78206751 Pathophysiology PUD

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GORDONCILLO, LEAH A. CI: Sir Lourdevick Poquiz BSN 4D Area: 3B PATHOPHYSIOLOGY BOOK BASE PATHOPYSIOLOGY Damage to mucosal wall alcohol abuse, smoking, use of NSAID’s Acid and pepsinogen release with chronic vagal response to increased stress Infection with Helicobacter Pylori Damage mucosal barrier Low of mucosal cells; low quality of mucous; less of tight junction between cells Imbalance of aggressive and defensive factor Infection gives increased gastrin and decrease somatostatin production

Transcript of 78206751 Pathophysiology PUD

Page 1: 78206751 Pathophysiology PUD

GORDONCILLO, LEAH A. CI: Sir Lourdevick PoquizBSN 4D Area: 3B

PATHOPHYSIOLOGY

BOOK BASE PATHOPYSIOLOGY

Damage to mucosal wall alcohol abuse,

smoking, use of NSAID’s

Acid and pepsinogen release with chronic

vagal response to increased stress

Infection with Helicobacter Pylori

Damage mucosal barrier

Low of mucosal cells; low quality of mucous; less of tight

junction between cells

Imbalance of aggressive and defensive factor

Infection gives increased gastrin and decrease somatostatin

production

Page 2: 78206751 Pathophysiology PUD

GORDONCILLO, LEAH A. CI: Sir Lourdevick PoquizBSN 4D Area: 3B

Erosive gastritis inflammation >> decrease acid and intrinsic factor

Mucosal ulceration, possible bleeding and

scaring

Damage mucosa could not secrete

enough mucus to act

Severe UlcerationSigns and Symptoms:

Epigastric painHematemesis/Melena

DyspepsiaPyrosis

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GORDONCILLO, LEAH A. CI: Sir Lourdevick PoquizBSN 4D Area: 3BPATIENT BASE PATHOPHYSIOLOGY

MODIFIABLE:

Use of NSAID’s and response to increased stress

Acid and Pepsinogen release

Damage mucosal wall

Imbalance of aggressive and defensive factor

Low of mucosal cells; low quality of mucous; less of

tight junction between cells

Infection gives increased gastrin and decreased

somatostatin production

Page 4: 78206751 Pathophysiology PUD

GORDONCILLO, LEAH A. CI: Sir Lourdevick PoquizBSN 4D Area: 3B

Erosive gastritis inflammation >> decreased

acid and intrinsic factor

Mucosal ulceration, possible bleeding and

scaring

A damaged mucosa could not secrete enough mucus to act as a barrier against

gastric acid

SEVERE ULCERATIONSigns and Symptoms:

Epigastric painMelena

DyspepsiaPyrosis

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GORDONCILLO, LEAH A. CI: Sir Lourdevick PoquizBSN 4D Area: 3B

PATHOPHYSIOLOGYPredisposing Factors Precipitating

Factors

• Age (40-60) >NSAIDs

• Gender > Gastric Hyperacidity

> Stress

• Familial tendency > Irritating Foods

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GORDONCILLO, LEAH A. CI: Sir Lourdevick PoquizBSN 4D Area: 3B

Page 7: 78206751 Pathophysiology PUD

GORDONCILLO, LEAH A. CI: Sir Lourdevick PoquizBSN 4D Area: 3B

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GORDONCILLO, LEAH A. CI: Sir Lourdevick PoquizBSN 4D Area: 3B

SCHEDULE OF ACTIVTIES

2:45 – 3:00 pm Checking of Attendance and Paraphernalia and Assigning of patients

3:00 – 3:30 pm Endorsement and Rounds

3:30 – 4:00 pm Browsing of the Chart, Preparation of medications and starting the Nursing care

4:00 – 5:00 pm Vital Signs monitoring, Giving of medications Plotting the V/S in the TPR

5:00 – 6:00 pm Nursing Care

6:00 – 6:30 pm Feeding the patient

6:30 - 7:30 pm Break time

7:30 – 8:30 pm Vital Signs monitoring, Giving of Medications, Finalization of Nursing care

8:30 – 9:30 pm I and O monitoring, Accomplishing the TPR sheet and the chart

9:30 – 10:00 pm Rechecking of the Nursing Care Plan and the Chart by our clinical instructor

10:00 – 10:30 pm Endorsement

10:30 – 11:00 pm Post conference followed by dismissal