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    EMERGENCY NURSINGRoberto M. Salvador Jr. RN MD

    Is a specialized education, training and experience to gain expertise in assessing andidentifying patients health care problems in crisis situations.

    Emergency nurse establish priorities, monitors and continuously assesses acutely illand injured patients, supports and attends to families, supervise allied health personneland teaches the patient and families within a time limited, high pressured careenvironment.

    Issues in Emergency Nursing Care1. Documentation of consent.2. Limiting exposure to health risk.

    Providing holistic care

    a. Patient focused interventionb. Family focused intervention1. Anxiety and denial2. Remorse and guilt3. Anger4. grief

    Helping family members cope Take the family members to a private place Talk to the family together Reassure the family that everything possible was done Encourage family members to support each other Encourage the family to view the body if they wish Spend time with the family members, listening to them and identifying any needs Avoid unnecessary information Care given to clients with urgent and critical needs Care must be rendered without delay

    Diversified situations Consent (unless unconscious and without S.O.) Common clients (elderly, stomach pain, chest pain, fever, drug related, wound)

    Disaster Nursing (terrorism)Principle: TRIAGE

    Triage- a process use in sorting victims into categories of priority for care and transport based

    on severity of injuries and medical emergencies.

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    TRIAGEFrench word trier to sortSorting of clients based on the severity of health problemsHierarchy based on the potential for life loss

    Advanced skills

    Principles of tactical triage1. Accomplish the greatest good for the greatest number of casualties2. Employ the most efficient use of available resources3. Return personnel to duty as soon as possible

    TRIAGE3 categories of TRIAGE (Berners)

    1. Emergent2. Urgent3. Non-urgent

    TRIAGEI Emergent

    Highest priority

    Life threatening conditions, limbs

    Must be treated immediatelya. Airway compromiseb. Cardiac arrestc. Shockd. Strokee. Major Burns

    TRIAGEII Urgent

    Threatening conditions

    Not immediate

    Must be seen within 1 houra. Feverb. Minor Burnsc. Lacerations

    TRIAGE

    III Non-urgent Can be addressed within 24 hours

    Chronic conditionsa. Dental problemsb. Missed Menses

    4th categoryFast track simple first aid

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    TRIAGE

    Assess and Intervene (Primary survey)A airwayB breathing

    C circulationD disabilityE expose

    QUICK ASSESSMENTHEADMOUTH , LIPS & TEETHEYESNOSE & EARSFACESPINE & TRUNK

    LIMBS

    Glasgow Coma Scale

    Secondary Surveydone after the priorities has been addressed.

    a. Complete History and PEb. Diagnostic and laboratory testing

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    c. ECG, Arterial lines, urinary cathetersd. Splinting of suspected fracturese. Cleaning and dressing of woundsf. other necessary interventions

    WOUNDSLaceration skin tear with irregular edgesAvulsion tearing away from supporting structureAbrasion denuded skinEcchymosis/contusion blood trappedHematoma tumorlike under the skin mass of blood trapped under the skinStab incision with well defined edgesStab wound with eviscerationGun shot woundEntryExit

    Management: wound cleansing wound closure primary closure delayed primary closure Tetanus prophylaxis antibiotics Wound closure Primary closure

    Delayed primary closure

    Hemorrhage Stopping bleeding is essential to the care and survival Primary cause of shock

    Signs & Symptoms of Shock: Cool moist skin Falling blood pressure Increasing heart rate Delayed capillary refill Decreasing urine volume

    Management:fluid replacement control of external bleeding control of internal bleeding Fluid replacement & Blood replacement Control of external hemorrhage: Direct pressure

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    a. Temporalb. Facialc. Carotidd. Subclaviane. Brachial

    f. Radial & Ulnarg. Femoralh. Pressure dressingi. Tourniquets (last resort)

    Control of Internal Bleeding

    Signs & Symptoms: tachycardia Falling blood pressure Thirst

    Apprehension Cool & moist skin Delayed capillary refill

    Management Packed Red Blood Cell transfusion Surgery Pharmacologic therapy

    SHOCK

    Signs and SymptomsEarly stage Restless, confusion increase pulse rate, RR cold, moist skin decreased pulse pressure pallor thirst, dry mucous membrane diaphoresis oliguria

    Late stage shallow respiration Dec. BP Oliguria, anuria Cool, clammy skin ( hypovolemic, cardiogenic, septic) Cool, mottled skin ( neurogenic, vasogenic)

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    Lethargy Cyanosis Dilated pupils

    Nursing problems:

    a. altered tissue perfusion related to failing circulationb. impaired gas exchange related to ventilation-perfusion imbalancec. decreased cardiac output related to decreased circulating blood volume

    Management:1. Promoting fluid balance and cardiac output whole blood and blood products colloid solutions (albumin, plasma) plasma expanders crystalloids solution Isotonic solutions plain LR

    2. Assisting cardiac support modified trendelenburg position assisting with respiratory supports oxygen therapy mechanical ventilation suctioning deep breathing,coughing exercise

    3. Assisting with renal support monitor urine output bun, crea

    4. assisting GI support histamine blockers, antacids NGT

    5. promoting safety restraints strict asepsis technique

    Trauma Unintentional or intentional wound or injury 4th leading cause of death in the US Leading cause of death in children & young adults < 44 years of age Injury prevention ( only way to reduce incidence of trauma)a. Educationb. Legislationc. Automatic protection

    TRAUMAStab Wound1. Intra-abdominal injuries:

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    Penetrating abdominal injuries Gunshot wound, Stab wounds Serious & requires surgery Liver ( most frequently injured solid organ)

    All abdominal gunshot wounds require surgical exploration Stab wounds may be managed non-operatively

    Blunt TraumaBlunt Abdominal Injury Result from motor vehicle crashes, falls, blows or explosions Injuries may be hidden or difficult to detect Involves the liver, kidneys, spleen, blood vessel

    Assessment & Diagnostic Findings History & PE

    Lab studies: Urinalysis serial Hct. level WBC count Serum amylase analysis

    Internal Bleeding Inspection ( front of the body, flanks & back) Bluish discoloration, asymmetry, abrasion, contusion Abdominal CT Scan Abdominal Ultrasound Left shoulder pain ( ruptured spleen) Right shoulder pain (liver laceration)

    Intraperitoneal Injury Assess for tenderness, rebound tenderness, guarding, rigidity, spasm, increasing distention & pain Referred pain ( intraperitoneal injury)

    Diagnosis:1. abdominal ultrasound2. abdominal CT scan3. Diagnostic peritoneal lavage

    1 L LRS/ NSS

    400 ml return

    RBC > 100,000/mm3

    WBC ct > 500/mm3

    Bile, feces, food

    Sinography ( detection of peritoneal penetration)

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    Purse string

    Small catheter

    Contrast agentX-ray

    Intraabdominal Injury Management: Resuscitation procedure Occlusion of chest wound Direct pressure Intravenous fluid replacement Immobilization of the spine Cervical spine immobilization Tetanus prophylaxis Broad spectrum antibiotics

    Multiple Casualty Incident

    MCI is defined as an event involving a number and/or severity of casualties,which isbeyond the capabilities of available care teams and facilities.

    MINIMAL (GREEN TAG) Also known as the walking wounded Examples include but are not limited to small burns, lacerations, abrasions, and

    small fractures. These casualties have minor injuries and can usually care for themselves with

    self-aid or buddy aid. These casualties should still be employed for mission

    requirements (e.g., scene security).

    DELAYED (YELLOW TAG) The delayed category includes wounded casualties who may need surgery, but

    whose general condition permits a delay in surgical treatment without undulyendangering life or limb. Medical treatment (splinting, pain control, etc.) will berequired but it can wait.

    Examples include but are not limited to casualties with no evidence of shockwho have large soft tissue wounds, fractures of major bones, intra-abdominal orthoracic wounds, or burns to less than 20% of total body surface area.

    IMMEDIATE (RED TAG) The immediate category includes casualties who require immediate LSI and/orsurgery. Put simply, if medical attention is not provided, the patient will die. Thekey to successful triage is to locate these individuals as quickly as possible.

    Examples include but are not limited to hemodynamically unstable casualtieswith airway obstruction, chest or abdominal injuries, massive external bleeding,or shock.

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    EXPECTANT (BLACK TAG) Casualties in this category have wounds that are so extensive that even if they

    were the sole casualty and had the benefit of optimal medical resources, theirsurvival would be highly unlikely. Even so, expectant casualties should not beneglected. They should receive comfort measures, pain medications, if possible,

    and they deserve re-triage as appropriate. Examples include but are not limited to casualties with penetrating or blunthead wounds and those with absent radial pulses.

    TRIAGE TAGS Triage tags are designed to communicate the triage category, treatment

    rendered, and other medical information. By necessity, the information on thetag is brief. Triage tags are usually placed on the casualty by the triage officeralthough other members of the team may place or add information to the tags.

    PURPOSE

    To furnish the attending care provider during the evacuation of a casualty withessential information about the injury or disease and the treatment provided. The sole or initial medical record for the troops injured in combat. Each triage tag is coded with a unique sequential seven-character serial number

    used for identification and tracking of the casualty. The serial number is locatedon the top right and left diagonal tear-offs.

    Management:Determine the extent of injuryEstablish priority of treatmentNursing management in Sprain, Strain:1. Immobilize extremity and advise rest2. Apply cold packs initially then heat packs3. Compression bandage may be applied to relieve edema4. Assist in cast application5. Administer NSAIDS

    FRACTUREA fracture is a complete or incomplete break in the continuity of bone. This will be

    accompanied by varying degrees of injury to surrounding soft tissues.

    CLASSIFICATION OF FRACTURESBROAD CLASSIFICATION1. Complete fractureInvolves a break across the entire cross-section of the bone & is frequently displaced

    2. Incomplete fracture (usually in adults)The break occurs through only a part of the cross-section of the bone

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    Break in the continuity of the bone cause:DISPLACEMENT OF FRAGMENT CAUSES:DAMAGE TO THE SOFT PART CAUSES:

    Clinical Features of Fractures:

    1) pain and tenderness over the involved area2) loss of function3) deformity4) attitude ( shortening)5) abnormal mobility and crepitus (a grating sensation produced when bones rub each

    other)6) neurovascular injury ( localized swelling & discoloration of the skin)7) radiographic findings

    EMERGENCY MANAGEMENT OF FRACTURE

    1. Immobilize any suspected fracture by splinting2. Support the extremity above and below when moving the affected part from a vehicle3. Suggested temporary splints- hard board, stick, rolled sheets4. Apply sling if forearm fracture is suspected or the suspected fractured arm maybebandaged to the chest5. Open fracture is managed by covering a clean/sterile gauze to prevent contamination6. DO NOT attempt to reduce ( re-align) the fracture

    5 Ps in Fracture:P painP pallorP - paresthesiaP - pulselessnessP - Paralysis

    Nursing ConsiderationsAssessA airwayB breathingC circulation neurogenicD disabilityE expose

    always IMMOBILIZE the affected bonePrinciples of Fracture Treatment

    1. Reduction of fracture2. Maintenance of alignment3. Promote callus formation4. Restoration of function5. Prevent complications

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    ER Management

    1. Assess2. Immobilization3. A, B, C, D, E

    4. Control bleeding5. TT, TIG and TAT immunization6. Wound care7. Diagnostic and Lab Procedures8. Fracture Reduction

    Compartment Syndrome- a condition in which the circulation and function of tissues within a closed space are

    compromised by an increased pressure within that spaceS/Sx: 4 Ps - Pain / Pallor / Paralysis / Pulselessness

    * although none is pathognomonic, pain is the most important* best indicator: tissue pressure measurement

    - a surgical emergency (fasciotomy)

    Whitesides Technique* for measuring intracompartmental pressure

    * results in permanent neurovascular damage if not relieved in 4 to 6 hrs.* the normal tissue pressure within closed compartments is approximately 0 mmHg

    > pressures of within 10 to 30mmHg of a patients diastolic blood pressure - therewill be inadequate tissue perfusion and relative ischemia

    > if the pressure within a compartment equals or exceeds the patients diastolicblood pressure - there will be no effective tissue perfusion

    Compartment syndromeASSESSMENT FINDINGS1. Pain- Deep, throbbing and UNRELIEVED by opioidsPain is due to reduction in the size of the muscle compartment by tight castPain is due to increased mass in the compartment by edema, swelling or hemorrhage2. Paresthesia- burning or tingling sensation3. Numbness4. Motor weakness5. Pulselessness, impaired capillary refill time and cyanotic skin6. Edema unrelieved by elevation

    Compartment syndromeMedical and Nursing management1. Assess frequently the neurovascular status of the casted extremity2. Elevate the extremity above the level of the heart

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    3. Assist in cast removal and FASCIOTOMY

    Fat EmbolismOccurs usually in fractures of the long bonesFat globules may move into the blood stream because the marrow pressure is greater

    than capillary pressureFat globules occlude the small blood vessels of the lungs, brain kidneys and otherorgans

    Onset of s/sx of fat embolism is rapid, (within 24-72 hours)

    ASSESSMENT FINDINGS1. Sudden dyspnea and respiratory distress2. tachycardia3. Chest pain4. Crackles, wheezes and cough

    5. Petechial rashes over the chest, axilla and hard palate

    Nursing ManagementSupport the respiratory function

    Respiratory failure is the most common cause of deathAdminister O2 in high concentrationPrepare for possible intubation and ventilator support

    Environmental Emergencies

    HEAT CRAMPSPeople at risk:Not acclimatized to heatElderly & very youngUnable to care for themselvesWith chronic & debilitating diseasesTaking certain medicationsCauses thermal injury at the cellular level ( heart, liver, kidney, blood coagulation)

    Management:To reduce high temperature ASAPcool sheets & towels, TSBIce packCooling blanketsIced Saline LavageImmersion in cold water bathMassage ( promote circulation)Pt monitoring ( VS, ECG, CVP)Oxygenation (100%)

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    IV infusion therapyMonitor urine outputPatient education

    Frostbite

    Trauma from exposure to freezing temperatureActual freezing of tissue fluidsResults in cellular & vascular damageFeet, hand, nose, ears

    Assessment:History of exposure to coldFrozen extremity, hard, cold , insensitive to touch

    Management:Restore normal body temperature

    Circulating back of 37 40 CSterile gauze or cotton in between fingers & toesMassage is contraindicatedWhirlpool bathEscharotomyFasciotomy

    HypothermiaThe core (internal) temperature is 35 C or less

    Assessment and Findings:Progressive deterioration

    ApathyPoor judgement

    AtaxiaDysarthriaDrowsinessPulmonary edemaCoagulopathy

    Management:Monitoring VS, CVP, UO, ABG, Blood chem., ECG, Chest X-rayRewarminga. core rewarming method, CP bypass, warm fluid, warmhumidified oxygen, warm peritoneal lavageb. Passive external rewarming, warm blankets over the bed heatersSupportive Care

    Near DrowningSurvival for at least 24 hours after submersion

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    Hypoxemia ( most common consequence)Leading cause of unintentional death in children younger than 14 years old

    Factors:Alcohol ingestion

    Inability to swimDiving injuriesHypothermiaExhaustionFresh water aspiration (loss of surfactant)Salt water aspiration (pulmonary edema)

    Management:Maintain cerebral perfusion

    Adequate oxygenationImmediate CPR

    Monitor temperature by rectal probeRewarming proceduresECG monitoringIndwelling urinary catheterNGT.Decompression Sickness (DCS)

    Also called The BendsDiving, high altitude flying or flying in commercial aircraft within 24 hours after divingResults from nitrogen bubbles trapped in the bodyMusculoskeletal pain, numbness/hypesthesiaNitrogen bubbles become air emboli, stroke, paralysis, death

    Assessment & Diagnosis:Detailed historyRapid ascent, loss of air in the tank, buddy breathing, recent alcohol intake, lack ofsleep or flight within 24 hours

    Management:Patent airway

    Adequate ventilationOxygenation (100%)Hyperbaric chamber

    Anaphylactic ReactionAcute systemic hypersensitivity reactionOccurs within seconds or minutes after exposure to certain foreign substancesMedications

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    Insect stingsFoodsImmunoglobulin E (IgE)

    Diagnosis:

    Respiratory symptomsDOBStridor secondary to laryngeal edemaFainting, itching, swelling of mucus membraneSudden drop in BP

    Management:Patent airway & ventilationET intubation

    Aqueous epinephrineCrichothyroidotomy

    AntihistaminesAminophylinesAlbuterol inhalersIsoproterenol or DopamineIV Benzodiazepines

    Latex AllergyAffects healthcare providers who uses this productManagement: Latex free products

    . Injected Poisons: Stinging InsectsVenoms of the hymenoptera (bees, hornets, yellow jackets, fire ants, wasps)Venom allergy ( IgE mediated reaction)StingingClinical Manifestations:Generalized urticariaItchingMalaise

    AnxietyBronchospasmShockDeath

    Management:Stinger removalWound care with soap & waterIce applicationOral Antihistamines & analgesic

    Aqueous epinephrine SQDesensitization therapy

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    Snake Bites

    Affects ages 1- 9 yearsPit vipers (most frequent poisonous snake in the US)Cobra ( Philippines)

    Upper extremity (most common site)Envenomation (injection of a poisonous material by sting, spine, bite)

    Medical emergencyManagement:Have victim lie downRemove constrictive itemsProvide warmthCleanse & cover the woundImmobilize the injured part below the level of the heartIce & tourniquet is contraindicated

    Corticosteroids are contraindicated in the first 6-8 hours after biteObserve for at least 6 hoursAdministration of antivenin within 12 hours after the biteChildren requires more antivenin than adultsSkin or eye test to detect allergy to antiveninMeasurement of circumference of the affected partbefore administration of antivenin and every 15 minutes thereafter

    After symptoms decrease, every 30-60 minutes for the next 48 hoursDone to detect compartment syndrome (swelling, loss of pulse, increase pain,paresthesia)Diphenhydramine & CemetidineToo rapid infusion ( most common caused of allergic reaction)

    Common Household Poisons:First Aid Management

    Absorbed Poisons - a poison that enters the body through the skin.

    Injected Poisons - a poison that enters the body through a bite, stings, or syringe

    Ingested or Swallowed Poisons (Corrosive)Alkaline or acid agents caused tissue destruction after in contact with mucus membrane

    Management:Airway, ventilation, oxygenationWater or milk to drink for dilutionSyrup of Ipecac, Gastric lavage, Activated charcoal and Catharsis are allContraindicated.

    Antidote as early as possibleMonitor VS, CVP, Fluid & Electrolytes

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    Psychiatric consultation

    Inhaled Poisons : Carbon Monoxide PoisoningResult of industrial or household incidence or attempted suicideCarbon monoxide exerts its toxic effect by binding to circulating hemoglobin thereby

    reducing O2 carrying capacity of the blood

    Carboxyhemoglobin does not transport oxygenHgb has 200x more affinity than oxygen

    Signs & Symptoms :HeadacheMuscle weaknessPalpitationDizzinessConfusion

    CyanosisComa

    ManagementReverse cerebral and myocardial hypoxia and to hasten elimination of carbon monoxideCarry the patient to fresh air immediately and open all windows and doorsLoosen all tight clothingInitiate CPR, 100% O2

    Food PoisoningAfter ingestion of contaminated food or drinks

    Botulism ( serious form of food poisoning)Management:Determine the source & type of food poisoningFood, gastric contents, vomitus, serum, feces are examinedFluid & electrolyte correction

    Antiemetic medicationElicit informationHow soon after eating did the symptom occursWhat was eaten and did the food have an unusual smellDid anyone else become ill eating the same foodDid vomiting or diarrhea occursNeurologic symptomsWhat is the patient appearance

    Substance AbuseMisused of specific substances to alter mood or behaviorDrug & alcohol

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    Acute Alcohol IntoxicationAffects young adults or people older than 60 years of ageIt is a psychotropic drugs

    Alcohol or ethanol is a direct multisystem toxin & CNS depressant:Drowsiness

    IncoordinationSlurring of speechSudden mood changes, Aggression, belligerence, grandiosityUninhibited behavior

    Management:Detoxification of the acute poisoning, recovery, rehabilitationDenial & defensiveness

    Approach patient in a calm or non-judgemental manner

    Alcohol Withdrawal Syndrome/Delirium Tremens

    Acute toxic state that occurs as a result as a cessation of alcohol intake

    Signs & symptoms:AnxietyUncontrollable fearTremorIrritability

    AgitationInsomniaIncontinenceVisual, tactile, auditory, olfactory hallucination

    Diagnostic Testing1. Most commonly used tests include

    a. Urine Drug Screen (UDS)b. Blood Alcohol Level (BAL)

    1. Legal intoxication is 0.10%a. Clumsinessb. Impaired reaction time

    2. 0.20% brain is depressed, ataxia3. May experience withdrawal symptoms if BAL is high

    2. Length of time drugs can be found in urine and blood varies with dosage andmetabolic properties of drug

    Management:Adequate sedation & supportAllow pt to rest and recoverPlace pt in a calm, nonstressful environment

    Alcohol free environmentRefer pt to self help groups such as AA

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    Negative conditioning with Disulfiram(Antabuse)Naltrexone HCL (antidote)Drug OverdoseNursing DiagnosisRisk for Injury

    1. Determine disorientation, level of agitation, risk for suicide or harm to self orothers2. Protective environment, frequent observation3. Vital signs q 15 minutes: feedback for symptoms of withdrawalIneffective Individual Coping1. Limit setting; encourage expression of feelings, fears2. Teach alternative ways of dealing with stress

    Altered Nutrition: Less than Body Requirements1. Referral to dietician; nutritional assessment including blood work2. Client modification of diet, goal setting for weight according to needSelf-Esteem Disturbance

    1. Acceptance of person2. Focus on strength and accomplishments

    BURNS

    MAJORITY OF BURN CASESARE DUE TO NEGLIGENCESO HAZARD PRECAUTIONSMUST BE OBSERVED.pinabayaan ng NANAYCarelessness with matchScald from hot liquidDefective electrical equipmentImmersion in overheating bath waterUse of chemicals

    SafetyDont panicDrop to the floorLook for the exitCover face with wet clothImmerse into cool water or running water immediately if you get burned to preventfurther injury.Extinguish any remaining fire by dropping and rolling onto the floor.

    ASSESSMENTAIRWAYBREATHING

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    CIRCULATIONDISABILITIESEXPOSE

    Expose cont

    A airway - check nose, face and neck (priority) singed and sooty hair of the noseB breathing rise and fall of chestC circulation - if there is no breathing and circulation start CPRD check for disability and manage accordinglyE expose to determine extent of injury

    Types of BurnsThermal dry flames, moist and heatMechanical friction or abrasionChemical acid or alkaliElectrical most fatal

    Radiation sunlightClassification of BurnsBurn classification as to depth

    Superficial Partial thickness(1st degree)

    Outer layer of dermisErythema, pain up to 48 hrsHealing 1-2 wks [sunburn]Burn classification as to depth

    Deep Partial thickness(2nd degree)Epidermis & dermis involvedBlisters & edema, frequently quite painfulHealing 14-21 daysBurn classification as to depth

    Full thickness (3rd degree)Epidermis, dermis, subcutaneous fat are involvedDry, pearly white or charred in appearanceNot painfulEschar must be removed; may need grafting

    ABCDE assessmentAirway and fluid resuscitation (priority)Give TIG or TAT and TTProphylactic antibiotic

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    Sterile dressing for wound

    Thermal BurnsManagement:1st and 2nd degree

    - relieve pain by immersing in cold water or applying cold cloth- Cover the burn with dry, non-sticking sterile dressing

    3rd

    degree- cover with dry non-sticking sterile dressing- treat victim for shock and keep warm

    Chemical Burns- remove the chemical by flushing with water- flush for 20 min or longer- cover with dry dressing

    Electrical Burns- unplug or turn off power- check ABC- treat for shock

    INHALATION INJURIESHeat Inhalation-HOT AIR OR FLAMESSystemic Toxins-ENCLOSED FIRE-CO IS INHALEDSmoke Inhalations-FREQUENTLY HIDDEN BY MORE VISIBLE INJURIES (60-80% FATALITIES)

    Indications of inhalation injuryusually appears within 2-48 hours after the burn occurred. Indications may include:The patient faintsFire or smoke present in a closed areaEvidence of respiratory distress or upper airway obstructionSoot around the mouth or noseNasal hairs (SCORCHED HAIR), eyebrows, eyelashes have been singedBurns around the face or neckCriteria for classification of extent of burns

    Minor Burn- 2nd degree burn

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    - 3rd degree 25% TBSA in adults or 20% in children- all burns involving the critical areas

    Critical areasFaceHandsFeetPerineumChest

    ESTIMATION of BURNSVarious methods are utilized for estimating the extent of burn injury1. The Rule of Nines in adults

    Head and Neck- 9%Anterior trunk- 18%Posterior trunk- 18%Upper arms- 18% ( 9% each x 2)Lower ext- 36% ( 18% EACH X 2)Perineum- 1%

    Fluid replacementConsensus formulaLRS 2-4ml x BW (kg) x %TBSAHalf given in 1

    st8 hrs, then half for 16 hrs

    Evans formula- colloid: 1ml x BW x TBSA- electrolytes 1ml x BW x TBSA- Glucose (D5W5%) 200ml for IWL

    Parkland Formula(4ml x TBSA x BWkg)

    1st 8H give ,2nd 8H give and for the3rd 8H give the last part

    Burn Management

    1.EMERGENT PHASEBegins at the time of injury and ends with the restoration of the capillary permeability (with 48-72 hours)

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    The GOAL is to PREVENT hypovolemic shock and preserve the vital body organfunctionEmergency and pre-hospital care1st PhaseFluid Accumulation

    IV to IT and IC

    most critical period36-48H post burn, FVD or hypovolemia3rd fluid shiftedema on the injured area (IV to IT)fatal form is circumferential edema from chest injury1

    stPhase Cont

    c. edema and p. edema (IV to IC)hyponatremia (IV to outside from it)hyperkalemia (cell injury)

    1

    st

    Phase ContBVcurlings ulcer or paralytic ileus (dec. BV), NPO, NGT lavage, TPNInfection may set in (isolation)Fluid ResuscitationBlood MonitoringETT InsertiomPulse Carbon Monoxide Oximetry

    Arrhythmias MonitoringBurn Management

    2.RESUSCITATIVE PHASE

    Begins with the initiation of fluids and ENDS when capillary integrity returns to near-normal and large fluid shifts have decreasedThe GOAL is to prevent shock by maintaining adequate circulating blood volume tomaintain vital organ perfusion

    2nd PhaseFluid RemobilizationIT and IC to IVMay last 48-60HFVE (CHF)HypokalemiaDiuresis phase (oliguria may signifies RF)ISC IVCHemodilution2ndPhase ContHyponatremia due to fluid loss from diuresis phaseInfection may set in (isolation)

    Anemia may linger up to recovery periodComplications from immobility may set in (Circulo-O-electric bed)

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    Anemia may lingerBurn Management

    3.ACUTE PHASEBegins when the client is HEMODYNAMICALLY stable, capillary permeability is

    restored and DIURESIS has begunEmphasis is placed on restorative therapy and the phase continues until wound closureis achievedThe FOCUS is on infection control, wound care, wound closure, nutritional support, painmanagement and physical therapy

    3rd Phase to Recovery PeriodInfection may set in (isolation, Sulfadiazine application)Healing process to scar formation and contracturesSurgery (Reconstructive or Plastic) STSG auto-graft3

    rdPhase Cont

    Debridement and EscharotomyDiet: high caloric high CHONPsychological Aspect: dec. self esteem, stigma, perceived body changes, isolation,depression, loss of identity these are all related to physical disfigurement.

    Burn Management

    4.REHABILITATIVE PHASEThe final phase of Burn care, restoration of functions, cosmetic surgeryGoals of this phase patient independence and restoration of maximal function

    Infection PreventionSilver sulfadiazine- bactericidal- minimal penetration to eschar

    Silver Nitrate- bacteriostatic and fungicidal- does not penetrate eschar

    Mafenide acetate- Gram (-) and (+)-diffuses rapidly to eschar

    Nursing Management1. Emergent phase (time of injury)Remove person from source of burn.1) Thermal: smother burn beginning with the head.2) Smoke inhalation: ensure patent airway.

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    3) Chemical: remove clothing that contains chemical; lavage area with copious amountsof water.4) Electrical: note victim position, identify entry/exit routes, maintain airway.

    Nursing Management

    1. Emergent phase (time of injury)Cool the burn for several minutes. DONT USE ICE!!Wrap in dry, clean sheet or blanket to prevent further contamination of wound andprovide warmth and conserve body heat.

    Assess how and when burn occurred.

    Nursing Management1. Emergent phase (time of injury)Remove constricting clothes and jewelryCover the wound with a sterile dressing or clean, dry clothProvide IV route only if possible

    Transport immediately to a hospital or burn facilityNursing Management2. Resuscitative and Shock phase (first 2448 hours)Provide appropriate fluid resuscitation based on the Parkland formula4 mL Plain LR x %TBSA of burns x kg body weightNursing Management3. Fluid remobilization or diuretic phase (25 days post burn)Monitor and treat potential complications like acute renal failure, paralytic ileus,Curlings ulcer and hypokalemiaNursing Management4. Convalescent phasea. Starts when diuresis is completed and wound healing and coverage begin.

    GENERAL NURSING INTERVENTIONS IN THE HOSPITAL1. Provide relief/control of pain.a. Administer morphine sulfate IV and monitor vital signs closely.b. Administer analgesics/narcotics 30 minutes before wound care.c. Position burned areas in proper alignment

    GENERAL NURSING INTERVENTIONS IN THE HOSPITAL2. Monitor alterations in fluid and electrolyte balance.a. Assess for fluid shifts and electrolyte alterationsb. Monitor Foley catheter output hourly (30 cc per hour desired).c. Weigh daily.d. Monitor circulation status regularly.e. Administer/monitor crystlloids/colloids

    GENERAL NURSING INTERVENTIONS IN THE HOSPITAL3. Promote maximal nutritional status.a. Monitor tube feedings if Peripheral Nutrition is ordered.

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    NPO immediately after injury!!! ONLY when oral intake permitted, provide high-calorie,high-protein, high- carbohydrate diet with vitamin and mineral supplements .c. Serve small portions.d. Schedule wound care and other treatments at least 1 hour before meals.GENERAL NURSING INTERVENTIONS IN THE HOSPITAL

    4. Prevent wound infection.a. Place client in controlled sterile environment.b. Use hydrotherapy for no more than 30 minutes to prevent electrolyte loss.Observe wound for separation of eschar and cellulitis.GENERAL NURSING INTERVENTIONS IN THE HOSPITAL5. Prevent GI complications.a. Assess for signs and symptoms of paralytic ileus.b. Assist with insertion of NG tube to prevent/control Curlings/stress ulcer; monitorpatency/drainage.GENERAL NURSING INTERVENTIONS IN THE HOSPITAL5. Prevent GI complications.

    c. Administer prophylactic antacids through NG tube and/or IV cimetidine (Tagamet) orranitidine (Zantac) (to prevent stress ulcer).d. Monitor bowel sounds.e. Test stools for occult blood.

    RehabilitationMethods of coping and re-socializationEnsure optimum nutritionInitiate physical therapy to regain and maintain optimal range of motion and achievewound coverageProvide psychosocial support to promote mental health

    RehabilitationProvide family-centered care to promote integrity of the family as a unitEncourage post-discharge follow-up for several yearsEnsure appropriate referral to cosmetic surgeon, psychiatrist, occupational therapist,nutritionist and physical therapist

    Drugs for BurnsMafenide (Sulfamylon)1) Administer analgesics 30 minutes before application.2) Monitor acid-base status and renal function studies. SIDE EFFECT: LACTIC

    ACIDOSIS3) Provide daily BATH for removal of previously applied cream.

    Drugs for BurnsSilver sulfadiazine (Silvadene)1) Administer analgesics 30 minutes before application.2) Observe for and report hypersensitivity reactions (rash, itching)3) Store drug away from heat

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    4) Disadvantage: poor eschar penetration

    Drugs for BurnsSilver nitrate1) Handle carefully; solution leaves a gray or black stain on skin, clothing, and utensils.

    2) Administer analgesic before application.3) Keep dressings wet with solution; dryness increases the concentration and causesprecipitation of silver salts in the wound.

    Drugs for BurnsPovidone-iodine (Betadine)

    Administer analgesics before application.Assess for metabolic acidosis/renal function

    Gentamicin

    Assess vestibular/auditory and renal functions at regular intervals.

    CimetidineGiven to prevent Curlings ulcerWound debridement (ESCHAROTOMY)

    Skin graftingAutograftHomograft- from living or recently deceased

    Heterografts from animalsBiosynthetic biobraneDermal substitute integra, allodermSkin Grafting

    Donts in burnsDO NOT apply ointment, butter, ice, medications, fluffy cotton dressing, adhesivebandages, cream, oil spray, or any household remedy to a burn. This can interfere withproper healing.DO NOT allow the burn to become contaminated. Avoid breathing or coughing on theburned area.DO NOT disturb blisters or dead skin.DO NOT apply cold compresses and DO NOT immerse a severe burn in cold water.This can cause shock.DO NOT place a pillow under the victim's head if there is an airway burn and they arelying down. This can close the airway.

    Violence, Abuse, NeglectFamily Violence, Abuse & NeglectDomestic violence is the leading cause of death for young African American WomenMen & persons with disabilities are also victims of domestic violence

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    Elder abuse results physical, psychological abuse, neglect, vilations of personal rights &financial abuse

    Clinical Manifestation:Unexplained bruises, laceration, abrasion, head injuries & fractures

    Malnutrition & Dehydration (most common in neglect)

    Assessment:Early detection & InterventionCareful history

    Management:Primary concern safety & welfare of the pt.Separation of the pt with the abuserMandatory reporting laws

    Sexual AssaultRape is force sexual actVictims may either be male or female

    Crisis Intervention:Assessment & diagnostic findingsrape trauma syndromephases of psychological reactionacute disorganization phase ( shock, disbelief, fear, guilt, humiliation, anger)Denial Phase: (anxiety, fear, flash backs, sleep disturbances, hyperalertness &psychosomatic reactions)Phase of Reorganization: (Recovery)Physical examinationInformed and written consentFocus onExternal evidence of traumaDried semen stainsTreat potential STDPostcoital contraceptive medicationOvral _ 12-24hrs not later than 72 hrs

    Management:Give sympathetic supportReduce emotional traumaGather available evidenceRespect patient privacy and sensitivityGoal: have pt. regain control over her/his life

    . Violence in the Emergency Department

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    Pts & families waiting for assistance at the ED are sometimes dissatisfied resulting inviolenceManagement:Safety is the first priority

    Psychiatric EmergenciesIs an urgent, serious disturbance of behavior, affect, or thought that makes the pt.unable to cope with life situations & interpersonal relationshipsConcern: Determining whether pt is at risk for injuring self or others

    Aim: Maintain pt self esteem while providing care.Overactive Patients

    Display disturbed, uncooperative & paranoid behaviorManagement:Reliable history about mental illness, hospitalization, injuries, illnesses, use of alcohol ordrugs

    Immediate goal: Gain control of the situationRestraint is used as the last resortPsychotropic agent : Chlorpromazine, (Thorazine), Haloperidol (Haldol)

    Violent BehaviorUsually episodicMeans of expressing feelings of anger, fear, or hopelessnessManagement:Goal : bring the violence under controlUse calm & noncritical approachCrisis interventionSedativeRestraint

    Post Traumatic Stress Disorder (PTSD). Development of characteristic symptoms after a psychologically stressful eventSymptoms include intrusive thoughts & dreams, phobic avoidance reaction, heightenedvigilance, exaggerated startle reaction, generalized anxiety, societal withdrawal

    Assessment:Evaluation of the pts pretrauma history, the trauma itself & posttrauma functioningManagement:Crisis interventionEstablish a trusting & sharing relationshipEducation of the pt and family

    Underactive or Depressed PatientDepression may be masked by anxiety & somatic complaintsClinical manifestations:Sadness

    Apathy

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    Feeling of worthlessnessSelf-blameSuicidal thoughts

    Anorexia, Weight lossDecrease interest in sex

    SleeplessnessManagement:Ventilating personal feelingsSuicidal precaution

    Antidepressant & antianxiety agentsPsychiatric consultation

    Suicidal Patients. Attempted suicide is an act that stems from depressionViewed as a cry for help or interventionWeight loss

    Sleep disturbancesSomatic complaintsSuicidal preoccupationManagement:Treat the consequences of suicidal attempt & prevent further self injuryCrisis intervention

    Myxedematous coma1. Life-threatening complication of long-standing and untreated hypothyroidism2. Hyponatremia, hypoglycemia, acidosis3. Precipitated by stressors, failure to take thyroid replacement meds4. Treatment includes restoring balance throughout systems and increasing thyroidhormone levels

    Diagnostic Testsa. Serum thyroid antibodies (TA): antibodies in Hashimotos Thyroiditisb. TSH test: (from pituitary) elevated with primary hypothyroidismc. T3 and T4: decreased for diagnosis of hypothyroidismd. T3 uptake test; decreased with hypothyroidismRAI uptake test1. Oral or intravenous dose of radioactive iodine (131I or 123I) given to client2. Thyroid scanned after 24 hours3. Uptake decreased with hypothyroidism4. Size and shape of gland revealedf. Serum cholesterol is elevated

    DISORDERS OF the THYROID GLANDNURSING INTERVENTIONS1. Monitor VS especially HR

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    2. Administer hormone replacement: usually Levothyroxine( Synthroid)-should be takenon an empty stomachDISORDERS OF the THYROID GLANDNURSING INTERVENTIONS3. Instruct patient to eat LOW calorie, LOW cholesterol and LOW fat diet

    4. Manage constipation appropriately5. Provide a WARM environmentDISORDERS OF the THYROID GLANDNURSING INTERVENTIONS6. Avoid sedatives and narcotics because of increased sensitivity to these medications7. Instruct patient to report chest pain promptlyNursing Diagnosesa. Decreased Cardiac Outputb. Constipationc. Risk for Impaired Skin Integrity: due to over all edema high risk for skinbreakdown: preventative interventions

    DISORDERS OF the THYROID GLANDThyroid storm

    An acute LIFE-threatening condition characterized by excessive thyroid hormoneDISORDERS OF the THYROID GLAND

    Thyroid stormCAUSE: Manipulation of the thyroid during surgery causing the release of excessivehormones in the bloodDISORDERS OF the THYROID GLAND

    ASSESSMENT Findings for Thyroid Storm1. HIGH fever2. Tachycardia and Tachypnea3. Systolic HYPERtensionDISORDERS OF the THYROID GLAND

    ASSESSMENT Findings for Thyroid Storm4. Delirium and coma5. Severe vomiting and diarrhea6. Restlessness, Agitation, confusion and SeizuresDISORDERS OF the THYROID GLANDNURSING INTERVENTIONS1. Maintain PATENT airway and adequate ventilation2. Administer anti-thyroid medications such as Lugols solution, Propranolol, andGlucocorticoids

    DISORDERS OF the THYROID GLANDNURSING INTERVENTIONS3. Monitor VS

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    4. Monitor Cardiac rhythms5. Administer PARACETAMOL ( not Aspirin) for FEVERDISORDERS OF the THYROID GLANDNURSING INTERVENTIONS6. Manage Seizures as required.

    7. Provide a quiet environment

    Diabetic KetoacidosisThis is cause by the absence of insulin leading to fat breakdown and production ofketone bodiesThree main clinical features:1. HYPERGLYCEMIA2. DEHYDRATION & electrolyte loss3. ACIDOSISDKA

    PATHOPHYSIOLOGY

    No insulin

    reduced glucose breakdown and increased liver glucose production

    HyperglycemiaDKA

    PATHOPHYSIOLOGYHyperglycemia kidney attempts to excrete glucose increased osmotic load diuresis DehydrationDKA

    PATHOPHYSIOLOGYNo glucose in the cell fat is broken down for energy ketone bodies are producedKetoacidosis

    DKARisk factors1. infection or illness- common2. stress3. undiagnosed DM4. inadequate insulin, missed dose of insulinDKA

    ASSESSMENT FINDINGS1. 3 Ps2. Headache, blurred vision and weakness3. Orthostatic hypotensionDKA

    ASSESSMENT FINDINGS4. Nausea, vomiting and abdominal pain5. Acetone (fruity) breath6. Hyperventilation orKUSSMAULs breathingHYPERGLYCEMIAHyperglycemiaDKALABORATORY FINDINGS

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    1. Blood glucose level of 300-800 mg/dL2. Urinary ketonesDKALABORATORY FINDINGS3. ABG result of metabolic acidosis- LOW pH, LOW pCO2 as a compensation, LOW

    bicarbonate4. Electrolyte imbalances- potassium levels may be HIGH due to acidosis anddehydrationDKANURSING INTERVENTIONS1. Assist in the correction of dehydrationUp to 6 liters of fluid may be ordered for infusion, initially NSS then D5WMonitor hydration statusMonitor I and OMonitor for volume overloadDKA

    NURSING INTERVENTIONS2. Assist in restoring ElectrolytesKidney function is FIRST determined before giving potassium supplements!DKANURSING INTERVENTIONS3. Reverse the AcidosisREGULAR insulin injection is ordered IV bolus 5-10 unitsThe insulin is followed by drip infusion in units per hourBICARBONATE is not used!

    HHNSA serious condition in which hyperosmolarity and extreme hyperglycemia predominateKetosis is minimalOnset is slow and takes hours to days to developHHNSPATHOPHYSIOLOGYLack of insulin action or Insulin resistance hyperglycemiaHyperglycemia osmotic diuresis loss of water and electrolytesHHNSPATHOPHYSIOLOGYInsulin is too low to prevent hyperglycemia but enough to prevent fat breakdownOccurs most commonly in type 2 DM, ages 50-70HHNSPrecipitating factors1. Infection2. Stress3. Surgery4. Medication like thiazides5. Treatment like dialysis

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    HHNSASSESSMENT FINDINGS1. Profound dehydration2. Hypotension3. Tachycardia

    4. Altered sensorium5. Seizures and hemiparesisHHNSDIAGNOSTIC TESTS1. Blood glucose- 600 to 1,200 mg/dL2. Blood osmolality- 350 mOsm/L3. Electrolyte abnormalitiesHHNSNURSING INTERVENTIONS

    Approach is similar to the DKA1. Correction of Dehydration by IVF

    2. Correction of electrolyte imbalance by replacement therapyHHNSNURSING INTERVENTIONS3. Administration of insulin injection and drips4. Continuous monitoring of urine outputMACROVASCULAR CXNursing management1. Diet modification2. ExerciseMACROVASCULAR CXNursing management3. Prevention and treatment of underlying conditions such as MI, CAD and stroke4. Administration of prescribed medications for hypertension, hyperlipidemia and obesity

    Myocardial infarctionDeath of myocardial tissue in regions of the heart with abrupt interruption of coronaryblood supplyMyocardial infarctionETIOLOGY and Risk factors1. CAD2. Coronary vasospasm3. Coronary artery occlusion by embolus and thrombus4. Conditions that decrease perfusion- hemorrhage, shockMyocardial infarctionRisk factors1. Hypercholesterolemia2. Smoking3. Hypertension4. Obesity5. Stress

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    6. Sedentary lifestyleMyocardial infarction

    PATHOPHYSIOLOGYInterrupted coronary blood flow myocardial ischemiaanaerobic myocardialmetabolism for several hours myocardial death depressed cardiac function

    triggers autonomic nervous system response

    further imbalance of myocardial O2demand and supply

    Myocardial infarctionASSESSMENT findings

    1. CHEST PAINChest pain is described as severe, persistent, crushingsubsternal discomfortRadiates to the neck, arm, jaw and backMyocardial infarction

    ASSESSMENT findings1. CHEST PAIN

    Occurs without cause, primarily early morningNOTrelieved by rest or nitroglycerinLasts 30 minutes or longerMyocardial infarction

    Assessment findings2. Dyspnea3. Diaphoresis4. cold clammy skin5. N/V6. restlessness, sense of doom7. tachycardia or bradycardia8. hypotension9. S3 and dysrhythmiasMyocardial infarction

    Laboratory findings1. ECG- the ST segment is ELEVATED. T wave inversion, presence of Q wave2. Myocardial enzymes- elevated CK-MB, LDH and Troponin levels3. CBC- may show elevated WBC count4. Test after the acute stage- Exercise tolerance test, thallium scans, cardiaccatheterizationMyocardial infarction

    Nursing Interventions1. Provide Oxygen at 2 lpm, Semi-fowlers2. Administer medicationsMorphine to relieve painnitrates, thrombolytics, aspirin and anticoagulants

    Stool softener and hypolipidemics3. Minimize patient anxietyProvide information as to procedures and drug therapyMyocardial infarction

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    4. Provide adequate rest periods5. Minimize metabolic demandsProvide soft dietProvide a low-sodium, low cholesterol and low fat diet6. Minimize anxiety

    Reassure client and provide information as neededMyocardial infarction7. Assist in treatment modalities such as PTCA and CABG8. Monitor for complications of MI- especially dysrhythmias, since ventriculartachycardia can happen in the first few hours after MI9. Provide client teachingMI

    Medical Management1. ANALGESICThe choice is MORPHINEIt reduces pain and anxietyRelaxes bronchioles to enhance oxygenationMIMedical Management2. ACEPrevents formation of angiotensin IILimits the area of infarctionMIMedical Management3. ThrombolyticsStreptokinase, AlteplaseDissolve clots in the coronary artery allowing blood to flow

    PURPOSEDfunctionissolve and lyze the thrombus (thrombolysis)

    Allowing blood to flow again (reperfusion)Minimizing the size of infarctionPreserving ventricular

    Absolute ContraindicationActive bleedingKnown bleeding disorderHistory of hemorrhagic strokeHistory of intracranial vessel malformationRecent major surgery or trauma

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    Uncontrolled hypertensionPregnancy

    Nursing ConsiderationMinimize skin puncture

    Avoid IM injectionDraw blood for laboratory test when starting IV lineStart Iv line prior to thrombolytic therapyMonitor for dysrhythmias, hypotension, and allergic reactionMonitor for reperfusion, resolution of angina or acute ST segment changesCheck for signs and symptoms of bleeding, < Hgb, Hct, < BP, >HR, oozing or bulging atthe site, change in LOC

    Apply direct pressure

    AnticoagulantHeparin

    - prevents formation of thrombin- monitor PTT- Protamine Sulfate

    Warfarin- Suppresses formation of prothrombin- monitor PT- Vit K

    Myocardial infarction

    NURSING INTERVENTIONS AFTER ACUTE EPISODE1. Maintain bed rest for the first 3 days2. Provide passive ROM exercises3. Progress with dangling of the feet at side of bedMyocardial infarctionNURSING INTERVENTIONS AFTER ACUTE EPISODE4. Proceed with sitting out of bed, on the chair for 30 minutes TID5. Proceed with ambulation in the room toilet hallway TIDMyocardial infarctionNURSING INTERVENTIONS AFTER ACUTE EPISODECardiac rehabilitationTo extend and improve quality of lifePhysical conditioningPatients who are able to walk 3-4 mph are usually ready to resume sexual activities Treatments for coronary disease - angioplastyCoronary angioplasty involves inserting a balloon into a diseased (blocked/narrowed)coronary artery through an artery in the groin or arm.Commonly a metal support (stent) is inserted into the artery to help keep it open.

    A close up of a Stent.Angina Pectoris

    NURSING MANAGEMENT

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    1. Administer prescribed medicationsNitrates- to dilate the coronary arteries

    Aspirin- to prevent thrombus formationBeta-blockers- to reduce BP and HRCalcium-channel blockers- to dilate coronary artery and reduce vasospasm

    Basic Life SupportThis is a strategy which aims to improve the outcome for victims of Cardiopulmonaryarrest and is now being adopted internationallyIt involves a series of events which are interconnected to each other like the links of achain

    HOW DOES CPR WORK?All the living cells of our body need a steady supply of oxygen to keep us aliveCPR works because you can breathe air into the victims lungs to provide oxygen intothe blood. Then, when you press on the chest, you move oxygen-carrying blood throughthe body.

    WHEN WILL YOU DO CPR?CPR must be started as soon as possible when the carotid pulse is not appreciated or

    if breathing either stops or ineffective.In case of doubt, do CPR. Any delay in starting CPR reduces the chances of survival.

    In addition, the brain cells begin to die after four to six minutes without oxygen.