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    ger code - Wikipedia, the free encyclopedia

    ://en.wikipedia.org/wiki/Berger_code(1 of 5) [2/20/2008 3:41:43 AM]

    Berger codeFrom Wikipedia, the free encyclopedia

    In telecommunication, a Berger code is an unidirectional error detecting code, named

    after its inventor, J. M. Berger. Berger codes can detect all unidirectional errors.

    Unidirectional errors are errors that only flip ones into zeroes or only zeroes into ones, such

    as in asymmetric channels. The check bits of Berger codes are computed by summing all

    the zeroes in the information word, and expressing that sum in natural binary. If the

    information word consists ofn bits, then the Berger code needs

    "check bits", giving a Berger code of length k+n. (In other words, the k check bits are

    enough to check up to n = 2k

    1 information bits). Berger codes can detect any

    number of one-to-zero bit-flip errors, as long as no zero-to-one errors occurred in the same

    code word. Berger codes can detect any number of zero-to-one bit-flip errors, as long as

    no one-to-zero bit-flip errors occur in the same code word. Berger codes cannot correct

    any error.

    Like all unidirectional error detecting codes, Berger codes can also be used in delay-

    insensitive circuits.

    As stated above, Berger codes detect any number of unidirectional errors. For a

    given code word , if the only errors that have occurred

    are that some (or all) bits with value 1 have changed to value 0, then this transformation

    will be detected by the Berger code implementation. To understand why, consider that

    there are three such cases:

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    [edit]Unidirectional error detection

    ArticleDiscussionEdit this pageHistory

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    For case 1, the number of 0-valued bits in the information section will, by definition of the

    error, increase. Therefore, our berger check code will be lower than the actual 0-bit-count

    for the data, and so the check will fail.

    For case 2, the number of 0-valued bits in the information section have stayed the same,

    but the value of the check data has changed. Since we know some 1s turned into 0s, but

    no 0s have turned into 1s (that's how we defined the error model in this case), the

    encoded binary value of the check data will go down (e.g, from binary 1011 to 1010, or to

    1001, or 0011). Since the information data has stayed the same, it has the same number

    of zeros it did before, and that will no longer match the mutated check value.

    For case 3, where bits have changed in both the information and the check sections, notice

    that the number of zeros in the information section has gone up , as

    described for case 1, and the binary value stored in the check portion has gone

    down , as described for case 2. Therefore, there is no chance that the two will end

    up mutating in such a way as to become a different valid code word.

    A similar analysis can be performed, and is perfectly valid, in the case where the only

    errors that occur are that some 0-valued bits change to 1. Therefore, if all the errors that

    occur on a specific codeword all occur in the same direction, these errors will be detected.

    For the next code word being transmitted (for instance), the errors can go in the opposite

    direction, and they will still be detected, as long as they all go in the same direction as

    1. Some 1s bits in the information part of the code word have changed to 0s.

    2. Some 1s bits in the check (or redundant ) portion of the

    code word have changed to 0s.

    3. Some 1s bits in both the information and check portions have changed to 0s.

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    ger code - Wikipedia, the free encyclopedia

    Navigation://en.wikipedia.org/wiki/Berger_code(3 of 5) [2/20/2008 3:41:43 AM]

    each other.

    Unidirectional errors are common in certain situations. For instance, in flash memory, bits

    can more easily be programmed to a 0 than can be reset to a 1.

    [edit]References

    J. M. Berger, "A note on an error detection code for asymmetric channels",

    Information and Control , vol

    4, pp. 68-73, March 1961.

    Subhasish Mitra and Edward J. McCluskey, "Which concurrent error detection scheme

    to choose?", Center for Reliable Computing, Stanford University, 2000.

    This computer science -

    related article is a

    stub . You can help

    Wikipedia by expanding it .

    "Delay-Insensitive Codes -- An Overview" by Tom Verhoeff

    Categories: Coding theory | Error detection and correction | Computer science stubs

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    ger code - Wikipedia, the free encyclopedia

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    Why the 4 code Genetic Primer Should Have 6 Nucleotide Codes

    Inosine controls the "degradation" process

    Inosine fulfills the first law of thermodynamics which states that energy or

    matter is neither lost or gained in a chemical reaction but changes states

    through phasesInosine two critical roles (IMP to ATP synthesis) plus responsible for the

    entire degradation and recycling process qualifies it to be uracil'snucleotide

    partner and the sixth and final nucleotide code

    Inosine, itself, is the major "recycler" of "spent" amino acids (bases, sugars,

    phosphodiesters)

    IMP is the initiator of the nine step process which results in the synthesis ofATP (Adeno-Tri-Phosphate)

    Inosine through its IMP (Inosine Mono-Phosphate) compound is the first purine tobe completed

    we submit Inosine replaces adenosine when a new stem, branch or shape is

    requiredironically dr. ferry won a nobel prize in 1953 for his discovery of thewobble position in the tRNA amino acid assembly process

    Inosine takes the place of adenosine when the amino acid chain begins anon-linear "cloverleaf" directional change in the third anti-codon or z (height)

    position, Inosine bonds with uracil, guanosine and cytosineThe substitution of U for T keeps the same number of genetic nucleotide codes

    (i.e.) equal when DNA combines with RNA to form one of the twenty amino acids

    found in every protein

    The net difference in the substitution of U for T is the loss of 2 hydrogen

    protons and the gain of one oxygen proton

    The second major difference between d (deoxy-ribonucleic acid) DNA and

    r(ribonucleic acid) RNA is RNA has one more furanose pentagonal ribose sugar

    molecule

    The net difference is H3-H1 = H2 or 2 hydrogen protons (H+)Thymine has a CH3 on position 6 of the C4N2 guanosine base while uracil hasonly a CH1

    Inosine never bonds with thyminePrescription medications are the fourth leading cause of death in the United

    States

    300% more people die from medicines prescribed by their physician than in

    automobile and motorcycle accidents

    The current 4 dna nucleotide genetic codes are :1. (Adenosine=A), 2.

    (Thymine=T), 3. (Guanosine=G) and 4. (Cytosine=C)

    The current 4 rna nucleotide genetic codes are :1. (Adenosine=A), 2. (Uracil

    = U), 3. (Guanosine=G) and 4. (Cytosine=C)

    Uracil substitutes for Thymine in the current 4 code dna/rna amino acidsynthesis process

    The DNA molecule resides in the nucleus of most if not all cell types

    The RNA molecule resides and performs its functions outside the nucleus but

    within the cellular walls

    the future of medicine is based on the foundation of the double helix model

    discovered in 1953 by waston and crick

    all current genomic technologies are based on a four (4) code genetic primer

    (dna = ATGC), (rna=AUGC)

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    ATGC = dna , AUGC = rna; AGC remain the same symbols in the same positions in

    the current dna/rna transcription, translation and Transformation processes

    The key differences between uracil and thymine are two fold:the substitution of u for t keeps the same number of genetic nucleotide codes

    (i.e.4) equal when dna combines with rna to form one of the twenty amino acids

    found in every protein

    the net difference in the substitution of u for t is a. the loss of 2 hydrogen

    protons and b. the gain of one ribose sugar molecule

    the second major difference between d (deoxy-ribonucleic acid) dna and

    r(ribonucleic acid) rna is obviously rna has one more furanose pentagonal ribose

    sugar molecule or one more "dose" of energy

    thymine has a CH3 on position 6 of the C4N2 guanosine base while uracil hasonly a CH1 - the net difference is H3-H1 = H2 or 2 hydrogen protons (H+)

    In 1966 the "almost" universal code was finally "solved" to the satisfaction of

    the international life sciences scientific community

    the "almost " universal genetic code contains 64 unique 3 bit nucleotide codes

    each of the twenty amino acid is composed of 3 nucleotide codes

    there are 4 total nucleotide codes; only 3 are used to "code" for one of the

    twenty amino acids since 2 is not equal to 3 then symmetry, equilbrium or charge

    neutrality does not exist for every two (2) A-T pairs, there are three (3) (G-C)

    pairsthe guanosine - cytosine pairing occurrs 50% more frequently than the

    adenosine-thymine pairing

    in rna adenosine is always covalently paired with uracil (remember u

    substitutes for t in dna to rna) and guanosine remains paired with cytosine

    Per Chargraff dna bond pairing in dna adenosine always is bonded to thymine

    and guanosine is bonded to cytosine

    Adenosine, Uracil, Guanosine, Cytosineevery polypeptide is connected by the sulfur amino acid cysteine through its

    disulfide bonds

    Alternative interpretation of the scientific facts

    scientist discover five different nucleotides (Adenosine, Thymine,

    Guanosine, Cytosine and UracilThe number 5 position or structure took the place of the number 2 position or

    structure

    There are 5 known nucleotides at this point in scientific time (A,T,G,C,U)

    the number 2 (thymine) and number 5 (uracil) molecular structures are not thesame, they are not equivalent they are not interchangeable parts

    remember in this substitution of U for T, 2 hydrogen protons (H2+) are lost and

    one ribose sugar is gained

    The decision was made to substitute the fifth (5) =( Uracil = U) nucleotide

    for the second nucleotide (Thymine = T)These 5 nucleotides represent 5 different positions in the 230 space crystalline

    lattice structure

    These 5 nucleotides represent 5 different crystalline molecular structures

    thymine is in reality a sulfur thioester (thiamine) which mutated to its finalstate by an ultraviolet light b (nm < 200nm)mutation which created an aptamer or

    two thymineMathematical Operators

    Inosine is the best and most likely candidate for the uracil's bonding partnerand sixth nucleotidein the wobble rules

    uracil's partner would have to be able to interact effectively with the twoother pairs of nucleotides (A-T,G-C)

    IMP is the starter to ATP; it provides the same function as a "starter" in a

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    Car once the engine "starts" the starter or IMP role becomes less critical andis observed less frequently in intermediate metabolic processes

    ATP is produced by oxidative phosphorylation in the Mitochondria, the power

    generating plant organelle, in all plants and animals

    would have to provide mission surival critical functionality

    ATP is the universal energy molecule for all carbon based life forms

    the genetic primer's 3 codon pairs specify the x (breadth), y(length) and z

    (height) of the amino acid, polypeptide protein to be made

    the main purpose of the genetic primer is to specify the generic type of stem

    cell for exact shape and size and the exact atomic and molecular compound

    compositions

    the site on which the amino acid will be assembled by the ribosome

    would be the third purine in joining adenosine and guanosinein the last codon position (x,y,z)

    in the z position of the current watson crick bonding arrangements A-U is the

    covalent pairing

    would have to be able to bond to cytosine and uracil but not thyminetRNA the last of the three phases, insures the transfer and validity of the

    codon and anticodon strands to be assembled in the ribosomes

    ribosomal rna which is the actual physical interface of the biochemical

    molecules (nucleotides) to the crystalline molecular substrate or base of the

    amino acid assembly

    messenger rna which decodes the dna instruction set, specifies

    the exact sequence of codons

    when the double helix hydrogen bonds which bond the

    nucleotides together (A-T, G,C, I,U) is hydrolyzed

    would have to provide a non-linear solution to amino acid sequence growth and

    thus "budding"

    uracil's missing partner would have to have these characteristics orproperties

    Inosine and the "wobble effect" or wobble rules" were formulated by Dr. Francis

    Crick, of double helix fame

    what criteria might be used to determine the identity ofuracil

    partner and

    sixth nucleotide

    before the substitution of U for T there were five total genetic codes

    (A,T,G,C,U)

    the third pairing would be uracil and ? the sixth and final nucleotidewhat advantage would a 6 code genetic primer have over a 4 code genetic primer

    what would be the result of such a change in the current 4 code genetic primer

    uracil's partner would have to be involved in the major metabolic processesand

    amino acid synthesis

    uracil's partner would have to provide essential, survival level functions andservices

    since this is not how human life or any organic life increases the number in the

    species in order to IMProve the chances of continued existence then it isfallacious to expect nature to use substitution and not addition in the dna to

    rna transformation

    in sexual reproduction addition not substitution is the mathematical process at

    work

    addition is nature's and therefore "the creators" algorithm for organic life,

    growth and evolution

    why would addition be more likely than substitution in the dna rna

    transformation

    what if addition were used instead of substitution as the primary dna to rna

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    transformation

    when uracil was found, what if science had found a partner for uracil insteadof

    substituting uracil for thymine

    if a partner were found for uracil that would make 6 and not 4 genetic codes

    uracil's partner would certainly have to be found in amino acids and proteins

    which are "downstream" from the nucleotide phasethe second pairing would be guanosine and cytosine (G-C)

    the first pairing would be adenosine and thymine (A-T)Addition is more predictable than substitution

    the purpose of the genetic code is to conserve and "pass down" successful

    protein building plans which will increase the odds of survival and reproduction

    the genetic code is a "blueprint" or master plan for constructing three

    dimensional proteins

    the final product of the nucleotide/dna/rna/amino acid synthesis process is the

    construction of proteins and enzymes

    addition would provide greater reliability and fidelity in conserving the

    optimized protein building specifics such as location, composition and function

    the use of substitution as the most reliable way to conserve the "wisdom of the

    genes" as accumulated over 5 billion years of evolutionary "trial and error"learning is highly unlikely

    a rational and logical designer would use addition and not substitution to

    conserve the dna encoded survial lessons

    one male sperm plus one female egg combine to create one unique life form

    the male and female, numbered two

    after the "offspring" was born there were now three human beings not two

    if substitution was used for reproduction or genetic encoding then one of the

    parents would be extincted and there would remain two unique life forms: (i.e.

    one of the original parents and the offspring)

    The substitution mathematical operator (i.e. Uracil (U) for Thymine (T) is asecondary mathematical process or genetic algorithm

    The four fundamental, basic and most elemental math operators are 1. addition,

    2. subtraction, 3. multiplication and 4. divisionthe substitution of uracil = U for Thymine = T is the only "code" differencebetween the dna and rna genetic codes

    The substitution of uracil = U for Thymine = T is the only "code" differencebetween the DNA and RNA genetic codes

    Every single prescription drug ever made has side effects

    The current 4 RNA nucleotide genetic codes are :1. ((Adenosine=A), 2. (Uracil=

    U), 3. (Guanosine=G) and 4. (Cytosine=C)

    The current 4 DNA nucleotide genetic codes are :1. (Adenosine=A), 2.

    (Thymine=T),

    3. (Guanosine=G) and 4. (Cytosine=C)

    The future of medicine is based on the foundation of the double helix modeldiscovered in 1953 by Watson and crick

    The "almost " universal genetic code contains 64 unique 3 bit nucleotide codes

    called codons or 192 total nucleotide bases

    All current genomic technologies are based on a four (4) code genetic primer

    (DNA = ATGC), (RNA=AUGC)

    Violates Inheritance Laws

    Violates Inheritance Laws

    Purines

    Guanosine Monophosphate

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    GMP

    Adenosine MonophosphateAMP

    Adenosine Triphosphate ATP is the energy molecule which powers over 96% of allmetabolic reactions

    Evolution Missing Purine Genetic Code

    ADARADA

    Xanthine Hydrolase

    Xanthosine Oxidase

    IMPDH2

    IMPDH1HPGRT

    IMPPurine Catabolic Degradation

    Purine Synthesis Salvage

    Pyrmidine Metabolism

    First and Last Metabolic Process Steps in DNA and RNA Metabolism are certainly

    critical metabolic roles

    Inosine Family (XMP -xanthine oxidase) is final step purine catabolicmetabolism; removes toxic ammonia from central nervous system - especially

    glutamate - the major excitatory nuerotransmitter in human brain

    Inosine family (IMP) starts purine nucleotide synthesis which ultimately resultsin the RNA and DNA Molecules

    Purines

    Purine Nucleotide Anabolic Metabolism

    Conversion of IMP to AMP and GMP in Purine Anabolic Metabolism from the firstclosed purine ring to ATP

    Nucleotide Side Chains, Oxidation Initiation and Metabolic Cycle Patterns

    Purine biosynthesis

    Inosine Mono Phosphate

    Urea Cycle Inherited Diseases, Arginine andInosine

    Wobble CodonThere should be an orange purine in the genetic code especially since Inosine is

    parent to ATP and GTP

    The Wobble Codons are Key Metabolic Pathway Switches

    The Necessity of the IMP Purine Nucleotide FamilyPurine ring wobble photonic diffraction Vectors

    Purine Metabolism Map Color Zones and Wobble Codons

    Arginine Inosine Wobble Diseases and the Citric Acid Cycl

    Consequences omitting Parent Purine from Genetic Code

    Conversion of IMP to AMP and GMP, IMP Parent Nucleotide, Belongs Genetic CodeFirst Purine Base and Closed Ring

    First Purine Catalytic Enzymes Inosine

    Fractal oxidation methyl group

    Genetic code nucleotides and missing purine parent

    Genetic code wrong omitted parent purine nucleotide base

    IMP begins oxidation with AMP with no Oxygens

    IMP building blocks purine synthesis

    IMP First Enzyme Parent Purine

    IMP first purine enzyme for purine synthesis

    IMP initiates urea cycle stopping amino acid synthesis

    IMP is clearly the parent purine and precursor of AMP and GMP

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    IMP Root Purine Nucleotide Molecular Structure

    IMPDH Inhibits RNA Synthesis

    Initiator IMP nucleotide synthesis initiator

    Inosine and side effects

    Inosine anticodon position 34 and metabolic pathway-switching node

    Inosinehas standard physical properties just like the other five nucleotide

    genetic codes

    Inosine key to decoding secrets of genetic script

    Inosine Metabolic Functional Space used Purine Synthesis and Ammonia Removal

    Inosine N34 stops amino acid and starts urea cycles removal toxic ammonia

    Inosine orange functional boundaries

    Inosine parent purine nucleotide

    Inosine parent purine and triple helix genetic primer

    Inosine Parent Purine Synthesis

    Inosine parent purine to adenosine and guanosine

    Inosine Purine Synthesis Central Internodal Connections

    Inosine wobble codes and functional areas degraded in purine metabolismInosine Wobble, Arginine, and Urea Related Diseases

    Nucleotides Compose Nucleic Acid Molecules

    Nucleotides Purine Metabolism and Wobble Codes

    Nucleotides, amino acids and Inosine wobble codes

    Nucleotides, Genetic Code, Amino Acids and Inosine Wobble

    Nucleotides, Purines, Inosine and the Missing Genetic Code

    Omitting the Parent Purine Nucleotide Negates Evolutionary Inheritance for A and

    G

    Orange Inosine is parent to red adenosine and green guanosine

    Oxygen and Inosine atomic molecular self-identity

    Parent purine structure always remains in the derivative molecular compound

    Purine anabolic and catabolic cycle start, stop and metabolic pathwayinterchanges

    Purine closed ring molecular atomic donors and IMP synthesis

    Purine Hierarchical Relationships Inosine Parent

    Inosine Starter of Organic Purine Synthesis

    AMP and GMP are made from IMP

    AMP and GMP are formed from IMP the parent purine

    Inosine Family

    Inosine and IMP

    amino acids coded by Inosine from the N34 position in the peptide chain

    Urea Cycle Inherited Diseases, Arginine and Inosine Wobble Codon

    The Wobble Codons are Key Metabolic Pathway SwitchesThe Logic of Nucleotide Molecular Inheritance

    Purine Nucleotide Hiearchical Root Molecular Structure

    Purine Ring Formation, IMP and InheritanceOmitting The Parent Purine Nucleotide Negates Evolutionary Inheritance for A and

    G

    Diseases from Missing Wobble Codon Amino Acids Arginine Serine Alanine

    Inosine Wobble Genetic Codes and Amino Acid Codons

    Current 5 Genetic Code Nucleotides Omitting Parent Purine IMP

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    ATP & IMP mRNA Switch TCA to Urea CyclesPurine Nucleotide Anabolic to Catabolic Cycle Switch A to I Deaminase

    Wobble Switch

    IMP Starts ATP Synthesis

    Inosine Parent Purine

    The Purine Hexagonal Closed Ring Molecular Structure as the Foundation of The

    Genetic CodeIMP is Parent Nucleotide Purine Molecular Structure of ATP

    IMP Parent Purine

    Inosine Family Genes, Enzymes, Proteins, and Wobble Amino Acids

    DNA Hydrolysis by DNA deoxyInosine ( 3.2.2.15) glycosidase

    Biosynthetic End Products from Alpha Ketoglutarate and Arginine

    Xanthine Enzyme Multi-Function Nucleotide Compounds

    Xanthine Oxidase Oxidation Mechanism and Uric Acid Eliminiation

    Omitting The Parent Purine Nucleotide Base IMP is The Second Fatal Flaw of theCurrent Genetic Code

    Natures Ingenious 3 bit Codon even Codes for bi-lateral Symmetry (i.e. left

    and

    right hands) Using the Dominant (5) and Recessive (3) NucleotideComplementary

    Pairing made Famous by Watson and Crick

    Each Purine-Pyrmidine Nucleotide Base Pairing (IMP-UMP, AMP-TMP, GMP-CMP) isthe X (width), Y (length) or Z (height) specification for making each of the 20

    Standard Protein Amino Acids

    Position Three is for the Z or Height Dimension and Contains three or

    tri-phosphate esters with three hydrogen bonds

    The Third Codon is Quite IMPortant Contrary to Conventional Genetic Code WisdomThe GMP-CMP Nucleotide Base Pair is the Third and Last Covalent Partnering of

    the Novagon DNA Triple Helix 6 Code Genetic Primer

    Position Two specifies the Y or length dimension and the Di-Phosphate Nucleotide

    The Second Genetic Code Nucleotide Base Pair is AMP Covalently Bonded to TMP

    with two hydrogen bonds

    Position One of the Codon specifies the X dimension and one mono phosphate

    The First Genetic Code Nucleotide Base Pair is IMP and UMP with one hydrogenbond

    IMP is the Evolutionary Parent Purine Nucleotide for ITP, GTP and ATP

    ATP had not been produced through phosphorylation before IMP began the PurineNucleotide Synthesis de novo Process

    ATP is The Universal Energy Molecular Structure which Powers 96% of all

    Metabolic Cycles

    Inosine Mono Phosphate (IMP) - Was Natures First Purine Nucleotide MolecularStructure

    Inosine Enzyme Nucleotide Compounds

    Inosine Thermodynamic Parameters for Binding Drugs to DNA

    Inosine Metal Binding and Ligand Stability Constants

    IMP & XMP Metal Binding CofactorsPurine Nucleotide Family Inheritance Tree

    Conversion of IMP to AMP and GMP is a Classic Parent Child InheritanceRelationship

    IMP parent purine base nucleotide of ATP & GTP

    Interconversion Purine Nucleotides by IMP Catalytic Starter ATP Synthesis

    The IMP Purine Parent with his XMP Partner

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    Purine Nulceotide Catabolic Metabolism

    Metabolism of Ammonia

    Eliminating Toxic Ammonia is a Survival Level Functions and Representation in

    the Genetic Code

    Purine Catabolic Degradation and Recycling

    Purine Catabolic Metabolism and Uric Acid Toxic Ammonia Elimination

    Purine Salvage and IMP Parent Purine Nucleotide Omitted in the Current GeneticCode

    Urea Cycle, Toxic Ammonia Removal and Xanthine Oxidase Enzyme

    Inosine Mono Phosphate

    Urea Cycle Inherited Diseases, Arginine and Inosine Wobble Codon

    There should be an orange purine in the genetic code especially since Inosine is

    parent to ATP and GTP

    The Wobble Codons are Key Metabolic Pathway Switches

    The Necessity of the IMP Purine Nucleotide FamilyPurine ring wobble photonic diffraction Vectors

    Purine Metabolism Map Color Zones and Wobble Codons

    Arginine Inosine Wobble Diseases and the Citric Acid Cycl

    Consequences omitting Parent Purine from Genetic Code

    Conversion of IMP to AMP and GMP, IMP Parent Nucleotide, Belongs Genetic CodeFirst Purine Base and Closed Ring

    First Purine Catalytic Enzymes Inosine

    Fractal oxidation methyl group

    Genetic code nucleotides and missing purine parent

    Genetic code wrong omitted parent purine nucleotide base

    IMP building blocks purine synthesis

    IMP First Enzyme Parent Purine

    IMP first purine enzyme for purine synthesis

    IMP initiates urea cycle stopping amino acid synthesis

    IMP is clearly the parent purine and precursor of AMP and GMP

    IMP Root Purine Nucleotide Molecular Structure

    IMPDH Inhibits RNA Synthesis

    Inosine and side effects

    Inosine anticodon position 34 and metabolic pathway-switching node

    Inosine has standard physical properties just like the other five nucleotide

    genetic codes

    Inosine key to decoding secrets of genetic script

    Inosine Metabolic Functional Space used Purine Synthesis and Ammonia Removal

    Inosine N34 stops amino acid and starts urea cycles removal toxic ammonia

    Inosine orange functional boundaries

    Inosine parent purine nucleotide

    Inosine parent purine and triple helix genetic primer

    Inosine Parent Purine Synthesis

    Inosine parent purine to adenosine and guanosine

    Inosine Purine Synthesis Central Internodal Connections

    Inosine Starter of Organic Purine Synthesis

    Inosine wobble codes and functional areas degraded in purine metabolism

    Inosine Wobble, Arginine, and Urea Related Diseases

    Purine Hierarchical Relationships Inosine Parent

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    Purine closed ring molecular atomic donors and IMP synthesisPurine anabolic and catabolic cycle start, stop and metabolic pathway

    interchanges

    Nucleotides Compose Nucleic Acid Molecules

    Parent purine structure always remains in the derivative molecular compound

    Orange Inosine is parent to red adenosine and green guanosine

    Omitting the Parent Purine Nucleotide Negates Evolutionary Inheritance for A andG

    Nucleotides, Purines, Inosine and the Missing Genetic Code

    Nucleotides, Genetic Code, Amino Acids and Inosine Wobble

    Nucleotides, amino acids and Inosine wobble codes

    Nucleotides Purine Metabolism and Wobble Codes

    Oxygen and Inosine atomic molecular self-identity

    Initiator IMP nucleotide synthesis initiator

    AMP and GMP are made from IMP

    IMP begins oxidation with AMP with no Oxygens

    AMP and GMP are formed from IMP the parent purinePurine Nucleotide Anabolic Metabolism

    Conversion of IMP to AMP and GMP in Purine Anabolic Metabolism from the firstclosed purine ring to ATP

    Nucleotide Side Chains, Oxidation Initiation and Metabolic Cycle Patterns

    Purine biosynthesis

    Purine Nulceotide Catabolic Metabolism

    Metabolism of Ammonia

    Eliminating Toxic Ammonia is a Survival Level Functions and Representation in

    the Genetic Code

    Purine Catabolic Degradation and Recycling

    Purine Catabolic Metabolism and Uric Acid Toxic Ammonia Elimination

    Purine Salvage and IMP Parent Purine Nucleotide Omitted in the Current GeneticCode

    Urea Cycle, Toxic Ammonia Removal and Xanthine Oxidase Enzyme

    5-phosphoribosylamineClosed Ring Purine Synthesis de novo

    IMP The First Purine Nucleotide "closed" ring Molecular StructureFirst Purine Nucleotide Closed Ring Structure and Purine Nucleotide Synthesis

    First Closed Purine Nucleotide Ring IMPThe First Closed Purine Nucleotide Molecule Paved The Way for DNA and RNA to

    Evolve

    IMP The First Purine Nucleotide "closed" ring Molecular StructureFirst Purine Nucleotide Closed Ring Structure and Purine Nucleotide Synthesis

    First Closed Purine Nucleotide Ring IMPThe First Closed Purine Nucleotide Molecule Paved The Way for DNA and RNA to

    Evolve

    Purine Nucleotide Family Inheritance Tree

    Conversion of IMP to AMP and GMP is a Classic Parent Child InheritanceRelationship

    IMP parent purine base nucleotide of ATP & GTP

    Interconversion Purine Nucleotides by IMP Catalytic Starter ATP Synthesis

    The IMP Purine Parent with his XMP Partner

    Inosine Mono Phosphate

    Urea Cycle Inherited Diseases, Arginine and Inosine Wobble Codon

    There should be an orange purine in the genetic code especially since Inosine is

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    parent to ATP and GTP

    The Wobble Codons are Key Metabolic Pathway Switches

    The Necessity of the IMP Purine Nucleotide FamilyPurine Metabolism Map Color Zones and Wobble Codons

    Purine ring wobble photonic diffraction Vectors

    Purine Hierarchical Relationships Inosine Parent

    Purine closed ring molecular atomic donors and IMP synthesisPurine anabolic and catabolic cycle start, stop and metabolic pathway

    interchanges

    Parent purine structure always remains in the derivative molecular compound

    Oxygen and Inosine atomic molecular self-identity

    Orange Inosine is parent to red adenosine and green guanosineOmitting the Parent Purine Nucleotide Negates Evolutionary Inheritance for A and

    G

    AMP and GMP are made from IMP

    Arginine Inosine Wobble Diseases and the Citric Acid Cycl

    Consequences omitting Parent Purine from Genetic Code

    Conversion of IMP to AMP and GMP, IMP Parent Nucleotide, Belongs Genetic Code

    First Purine Base and Closed RingFirst Purine Catalytic Enzymes Inosine

    Fractal oxidation methyl group

    Genetic code nucleotides and missing purine parent

    Genetic code wrong omitted parent purine nucleotide base

    IMP begins oxidation with AMP with no Oxygens

    IMP building blocks purine synthesis

    IMP First Enzyme Parent Purine

    IMP first purine enzyme for purine synthesis

    IMP initiates urea cycle stopping amino acid synthesis

    IMP is clearly the parent purine and precursor of AMP and GMP

    IMP Root Purine Nucleotide Molecular Structure

    IMPDH Inhibits RNA SynthesisInitiator IMP nucleotide synthesis initiator

    Inosine and side effects

    Inosine anticodon position 34 and metabolic pathway-switching node

    Inosine has standard physical properties just like the other five nucleotide

    genetic codes

    Inosine key to decoding secrets of genetic script

    Inosine Metabolic Functional Space used Purine Synthesis and Ammonia Removal

    Inosine N34 stops amino acid and starts urea cycles removal toxic ammonia

    Inosine orange functional boundaries

    Inosine parent purine nucleotide

    Inosine parent purine and triple helix genetic primer

    Inosine Parent Purine Synthesis

    Inosine parent purine to adenosine and guanosine

    Inosine Purine Synthesis Central Internodal Connections

    Inosine Starter of Organic Purine Synthesis

    Nucleotides, Purines, Inosine and the Missing Genetic Code

    Inosine wobble codes and functional areas degraded in purine metabolism

    Inosine Wobble, Arginine, and Urea Related Diseases

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    Nucleotides Compose Nucleic Acid Molecules

    Nucleotides Purine Metabolism and Wobble Codes

    Nucleotides, amino acids and Inosine wobble codes

    Nucleotides, Genetic Code, Amino Acids and Inosine Wobble

    AMP and GMP are formed from IMP the parent purine

    Inosine Family Genes, Enzymes, Proteins, and Wobble Amino Acids

    Inosine Enzyme Nucleotide Compounds

    Inosine Thermodynamic Parameters for Binding Drugs to DNA

    Inosine Metal Binding and Ligand Stability Constants

    IMP & XMP Metal Binding Cofactors

    DNA Hydrolysis by DNA deoxyInosine ( 3.2.2.15) glycosidase

    Biosynthetic End Products from Alpha Ketoglutarate and Arginine

    Xanthine Enzyme Multi-Function Nucleotide Compounds

    Xanthine Oxidase Oxidation Mechanism and Uric Acid Eliminiation

    Omitting The Parent Purine Nucleotide Base IMP is The Second Fatal Flaw of theCurrent Genetic Code

    IMP is the Evolutionary Parent Purine Nucleotide for ITP, GTP and ATP

    ATP did not Exist beforeIMP

    began the Purine Nucleotide Synthesis Process

    ATP is The Universal Energy Molecular Structure which Powers 96% of all

    Metabolic Cycles

    Inosine Mono Phosphate (IMP) - Was Natures First Purine Nucleotide MolecularStructure

    Each Purine-Pyrmidine Nucleotide Base Pairing (IMP-UMP, AMP-TMP, GMP-CMP) isthe

    X (width), Y (length) or Z (height) specification for making each of the 20

    Standard Protein Amino Acids

    Position Three is for the Z or Height Dimension and Contains three or

    tri-phosphate esters with three hydrogen bonds

    The Third Codon is Quite IMPortant Contrary to Conventional Genetic Code WisdomThe GMP-CMP Nucleotide Base Pair is the Third and Last Covalent Partnering of

    the Novagon DNA Triple Helix 6 Code Genetic PrimerPosition Two specifies the Y or length dimension and the Di-Phosphate Nucleotide

    The Second Genetic Code Nucleotide Base Pair is AMP Covalently Bonded to TMP

    with two hydrogen bonds

    Position One of the Codon specifies the X dimension and one mono phosphate

    The First Genetic Code Nucleotide Base Pair is IMP and UMP with one hydrogenbond

    Natures Ingenious 3 bit Codon even Codes for bi-lateral Symmetry (i.e. left

    and

    right hands) Using the Dominant (5) and Recessive (3) Nucleotide

    Complementary

    Pairing made Famous by Watson and Crick

    Pyrmidines

    Pyrmidine Nucleotides

    Pyrmidine Nucleotide Anabolic Metabolism

    Pyrmidine Nucleotide Catabolic Metabolism

    Interconversions and Degradation of Pyrmidine Nucleotides and XMP Ribonucleases

    Pyrmidine Metabolism, Thioredoxin Reduction and Nucleic Acid Polymerases

    Genetic Code Violates Inheritance Laws - Omits Parent Purine Nucleotide -

    Inosine Monophosphate (IMP) - Purine Synthesis De Novo " from scratch" -Purine and Pyrmidine "de novo" Metabolism Synthesized The First Evolutionary

    Nucleic Acids (RNA & DNA)

    "Closed Ring" allowed Photosynthetic Photons (electrons & protons) to be

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    captured by closed crystalline molecular structure

    IMP - First " Closed Purine Ring" Nucleotide made by NaturePurine Synthesis "de novo" -Most complex molecular structure synthesis in nature

    - 14 dufferent enzymatic intermediate structures involved

    Omits Parent Purine Nucleotide - Inosine Monophosphate (IMP)

    IMP Begins Purine Synthesis De Novo

    Purine Nucleotide Synthesis De Novo Makes Molecular Structures for DNA and RNAMolecules

    AMP and GMP are Formed From IMPNucleotides

    Thioester Phosphates

    Pentose Sugars

    Nitrogen Bases

    Structures of Nucleotides and Nucleosides

    ATP is the energy currency of metabolic reactions in cellular operations by

    cellular organelles i.e. mitochdondria, ribosomes

    Structures of Nucleotides and Nucleosides

    Scientist discovered five different nucleotides (Adenosine, Thymine,

    Guanosine,

    Cytosine and Uracil)Remember in this substitution of U for T, 2 hydrogen protons (H2+) are lost and

    one oxygen is gained

    The number 2 (thymine) and number 5 (uracil) molecular structures are not thesame, they are not equivalent they are not interchangeable parts

    The number 5 position or structure took the place of the number 2 position or

    structure

    The decision was made to substitute the fifth (5) =( Uracil = U) nucleotidefor

    the second nucleotide (Thymine = T)These 5 nucleotides represent 5 different positions in the 230 space crystalline

    lattice structure

    These 5 nucleotides represent 5 different crystalline molecular structures

    Nucleotides

    Adenosine

    Guanosine

    Inosine

    Xanthosine

    Purine Nucleosides are composed of four major molecular structures

    Inosine Purine Parent

    Purine Bases

    Pyrmidine Bases

    Nucleosides are made up of nitrogen bases

    Purine Nucleosides

    Pyrmidine Nucleosides

    Nucleotides are made up of nucleosidesPurine Nucleotide Bases

    IMPAMP

    GMP

    IMP FamilyThe entire concept of inheritance and evolution rests on "new generations" (i.e.

    GMP and AMP) of molecules starting with " a template" or organic molecular

    structure; since Inosine was the first purine to possess the critical "closed

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    ring" molecular structure and GMP and AMP are derived from IMP why wouldn't thefirst purine molecule be the first purine genetic code

    IMP (E.C. = 1.1.1.1.250 is the lowest purine enzyme classification number whichmeans it came before all other purine enzymes;

    Besides being the first enzyme, the first purine closed ring Inosine also has

    the perfect molecular structure to start an oxidation-reduction biochemical

    reaction between two chemical species i.e.Adenosine has no oxygen but only aNH2

    (ammonium side radical)

    ATP would never have evolved into it's present state and critical molecular

    function if the parent IMP did not develop the closed purine ring

    Omitting Inosine from the genetic code means eitherAdenosine or Guanosine istaking

    its legitimate place in the linear sequence of amino acids/codons;

    When A replaces I then the covalent pair lose their ability to be oxidized at a

    lower temperature or energy state; this makes the cell take valuable energy from

    other critical redox enzyme proteonic reactions

    If Guanosine is substituted into a specific atomic amino acid cell site thenthe

    molecule gets the O2 molecule for oxidation but also adds an NH2 or ammonium ion

    which causes an imbalance in the ammonia/urea cycle elemination process which is

    crucial for cellular heath; besides oxidation, ammonia is the dangerous threat

    to the brain master control center

    One has to assume in over 3.6 billion years that each nucleotide base code has

    only one amino acid associated with it; the genetic code is not degenerate; it

    is man's inability to use the more specific genetic information encoded in the

    atomic genetic primer which is atom specific; ie. there are 230 possible node or

    connecting points in the various three dimensional crystalline lattice

    structures which make up the "frame " of the gene; since 1 helical spiral has 10

    turns between purine base pairs then 230/10 = 23 chromosomes

    DNA is half protein (histones) and half dna ; it should be possible to "map" the

    exact location of the twenty three chromsomes over the three dimensional space

    the crystalline structure extends; we have calculated the maxium number of basepairs that each of six nodes can have for the purine/pyrmidine nucleotides

    If A and G, the children of I (IMP = GMP, AMP two different pathways); if the 2

    children are in the genetic code then their common parent, Inosine or IMP mustalso be in the genetic code primer

    Inosine is precursor IMP nucleotide to GMP and AMP or GTP and ATP ; two most

    IMPortant energy sources for internal (GMP = dna /rna transcription andtranslation processes); ATP external or extrons to invidivual cell; is a network

    charger = ATP

    These are the "wobble" nucleotide base pairings Inosine forms (I) - (U,C,A) and

    the three metabolic wobble "switched" anabolic and catabolic pathways

    Purine Catabolic, Decomposition and Recycling Cycle (salvaging "left over"

    purine bases, ribose sugar, and phosphodiester organic atoms for beginning IMPto ATP 9 step anabolic reaction

    Inosine and the wobble position - anti-codon branching point for beginning of

    catabolic purine process AMP + asparate = Inosine = IUG ; stop protein

    synthesis; begin recycling and new set up for different amino acid;

    Inosine can bond with Cystosine (first choice), Uracil (second choice) and take

    the place ofAdenosine (adenosine deaminase = mRNA editing) - violationwobble tRNA

    rule and central dogma linear model; numerous instances where both positive and

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    negative feedback processes regulate macromolecular anabolic and catabolic

    reactions

    Inosine only purine base to be able to bond to three different partners; maximum

    flexibility and IMProves purine pathways from 2 to 3; allows for feedbacksytems

    to develop; one needs three objects or processes to make a comparison and

    adjustmentThis is a metabolic map of purine metabolism showing the criticality of Inosine,

    IMP, Xanthine, Hypoxanthine and all the "orange" metabolic nodes

    Inosine Mono Phosphate - first purine "closed ring" hexagonal heterocyclic

    benzymatic molecular structure; fundamental nucleotide of genetic code protein

    primer

    IMP is the precursor purine parent to ATP and GTP

    IMP is first totally closed purine base ring

    Inosine Mono Phosphate Parent Purine

    IMP, precursor biochemical parent of AMP and GMP has the Perfect MolecularStructure and Optimal Number and Type of Purine Ring Side Atomic Molecular

    Structures

    There are two major metabolic cycles IMP or Inosine, hypoxanthine (base),xanthine (base), Inosine (nucleoside), Inosine monophosphate (nucleotide)

    IMP is made from: 1. three amino acids (glycine, aspartate, and gluamine), 2. atetrahydrofolate cofactor (10 Formyl Tetrahydrofolate), 3. a Carbon Dioxide

    (CO2), 4. PRPP (Phosphoribophosphate) - ribose sugar) with the IMPDH (inosinicacid -dehydrogenase enzyme E.C. = 1.1.1.1.250)

    IMP has only "one" oxygen on its side chain; AMP has no Oxygen Atoms in the

    Adenosine base Molecular Structure; since it has no oxygen's a self-bonding ofA-A

    has two NH2 side chains but no Oxgen; it has to wait for the pentose/ribose

    nucleoside sugar to connect to the adenosine base before it can becomemetabolically active

    If the Two Purine Nucleotide Molecular Strutures of the "Children" of InosineMono Phosphate (1.1.1.1.250) are in the Present Genetic Code Then the Parent

    Purine Nucleotide Molecular Structure must also be in that Same Genetic Code

    IMP is the first purine nucleotide to have a "closed ring structure"

    IMP is formed in a nine step synthesis from the following 6 different atomicmolecular donors

    Glutamine, Glycine, Aspartate, N10 Formyltetrahydrofolate, CO2, PRPP

    IMP, IDP, ITP

    IMP begins purine metabolism which terminates with the production of thetriphosphates i.e. ATP, GTP, ITP

    AMP Family

    AMP-S, AMP, ADP, ATP,

    ATP and GTP along with ITP are the purine nucleotides of the Genetic Primer

    GMP Family

    Purine Anabolic, Growth and Synthesis Cycle

    Purine Nucleotide Synthesis

    XMP, GMP, GDP,GTP

    Urea Cycle

    Arginine

    Purine Nucleotides are composed of three major molecular structures with 3 or 4

    isotopic alleles

    These are the 12 nucleosides (6) and nucleotides (6) which make up the genetic

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    codes which specify and coordinate protein synthesis

    Purine Anabolic and Catabolic Biomolecular Metabolic Redox Reactions

    Methyl- Inosine

    Thio-Inosine

    Hypoxanthine

    Xanthine Oxidase

    Inosine and IMP = Hydrogenase = 1.1.1.1.250Glutamine

    Glycine

    Aspartate 1

    N10 Formyl

    Tetrahydrofolate 1

    CO2

    Aspartate 2

    N10 Formyl

    Tetrahydrofolate 2

    PRPP

    Purine Nucleotide Closed Ring

    IMP

    XMPGMP

    GDP

    GTP

    AMP-S

    AMP

    ADP

    ATP

    DNA

    Nucleic Acid

    ATPase

    Water

    Ammonia

    AMP

    IMPXMP

    Xanthine Oxidase

    Uric Acid

    Urate

    Ammonia

    Eliminated

    GMP

    RNA

    Nucleic Acid

    RNA Genetic Code

    A2 - U1

    G2- C2

    ATPNucleotide

    UTP

    Nucleotide

    Adenosine Base

    Uracil BaseDNA Genetic Code

    A1 - T1

    G1- C1

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    ATP

    Nucleotide

    TTP

    Nucleotide

    Adenosine Base

    Thymine Base

    A purine nucleotide with no side groups but hydrogens has 9 different and uniquebonding positions; even though a 6 and 5 sided (hexagon + pentagon) are fused

    together, they share a common carbon bond thus substracting 2 sides, thus 11 -2

    = 9 thus in the purine electrochemical circuit their are nine different

    potential oxidation states each corresponding to sulfur's

    (+6,+5,+4,+3,+2,+1,0,-1,-2 ); it is interesting to note that the maximum

    positively c

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    CBCInnovis CBCInnovis Phone: 8002163463875 GRENTREE ROAD Fax:8 PARKWAY CENTERPITTSBURGH PA 15220

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    APPLICANT

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    1 FACT ACT:CURRENT FILE ADDRESS DOES NOT MATCH INPUT ADDRESS(ES) TRU01

    1 FACT ACT:THE NUMBER OF INQUIRIES ON THE CONSUMERS CREDIT FILE HAS ADVERSELY TRU01

    AFFECTED THE CREDIT SCORE WITH NO DEROGATORY INFORMATION FOUND ON THE FILE

    PUBLIC RECORD INFORMATION

    NO PUBLIC RECORDS FOUND

    CREDIT HISTORY

    PRESENT STATUS HISTORICAL STATUS

    E CREDITOR LIMIT OR NO

    C ACCOUNT NO LAST HIGHEST BALANCE AMOUNT TERMS TYPE/RATE MOS 30 60 9

    O RPTD ACT OPND CREDIT OWING PAST PYMT AMT VENDOR HIST DAYS DAYS DAA DUE REV

    1 GMAC MORT. 09/07 09/07 04/05 110800 108145 0 360 M 1194 MTG 01 29 2 1

    #685451145 REAL ESTATE 111111111111

    3451 HAMMOND AVE TRU 01 322111111111

    WATERLOO IA 50704 09/06 3 08/06 2

    (800) 7664622 09/06 3

    1 CITI 10/07 09/07 03/88 18800 1721 0 V 74 REV 01 48 0 0

    #542418070850 CREDIT CARD 111111111111

    POB 6241 TRU 01 111111111111

    SIOUX FALLS SD 57117

    (800) 8430777

    1 RMI/MCSI 07/04 05/04 100 100 100 100E OPN 09 0

    #80223

    3348 RIDGE RD TRU 01

    LANSING IL 60438

    (708) 8958522 COLLECTION CITY OF AURORA

    1 RMI/MCSI 10/04 07/04 100 100 100 100E OPN 09 0

    #02969

    3348 RIDGE RD TRU 01

    LANSING IL 60438

    (708) 8958522 COLLECTION CITY OF AURORA

    1 RMI/MCSI 07/04 05/04 100 100 100 100E OPN 09 0

    #80224

    3348 RIDGE RD TRU 01

    LANSING IL 60438(708) 8958522 COLLECTION CITY OF AURORA

    1 NCO FIN/22 11/07 07/05 101 91 91 91E OPN 09 0

    #7395288

    POB 41448 TRU 01

    PHILADELPHIA PA 19101

    (800) 2736816 COLLECTION NCO ASSIGNEE OF VERIZON WS

    PAGE 2

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  • 8/6/2019 Berger Visio Nee Ring Concept Images (2)

    50/82

    APPLICANT

    NAME: JOHN BERGER

    S.S.N : 430960430 Date Received: 11/15/07

    CREDIT HISTORY

    PRESENT STATUS HISTORICAL STATUS

    E CREDITOR LIMIT OR NOC ACCOUNT NO LAST HIGHEST BALANCE AMOUNT TERMS TYPE/RATE MOS 30 60 9

    O RPTD ACT OPND CREDIT OWING PAST PYMT AMT VENDOR HIST DAYS DAYS DA

    A DUE REV

    1 HARRIS 07/07 08/06 75 75 75 75E OPN 09 0

    #30844741

    600 W JACKSON TRU 01

    CHICAGO IL 60661

    (312) 2512300 COLLECTION 04 DUPAGE COUNTY CIRCUIT COURT

    1 CB USA INC 01/07 07/04 60 60 60 60E OPN 09 0

    #826724

    5252 HOHMAN TRU 01

    HAMMOND IN 46325

    (219) 9321000 COLLECTION MEDICAL

    1 NICOR GAS 10/07 09/07 09/89 0 34 0 34E OPN 01 48 0 0

    #312792 111111111111

    1844 FERRY ROAD TRU 01 1111XXX1111

    NAPERVILLE IL 60563

    (630) 3059500 UTILITY COMPANY

    1 CITISHELL 06/07 10/06 11/82 800 23 0 V 10 REV 01 48 0 0

    #48143 CREDIT CARD 111111111111

    CREDIT BUREAU DISP TRU 01 111111111111

    HAGERSTOWN MD 21747

    (800) 9505114

    1 CAPSTEAD INC 02/99 02/99 11/92 133900 0 0 360 M 1275 MTG 01 0 0 0

    #652691601 REAL ESTATE8401 N CENTRAL EXP TRU 01

    DALLAS TX 75225 TRANSFERRED

    2 DISCOVER FIN 12/03 11/03 11/94 5500 0 0 REV 01 44 0 0

    #601100745452 CREDIT CARD 111111111111

    POB 15316 TRU 01 111111111111

    WILMINGTON DE 19850 CLOSED CONSUMER

    1 GMAC MORT. 04/05 04/05 11/92 133900 0 0 360 M 1411 MTG 01 48 2 0

    #306342926 REAL ESTATE 111111111211

    3451 HAMMOND AVE TRU 01 111111111111

    WATERLOO IA 50704 07/04 2

    CLOSED(800) 7664622

    1 TNBTARGET 10/07 01/07 07/04 300 0 0 REV 01 39 0 0

    #29741 CHARGE ACCOUNT 111111111111

    3701 WAYZATA BLVD TRU 01 111111111111

    MINNEAPOLIS MN 55416 CLOSED

    1 VERIZON 07/05 04/03 01/98 101 0 0 OPN 09 0

    #630531412

    NATINAL RECOVERY D TRU 01

    FOLSOM CA 95630

    (800) 8521922 PURCHASED BY ANOTHER LENDER UTILITY COMPANY

    PAGE 3

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IZON&acctno=630531412&phoneno=(800)%20852-1922&balance=0&designator=1&address1=%3Cbr%3ENATINAL%20RECOVERY%20D&address2=&csz=%3Cbr%3EFOLSOM%20CA%2095630&rptd=07/05&lastact=04/03&open=01/98&limit=101&pastdue=0&terms=&estpay=&remarks=PURCHASED%20BY%20ANOTHER%20LENDER%20UTILITY%20COMPANY&monthpay=&accttype=OPN&mop=09&derog=*&months=0&30day=&60day=&90day=&vendors=TRU&loantype=&p1date=&p1rate=&p2date=&p2rate=&p3date=&p3rate=&ddate=&drate=&pprofile=&keydataid=1TRU0100107https://www.creditbureaureports.com/servlet/Presenter?command=statusService&type=advancedUpdate&request_id=API_80103533934_430960430_IN&name=VERIZON&acctno=630531412&phoneno=(800)%20852-1922&balance=0&designator=1&address1=%3Cbr%3ENATINAL%20RECOVERY%20D&address2=&csz=%3Cbr%3EFOLSOM%20CA%2095630&rptd=07/05&lastact=04/03&open=01/98&limit=101&pastdue=0&terms=&estpay=&remarks=PURCHASED%20BY%20ANOTHER%20LENDER%20UTILITY%20COMPANY&monthpay=&accttype=OPN&mop=09&derog=*&months=0&30day=&60day=&90day=&vendors=TRU&loantype=&p1date=&p1rate=&p2date=&p2rate=&p3date=&p3rate=&ddate=&drate=&pprofile=&keydataid=1TRU0100107https://www.creditbureaureports.com/servlet/Presenter?command=statusService&type=advancedUpdate&request_id=API_80103533934_430960430_IN&name=TNB-TARGET&acctno=29741&phoneno=&balance=0&designator=1&address1=%3Cbr%3E3701%20WAYZATA%20BLVD&address2=&csz=%3Cbr%3EMINNEAPOLIS%20MN%2055416&rptd=10/07&lastact=01/07&open=07/04&limit=300&pastdue=0&terms=&estpay=&remarks=CLOSED&monthpay=&accttype=REV&mop=01&derog=&months=39&30day=0/&60day=0/&90day=0&vendors=TRU&loantype=CHARGE%20ACCOUNT&p1date=&p1rate=&p2date=&p2rate=&p3date=&p3rate=&ddate=&drate=&pprofile=111111111111111111111111111&keydataid=1TRU0100108https://www.creditbureaureports.com/servlet/Presenter?command=statusService&type=advancedUpdate&request_id=API_80103533934_430960430_IN&name=GMAC%20MORT%20&acctno=306342926&phoneno=(800)%20766-4622&balance=0&designator=1&address1=%3Cbr%3E3451%20HAMMOND%20AVE&address2=&csz=%3Cbr%3EWATERLOO%20IA%2050704&rptd=04/05&lastact=04/05&open=11/92&limit=133900&pastdue=0&terms=360%20M%201411&estpay=&remarks=CLOSED&monthpay=&accttype=MTG&mop=01&derog=*&months=48&30day=2/&60day=0/&90day=0&vendors=TRU&loantype=REAL%20ESTATE&p1date=&p1rate=&p2date=&p2rate=&p3date=&p3rate=&ddate=07/04&drate=2&pprofile=111111111211111111111111111&keydataid=1TRU0100205https://www.creditbureaureports.com/servlet/Presenter?command=statusService&type=advancedUpdate&request_id=API_80103533934_430960430_IN&name=DISCOVER%20FIN&acctno=601100745452&phoneno=&balance=0&designator=2&address1=%3Cbr%3EPOB%2015316&address2=&csz=%3Cbr%3EWILMINGTON%20DE%2019850&rptd=12/03&lastact=11/03&open=11/94&limit=5500&pastdue=0&terms=&estpay=&remarks=CLOSED%20-%20CONSUMER&monthpay=&accttype=REV&mop=01&derog=&months=44&30day=0/&60day=0/&90day=0&vendors=TRU&loantype=CREDIT%20CARD&p1date=&p1rate=&p2date=&p2rate=&p3date=&p3rate=&ddate=&drate=&pprofile=111111111111111111111111111&keydataid=1TRU0100206https://www.creditbureaureports.com/servlet/Presenter?command=statusService&type=advancedUpdate&request_id=API_80103533934_430960430_IN&name=CAPSTEAD%20INC&acctno=652691601&phoneno=&balance=0&designator=1&address1=%3Cbr%3E8401%20N%20CENTRAL%20EXP&address2=&csz=%3Cbr%3EDALLAS%20TX%2075225&rptd=02/99&lastact=02/99&open=11/92&limit=133900&pastdue=0&terms=360%20M%201275&estpay=&remarks=TRANSFERRED&monthpay=&accttype=MTG&mop=01&derog=&months=0&30day=0/&60day=0/&90day=0&vendors=TRU&loantype=REAL%20ESTATE&p1date=&p1rate=&p2date=&p2rate=&p3date=&p3rate=&ddate=&drate=&pprofile=&keydataid=1TRU0100207https://www.creditbureaureports.com/servlet/Presenter?command=statusService&type=advancedUpdate&request_id=API_80103533934_430960430_IN&name=CITI-SHELL&acctno=48143&phoneno=(800)%20950-5114&balance=23&designator=1&address1=%3Cbr%3ECREDIT%20BUREAU%20DISP&address2=&csz=%3Cbr%3EHAGERSTOWN%20MD%2021747&rptd=06/07&lastact=10/06&open=11/82&limit=800&pastdue=0&terms=V%2010&estpay=&remarks=&monthpay=10&accttype=REV&mop=01&derog=&months=48&30day=0/&60day=0/&90day=0&vendors=TRU&loantype=CREDIT%20CARD&p1date=&p1rate=&p2date=&p2rate=&p3date=&p3rate=&ddate=&drate=&pprofile=111111111111111111111111111&keydataid=1TRU0100204https://www.creditbureaureports.com/servlet/Presenter?command=statusService&type=advancedUpdate&request_id=API_80103533934_430960430_IN&name=NICOR%20GAS&acctno=312792&phoneno=(630)%20305-9500&balance=34&designator=1&address1=%3Cbr%3E1844%20FERRY%20ROAD&address2=&csz=%3Cbr%3ENAPERVILLE%20IL%2060563&rptd=10/07&lastact=09/07&open=09/89&limit=0&pastdue=0&terms=&estpay=E&remarks=UTILITY%20COMPANY&monthpay=34&accttype=OPN&mop=01&derog=&months=48&30day=0/&60day=0/&90day=0&vendors=TRU&loantype=&p1date=&p1rate=&p2date=&p2rate=&p3date=&p3rate=&ddate=&drate=&pprofile=1111111111111111XXX11111111&keydataid=1TRU0100201https://www.creditbureaureports.com/servlet/Presenter?command=statusService&type=advancedUpdate&request_id=API_80103533934_430960430_IN&name=CB%20USA%20INC&acctno=826724&phoneno=(219)%20932-1000&balance=60&designator=1&address1=%3Cbr%3E5252%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    APPLICANT

    NAME: JOHN BERGER

    S.S.N : 430960430 Date Received: 11/15/07

    INQUIRIES

    DATE ECOA KOB MEMBERNO

    11/15/07 1 Z 00287010 CBCINNOVIS TRU01

    02/16/07 1 Y 06141825 TRUELOGIC FI TRU01

    11/29/06 1 Y 01345541 HARRIS AND H TRU01

    ADDITIONAL INFORMATION

    1 MISCELLANEOUS CBC99

    A SCORE DISCLOSURE LETTER WAS MAILED TO THE APPLICANT ON 11/15/2007.

    THE REPOSITORIES ACCESSED AND PRINTED WERE: TRU

    CONSUMER REFERRAL INFORMATION

    TRANSUNION TRU06CH

    WWW.TRANSUNION.COM/DIRECTP.O. BOX 1000

    CHESTER, PA 19022

    8008884213

    ECOA Key: 1=Borrower, 2=Joint, 3=Authorized User, 4=Joint, 5=CoMaker, 7=Maker, 8=CoBorrower, 9=Terminated, 0=Undesignated

    DISCLOSURE

    NATIONAL CREDIT REPOSITORY (IES) ACCESSED FOR THIS CREDIT REPORT:TRANS UNION

    Reporting bureau certifies with contractual requirements governing check of public records as well as credit information.

    END OF REPORT

    PAGE 4

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    CBCInnovis CBCInnovis Phone:875 GRENTREE ROAD Fax:8 PARKWAY CENTERPITTSBURGH PA 15220

    SCORE DISCLOSURE

    PREPARED FOR: DATE RECEIVED:11/15/07KEMPER MORTGAGE INC475 N MARTINGALE RD #820SCHAUMBURG IL 60173 COMPUTER ID #: DATE COMPLETED:11/15/07

    413531917480700ATTENTION: LENDER CASE #:

    KMPRILSZ REPORT TYPE:INDIVIDUAL XXXXXXXXX

    APPLICANT

    INPUT INFORMATION:

    NAME:JOHN BERGER

    7 S 412 CREEK DR DEPENDENTS:0NAPERVILLE IL 60540

    EMPLOYMENT INFORMATION

    "NOTICE TO THE HOME LOAN APPLICANT"

    In connection with your application for a home loan, the lender must disclose to you the score that a consumer reporting agency distributed to users and the lender used

    in connection with your home loan, and the key factors affecting your credit scores.

    The credit score is a computer generated summary calculated at the time of the request and based on information that a consumer reporting agency or lender has on file.

    The scores are based on data about your credit history and payment patterns. Credit scores are important because they are used to assist the lender in determining whether

    you will obtain a loan. They may also be used to determine what interest rate you may be offered on the mortgage. Credit scores can change over time, depending on your

    conduct, how your credit history and payment patterns change, and how credit scoring technologies change.

    Because the score is based on information in your credit history, it is very important that you review the creditrelated information that is being furnished to make sure it is

    accurate. Credit records may vary from one company to another.

    If you have questions about your credit score or the credit information that is furnished to you, contact the consumer reporting agency at the address and telephone number

    provided with this notice, or contact the lender, if the lender developed or generated the credit score. The consumer reporting agency plays no part in the decision to take

    any action on the loan application and is unable to provide you with specific reasons for the decision on a loan application.

    If you have questions concerning the terms of the loan, contact the lender.

    Acknowledgement

    I have received a Notice to The Home