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    Pressure Ulcers

    Definition

    Pressure Ulcers are localized areas of tissue

    necrosis that tend to occur when soft tissue is

    compressed between a bony prominence and

    an external surface for a prolonged period.

    These lesions are also called bedsores,

    decubitus ulcers and pressure sores

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    Pressure Ulcers

    Epidemiology

    1-3 million Americans are affected

    Health care expenditures: $ 5 billion/year

    More than 17,000 lawsuits related to pressureUlcers are filed annually

    1 in 4 persons in the USA who died in 1987had a dermal ulcer

    Pressure Ulcers develop primarily in elderlypatients

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    Pressure Ulcers

    Epidemiology

    Setting

    Hospital 60%

    Nursing homes 18%

    Home 18%

    1/3 of patients undergoing surgery for hip fracturedevelop a pressure ulcer

    The longer the patient stays in a nursing home, the

    greater the likelihood of developing a pressure ulcer

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    THERMODYNAMICS, METABOLISM

    AND PRESSURE

    Thermodynamic factors - skin/surfaceinterface

    As temperature increases, skin becomes more

    metabolically active and 02 demands increase With increased pressure, metabolic demands

    not able to be met and skin becomes hypoxic

    Hypoxic skin more susceptible to breakdown

    Adding frictionand shearto already fragileskin is perfect storm

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    THE 4 FORCES

    1. Pressure: Force applied to soft tissue between

    hard surface and bony prominence. When skin

    and the underlying tissues are trapped between

    bone and a surface such as a wheelchair or bed,blood flow is restricted. This deprives tissue of

    oxygen and other nutrients -> tissue death.

    2. Friction: Resistance of one body sliding or rollingover another. Making skin more susceptible to

    pressure sores.

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    PRESSURE AND FRICTION

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    3. Shear: This occurs when skin moves in one

    direction, and the underlying bone moves in

    another. Sliding down in a bed or chair orraising the head of bed more than 30 degrees

    is especially likely to cause shearing, which

    stretches and tears cell walls and tiny bloodvessels. Especially affected are areas such as

    tailbone where skin is already thin and fragile.

    4. Strain: Tissue deformation in response topressure

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    Pathophysiology

    Prolong weight bearing and mechanical shear

    forces act on areas of soft tissue overlying

    bony prominence > when this pressure

    exceeds normal capillary perfusion pressure(32 mm Hg) > occlusion & tearing of small

    blood vessels > reduced tissue perfusion >

    ischaemic necrosis > Pressure sore.

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    Risk factors

    Age.Older adults tend to have thinner skin,

    making them more susceptible to damagefrom minor pressure. They have less natural

    cushioning over their bones. And poor

    nutrition, delays wound healing.

    Lack of pain perception.Spinal cord

    injuries and some diseases cause a loss ofsensation > bedsore is forming.

    http://www.mayoclinic.com/health/bedsores/DS00570/DSECTION=risk-factorshttp://www.mayoclinic.com/health/bedsores/DS00570/DSECTION=risk-factors
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    Natural thinness or weight loss.Muscle atrophy andwasting are common in people living with paralysis. If

    you lose fat and muscle there is no cushion over yourbones.

    Malnutrition. Pressure sores develops if you have apoor diet, especially one deficient in protein, zinc andvitamin C.

    Urinary or fecal incontinence.Problems with bladdercontrol can greatly increase risk of pressure soresbecause skin stays moist, making it more likely tobreak down. And bacteria from fecal matter not only

    can cause serious local infections but also can lead tolife-threatening systemic complications such as sepsis,gangrene and, rarely, necrotizing fasciitis, a severeand rapidly spreading infection.

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    Other medical conditions. diabetes and

    vascular diseaseaffect circulation > tissue

    damage. Smoking.Smokers tend to develop more

    severe wounds and heal more slowly, mainly

    because nicotine impairs circulation andreduces the amount of oxygen in blood.

    Decreased mental awareness.People whose

    mental awareness is lessened by disease,

    trauma or medications are often less able to

    take the actions needed to prevent or care for

    pressure sores.

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    Risk Factors

    Spinal cord injuries

    Traumatic brain injury

    Neuromuscular disorders Immobility

    Malnutrition

    Fecal and urinaryincontinence

    Altered level ofconsciousness

    Chronic systemic illness

    Fractures

    Aging skin decreased epidermal

    turnover

    dermoepidermaljunction flattens

    fewer blood vessels Decreased pain

    perception

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    Pressure ulcerscommonly occur

    over the :

    Sacrum

    Greatertrochanter

    Ischial tuberosity

    Malleolus

    Heel Fibular head

    Scapula

    Site

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    While on a wheelchair a pressure sore develop

    on:

    - tailbone or buttocks

    - shoulder blades and spine

    - The backs of arms and legs where they

    rest against the chair

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    Stages/ classification

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    Classification Stage I

    1. 1. most superficial,

    2. non blanchableredness,does not subside

    after pressure is relieved.

    3. The skin may be hotter or cooler than normal

    4. have an odd texture, or

    5. perhaps be painful to the patient.

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    STAGE I

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    Stage IIis damage to the epidermisextending

    into, but no deeper than, the dermis. In this

    stage, the ulcer may be referred to as a blisteror abrasion.

    The ulcer is superficial and manifest clinically

    as an abrasion, blister or shallow crater

    http://en.wikipedia.org/wiki/Epidermis_(skin)http://en.wikipedia.org/wiki/Dermishttp://en.wikipedia.org/wiki/Blisterhttp://en.wikipedia.org/wiki/Abrasion_(medical)http://en.wikipedia.org/wiki/Abrasion_(medical)http://en.wikipedia.org/wiki/Blisterhttp://en.wikipedia.org/wiki/Dermishttp://en.wikipedia.org/wiki/Epidermis_(skin)
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    STAGE II

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    STAGE II

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    Stage IIIinvolves the full thickness of the skinand may extend into the subcutaneous tissue

    layer. This layer has a relatively poor bloodsupply and can be difficult to heal.

    The ulcer manifests clinically as a deep crater

    with or without undermining of adjacent

    tissue

    http://en.wikipedia.org/wiki/Subcutaneous_tissuehttp://en.wikipedia.org/wiki/Subcutaneous_tissue
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    STAGE III

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    STAGE III

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    Stage IVis the deepest, extending into themuscle, tendonor even bone.

    Full thickness tissue loss with exposed bone,

    tendon or muscle. Slough or eschar may bepresent on some parts of the wound bed.Often include undermining and/or tunneling

    Depth varies according to anatomic location Exposed bone/tendon usually directly visible

    and/or palpable

    http://en.wikipedia.org/wiki/Musclehttp://en.wikipedia.org/wiki/Tendonhttp://en.wikipedia.org/wiki/Bonehttp://en.wikipedia.org/wiki/Bonehttp://en.wikipedia.org/wiki/Tendonhttp://en.wikipedia.org/wiki/Muscle
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    STAGE IV

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    B

    A

    Stage 4

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    STAGE IV

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    Complications

    Cellulitis.This causes pain, redness and swelling, all of

    which can be severe. Cellulitis can also lead to life-

    threatening complications, including sepsis and

    meningitis.

    Bone and joint infections.These develop when the

    infection from a bedsore burrows deep into joints and

    bones. Joint infections (septic or infectious arthritis) can

    damage cartilage and tissue, whereas bone infections(osteomyelitis) may reduce the function of joints and

    limbs.

    http://www.mayoclinic.com/health/bedsores/DS00570/DSECTION=complicationshttp://www.mayoclinic.com/health/bedsores/DS00570/DSECTION=complications
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    Sepsis.It occurs when bacteria enters

    bloodstream through the broken skin and

    spreads throughout the body a rapidly

    progressing, life-threatening condition thatcan cause shock and organ failure.

    Cancer.This is usually an aggressive carcinoma

    affecting the skin's squamous cells.

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    Tests and diagnosis

    Bedsores are usually unmistakable, even in

    the initial stages, but doctor is likely to order

    blood tests to check nutritional status and

    overall health. Depending on thecircumstances, there may have other tests.

    Wound swabC/S

    Incision biopsyif malignancy is suspected.

    http://www.mayoclinic.com/health/bedsores/DS00570/DSECTION=tests-and-diagnosishttp://www.mayoclinic.com/health/bedsores/DS00570/DSECTION=tests-and-diagnosis
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    Treatments and drugs

    Treating bedsores is challenging. Openwounds are slow to heal, and because skinand other tissues have already been

    damaged or destroyed, healing is neverperfect.

    Requires a multidisciplinary approach

    nurses, physician, social worker, physicaltherapist, urologist or gastroenterologist, aneurosurgeon, orthopedic surgeon andplastic surgeon.

    http://www.mayoclinic.com/health/bedsores/DS00570/DSECTION=treatments-and-drugshttp://www.mayoclinic.com/health/bedsores/DS00570/DSECTION=treatments-and-drugs
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    TREATMENT OBJECTIVES Identification of problem

    Debridement of necrotic tissue

    Moist wound care without maceration

    Control of infection/bioburden

    Management of pain

    Pressure redistribution/Offloading

    Choice of wound care products is individualpreference as long as above objectives met.

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    A) Conservative treatment

    Although it may take some time, most stage I

    and stage II sores will heal within weeks with

    conservative measures. But stage III and stageIV wounds, which are less likely to resolve on

    their own, may require surgery.

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    1. Changing positions often.Carefully follow the

    schedule for turning and repositioning approximately

    every 15 minutes if in a wheelchair and at least once

    every two hours when in bed. If unable to change

    position on own, a family member or other caregiver

    must be able to help.

    2. Using support surfaces.These are special cushions,

    pads, mattresses and beds that relieve pressure on anexisting sore and help protect vulnerable areas from

    further breakdown.

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    PRESSURE REDISTRIBUTION

    http://www.nationalwound.com/images/products/group1/group1_nwc400new.jpg
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    3. Cleaning.It's essential to keep woundsclean to prevent infection. A stage I woundcan be gently washed with water and mildsoap, but open sores should be cleaned with a

    saltwater (saline) solution each time thedressing is changed. Avoid antiseptics such ashydrogen peroxide and iodine, which candamage sensitive tissue and delay healing.

    4. Controlling incontinenceas far as possibleis crucial to helping sores

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    5. Removal of damaged tissue (debridement).Toheal properly, wounds need to be free of damaged,

    dead or infected tissue. This can be accomplished inseveral ways -

    a. Autolytic debridementis autolysiswith the body'sown enzymes.

    b. Biological debridement, or maggot debridementtherapy,

    c. Chemical debridement, or enzymatic debridement

    d. Mechanical debridement

    e. Sharp debridementis the removal of necrotic tissuewith a scalpel or similar instrument.

    f. Surgical debridement

    g. Ultrasound-assisted wound therapy

    http://en.wikipedia.org/wiki/Autolysishttp://en.wikipedia.org/wiki/Maggot_therapyhttp://en.wikipedia.org/wiki/Maggot_therapyhttp://en.wikipedia.org/wiki/Maggot_therapyhttp://en.wikipedia.org/wiki/Maggot_therapyhttp://en.wikipedia.org/wiki/Autolysis
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    6. Dressings.

    7. Oral antibiotics.

    8. Healthy diet.

    9. Muscle spasm relief

    10. Educating the caregiver

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    B) Surgical repairby - tissue flap, free flap,

    Negative Pressure Wound Therapy

    C) Other treatment options

    Researchers are searching for more effective

    bedsore treatments. Under investigation are

    hyperbaric oxygen, electrotherapyand thetopical use of human growth factors.

    http://en.wikipedia.org/w/index.php?title=Tissue_flap&action=edit&redlink=1http://en.wikipedia.org/wiki/Free_flaphttp://en.wikipedia.org/wiki/Negative_Pressure_Wound_Therapyhttp://en.wikipedia.org/wiki/Negative_Pressure_Wound_Therapyhttp://en.wikipedia.org/wiki/Free_flaphttp://en.wikipedia.org/w/index.php?title=Tissue_flap&action=edit&redlink=1
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    Prevention

    Bedsores are easier to prevent than to treat,

    but that doesn't mean the process is easy or

    uncomplicated. Although wounds can develop

    in spite of the most scrupulous care, it'spossible to prevent them in many cases.

    http://www.mayoclinic.com/health/bedsores/DS00570/DSECTION=preventionhttp://www.mayoclinic.com/health/bedsores/DS00570/DSECTION=prevention
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    Position changesChanging position frequently and consistentlyis crucial to preventing bedsores. Expertsadvise shifting position about every 15

    minutes that you're in a wheelchair and atleast once every two hours, even during thenight, if you spend most of your time in bed.

    Skin inspectionDaily skin inspections for pressure sores are anintegral part of prevention.

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    Nutrition

    A healthy diet is important in preventing skin

    breakdown and in aiding wound healing

    Lifestyle changes

    -Quitting smoking,

    - Exercise - Daily exercise improves

    circulation