Lecture 9.3 rad240 pathology
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Transcript of Lecture 9.3 rad240 pathology
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Lecture 9.3rad240 pathology
Dr shai’
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POLYPS• ANY mucosal bulging, blebbing, or bump
•HYPERPLASTIC (NON-NEOPLASTIC)
• HAMARTOMATOUS (NON-NEOPLASTIC)
•ADENOMATOUS (TRUE NEOPLASM, and regarded by many as “potentially” PRE-MALIGNANT as well)
• SESSILE vs. PEDUNCULATED• TUBULAR vs. VILLOUS
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POLYPS
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PEDUNCULATED vs VILLOUS vs SESSILE
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BENIGN vs. MALIGNANT• Usual, atypia, pleo-, hyper-, mitoses, etc.• Stalk invasion!!!
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HPERPLASTIC POLYP
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ADENOMATOUS POLYP (TUBULAR)
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ADENOMATOUS POLYP (VILLOUS)
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“FAMILIAL” NEOPLASMS• 1) POLYPOSIS (NON-NEOPLASTIC,
hamartomatous)• 2) POLYPOSIS (NEOPLASTIC, i.e.,
cancer risk)• 3) HNPCC: (Hereditary Non
Polyposis Colorectal Cancer)
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MUCINOUS CYSTADENO(CARCINO)MA
ADENOMA CARCINOMA
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PERITONEUM• Visceral, Parietal: all lined by mesothelium• Peritonitis, acute:– Appendicitis, local or with rupture– Peptic ulcer, local or ruptured– Cholecystitis, local or ruptured– Diverticulitis, local or with rupture– Salpingitis gonococcal or chlamydial, retrograde
or perforated– Ruptured bowel due to any reason– Perforating abdominal wall injuries
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PERITONITIS• E. coli• STREP• S. aureus• ENTEROCOCCUS
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PERITONITIS, outcomes:
• Complete RESOLUTION•Walled off ABSCESS
•ADHESIONS
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SCLEROSING RETROPERITONITIS
• Unknown cause (autoimmune?)• Generalized retroperitoneal fibrosis,
progressive hydronephrosis
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TUMORS• MESOTHELIOMAS (solitary nodules or
diffuse constricting growth pattern, also asbestos caused)• METASTATIC, usually diffuse, often
looking very much like pseudomyxoma peritoneii, but containing tumor cells, usually adenocarcinoma