F:\Powerpoints\Pressure Ulcer Presentation Nursing Orientaiton 10 Update
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Transcript of F:\Powerpoints\Pressure Ulcer Presentation Nursing Orientaiton 10 Update
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Pressure ulcers
Katherine Constable, MSN, CWONPatty Brown, BSN, CWOCN
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Beauty, to me, is about being comfortable in your own skin. That, or a kick-ass red lipstick.
Gwyneth Paltrow
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Skin Facts•Largest organ of the body covers approximately 3000 square inches receives 1/3 circulating blood volume.
•From birth to maturity, the skin will undergo a sevenfold expansion.
•Weighs about 6 pounds
•1cm of skin has 15 sebaceous glands, 3 yards of blood vessels, 100 sweat glands, 3,000 sensory cells, 4 yards of nerves, 300,000 epidermal cells and 10 hair follicles
•This organ is capable of self-generation and can withstand limited mechanical and chemical assault.
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Factors
Decreased sensation Decreased mobility Nutritional challenged Incontinence of urine and stool Decreased perception Shear and Friction
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Assessment
VISUAL INSPECTION OF THE TAIL
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Assessment
Conduct skin assessment within 4 hours admission
Inspect skin daily Use the 7 day principle Use a risk assessment scale Braden Note moisture, pressure, shear, friction Document
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Perineal Dermatitis
Skin problems experience by our patients with wound drainage, fecal and urinary incontinence or offending chemical exposure to the perinium causing excoriation, irritation, frequently with satellite lesion (ie yeast).
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6 factors identified
Chronic exposure to moisture Fecal and urinary incontinence Limit amount of pads Alkaline ph Overgrowth or infection with pathogen Friction or shearing
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Treatment Cleanse area with warm water Pat dry and use 3M wipes Use nystatin powder next to skin Cover with extra protective cream w/
antifungal use q12 hours or prn Stop the stooling/urine i.e. butt bag or fecal
management system. Use dri flows under patient- chux trap body heat! Get a low air loss overlay.
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A bit of lusting after someone does wonders for the skin.
Elizabeth Hurley
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Scope Of The Problem
2.5 million patients treated in acute-care facilities annually.
Pressure ulcer incidence range U of L Hospital is 5.3% (compared 7-9%).
Estimated cost $40,000. Treatment cost is estimated $11 billion.
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Pathophysiology
Prolong pressure Sudden impact Shear and friction
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Shear
Diminishes circulation to tissue and damages tissue and blood vessel integrity
Skeleton moves but the skin remains fixed to the surface
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Moisture Speeds up decomposition of
tissue. Ph balance → tissue fragility Macerated tissue is prone to
more erosion. Incontinence greater risk of PU.
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Nutritional Needs
% total weight loss
Complications mortality
10% Impaired immune fx,
Increased infection
20%
30% Weakness, pneumonia poor healing, too weak to sit, no healing
50%
40%
Death (pneumonia)
100%
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Stage I
Intact skin with non-blanchable redness of a localized area usually over a bony prominence. Darkly pigmented skin may not have visible blanching; its color may differ from the surrounding area.
Further description: The area may be painful, firm, soft, warmer or cooler as compared to adjacent tissue. Stage I may be difficult to detect in individuals with dark skin tones. May indicate “at risk” persons (a heralding sign of risk).
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Stage II
Partial thickness loss of dermis presenting as a shallow open ulcer with a red pink wound bed, without slough. May also present as an intact or open/ruptured serum-filled blister.
Further Description: Presents as a shiny or dry shallow ulcer without slough or bruising.* This stage should not be used to describe skin tears, tape burns, perineal dermatitis, maceration or excoriation.
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After 3 weeks
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Difference between Stage I and II
Stage I
Red non-blanching
Skin intact
Stage II
Partial thickness Shallow crater
Fluid filled blister
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Mattress Selection
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Stage III
Full thickness tissue loss. Subcutaneous fat may be visible but bone, tendon or muscle are not exposed. Slough may be present but does not obscure the depth of tissue loss. May include undermining and tunneling.
Further Description: The depth of a stage III pressure ulcer varies by anatomical location. The bridge of the nose, ear, occiput and malleolus do not have subcutaneous tissue and stage III ulcers can be shallow. In contract, areas of significant adiposity can develop extremely deep stage III pressure ulcers. Bone/tendon is not visible or directly palpable.
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Difference between Stage II and III
Stage II
Partial thickness Shallow crater
Fluid filled blister
Stage III
Full thickness with subqExposure may have
undermining and/or tunneling
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Stage IV
Full thickness tissue loss with exposed bone, tendon or muscle. Slough or eschar may be present on some parts of the wound bed. Often include undermining and tunneling.
Further Description: The depth of a stage IV pressure ulcer varies by anatomical location. The bridge of the nose, ear, occiput and malleolus do not have subcutaneous tissue and these ulcers can be shallow. Stage IV ulcers can extend into muscle and/or supporting structures (e.g., fascia, tendon or joint capsule) making osteomyelitis possible. Exposed bone/tendon is visible or directly palpable.
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Unstageable
Full thickness tissue loss in which the base of the ulcer is covered by slough (yellow, tan, gray, green or brown) and/or eschar (tan, brown or black) in the wound bed.
Further Description: Until enough slough and/or eschar is removed to expose the base of the wound, the true depth, and therefore stage, cannot be determined. Stable (dry, adherent, intact without erythema or fluctuance) eschar on the heels serves as “the body’s natural (biological) cover” and should not be removed.
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(Suspected) Deep Tissue Injury
Purple or maroon localized area of discolored intact skin or blood-filled blister due to damage of underlying soft tissue from pressure and/or shear. The area may be preceded by tissue that is painful, firm, mushy, boggy, warmer or cooler as compared to adjacent tissue.
Further Description: Deep tissue injury may be difficult to detect in individuals with dark skin tones. Evolution may include a thin blister over a dark wound bed. The wound may further evolve and become covered by thin eschar. Evolution may be rapid exposing additional layers of tissue even with optimal treatment.
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After 2 weeks
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After 4 weeks
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Difference between Stage I and DTI
Stage I
Recovers within 24 hours with pressure relief.
Red or eggplant color
Skin intact
DTI
Doesn’t recover within 24 hours with pressure relief
Develops rapidly into Stage II
Purple/ischemic looking
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Practice Time
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