Conflicts of interest: None Problematic Peds Employer: Idaho … · Intubation Tips Ways to select...
Transcript of Conflicts of interest: None Problematic Peds Employer: Idaho … · Intubation Tips Ways to select...
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Problematic Peds
Lucus Christoffersen, MSN, RN, CEN, CPEN, CCRN
Assistant Clinical Professor
Idaho State University
Disclaimer
Conflicts of interest: None
Employer: Idaho State University and Bingham Memorial Hospital
Sponsorship/Commerical support: None
Background
Clinical Assistant Professor since Jan 2016
Teach Adult Nursing Health I and II, along with clinical components
House Supervisor at Bingham Memorial Hospital in Blackfoot, Idaho
Utah State Emergency Nurses Association President, 2016-2018
Journal of Emergency Nursing
Reviewer
Emergency Nurses Association
Test Item Writer and Reviewer/Publication Writer and Reviewer
8 years of Critical Care, Emergency, and Trauma Nursing
Level 1 trauma centers to rural community hospitals
ICU, ED, LifeFlight and Trauma Resuscitation
Emergency Department Manager/Educator
Board Certified in Emergency, Pediatric Emergency, and Critical Care
Objectives
Understand normal pathophysiologic changes associated with pediatric
patients
Discuss and understand common complications seen with pediatric patients
Recognize interventions that will improve the care of pediatric patients that
are related to the disease processes
Terminology
Let’s take a few moments to discuss terms:
Neonate – birth to 28 days
Term – 37-40 weeks of pregnancy
Pre-term - <37 weeks pregnant
A pre-term infant is considered a neonate (newborn) until the expected due date plus 28
days
Infant – 1-12 months
Toddler 13 months to 3 years
Preschoolers – 3 – 5 years
School-Aged Child – 5-11 years
Adolescent – 11-18 years
Pediatric Patients Pathophysiology
There are many characteristics that make pediatric patients different than
adults
Remember that pediatric patients are NOT small adults!
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A–Airway and Alertness
B-Breathing
C-Circulation
Obligate nose breathers
Smaller airway diameter
Tongue is larger in proportion
Respiratory rates are faster
Increased metabolic rate
Lower tidal volumes
Alveoli fewer and smaller
Flat diaphragm
Use abdominal muscles for breathing
Largely comprised of water
Myocardium less compliant
Poor tissue perfusion
Tachycardia
Delayed capillary refill
Head proportionately larger
Smaller subarachnoid space
Cranial sutures and open fontanels
Normal intracranial pressures
Larger body surface area to body mass ratio
Limited ability to regulate temperature
Less body fat
Thinner skin
Hypothermia
D-Disability
E-Environment
Assessment of Pediatric Patient
Remember ABCDE’s
A-Airway
B-Breathing
C-Circulation
D-Disability (Neuro Functioning)
E-Environment
S-Safety
PAT – Pediatric Assessment Triangle
Across the room observation, looking at general appearance, work of breathing,
skin color/circulation status
Gives a quick understanding of the pediatric patient’s compensation and risk for
deterioration
General Appearance
Includeso Interactiono Muscle toneo Consolabilityo Look or gazeo Speech or cry
Reflectso Oxygenationo Ventilationo Perfusion of vital
organs
Work of Breathing
More accurate indicator of
oxygenation and ventilation
Look and listen for:
o Abnormal airway sounds
o Coughing
o Abnormal positioning
o Retractions/Nasal
flaring
o Rate and depth
Circulation to the Skin
Adequate circulation to the
skin reflects perfusion
Inspect the skin
o Color
Central areas
Lips
Mucous membranes
o Pallor, mottling, cyanosis
o Flushed, diaphoretic, dry
“Sick, Sicker, Sickest”
If all components of the PAT are stable, consider the child
sick based on concerns of caregiver
o Considered sick until proven otherwise by conducting
the focused assessment
Acute disruption in one component of the PAT, consider to
be sicker
Acute disruption in two or more components of the PAT,
consider sickest and should warrant immediate care by
the team!
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Clinical Red Flags
Airway – apnea, choking, drooling
Breathing – grunting, retractions, tachypnea/bradypnea, absence of
breath sounds, cyanosis
Circulation – cool or clammy skin, tachycardia/bradycardia, delayed
capillary refill, diminished peripheral pulses
Disability – change in level of consciousness (from baseline), sunken or
bulging fontanel
Exposure – petechiae, purpura, signs of potential child maltreatment
Full Set of Vital Signs – hypothermia, fever
Give Comfort – severe pain
History – chronic illness, return visit to ED within 24 hours
History
CIAMPEDS
C Chief complaint P Past medical history and Parent’s impression
I Immunizations and isolation
E Events surrounding illness or injury
A Allergies D Diet and diapers
M Medications S Symptoms associated with illness or injury
Respiratory Emergencies
Upper Airway
Croup
Epiglottitis
Bacterial tracheitis
Foreign bodies
Sleep apnea
Tracheomalacia
Lower Airway
Asthma
Bronchiolitis
Pertussis
Pneumonia
Foreign bodies
Bronchomalacia
Failure secondary to neurological disorder (muscular dystrophy) or anatomical issues (scoliosis/kyphosis)
Respiratory Distress vs.
Respiratory Failure
Respiratory distress o Increased respiratory rate
Tachypneao Increased respiratory effort
Nasal flaring
Use of accessory
muscles
Retractions
Respiratory failure
o Fatigue from excessive work of
breathing
o Inadequate oxygenation and/or
ventilation
Signs and Symptoms
of Respiratory Distress
Increased work of breathing
Tripod position
Paradoxical respirations
Change in skin color
Drooling
Fast respiratory rate
Fast heart rate
Upper airway sounds
Adventitious breath sounds
Signs and Symptoms of
Respiratory Failure
Change in level of consciousness
Decreasing PO2 and/or increasing
PCO2
Can exist with:
o Slow respiratory
rate
o Normal respiratory
rate
o Fast respiratory
rate
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Monitoring
Monitors
o Pulse oximetry
o Capnography
o Cardiopulmonary
monitor Poor perfusion may impede the
monitor’s ability to obtain an
accurate reading
Intubation Tips
Ways to select the size of endotracheal tube:
1. Length-basedresuscitation tape
2. (Age in years +16) / 4
3. Patient’s pinky finger
4. Patient’s naris
5. Half size smaller than the calculated uncuffed ETT diameter
Have both cuffed and uncuffed ETTs available
Alternate Airway
Laryngeal mask airway
Needle cricothyrotomyo When all other options have failed
Surgical cricothryroidotomyo Not recommended for children <12 years
Emergency tracheostomy is rarely indicated
Croup
Upper respiratory disorder- Commonly viral
Barky, seal-like cough – worse at night
Treatment is symptomatic/supportive
o Cool mist/oxygen, as indicated
o Consider corticosteroids
o Consider racemic epinephrine
(typically 2 hours relief)
Concurrent resting stridor
Observe for rebound effect
Epiglottitis
This is an emergency! Symptoms
o Acutely high fevero Muffled voiceo Severe sore throat − difficulty swallowingo Droolingo Stridoro Use of HIB vaccination has decreased the incidence in the pediatric
populationo Causes
o Haemophilus influenzae type Bo Streptococcus pneumoniae and Staphylococcus
o Lateral neck film – thumb signo NO STRESS! Keep the patient calm until airway stabilized
Asthma
Most common chronic childhood illness – 9.3% of children
in the U.S.
Confirmation of diagnosis delayed until the child has had
repeated episodes and is older than one year
A chronic inflammatory disorder of the airways, which is
characterized by:
o Hyperreactiveness of airway
o Widespread inflammatory changes
o Bronchospasm
o Mucous plugging
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Signs/Symptoms of Asthma
Wheezing on inspiration or expiration
Prolonged expiratory phase
Decreased or unequal breath sounds
Tachypnea
Retractions
Coughing, especially at night and in the early morning
Asthma: Interventions
Supplemental O2 & monitoring of O2 saturations
Obtain a peak expiratory flow
Administer medications as ordered
o Inhaled short-acting ß2-agonists and anticholinergic agents
o Steroids
Reassess after dose of inhaled bronchodilator and at least every
60 minutes thereafter
Prepare for hospital admission or transfer if the child’s
condition does not improve
Prepare for discharge only if child’s condition improved and
desired outcomes are achieved
Bronchiolitis
Acute viral infection
Involves the lower respiratory tract and is most commonly
caused by the respiratory syncytial virus (RSV)
Symptoms generally worsen for the first 3-5 days and then
gradually improve
Apnea is one of the most concerning complications of
bronchiolitis in young infants
History of prematurity or cardiac/pulmonary diseases at
greater risk for severe life-threatening manifestations
Signs/Symptoms of Bronchiolitis
Rhinorrhea
Pharyngitis
Coughing/sneezing
Tachypnea
Retractions
Wheezing/prolonged
expiratory phase
Decreased air entry or
exchange
Dehydration secondary to
decreased oral intake
Apnea spells
Low-grade fever
Bronchiolitis: Interventions
The most important intervention is nasal suctioning to decrease
secretions
Place in enhanced contact isolation
Obtain a nasopharyngeal specimen for RSV testing
Administer medications as ordered
Routine use of bronchodilator/corticosteroids controversial
Nebulized saline may reduce length of hospitalizations
Palivizumab (Synagis)
Causes of Pediatric Dysrhythmias
Unrecognized respiratory or
metabolic compromise
Congenital cardiovascular
abnormalities
Long QT syndrome
Marfan syndrome
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Sudden Cardiac Arrest: Causes
Structural defectso Cardiomyopathies
o Arrhythmogenic right ventricular cardiomyopathy
o Myocarditis
o Congenital coronary artery anomalies
o Marfan syndrome
Arrhythmogenic disorderso Congenital long QT syndrome
o Other channelopathies
Marfan Syndrome
Inherited genetic
mutation
Appears at any age
Complications
o Aneurysms
o Mitral valve prolapse
Medical supervision
Long QT Syndrome
Inherited Prolonged QT
o Exercisingo During severe emotionso When startled
Red flagso Chest paino Syncopeo Palpitations
Bradycardia: Interventions
No signs of decreased CO: No treatment needed Ventilate with 100% oxygen Obtain vascular access Begin chest compressions if pulse is <60 BPM or signs of
poor perfusion Lethargy, comatose, pallor, cyanotic, CRT >5 seconds
Look for potential causes Administer pharmacological agents
o Epinephrine (drug of choice in pediatric patients) Consider pacing
Sinus Tachycardia:
Signs and Symptoms
Poor perfusion
o Delayed capillary refill
o Pale cool extremities
o Mottled skin
o Decreased BP
Dry mucous membranes
Decreased number of wet
diapers
Increased stool/diarrhea or
vomiting
Increased work of breathing
Sinus Tachycardia: Interventions
Support ABCs
Look for cause of
tachycardia and treat
If due to hypovolemia:
o Obtain vascular access
o Fluid bolus 20 mL/kg
(unless cardiac cause)
If due to fever:
o Monitor and maintain
normal body
temperature
If due to pain:
o Treat with analgesics
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SVT: History
Previous SVT episode Wolff-Parkinson-White syndrome Congenital heart disease Poor feeding, fussiness Sudden onset of symptoms
SVT: Signs and Symptoms
Pale, cyanotic or mottled Hypotension Poor perfusion Altered level of consciousness Irritability Heart rate too fast to count Signs of congestive heart failure
SVT: Interventions
Vagal stimulation/
maneuvers
Support ABCs
Adenosine (Adenocard) 0.1-0.2 mg/kg
Synchronized
cardioversion 0.5-1 joule/kg
SYNC mode
Ventricular Tachycardia: Assessment
History
Structural heart disease
Myocarditis/ cardiomyopathy
Prolonged QT syndrome
Suspected overdose
Electrolyte imbalance
Signs and Symptoms
Poor perfusion
Ventricular rate > 120 bpm
Wide QRS (> 0.09 seconds)
Pulses may be absent
Palpitations
Increased work of breathing
Ventricular Tachycardia: Treatment
With a Pulse
Obtain immediate cardiology
consult
Support airway and
ventilation
Obtain ECG
Obtain venous access
Consider synchronized
cardioversion
Medications
o Adenosine (Adenocard)
o Amiodarone (Cordarone)
o Procainamide (Pronestyl)
Without a Pulse
• Defibrillate – follow VF
algorithm
Ventricular Fibrillation: Assessment
History
Electrocution
Recent viral illness
History of cardiac
surgery/transplant
Ingestion of toxin
Blunt impact to chest
Signs and Symptoms
Unresponsive child
Absence of palpable pulse
Chaotic, wavy lines
No P or T waves or QRS
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Ventricular Fibrillation: Treatment
Defibrillate at 2 J/kg
Resume CPR for 5 cycles after each shock
Subsequent shocks at 4 J/kg
Vascular access
Medications
Family presence
Used with permission of Physio-Control,
Inc.
Asystole: Assessment
History
Time patient was found pulseless
Events prior to pulselessness
Primary caregiver at time of event
Time of BLS initiation
Time of ALS initiation
Signs and Symptoms
Absent electrical activity
Apnea
Profound shock/poor peripheral
perfusion
Absent heart sounds
No palpable pulses
Asystole: Treatment
Treatment as outlined for PEA
BEST intervention is PREVENTION!
Confirm asystole in two leads
High quality CPR
Family presence with assigned staff
Anticipate need to terminate resuscitative efforts
Fever
Most common complaint
Normal physiologic response
Rectal temperature at least 38°C
(100.4°F)
Degree does not reflect severity
Fever: Diagnostics
and Interventions
Lab studies
Radiographic studies
Antipyretics
Septic work-up
Fluids
Dehydration
Definition and causes
o Fluid loss in excess of intake
o Most common cause is gastrointestinal sources
Pediatric characteristics
o Relatively more body water
o Higher metabolic rate leads to increased insensible fluid loss
o Greater body surface area to weight ratio
o Immature kidney function
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Dehydration: Interventions
Oral rehydration
Subcutaneous fluid replacement
Intravascular fluid replacement
Electrolyte replacement
Monitor weight, intake and output
Consider admission
Gastroenteritis
Definition: GI viral infection
Causes
o Rotavirus, adenovirus, enterovirus
Signs and symptoms
o Fever
o Nausea and vomiting
o Diarrhea
Additional history
Interventions
o Antiemetics
o Rehydration
Meningitis
Inflammation of the meninges Common bacterial organisms
o Neonate Group B streptococcus
o Older child Neisseria meningitidis Haemophilus influenzae (H flu)
Complicationso Seizureso SIADHo Neural damage
Meningitis: Signs and Symptoms
Fever Irritability Mental status changes Poor feeding/vomiting Seizures Bulging fontanel Headache and stiff neck Nonblanching rash
Meningitis: Diagnostics
and Interventions
Septic work-up o CBC with differential
o Electrolytes
o Glucose
o Urinalysis and urine culture
o CSF for cell count, culture,
protein, glucose, and gram stain
Isolation
IV, fluids, antibiotics
Meningococcemia
Definitiono Neisseria meningitidis sepsis
Signs and symptomso Fever
o Mental status changes
o Signs of shock
o Nonblanching rash
Prognosis - Poor
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Meningococcemia Interventions
Isolation
Antibiotic therapy
Monitoring
Clotting factor replacement,
platelet or plasma transfusion
Inotropes/vasopressors
Seizures
• Definition: o CNS dysfunction
• Causes o Fever
o Metabolic disturbance
o Toxin exposure
o Trauma
• Prevalence
• Types o Generalized
o Partial seizures
o Unclassified: febrile, neonatal
• Status epilepticus
Seizures: Diagnostics
and Interventions
Diagnostics
Safe environment
Oxygen and suction
IV access
Monitoring
Medications IV Ativan
Rectal Diazepam
Hydrocephalus
Definitiono Abnormal production, absorption,
or drainage of CSF
o Present with shunt obstruction,
infection
Signs and symptomso Mental status changes
o Signs of increased ICPo Lethargy, agitation, irritable
o Fever
o Bulging Fontanel
Diagnosticso Shunt series, CT, MRI
o CSF analysis, CBC, blood/urine
cultures
Interventionso Surgical repair
Constipation
Definitiono Infrequent or painful defecation
Age groupso Infantso Toddlers
School age Signs and symptoms
o Pain or bleedingo Common withholding behaviors
Squatting Crossed ankles/legs Body stiffening Flushing Sweating Crying
Diagnostics Interventions
Increase fluids, Mag Citrate
Appendicitis
Definitiono Inflammation of the vermiform
appendix
Signs and symptomso Abdominal pain, fever, vomiting,
anorexia
Diagnosticso Clinical exam
o Ultrasound
o CT
Interventions
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Diabetic Ketoacidosis
Presentationo Serum glucose > 300 mg/dLo Dehydrationo Serum pH of < 7.3o Ketonemia/ketonuriao Glycosuria
Signs and symptomso Kussmaul breathingo Mental status changeso Dehydrationo Electrolyte imbalanceo Cardiac dysrhythmiaso Acetone breath
Diabetic Ketoacidosis: Interventions
Correct dehydration
Serial laboratory studies
Correct acidosis
Correct electrolyte imbalances
Monitor neurologic status, vital signs, and ECG
Accurate intake and output
Coagulation Disorders
Hemophilia
von Willebrand
Signs of bleeding and bruising
Treatment
o Replacement of clotting
factor
o Immobilization of affected
joints
o Pain management
Coagulation Disorders
Idiopathic thrombocytopenic
purpura From Viral infections
Signs of bleeding and bruising,
petechiae and purpura
Treatment
o IgG
o Corticosteroids
o Limitation of activity
o Transfusion for severe
blood loss
Apparent Life-Threatening Events
Episode of apnea with o Color changeo Loss of muscle toneo Chokingo Gagging
Historyo Detailedo Infant o Family
Assess for:o Infection
o GERD
o Seizures
o Pulmonary assessment
o Metabolic laboratory
o Neuromuscular
o Assess for signs of child abuse
Burns
Integumentary disruption
Fluid shifts
Vasoconstriction
Tissue necrosis
Electrolyte imbalance
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Burn Assessment
Airway
• Stridor
• Hoarse voice or cough
• Carbonaceous sputum
• Drooling
• Edema
• Blisters
• Singed nasal hairs,
eyebrows, or lashes
Breathing
• Burns to chest
• Inhaled toxins
• Aspiration
Circulation
• Signs of shock
• Urine output
• Laboratory analysis
results
Burn Assessment
Depth o Superficial partial-thicknesso Deep partial-thicknesso Full-thickness
Extento Rule of Nineso Lund and Browdero Rule of Palm
Calculation of fluid needs Only needed for Deep-Partial and Full-thickness burns Parkland Formula Lactated Ringers
2-4 ml/kg X BSA burned 2 ml/kg – Pediatrics 3 ml/kg – Adults 4 ml/kg – All electrical burns
½ of total fluid given in first 8 hours of the initial burn time
Remainder given in next 16 hours Example – 4 year old patient with 50% burns who ways
15 kg from a chemical burn in father’s shop 2 ml of LR X 15 kg X 50% = 1500 ml of LR Fluid 750 ml given in first 8 hours = 94 ml/hr of fluid
Burn Management
Stop the burning process
Wound care
Gastric tube
Laboratory studies
Pain management
Transfer
Head Trauma Assessment
● Neurological assessmento Glasgow Coma Scale
o FOUR Score
o Pupils
● History: o Loss of consciousness
o Nausea and vomiting
o Abnormal behavior
o Seizure activity
● Cushing’s triado Bradycardia
o Hypertension
o Alteration in respiration
Head Trauma Assessment
● Inspect and Palpateo Lacerations
o Abrasions
o Step offs
o Edema
o Hemotympanum
o Periorbital/postauricular
ecchymosis
o Otorrhea/rhinorrhea
o Fontanel
● Assess neurological
functiono Gait
o Grip strength
o Reflexes
Head Trauma Interventions
Elevation of HOB
Oxygenation and ventilation
Sedation and analgesia
Diuretics
Maintain temperature
Trending of vital signs and neurological status
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Dental Trauma
Tooth fracture or avulsion
Interventions Baby tooth – follow up with
dentist
Adult teeth – clean off, stick
tooth back in socket and refer to
dentist If patient unconscious, place
tooth in glass of milk
Vertebral and Spinal Cord Trauma
Mechanism
o MVC
o Sports injuries
Anatomy and physiology
o Larger head
o Weak neck muscles/lax
ligaments
o Anteriorly wedged
vertebral bodies
o Growth plates
SCIWORA
o Spinal cord injury without
radiographic abnormality
Vertebral and Spinal Cord Trauma
Signs and Symptoms:
o Spinal deformity
o Neck pain
o Flaccid extremities
o Altered sensation
o Incontinence
o Absent sphincter tone
o Priapism
Vertebral and Spinal Cord Trauma
Interventions:
o Maintain full spinal
stabilization
o CT with radiologist
interpretation
o Neurosurgical consult
Cardiothoracic Trauma
Often component of multisystem
injury
Blunt vs. penetrating
Rib fractures
Cardiothoracic Trauma
Simple pneumothorax
Open pneumothorax
Tension pneumothorax
Hemothorax
Pulmonary contusion
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Abdominal Trauma
Mechanismo MVC
o Bicycle crashes
o Sports activities
Anatomy and physiologyo Thinner, weaker muscles
o Chest wall pliable
o Ribs horizontal
o Vascular duodenum
Abdominal Trauma Assessment
Location, quality and radiation of pain
Respiratory pattern and depth
Rigidity, guarding and distention
External soft tissue injury
Bloody urinary drainage
Abdominal Trauma Interventions
Cover open wounds with moist,
sterile dressings
FAST exam
Prepare for surgery
Musculoskeletal Trauma
Fracture
Sprain
Subluxation
Dislocation
Fracture Assessment
Deformity, shortening or
rotation
Edema or soft tissue injury
Tenderness
Reluctance or refusal to use
Neurovascular statuso Pallor
o Pain
o Pulselessness
o Paresthesia
o Paralysis
o Poikilothermia
Fracture Interventions
Compare circulation of affected extremity to the unaffected extremity
Proper alignment Immobilization Comparison radiographs Closed reduction Fluid replacement Spiral Fractures
Usually indicate child abuse – should report
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Summary
Pediatric patients have many compensatory mechanisms that may prevent
them form showing deterioration until later than expected
Many pediatric cardiac complications and codes are from respiratory
complications
Oxygen, Glucose, Thermoregulation are top three interventions for pediatric
patients
Immunizations help lower incidence of many childhood illnesses
Patient education and safety training are key to preventing many childhood
and pediatric problems
References
ENPC 4th edition Provider manual – Chapters 3, 4, 5, 7, 10-17