Dietary Modification in Various Imbalance of Endocrine Production
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Transcript of Dietary Modification in Various Imbalance of Endocrine Production
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DIETARY MODIFICATION IN VARIOUS IMBALANCE OF
ENDOCRINE PRODUCTION
( dr. Dimyati Burhanuddin, Msc)
The Deviation of normal metabolic pathway, result to
Abnormal production of one or more hormones
Deficiency of an enzymes
Modification of ecretion
! effect the balance of endocrine production
!diseases
"ypo#lycemia ! shoc$ (insuline shoc$)
%aused by &
'nsuline overdose
Decrease of available #lucose
o food
oss of food
*ver eercise
+pontaneous hypo#lycemia disorder of carbohydrate metabolism ( functional, or#anic)
'n functional hyperinsulinism ! stimulative type of hypo#lycemia ( no or#anic lesion )
%arbohydrate islet cells (hi#h respons)
"yperinsulinism
"ypo#lycemia
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'n or#anic lesion (post #astro intestimal operation)
%arbohydrate
di#estion
rapid #lucose absorption
sudden elevation of blood #lucose
islet cells
hyperinsulinism
hypo#lycemia
Dietary principle
carbohydrate & restricted to levels below -// #r (01 1/ # , if no improvment)
protein & hi#h protein diet (-2/ -3/ #r)
! available blood #lucose, protein decrease to 1/ 4
5at & when blood level of carbohydrate and protein have been estabilshed,
fat for the remainin# calories (in available need)
Dietary plannin#
6se food echan#e list
Ade7uate amount of fruits and ve#etables
Mil$ ! limited
+upplement %a
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Adrenocortical insuicienc!
(Addison8s disease)
Adrenal corte ! Atrophy (un$nown ori#in)
! impairment the function
+ome causes & tubercolosis
adrenal ectomy
hypophysectomy
! absence of adrenocortical hormones
9lucocorticoids
%ortisol re#ulates the metabolism of carbohydrate, protein and fat.
'n absence & rapid #lyco#en depletion, followed by hypo#lecemia (in a few
hours
After meals)
Mineralocorticoids
Aldosterone & maintainin# electrolite homeostatis (espec a : ;)
a retention
; ecretion
The production is not under the influence pituitary
Deficiency aldosterone &
'ncrease Naecretion
'ncrease " retention
ead to & lar#e salt loss < water
Dehydration
"ypotension e.t.c
Andro#enic hormones
=
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+timulate protein synthesis in absence &
>ei#ht loss
Tissue wastin#
?eduction of muscle stren#th
5ati7ue
Dietary modification
mild insuff &
"i#her salt inta$e
5re7uent meals
(1 @ times a day)
+evere insuff &
Minerals metabolism in s controlled by #ivin# D*%A
(dioycarticosterone acetate)
"i#h protein and low carbohdydrate reduces the
stimulation of insuline
5re7uent intervals of meals
Avoid simple carbohydrate candy, su#ar, e.t.c
Thyroid "ormones
?e#ulates the oidation rate within the cells to influences
hysical and mental #rowth
5unctionin# of the nervous : muscles tissues
%irculatory activity
utrient metabolism
'n the low level of blood thyro#lobulin
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Thyroid stimulatin# hormon (T+")
(hypophsis anterior)
+timulate
Activity of thyroid #land increase
! with draw circulatory iodide
! concentrate
! oidizes ! iodine
C
Thyro#lobuline di iodotyrosine incorporate totyrosine
%omple triiodotyronine to forms
thyroine
%irculatory
B' protein bindin# iodine
"yperthyroidism Eophtalmic #oiter
Thyrotoicosis
9rave disease
Basedow8s diseases
Disturbance of thyroid function due to the eessive secretion of thyroid #land with a
conse7uent increase in the metabolic rate
%hief symptomes &
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wei#ht loss
Eessive
rominence of the eyes
Enlar#ement of thyroid #land
'ncrease appetide
wea$ness
cardic failure
serum B' elevated
basal metabolic increase 1/ 4 or more
increase of nitro#en metabolism ! destruction of tissue protein
ecretion of %a and increase ! osteoporosis, bone structure
Dietary modification &
restore normal nutrition
3/// 1/// calories
rotein -// -21 #r
%a 2 = #rFday
+upplement vit D, vit A, B comple, vit %
"ypothroidism
Decrease production of thyroid hormon
;nown as myedema (adult), cretinism (infant)
#OINT DISEASE
Arthritis
@
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?heumatism
9out
?heumaticfever &
in child or youn# adult
in adu7uate treatment
permanent dema#e of heart
osteoarthritis (de#enerative arthritis)
especially women youn# adults
inflamation, paintful
cause (theory) &
infection
hypersensitivity
hereditary
metabolic disorders
sympotmes & fati7ue
pain
stiffness
deformity
the disease is pro#ressive
the symptomes may spontaneously dissapear but reappear
later
supportive diet
in #ood nutritional status ! normal diet
obese ! low calorie diet (wei#ht loss)
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in poor nutritional status ! hi#h calorie and protein diet (wei#ht #ain)
in steroid therapy, when a retention
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*idized
6ric acid
6ric acid is also synthesized in the body from simple carbon and nitro#en compound.
+tores in liver and tissues
Eerated in urine and via the bile into intestinal
tract
Dietary modification &
owFrestriction purine diet
urine content of blood (per -// #r)
9roup ' & (/ -1 m#)
ve#etables caviar
fruits nuts
mil$ butter
chesse #elatin
e##s su#ar
bread and cereal fish roe
9roup '' & (1/ -1/ m#)
meats dry beans
poultry peas
fish spinach
sea food oatmeal
9roup ''' & (-1/ m# over)
sweet breads $idney
sardines meat etract
liver brains
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purine restriction (#roup '', limited amount)
ener#y obese low calorie diet (-2// -@// cal) in interval time
protein restricted to about @/ #Fday
fruits G = l per day
coffe, tea ! allowed
Osteo$orosis
deficiency disease
mostly the result of endocrine disorders
hyperthyroidism
hyperparathyroidism
hyperadrenocortism
acrome#aly
immobilization
rheumatoid arthritis
sic$le cell anemia
nutritional deficiency
symptomes &
low bac$ pain
vertebral fracture
Metabolic chan#es &
characterized by a reduction in the total bone mass
ne#ative balance of %a, , and level in blood
Dietary treatment
estro#en and andro#en
(anabolic effect, mineral retention)
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%a is recommended (-,1 # Fday)
+upplement & %a
Jit D
rotein & (hi#h protein inta$e)
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