Chronic Inflammatory Response Syndrome · 2020-03-10 · Key Facts 95% •of CIRS occurs in...
Transcript of Chronic Inflammatory Response Syndrome · 2020-03-10 · Key Facts 95% •of CIRS occurs in...
Chronic Inflammatory Response Syndrome
Dr Sandeep Gupta
[photo of blue-green algae or infected
waterway]
Mold Illness Made Simple © Dr Sandeep Gupta2
[photo of intensive care unit]
Stages of Sepsis
Initial infection
Bloodstream spread
Inflammatory Cascade
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Chronic Inflammatory Response Syndrome
Biotoxin exposure Chronic inflammation
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[photo of water-damaged building and a
tick]
Mold Illness Made Simple © Dr Sandeep Gupta8
Predisposition to inflammatory illnesses
Specific gene types
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Key Facts
95%
• of CIRS occurs in genetically predisposed people
HLA
• Genetic glitches lead to chaotic inflammatory response on exposure to biotoxins
CSM
• An external binding agent such as cholestyramine can allow the body to rid itself of biotoxins.
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Inflammation
Redness
Pain Swelling
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Cytokines
Redness
PainSwelling
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CIRS Inflammation
Silent
Deep-seated
Chaotic and inefficient
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Poor oxygen delivery to
tissues
Immune dysfunction
Hormonal changes
Inflammation in CIRS
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Biotoxin Illness in AustraliaEpidemic due to:
• Structure of houses
• Use of particle board
• Sub ground level basements
• Lack of curing of slabs
• Frequency of flooding due to extreme weather conditions etc
• Outdoor lifestyle/abundance of rainforest
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Surviving Mould Down Under
Essays documenting exact companies and brands to use to
implement Dr Shoemaker’s protocol in Australia
Discusses which items covered by Medicare
PBS Criteria for various medications discussed
Australian resources for treatment of tick-borne infections
discussed
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Commonly Known Biotoxins
Mycotoxins (of fungal original)
Ochratoxin A
Aflatoxin
Trichothecene
Borrelia
Babesia
Ciguatera toxin
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Common known biotoxins
Mycotoxins (of fungal origina)
Ochratoxin A
Aflatoxin
Trichothecene
Bacterial toxins
Botulinum toxin
Tetanospasmin
Exfoliatin or Enterotoxin Type B
Borrelia and co-infections ?
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Summary
A very specific pathophysiology of CIRS including defective antigen presentation in those with impaired HLA genetics followed by hyperactivity of the innate immune system has been described by Shoemaker.
As the science and experience evolves theories are springing up to explain observations not easily explainable by the main theory. It may be that no one theory explains all pieces.
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