2.5 Ethambutol in the Treatment of Patients With Chronic Pulmonary Tuberculosis
Management of Patients With Chronic Pulmonary Disease.
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Transcript of Management of Patients With Chronic Pulmonary Disease.
![Page 1: Management of Patients With Chronic Pulmonary Disease.](https://reader035.fdocuments.us/reader035/viewer/2022081515/56649d935503460f94a7afe5/html5/thumbnails/1.jpg)
Management of Patients With Chronic Pulmonary
Disease
Management of Patients With Chronic Pulmonary
Disease
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COPD:
• Chronic Obstructive Pulmonary Disease• A disease state characterized by airflow limitation
that is not full reversible (GOLD).• COPD is the currently is 4th leading cause of death
and the 12th leading cause of disability. • COPD includes diseases that cause airflow
obstruction (emphysema, chronic bronchitis) or a combination of these disorders.
• Asthma is now considered a separate disorder but can coexist with COPD.
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Pathophysiology of COPD• Airflow limitation is progressive and is associated with
abnormal inflammatory response of the lungs to noxious agents.
• Inflammatory response occurs throughout the airways, lung parenchyma, and pulmonary vasculature.
• Scar tissue and narrowing occurs in airways.• Substances activated by chronic inflammation
damage the parenchyma. • Inflammatory response causes changes in pulmonary
vasculature.
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Chronic Bronchitis
• The presence of a cough and sputum production for at least 3 months in each of 2 consecutive years.
• Irritation of airways results in inflammation and hypersecretion of mucous.
• Mucous-secreting glands and goblet cells increase in number.
• Ciliary function is reduced, bronchial walls thicken, bronchial airways narrow, and mucous may plug airways.
• Alveoli become damaged, fibrosed, and alveolar macrophage function diminishes.
• The patient is more susceptible to respiratory infections.
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Pathophysiology of Chronic Bronchitis
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Emphysema:• Abnormal distention of air spaces beyond the terminal
bronchioles with destruction of the walls of the alveoli.• Decreased alveolar surface area causes an increase in
“dead space” and impaired oxygen diffusion.• Reduction of the pulmonary capillary bed increases
pulmonary vascular resistance and pulmonary artery pressures.
• Hypoxemia result of these pathologic changes. • Increased pulmonary artery pressure may cause right-
sided heart failure (cor pulmonale).
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Changes in Alveolar Structure with Emphysema
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Risk Factors for COPD
• Tobacco smoke causes 80-90% of COPD cases!• Passive smoking• Occupational exposure• Air pollution• Genetic abnormalities (2% of cases)– Alpha1-antitrypsin deficiency ( enzyme inhibitor
that protect the lung parenchyma from injury)
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Clinical Manifestation• 3 primary symptoms:1. Chronic cough2. Sputum production3. Dyspnea on exertion• Wt loss• Barrel chest (A-P diameter/ Transverse diameter : 2/1)• Retraction in the supraclavicular area on inspiration • Shrug shoulder• Abdominal muscle contraction on inspiration (paradox
respiration) .
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Normal Chest Wall and Chest Wall Changes with Emphysema
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Typical Posture of a Person with COPD
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Medical Management
• Risk reduction• Pharmacologic therapy• Management of exacerbation• O2 therapy• Surgical management • Pulmonary rehabilitation
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Nursing Process: The Care of Patients with COPD- Assessment
• Health history• Inspection and examination findings • Review of diagnostic tests
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Nursing Process: The Care of Patients with COPD- Diagnoses
• Impaired gas exchange • Impaired airway clearance• Ineffective breathing pattern• Activity intolerance• Deficient knowledge• Ineffective coping
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Collaborative Problems• Respiratory insufficiency or failure• Atelectasis• Pulmonary infection• Pneumonia• Pneumothorax• Pulmonary hypertension
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Nursing Process: The Care of Patients with COPD- Planning
• Smoking cessation• Improved activity tolerance• Maximal self-management• Improved coping ability• Adherence to therapeutic regimen and home
care• Absence of complications
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Improving Gas Exchange
• Proper administration of bronchodilators and corticosteroids
• Reduction of pulmonary irritants• Directed coughing, “huff” coughing• Chest physiotherapy• Breathing exercises to reduce air trapping– diaphragmatic breathing – pursed lip breathing
• Use of supplemental oxygen
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Improving Activity Tolerance• Focus on rehabilitation activities to improve
ADLs and promote independence.• Pacing of activities• Exercise training• Walking aides• Utilization of a collaborative approach
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Other Interventions• Set realistic goals• Avoid extreme temperatures• Enhancement of coping strategies• Monitor for and management of potential
complications
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Patient Teaching
• Disease process• Medications • Procedures • When and how to seek help• Prevention of infections • Avoidance of irritants; indoor and outdoor
pollution, and occupational exposure • Lifestyle changes, including cessation of
smoking