Advanced Cardiac Life Support
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Transcript of Advanced Cardiac Life Support
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ADVANCED CARDIAC LIFE SUPPORTBY
ANITA.F.LOPESMSN,BSN,RN.
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ACLS Systematic approach to assessment and
management of cardiopulmonary emergencies
Continuation of Basic Life Support Resuscitation efforts aimed at restoring
spontaneous circulation and retaining intact neurologic function
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ABCD
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THE AAA’S Assess the patient
Establish unresponsivenessCheck pulse, respiration
Activate EMSCall for help
AEDGet an AED (automated external
defibrillator)
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CIRCULATION Check for a pulse Start CPR
30 compressions/ 2 respirations
Compressions more important than respirations!
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AIRWAY Open the airway
Head tilt-chin lift Jaw thrust
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BREATHING Look, Listen and Feel
Give 2 rescue breaths
Watch for appropriate chest rise and fall
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DEFIBRILLATION Know your AED
Universal steps:1. Power ON2. Attach electrode pads3. Analyze the rhythm4. Shock (if advised)
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DEFIBRILLATION Most frequent initial rhythm in
witnessed sudden cardiac arrest is ventricular fibrillation (VF) or pulseless ventricular tachycardia (VT) which rapidly deteriorates into VF
The only effective treatment for VF is electrical defibrillation
VF rapidly converts to asystole if not treated
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EARLY DEFIBRILLATION = INCREASED SURVIVAL
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SECONDARY SURVEY (ACLS) Airway Breathing Circulation Differential Diagnosis
Assess and manage at each step before moving on!
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AIRWAY Maintain airway patency
Head tilt-chin lift/jaw thrustOro- or nasopharyngeal airway
Advanced airway managementETTCombitubeLMA
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COMBITUBE AND LMA
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BREATHING Assess adequacy of oxygenation and
ventilation Provide supplemental oxygen Confirm proper airway placement Secure tube
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CIRCULATION Assess/monitor cardiac rhythm Establish IV or IO access Give medications as appropriate for
rhythm and BP Fluid resuscitation Minimize interruption of compressions
to maximize survival.
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DIFFERENTIAL DIAGNOSIS Look for and treat any reversible cause
of arrest
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THE H’S AND T’S Hypovolemia Hypoxia Hydrogen ion
(acidosis) Hyper-/
hypokalemia Hypothermia
Toxins Tamponade Tension
pneumothorax Thrombosis
(coronary or pulmonary)
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BASIC RHYTHM ANALYSIS
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BASIC RHYTHM ANALYSIS Rate – too fast or too slow? Rhythm – regular or irregular? Is there a normal looking QRS? Is it
wide or narrow? Are P waves present? What is the relationship of the P waves
to the QRS complex?
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RHYTHM ANALYSIS
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Lethal vs non-lethal?
Shockable vs. non-shockable? Too fast vs too slow?
Symptomatic vs. asymptomatic?
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LETHAL RHYTHMS Shockable (Defibrillation)
Ventricular fibrillationPulseless ventricular tachycardia
Non-shockableAsystolePulseless electrical activity
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NON-LETHAL RHYTHMS Too fast (tachycardias)
SinusSupraventricular (including a-fib/flutter)Ventricular
Too slow (bradycardias)SinusHeart block (1°, 2°, 3° AV block)
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WHAT IS A SYMPTOMATIC DYSRHYTHMIA? Any abnormal rhythm that produces
signs or symptoms of hypoperfusionChest Pain/ischemic EKG changesShortness of BreathDecreased level of consciousnessSyncope/pre-syncopeHypotensionShock - decreased UO, cool extremities,
etc.Pulmonary Congestion/CHF
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NAME THAT RHYTHM…
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63 YR MAN WITH A WITNESSED COLLAPSE WHILE MOWING THE LAWN
What is the rhythm?What is the management?
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VENTRICULAR FIBRILLATION
Rapid and irregular No normal P waves or QRS complexes
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VF / PULSELESS VT
26Primary Survey - ABC
Secondary Survey - ABC
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79YR MAN S/P NSTEMI
What is the rhythm?What is the management?
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VENTRICULAR TACHYCARDIA
Rapid and regular No P waves Wide QRS complexes
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VENTRICULAR TACHYCARDIA• Monomorphic VT
• Polymorphic VT
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VENTRICULAR TACHYCARDIA Assume any wide complex tachycardia
is VT until proven otherwiseSVT with aberrant conduction may also
have wide QRS complexes Attempt to establish the diagnosis
Ischemia risk and VT go together
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TREATMENT OF VT If pulseless - follow VF algorithm If stable try anti-arrhythmics
AmiodaroneLidocaineProcainamide?
If patient has a pulse, but is unstable or not responding to meds - shock
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TREATMENT OF VT Anti-arrhythmics are also pro-
arrhythmic One antiarrhythmic may help, more
than one may harm Electrical cardioversion should be the
second intervention of choice
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TACHYCARDIA
Lots of optionsbased on rhythm
Stable?
Shock
Unstable?
Evaluate Patien t
Treat the patient NOT the monitor!!!
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PULSELESS ELECTRICAL ACTIVITY Any organized (or semi-organized)
electrical activity in a patient without a detectable pulse
Non-perfusing
Treat the patient NOT the monitor
Find and treat the cause!!!!!
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PEA
Regular rate and rhythm Normal P waves and QRS No pulse
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ASYSTOLE
• Is it really asystole?• Check lead and cable connections.• Is everything turned on?• Verify asystole in another lead.• Maybe it is really fine v-fib?
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FIND AND TREAT THE CAUSE Non-shockable rhythm The most effective treatment is to find
and fix the underlying problem
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SO WHAT CAUSES PEA? #1 cause of PEA in adults is
hypovolemia #1 cause in children is
hypoxia/respiratory arrest
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THE H’S AND T’S Hypovolemia Hypoxia Hydrogen ion
(acidosis) Hyper-/
hypokalemia Hypothermia
Toxins Tamponade Tension
pneumothorax Thrombosis
(coronary or pulmonary)
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TREAT THE H’S AND T’S Hypovolemia
Volume – IVF, PRBC’s Hypoxia
Oxygenate/Ventilate Hydrogen ion
(acidosis) Sodium bicarbonate Hyperventilation
Hyper-/hypokalemia Sodium bicarbonate Insulin/glucose Calcium
Hypothermia Warm -- invasive
Toxins Check levels Charcoal Antidotes
Tamponade pericardiocentesis
Tension pneumothorax Needle decompression Tube thoracostomy
Thrombosis (coronary or pulmonary) Thrombolytics OR/cath lab
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SINUS BRADYCARDIA
Slow and regular Normal P waves and QRS complexes
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BRADYCARDIAS Many possible causes
Enhanced parasympathetic tone Increased ICPHypothyroidism Hypothermia Hyperkalemia Hypoglycemia Drug therapy
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BRADYCARDIAS Treat only symptomatic bradycardias
Ask if the bradycardia causing the symptoms
Recognize the red flag bradycardiasSecond degree type II blockThird degree block
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TRANSCUTANEOUS PACING Class I for all symptomatic bradycardias Always appropriate Doesn’t always work Technique
Attach pacer padsSet a rate to 80 bpmTurn up the (amps) until you get capture
Painful – may need sedation / analgesia
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TRANSVENOUS PACING Invasive Time-consuming to establish Skilled procedure Better long-term than transcutaneous May have better capture than
transcutaneous pacing
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BRADYCARDIA TREATMENT Medications
VagolyticAtropine
AdrenergicEpinephrineDopamine
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KNOW WHEN TO STOP With return of spontaneous circulation No ROSC during or after 20 minutes of
resuscitative effortsPossible exceptions include near-drowning,
severe hypothermia, known reversible cause, some overdoses
DNR orders presented Obvious signs of irreversible death
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TAKE HOME POINTS Assess and manage at every step before
moving on to the next step Rapid defibrillation is the ONLY
effective treatment for VF/VT Search for and treat the cause Treat the patient not the monitor Reassess frequently Minimize interruptions to chest
compressions
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