum. The drum is draped over this bone, which is visible through its top half, angled down and backwards. The part that is closest to the top of the drum is called the lateral process, and is generally most prominent. The tip at the bottom-most aspect is the umbo.

    LIGHT REFLEX Light originating from your scope will be reflected off the surface of the drum, making a triangle that is visible below the malleous

    ACUTE OTITIS EXTERNA ANATOMY

    CLINICAL NOTES

    INFECTIONS AND OTITIS MEDIA Pathogenic organisms can gain entrance to the middle ear by ascending through the auditory tube from the nasal part of the pharynx. Acute infection of the middle ear (otitis media) produces bulging and redness of the tympanic membrane.

    COMPLICATIONS OF OTITIS MEDIA Inadequate treatment of otitis media can result in the spread of the infection into the mastoid antrum and the mastoid air cells (acute mastoiditis). Acute mastoiditis may be followed by the further spread of the organisms beyond the confines of the middle ear. The meninges and the temporal lobe of the brain lie superiorly. A spread of the infection in this direction could produce a meningitis and a cerebral abscess in the temporal lobe. Beyond the medial wall of the middle ear lie the facial

  • TRANSCRIBED BY: FRED, GEORGE, RON, BILL, GINNY

    Page 10 of 13

    HEENT

    nerve and the internal ear. A spread of the infection in this direction can cause a facial nerve palsy and labyrinthitis with vertigo. The posterior wall of the mastoid antrum is related to the sigmoid venous sinus. If the infection spreads in this direction, a thrombosis in the sigmoid sinus may well take place. These various complications emphasize the importance of knowing the anatomy of this region.

    ACUTE OTITIS MEDIA ANATOMY

    AUDITORY ACUITY

    If the patient does not complain of hearing loss, this part of the exam is omitted.

    A crude assessment can be performed by asking the patient to close their eyes while you place your fingers a few centimeters from either ear. Rub the fingertips of first one hand and then the other. Make note of any obvious differences in hearing.

    Alternatively, you can stand behind the patient and whisper a few words in first one ear and then the other. Are they able to repeat the phrases back correctly?

    Does this seem to be equal on either side? These tests obviously are not very objective. Precise quantification requires sensitive equipment and is usually done by a trained audiologist.

    WEBER TEST (Test for Lateralization)

    Grasp the 512 Hz tuning fork by its stem and get it to vibrate by either striking the tines against your hand or by "snapping" the ends between your thumb and middle finger.

    Then place the stem towards the back of the patient's head, on an imaginary line equidistant from either ear. The bones of the skull will transmit this sound to the 8th nerve, which should then be appreciated in both ears equally. Remind the patient that they are trying to detect sound, not the buzzing vibratory sensation from the fork. If there is a conductive deficit (e.g. wax in the external canal), the sound will be heard better in that ear. This is because impaired conduction has prevented any competing sounds from entering the ear via the normal route. You can create a transient conductive hearing loss by putting a finger in one ear. Sound transmitted from the tuning fork will then be heard louder on that side.

    In the setting of a sensorineural abnormality (e.g. an acoustic neuroma, a tumor arising from the 8th CN), the sound will be best heard in the normal ear. If sound is heard better in one ear it is described as lateralizing to that side. Otherwise, the Weber test is said to be mid-line.

    RINNE TEST Strike the same tuning fork and place the stem on the mastoid bone, a bony prominence located just behind and below the ear. Bone conduction will allow the sound to be transmitted and appreciated.

    Instruct the patient to let you know as soon as they can no longer hear the sound.

    Then place the tines of the still vibrating fork right next to, but not touching, the external canal. They should again be able to hear the sound. This is because, when everything is functioning normally, transmission of sound through air is always better than through bone.

    This will not be the case if there is a conductive hearing loss (e.g. fluid associated with an infection in the middle ear), which causes bone conduction to be greater than or equal to air. BC> AC

    If there is a sensorineural abnormality (e.g. medication induced toxicity to the 8th CN), air conduction should still be better than bone as they will both be equally affected by the deficit. AC>BC

    NOSE EXAMINATION Anatomy and Physiology (from Bates): Upper third is supported by bone, lower 2/3 supported by cartilage. Flow of air: Media wall: nasal septum (supported by bone and cartilage)

    Vestibule only part lined by hair-bearing skin, not mucosa Turbinates curving bony structures that protrude into the nasal cavity (superior, middle, and inferior), below each is a meatus named accdng to the turbinate above it.

    Functions: cleansing, humidification, temp control of

    inspired air

    Inferior meatus: where the nasolacrimal duct drains

    Middle meatus: where most of the paranasal sinuses drain

  • TRANSCRIBED BY: FRED, GEORGE, RON, BILL, GINNY

    Page 11 of 13

    HEENT

    Paranasal sinuses air-filled cavities within the bones of the skull, line by mucous membrane (Only the maxillary and frontal sinuses are readily available for clinical exam)

    TECHNIQUES IN EXAMINATION

    Exam is generally omitted in the absence of symptoms First check to see if the patient is able to breathe through either nostril effectively.

    Push on one nostril until it is occluded and have them inhale. Then repeat on the other side. Air should move equally well through each nares.

    To look in the nose, have the patient tilt their head a little back. Push up slightly on the tip of the nose with the thumb of your left hand. Place the end of the speculum (it's OK to use the same one from the ear exam) into the nares under direct vision.

    NOTE THE FOLLOWING: The color of the mucosa. It can become quite reddened in the setting of infection.

    The presence of any discharge as well as its color (clear with allergic reactions; yellowish with infection).

    The middle and inferior turbinates, which are shelf-like projections along the lateral wall. Any polypoid growths, which may be associated with allergies and obstructive symptoms

    The other nostril is examined in a similar manner. Loss of smell (anosmia) is a relatively common problem, though often undiagnosed. In patients who make mention of this problem, olfaction can be crudely assessed using an alcohol pad sniff test as follows:

    Ask the patient to close their eyes so that they don't get any visual cues.

    Occlude each nostril sequentially, making sure that they can move air adequately thru both. Occlude one nostril and then present an alcohol pad to the other side, asking the patient to inform you when they are able to detect its smell.

    ASSESSMENT OF FRONTAL AND MAXILLARY SINUSES Paranasal sinuses

    o are air-filled cavities within the bones of the skull. o are lined with mucous membrane. o Act as voice resonators and reduce the weight of the skull. o Only the frontal and maxillary sinuses are readily accessible to

    clinical examination. (indirectly)

    TECHNIQUES IN THE EXAMINATION OF THE SINUSES

    1. Check for colored mucosal discharge o This is due to the fact that the maxillary sinuses drain into

    the nose via a passageway located under the middle turbinate.

    2. Directly palpate and percuss the skin overlying the frontal and maxillary sinuses.

    o Press up on the frontal sinuses from under the bony brows, avoiding pressure on the eyes.

    o Then check on the maxillary sinuses by pressing the finger against the anterior wall of the maxilla below the inferior orbital margin

    o Local tenderness, together with symptoms such as pain, fever, and nasal discharge, suggest acute sinusitis

    3. With the use of an otoscope: First dim the room lights. o Place the lighted otoscope directly on the infraorbital rim

    (bone just below the eye). o Ask the patient to open their mouth and look for red dim

    glow through the mucosa of the upper mouth. o In the setting of inflammation, the maxillary sinus becomes

    fluid filled and will not allow this transillumination. There are specially designed transilluminators that may work better for this task, but are not readily available.

    4. Using a tongue depressor,: o Tap on the teeth which sit in the floor of the maxillary sinus.

    This may cause discomfort if the sinus is inflamed because the pain is referred to the jaw and teeth. Remember that the maxillary sinus is innervated by the infraorbital nerve.

    INFECTION OF THE NASAL CAVITY

    Infection may spread via the nasal part of the pharynx and the auditory tube to the middle ear.

    organisms may ascend to the meninges of the anterior cranial fossa, along the sheaths of the olfactory nerves through the cribriform plate, and produce meningitis.

  • TRANSCRIBED BY: FRED, GEORGE, RON, BILL, GINNY

    Page 12 of 13

    HEENT

    FOREIGN BODIES IN THE NOSE

    Foreign bodies in the nose are common in children.

    The presence of the nasal septum and the existence of the folded, shelflike conchae make impaction and retention of balloons, peas, and small toys relatively easy.

    NOSE BLEEDING Epistaxis, or bleeding from the nose, is a frequent condition.

    Most common cause: nose picking

    The bleeding may be arterial or venous, and most episodes occur on the anteroinferior portion of the septum.

    EXAMINATION OF THE MOUTH AND PHARYN Anatomy and Physiology (from Bates):

    Lips muscular folds that surround the entry to the mouth

    Gingiva (gums) firmly attached to the teeth and to the maxilla/mandible where they are seated

    Labial frenulum

    Connects each lip with the gingival

    Teeth composed of dentin, only the crown (enamel-covered) is exposed. Blood vessels and nerves enter the tooth via the apex and pass into the pulp canal and chamber (32 total, 16 in each jaw)

    Tongue dorsum covered by papillae, giving it a rough surface. Undersurface has no papillae Lingual frenulum connects the tongue to floor of mouth Ducts of submandibular glands (Whartons ducts) are located at the base of the tongue; they lie on each side of the lingual frenulum

    Anterior and posterior pillars, soft palate, and uvula form an arch

    above and behind the tongue

    Right and left tonsils are often small or absent, and are located in the tonsillar fossa in between the anterior and posterior pillars.

    Buccal mucosa lines the cheeks, where the parotid duct (Stensens duct) opens to (near the upper Second molar)

    TECHNIQUES IN THE EXAMINATION OF THE MOUTH Ask the patient to remove his/her dentures.

    Inspect the following: 1. Lips

    o Observe their color and moisture, and note any lumps, ulcers, cracking, or scaliness.

    2. Oral mucosa o Ask the patient to open his/her mouth and, with a good

    light and the help of a tongue blade, inspect o the oral mucosa for:

    color, ulcers, white patches nodules wavy white line on the adjacent buccal

    mucosa developed where the upper and lower teeth meet - related to irritation from sucking or chewing.

    3. Gums and Teeth o Note the color of the gums, normally pink. o Brown patches may be present, especially but not

    exclusively in black people.

    o Inspect the teeth. Are any of them: missing, discolored, misshapen abnormally positioned looseness

    4. Roof of the Mouth o Inspect the color and architecture of the hard palate.

    5. The Tongue and the Floor of the Mouth. o Ask the patient to put out his or her tongue. Inspect it for

    symmetrya test of the hypoglossal nerve (CN XII). o Note the color and texture of the dorsum of the tongue. o Inspect the sides and under surface of the tongue and

    the floor of the mouth, areas where cancer often develops.

    o Palpate any lesions. Ask the patient to protrude the tongue. With your right hand, grasp the tip of the tongue with a square of gauze and gently pull it to the patients left. Inspect the side of the tongue, and then palpate it with your gloved left hand, feeling for any induration. Reverse the procedure for the other side.

    o Tongue cancer is a common oral cancer, especially in: men older than 50 years smokers tobacco chewers alcohol drinkers

    TECHNIQUES IN THE EXAMINATION OF THE PHARYNX

    Ask the patient to say ah or yawn with the patients mouth open but the tongue not protruded so you can see the pharynx well. If not, press a tongue blade firmly down upon the midpoint of the arched tonguefar enough back to visualize the pharynx but not so far that you cause gagging.

    Note the rise of the soft palatea test of CN X (the vagal nerve). In CN X paralysis:

    soft palate fails to rise uvula deviates to the opposite side.

    EXAMINATION OF THE NECK May use the Sternocleidomastoid muscle as a landmark.

    LYMPH NODES

    Nodes are normally round or ovoid smooth

    Tonsillar node and supraclavicular nodes may be palpable.

    Palpate the lymph nodes: (Bates) Using the pads of your index and middle fingers, move the

    skin over the underlying tissues in each area. With neck flexed slightly forward can usually examine both sides at once. For the submental node, however, feel with one hand while

    bracing the top of the head with the other.

    Sequence of Lymph nodes palpation based from Bates:

  • TRANSCRIBED BY: FRED, GEORGE, RON, BILL, GINNY

    Page 13 of 13

    HEENT

    Lymph Node

    Location

    Drainage

    1. Preauricular In front of ear

    2. Posterior

    Auricular

    Superficial to the mastoid process

    3. Occipital

    At the base of the skull posteriorly

    4. Tonsillar

    Below thw Angle of the mandible

    Tonsillar and posterior pharyngeal regions

    5. Submandibular

    Midway between the angle and the tip of the mandible.

    Structures in the floor of the mouth

    6. Submental

    In the midline a few centimeters behind the tip of the mandible (below the chin)

    Teeth and Intra-oral cavity

    7. Superficial/ Anterior Cervical

    Superficial to the sternomastoid

    Internal structures of the throat

    Part of the posterior pharynx, tonsils, and thyroid gland

    8. Posterior Cervical

    Posterior to SCM, Along the anterior edge of the trapezius

    Skin on the back of the head

    9. Deep Cervical Chain

    Deep to the sternocleidomastoid and often inaccessible to examination

    10. Supraclavicular

    Deep in the angle formed by the clavicle and the sternomastoid

    Part of the thoracic cavity and Abdomen

    Describe: Tender Solitary, multiple Movable, non-movable Size

    Infected LN : Firm Tender Enlarged Warm

    Malignancy: Firm Non-tender Fixed Increasing size over time

    Stage 3 disease if present at cervical, axillary and inguinal areas.

    Characteristics of LN: Hard Kneecap Firm Nose Soft mons pubis

    TRACHEA

    Place your finger along one side of the trachea

    Note the space between it and the sternocleidomastoid. The spaces should be symmetric for both sides.

    THYROID

    Inspection

    Tip the patients head back a bit. Using tangential lighting directed downward from the tip of the patients

    chin, inspect the region below the cricoid cartilage for the gland.

    Observe the patient swallowing:

    o Ask the patient to sip some water and to extend the neck again and swallow. Watch for upward movement of the thyroid gland, noting its contour and symmetry.

    Palpation

    Find your landmarksthe notched thyroid cartilage and the cricoid cartilage below it.

    Locate the thyroid isthmus, usually overlying the second, third, and fourth tracheal rings

    Place the fingers of both hands on the patients neck so that your index fingers are just below the cricoid cartilage.

    Ask the patient to sip and swallow water as before. Feel for the thyroid isthmus rising up under your finger pads.

    Displace the trachea to the right with the fingers of the left hand; with the right-hand fingers, palpate laterally for the right lobe of the thyroid in the space between the displaced trachea and the relaxed sternocleidomastoid. Find the lateral margin.

    In a similar fashion, examine the left lobe.

    The anterior surface of a lateral lobe is approximately the size of the distal phalanx of the thumb and feels somewhat rubbery.

    Note the size, shape, and consistency of the gland and identify any nodules or tenderness.

    If the thyroid gland is enlarged, listen over the lateral lobes with a stethoscope to detect a bruit,a sound similar to a cardiac murmur but of noncardiac origin