024.bone loss and patterns of bone destruction
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Transcript of 024.bone loss and patterns of bone destruction
Dr Jaffar Raza Syed
Bone Loss and Patterns of Bone Destruction
Normal Anatomy Of Alveolar Bone
Bone Loss and Patterns of Bone Destruction
Normal Anatomy Of Alveolar Bone
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Bone Loss and Patterns of Bone Destruction
Dr Jaffar Raza Syed Page 2
Various Factors Determining Bone Morphology In Periodontal Disease (i) Normal variation in alveolar bone (ii) Exostoses (iii) Trauma from occlusion (iv) Buttressing bone formation (v) Food impaction (vi) Aggressive periodontitis
Dr Jaffar Raza Syed Page 3
Various Anatomic Features That Influence Bone Destructive
Patterns In Periodontal Disease. (i) Thickness, width and crestal angulation of the interdental septa (ii) Thickness of facial and lingual alveolar plates (iii) Presence of fenestration and dehiscence (iv) Alignment of teeth (v) Proximity with another tooth surfaces (vi) Root and root trunk anatomy (vii) Root position within alveolar bone
Dr Jaffar Raza Syed Page 4
Various Causes Of Bone Destruction (i) Extension of gingival inflammation (ii) Trauma from occlusion (iii) Systemic disorders. Various Systemic Disorders Which Cause Bone Destruction (i) Hyperparathyroidism (ii) Leukemia (iii) Langerhan’s cell histiocytosis
Dr Jaffar Raza Syed Page 6
Changes In The Bone Could Be As Follows:
Gingival inflammation ↓
Marrow spaces ↓
Replaced by leukocytes and fluid exudates, new blood vessels and proliferating fibroblasts
↓ Increase in osteoclasts and mononuclear cells
↓ Thinning of bone trabeculae and enlargement of the marrow spaces
↓ Destruction of the bone and reduction in bone height
↓ Replacement of fatty bone marrow with the fibrous type
(around the resorption areas)
Dr Jaffar Raza Syed Page 7
various mechanisms of bone destruction According to Hausmann, the various mechanisms of bone destruction are: (i) Direct action of plaque products on bone progenitor cells induces the differentiation of these cells into osteoclasts. (ii) Plaque products act directly on bone, destroying it through a noncellular mechanism. (iii) Plaque products stimulate gingival cells, causing them to release mediators, which in turn induce bone progenitor cells to differentiate into osteoclasts. (iv) Plaque products causes gingival cells to release agents that can act as cofactors in bone resorption. (v) Plaque products causes gingival cells to release agents that destroy bone by direct chemical action, without oseoclasts.
Dr Jaffar Raza Syed
Various Bone Destructive Patterns In Periodontal Disease (i) Horizontal bone loss (ii) Vertical/angular defects
Various Bone Destructive Patterns In Periodontal Disease
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Dr Jaffar Raza Syed
Osseous Defects • One walled osseous defects • Two walled osseous defects • Three walled osseous defects
: only one bony wall is present
: two bony walls are present
: three bony walls are present
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nt
: three bony walls are present
Dr Jaffar Raza Syed
(iii) Osseous craters They are concavities in the crest of the interdental bone confined within the facial and lingual walls
They are concavities in the crest of the interdental bone confined within the facial and lingual walls
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Dr Jaffar Raza Syed
(iv) Bulbous bony contours
caused by exostoses, adaptation
found more frequently in the maxilla than mandible
caused by exostoses, adaptation to function or buttressing bone formation.
more frequently in the maxilla than mandible
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to function or buttressing bone formation.
Dr Jaffar Raza Syed
(v) Reversed architecture
defects are produced by loss of interdental bone plates without loss of radicular bone (more common in maxilla)
loss of interdental bone, including the facial and lingual
without loss of radicular bone, thereby reversing the normal
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including the facial and lingual
, thereby reversing the normal architecture
Dr Jaffar Raza Syed
(vi) Ledges
They are plateau-like bone margins caused by resorption of thickened bony plates.
like bone margins caused by resorption of
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Dr Jaffar Raza Syed Page 15
Bone Destruction in Periodontal Disease • Bacteria mediated — LPS, lipoteichoic acid, lipoproteins and others. • Host mediated — prostaglandins, leukotrienes, cytokines and others. • Combination of both. Diagnosis of Osseous Defects a. Clinical examination — transgingival probing. b. Radiographs — not very reliable, cannot reveal the extent of involvement and presence/absence of bony walls. c. Surgical exposure — during flap operations, it is the only reliable method for determining the true architecture of a bony defect